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PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, pp. e54014 - e54014
Purpose: The receptors for hepatocyte and vascular endothelial cell growth factors (MET and VEGFR2, respectively) are critical oncogenic mediators in gastric... 
ACTIVATION | AMPLIFICATION | GENE | MULTIDISCIPLINARY SCIENCES | C-MET | ACQUIRED-RESISTANCE | SUNITINIB | PROGNOSTIC MARKER | CARCINOMA | EXPRESSION | VASCULAR ENDOTHELIAL GROWTH | Anilides - therapeutic use | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Humans | Middle Aged | Male | Stomach Neoplasms - pathology | Treatment Outcome | Antineoplastic Agents - therapeutic use | Stomach Neoplasms - drug therapy | Quinolines - pharmacology | Xenograft Model Antitumor Assays | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Neoplasm Metastasis | Animals | Anilides - pharmacology | Aged, 80 and over | Adult | Female | Aged | Antineoplastic Agents - pharmacology | Mice | Quinolines - therapeutic use | Hypertension | Cancer patients | Aspartate | Metastasis | Drug therapy | Stomach cancer | Cancer | Endothelium | Adenocarcinoma | Schedules | Carcinoma | Oncology | Kinases | Axl protein | Metastases | c-Met protein | Liver cancer | Receptors | Cell growth | Tumorigenesis | Bioindicators | MET protein | Safety | Vascular endothelial growth factor | Growth factors | Gastric cancer | Medical research | Dosing | Receiving | Pharmacology | Patients | Endothelial cells | Ron protein | Amplification | Signaling | Gene amplification | Inhibitors | Correlation analysis | Medical prognosis | Biomarkers | Aspartate aminotransferase | Mutation | Pharmacokinetics | Tumors | Index Medicus
Journal Article
Cell Reports, ISSN 2211-1247, 03/2016, Volume 14, Issue 10, pp. 2476 - 2489
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2016, Volume 11, Issue 2, pp. e0148075 - e0148075
The epithelial-mesenchymal transition (EMT) is a complex cancer progression that can boost the metastatic potential of transformed cells by inducing migration,... 
EPITHELIAL-MESENCHYMAL TRANSITION | MATRIX METALLOPROTEINASES | SCATTER FACTOR | PROTEIN-FREE CULTURE | SIGNALING PATHWAY | MULTIDISCIPLINARY SCIENCES | HEPATOCYTE GROWTH-FACTOR | C-MET | CHO-CELLS | CANCER PROGRESSION | KINASE ACTIVATION | Proto-Oncogene Proteins c-met - metabolism | Cell Adhesion - genetics | Gene Expression | Phosphorylation | Signal Transduction | Sialyltransferases - genetics | Ectopic Gene Expression | Epithelial-Mesenchymal Transition - genetics | Autocrine Communication - genetics | Phenotype | Animals | Models, Biological | Dogs | Madin Darby Canine Kidney Cells | Hepatocyte Growth Factor - genetics | Matrix Metalloproteinase 1 - metabolism | Matrix Metalloproteinase 1 - genetics | Physiological aspects | Cell receptors | Genetic aspects | Research | Gene expression | Stem cells | Cell proliferation | Biotechnology | Motility | Transcription | Leukocyte migration | Mesenchyme | Laboratories | Oncology | Metastasis | Matrix metalloproteinase | Kinases | Cell adhesion & migration | Metastases | c-Met protein | Signal transduction | Transformed cells | Cell adhesion | Cell cycle | Extracellular matrix | Protein-tyrosine kinase receptors | Metalloproteinase | Autocrine signalling | Growth factors | Protein-tyrosine kinase | Deoxyribonucleic acid--DNA | Tyrosine | Medical research | Enzymes | Secretion | Hepatocyte growth factor | Research & development--R&D | Cloning | DNA microarrays | Cell lines | Genetic engineering | Kidney diseases | Diabetes | Cell migration | Cancer | Index Medicus | Research & development | Deoxyribonucleic acid | R&D | DNA
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 01/2013, Volume 31, Issue 2, pp. 181 - 186
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2012, Volume 7, Issue 8, pp. e42717 - e42717
The Hepatocyte Growth Factor (HGF)/c-Met signaling pathway regulates hepatocyte proliferation, and pathway aberrations are implicated in the invasive and... 
