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PLoS ONE, ISSN 1932-6203, 12/2008, Volume 3, Issue 12, pp. e3908 - e3908
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2012, Volume 109, Issue 22, pp. 8699 - 8704
The mechanisms underlying tumor dormancy have been elusive and not well characterized. We recently published an experimental model for the study of human tumor... 
Dormancy | Tumor cell line | Cell growth | Secretion | Cell lines | Breast cancer | Down regulation | T tests | Endothelial cells | Tumors | NFκB | STAT3 | APOPTOSIS | ANGIOGENIC SWITCH | MULTIDISCIPLINARY SCIENCES | FIBROBLAST-GROWTH-FACTOR | PROLIFERATION | MECHANISMS | HUMAN TUMOR DORMANCY | ENDOTHELIAL-CELLS | EXPRESSION SIGNATURE | HEAT-SHOCK-PROTEIN-27 | NF kappa B | MELANOCYTES | Cell Proliferation | Human Umbilical Vein Endothelial Cells - metabolism | Oligonucleotide Array Sequence Analysis | Humans | Gene Expression Regulation, Neoplastic | Male | Transplantation, Heterologous | Gene Expression Profiling | Vascular Endothelial Growth Factor A - metabolism | Mammary Neoplasms, Experimental - metabolism | Vascular Endothelial Growth Factor A - genetics | Mammary Neoplasms, Experimental - genetics | Breast Neoplasms - metabolism | HSP27 Heat-Shock Proteins - genetics | Time Factors | Mammary Neoplasms, Experimental - pathology | Fibroblast Growth Factor 2 - metabolism | Female | Down-Regulation | Cells, Cultured | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Vascular Endothelial Growth Factor C - genetics | Animals | Breast Neoplasms - genetics | Fibroblast Growth Factor 2 - genetics | Breast Neoplasms - pathology | Vascular Endothelial Growth Factor C - metabolism | Cell Line, Tumor | HSP27 Heat-Shock Proteins - metabolism | Neovascularization, Pathologic - genetics | Mice | Heat shock proteins | Development and progression | Care and treatment | Properties | Angiogenesis | Biomarkers | Gene expression | Index Medicus | Biological Sciences
Journal Article
Cancer Cell, ISSN 1535-6108, 2010, Volume 17, Issue 6, pp. 547 - 559
In mice, deletion and activation of results in lung tumors with a high penetrance of lymph node and distant metastases. We analyzed these primary and... 
CELLCYCLE | SIGNALING | EPITHELIAL-MESENCHYMAL TRANSITION | ONCOGENIC K-RAS | CANCER-CELLS | SUPPRESSOR | GENE | ONCOLOGY | SRC | KINASE INHIBITOR | EXPRESSION PROFILES | MUTATIONS | TUMORIGENESIS | Lung Neoplasms - drug therapy | Protein-Serine-Threonine Kinases - deficiency | Protein-Tyrosine Kinases - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Genomics | Humans | Lung Neoplasms - metabolism | Gene Expression Profiling | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Cell Movement - genetics | Phosphorylation - genetics | RNA Interference | Gene Expression Regulation, Neoplastic - genetics | MAP Kinase Kinase 1 - antagonists & inhibitors | Carcinoma, Non-Small-Cell Lung - metabolism | Signal Transduction - genetics | Enzyme Inhibitors - therapeutic use | Focal Adhesion Protein-Tyrosine Kinases - antagonists & inhibitors | Focal Adhesion Protein-Tyrosine Kinases - genetics | Focal Adhesions - genetics | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | Mice | TOR Serine-Threonine Kinases | src-Family Kinases - genetics | Protein-Tyrosine Kinases - antagonists & inhibitors | ras Proteins - genetics | Lung Neoplasms - pathology | Cell Transdifferentiation - genetics | Protein-Tyrosine Kinases - genetics | Neoplasm Metastasis - drug therapy | Mice, Mutant Strains | Protein-Serine-Threonine Kinases - antagonists & inhibitors | src-Family Kinases - metabolism | Female | Drug Therapy, Combination | Lung Neoplasms - genetics | Cell Adhesion - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | src-Family Kinases - antagonists & inhibitors | Protein-Serine-Threonine Kinases - genetics | Proto-Oncogene Proteins - genetics | Up-Regulation - genetics | Xenograft Model Antitumor Assays | Neoplasm Metastasis - genetics | Animals | MAP Kinase Kinase 2 - antagonists & inhibitors | Protein Kinase Inhibitors - therapeutic use | Focal Adhesions - metabolism | Proteomics | Protein Kinase Inhibitors - pharmacology | Oncology, Experimental | Analysis | Lung cancer | Development and progression | Metastasis | Research | Cancer | Index Medicus
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 09/2012, Volume 18, Issue 18, pp. 4973 - 4985
Journal Article
PLOS ONE, ISSN 1932-6203, 12/2008, Volume 3, Issue 12, p. e3908
Background: Inhibition of the epidermal growth factor receptor ( EGFR) has shown clinical success in patients with advanced non-small cell lung cancer (NSCLC).... 
