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Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 138, Issue 3, pp. 729 - 739.e11
Journal Article
Thorax, ISSN 0040-6376, 02/2014, Volume 69, Issue 2, pp. 152 - 160
Journal Article
Journal Article
Current Pharmaceutical Design, ISSN 1381-6128, 03/2010, Volume 16, Issue 9, pp. 1091 - 1105
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 2016, Volume 20, Issue 11, pp. 2029 - 2035
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 2015, Volume 19, Issue 6, pp. 1346 - 1356
Idiopathic pulmonary fibrosis is the most devastating diffuse fibrosing lung disease of unknown aetiology. Compelling evidence suggests that both... 
bleomycin | protease‐activated receptors (PARs) | pulmonary fibrosis | Protease-activated receptors (PARs) | Bleomycin | Pulmonary fibrosis | MEDICINE, RESEARCH & EXPERIMENTAL | INJURY | DEATH | FIBROBLASTS | CELL BIOLOGY | protease-activated receptors (PARs) | RESPONSES | MODELS | TRANSACTIVATION | PROGRESS | LUNG INFLAMMATION | TISSUE FACTOR | NIH 3T3 Cells | Receptor, PAR-1 - metabolism | Humans | Receptor, PAR-2 - metabolism | Actins - metabolism | Peptide Fragments - pharmacology | Pulmonary Fibrosis - genetics | Extracellular Signal-Regulated MAP Kinases - metabolism | Receptor, PAR-2 - antagonists & inhibitors | Thrombin - pharmacology | Trypsin - pharmacology | Pulmonary Fibrosis - metabolism | Phosphorylation - drug effects | Fibroblasts - metabolism | Receptor, PAR-2 - genetics | Receptor, PAR-1 - antagonists & inhibitors | Signal Transduction | Hydroxyproline - metabolism | Mice, Inbred C57BL | Blotting, Western | Receptor, PAR-1 - genetics | Mice, Knockout | Collagen - metabolism | Animals | Fibroblasts - drug effects | Pulmonary Fibrosis - chemically induced | Mice | Thrombin | Respiratory tract diseases | Muscle proteins | Collagen | Analysis | Medical research | Interferon | Proteases | Rodents | Hydroxyproline | Proteinase | Smooth muscle | Antagonists | Inactivation | Proteins | Protease | Actin | Fibroblasts | Extracellular matrix | Inhibition | Histochemical analysis | Deactivation | Lung diseases | Muscles | Inflammation | Lungs | Fibrosis | Ligands | Laboratory animals | Original
Journal Article