GENE | SIGNALING PATHWAY | FACTOR SCATTER FACTOR | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION FACTOR EGR-1 | IN-VIVO | PTEN | CLASSIFICATION | MELANOMA-CELLS | UP-REGULATION | EXPRESSION | Proto-Oncogene Proteins c-met - metabolism | Transcription, Genetic - drug effects | Humans | Hepatocyte Growth Factor - pharmacology | Gene Knockdown Techniques | Matrix Metalloproteinase 9 - metabolism | Early Growth Response Protein 1 - genetics | Indoles - pharmacology | Liver Neoplasms - pathology | Gene Expression Regulation, Neoplastic - drug effects | Heparin - pharmacology | Matrix Metalloproteinase 2 - metabolism | Neoplasm Invasiveness | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Cell Adhesion - drug effects | Enzyme Activation - drug effects | Matrix Metalloproteinase 14 - metabolism | Sulfonamides - pharmacology | Piperazines - pharmacology | Cell Movement - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Early Growth Response Protein 1 - deficiency | Carcinoma, Hepatocellular - pathology | Cell Line, Tumor | Early Growth Response Protein 1 - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Heparin | Metastasis | Hepatoma | Phosphorylation | Transcription factors | Motility | Lung cancer | Hepatocellular carcinoma | Activation | Biology | Kinases | Cancer therapies | Metastases | Cell adhesion & migration | Anticancer properties | c-Met protein | Liver cancer | Angiogenesis | Signal transduction | Biological effects | Rodents | Transcription activation | Cell cycle | Growth factors | EGR-1 protein | Hepatocyte growth factor | Invasiveness | MAP kinase | Gelatinase B | Gelatinase A | Medicine | Signaling | Hypotheses | Molecular modelling | Medical prognosis | Aberration | Gene therapy | Cancer | Apoptosis | Tumors | Index Medicus
Journal Article
Cancers, ISSN 2072-6694, 11/2014, Volume 6, Issue 4, pp. 2313 - 2329
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, pp. e100586 - e100586
Background: Overexpression of Met tyrosine kinase receptor is associated with poor prognosis. Overexpression, and particularly MET amplification, are... 
CELL LUNG-CANCER | GASTRIC-CANCER | SIGNALING PATHWAY | ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | HEPATOCYTE GROWTH-FACTOR | C-MET | THERAPEUTIC TARGET | PROTEIN EXPRESSION | STORED PARAFFIN SLIDES | KINASE INHIBITOR | Proto-Oncogene Proteins c-met - metabolism | Stomach Neoplasms - genetics | Humans | Stomach Neoplasms - metabolism | Male | Stomach Neoplasms - pathology | Esophageal Neoplasms - pathology | Proto-Oncogene Proteins c-met - genetics | Immunohistochemistry - methods | Gene Amplification | Esophageal Neoplasms - genetics | Esophageal Neoplasms - metabolism | Female | Mass Spectrometry - methods | Proteins | Immunohistochemistry | Genes | Genetic research | Mass spectrometry | Formaldehyde | Tumors | Correlation | Copy number | Genomics | Fluorescence | Clinical trials | Oncology | Quantitation | Kinases | Assaying | Paraffin | Nuclei | Stomach cancer | c-Met protein | Animal tissues | Fluorescence in situ hybridization | Protein-tyrosine kinase receptors | MET protein | Growth factors | Protein-tyrosine kinase | Recombinant | Tyrosine | Medical research | Spectroscopy | Hematology | Mass spectroscopy | Stability analysis | Gene expression | Peptide mapping | Medicine | Amplification | Pathology | Gene amplification | Collaboration | Medical prognosis | Protein expression | Scientific imaging | Cancer | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2018, Volume 13, Issue 6, pp. e0199361 - e0199361
The presence of cancer stem cells (CSCs) and the induction of epithelial-to-mesenchymal transition (EMT) in tumors are associated with tumor aggressiveness,... 
BREAST-CANCER | LUNG-CANCER | METASTASIS | CLAUDIN-LOW | MULTIDISCIPLINARY SCIENCES | PANCREATIC-CANCER | EXPRESSION | EMT | GENE CLONING | PLASTICITY | CD133 | Neoplastic Stem Cells - drug effects | Antibodies, Monoclonal - biosynthesis | Humans | Lung Neoplasms - metabolism | Epithelial-Mesenchymal Transition - drug effects | Lung Neoplasms - pathology | Antineoplastic Agents - therapeutic use | Pyrazines - therapeutic use | Triple Negative Breast Neoplasms - drug therapy | Benzamides - therapeutic use | Neoplastic Stem Cells - metabolism | Triple Negative Breast Neoplasms - pathology | Hepatocyte Growth Factor - genetics | Neoplastic Stem Cells - pathology | Female | Antineoplastic Agents - pharmacology | Benzamides - pharmacology | Carcinoma, Non-Small-Cell Lung - pathology | Tumor Microenvironment - drug effects | Antibodies, Monoclonal - pharmacology | