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PLoS ONE, ISSN 1932-6203, 09/2016, Volume 11, Issue 9, pp. e0162866 - e0162866
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 11/2016, Volume 186, Issue 2, pp. 157 - 163
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Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2016, Volume 75, Issue Suppl 2, pp. 665 - 666
BackgroundRheumatoid Arthritis (RA) is a systemic autoimmune disease resulting in chronic inflammation of the synovium and consequent cartilage and bone... 
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NEURAL REGENERATION RESEARCH, ISSN 1673-5374, 01/2019, Volume 14, Issue 1, pp. 75 - 76
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Journal of Antimicrobial Chemotherapy, ISSN 0305-7453, 2017, Volume 72, Issue 4, pp. 1152 - 1162
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PLoS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, p. e0169956
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Autoimmunity Reviews, ISSN 1568-9972, 03/2018, Volume 17, Issue 3, pp. 276 - 283
Psoriatic arthritis (PsA) is a chronic inflammatory joint disease, seen in combination with psoriasis. Both genetic and environmental factors are responsible... 
Microbiota | Genetic markers | Phylogeny | Psoriatic arthritis | RHEUMATOID-ARTHRITIS | NATURAL-KILLER | SYNOVIAL-FLUID | PERIPHERAL-BLOOD | INFLAMMATORY ARTHRITIS | INNATE LYMPHOID-CELLS | ANKYLOSING-SPONDYLITIS | GUT MICROBIOTA | IMMUNOLOGY | UNFOLDED PROTEIN RESPONSE | T-CELLS
Journal Article
Journal Article
NEUROMOLECULAR MEDICINE, ISSN 1535-1084, 03/2018, Volume 20, Issue 1, pp. 1 - 17
Parkinson's disease (PD) is a complex neurodegenerative disorder characterized by a progressive loss of dopamine neurons of the central nervous system. The... 
Pharmacogenetics | Monoamine oxidase inhibitors | Parkinson's disease | IMPULSE CONTROL DISORDERS | COMT inhibitors | PEAK-DOSE DYSKINESIAS | Polymorphisms | TRIPLEX-FORMING OLIGONUCLEOTIDES | ANGIOTENSIN-CONVERTING ENZYME | NEUROSCIENCES | CATECHOL-O-METHYLTRANSFERASE | LEVODOPA-INDUCED DYSKINESIA | MONOAMINE-OXIDASE-B | Dopamine receptor agonists | Levodopa | DOPAMINE TRANSPORTER GENE | SINGLE NUCLEOTIDE POLYMORPHISMS | EXCESSIVE DAYTIME SLEEPINESS | Monoamine Oxidase Inhibitors - therapeutic use | Nerve Tissue Proteins - physiology | Monoamine Oxidase Inhibitors - adverse effects | Humans | Catechol O-Methyltransferase - physiology | Dopamine Agonists - therapeutic use | Biotransformation - genetics | Dopamine Agonists - pharmacokinetics | Treatment Outcome | Dopamine Agonists - adverse effects | Parkinson Disease - drug therapy | Parkinson Disease - genetics | Pharmacogenomic Testing | Catechol O-Methyltransferase - genetics | Antiparkinson Agents - adverse effects | Dopamine Plasma Membrane Transport Proteins - genetics | Nerve Tissue Proteins - genetics | Antiparkinson Agents - pharmacokinetics | Antiparkinson Agents - therapeutic use | Monoamine Oxidase Inhibitors - pharmacokinetics | Receptors, Dopamine - genetics | Dopamine - metabolism | Receptors, Dopamine - physiology | Dopamine Plasma Membrane Transport Proteins - physiology | Oxidases | Sleep disorders | Medical research | Tremor | Medicine, Experimental | Drug therapy | Genetic polymorphisms | Dopamine receptors | Antiparkinsonian agents
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2018, Volume 77, Issue Suppl 2, p. 670
BackgroundSystemic lupus erythematosus (SLE) is a chronic autoimmune disease with a complex pathogenesis in which genes and environmental factors interact... 
Lupus | Phenotypes | Immune response | Laboratories | Pathogenesis | Stat4 protein | Arthritis | Single-nucleotide polymorphism | Environmental factors | Autophagy | Risk factors | Systemic lupus erythematosus | Genotyping | Correlation analysis | Alleles | Anti-DNA antibodies | Remission | Phagocytosis
Journal Article
Immunologic Research, ISSN 0257-277X, 8/2017, Volume 65, Issue 4, pp. 811 - 827
Journal Article