MULTIDISCIPLINARY SCIENCES | Glucose metabolism | Epidermal growth factor | PET imaging | Erlotinib | Genetic aspects | Glucose | Comparative analysis | Lung cancer, Non-small cell | Dextrose
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States, ISSN 0027-8424, 01/2009, Volume 106, Issue 2, p. 474
Mutations in the HER2 kinase domain have been identified in human clinical lung cancer specimens. Here we demonstrate that inducible expression of the most... 
Care and treatment | Squamous cell tumors | Lung cancer | Development and progression | Rapamycin | Drug therapy, Combination | Drug therapy | Health aspects | Methods | Cancer
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 01/2009, Volume 106, Issue 2, p. 474
  Mutations in the HER2 kinase domain have been identified in human clinical lung cancer specimens. Here we demonstrate that inducible expression of the most... 
Lung cancer | Rodents | Mutation | Gene expression | Drug therapy | Kinases | Tumors
Journal Article
Cancer Research, ISSN 0008-5472, 12/2010, Volume 70, Issue 23, pp. 9827 - 9836
Genetic rearrangements of the anaplastic lymphoma kinase (ALK) kinase occur in 3% to 13% of non-small cell lung cancer patients and rarely coexist with KRAS or... 
NPM-ALK | GENE | ONCOLOGY | THERAPEUTIC-TARGET | SIGNALING PATHWAY | KINASE DOMAIN MUTATIONS | TYROSINE KINASE | ANAPLASTIC LYMPHOMA | SMALL-CELL-LUNG | CANCER | EGFR | Lung Neoplasms - drug therapy | Mitogen-Activated Protein Kinase Kinases - genetics | Oncogene Proteins, Fusion - metabolism | Adenocarcinoma - pathology | Paclitaxel - pharmacology | Protein-Tyrosine Kinases - metabolism | Humans | Molecular Sequence Data | Lung Neoplasms - pathology | Male | Gene Expression Profiling | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Mitogen-Activated Protein Kinase Kinases - metabolism | Protein-Tyrosine Kinases - genetics | Female | Adenocarcinoma - genetics | Antineoplastic Agents - pharmacology | HSP90 Heat-Shock Proteins - genetics | Lung Neoplasms - genetics | Amino Acid Sequence | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Mice, Transgenic | Pyrimidines - pharmacology | Adenocarcinoma - drug therapy | Receptor Protein-Tyrosine Kinases | Phosphatidylinositol 3-Kinases - genetics | Xenograft Model Antitumor Assays | Animals | Tumor Burden - drug effects | HSP90 Heat-Shock Proteins - antagonists & inhibitors | Oncogene Proteins, Fusion - genetics | Survival Analysis | Cell Line, Tumor | HSP90 Heat-Shock Proteins - metabolism | Mice | Carboplatin - pharmacology | Cluster Analysis | Protein-Tyrosine Kinases - antagonists & inhibitors | Index Medicus
Journal Article
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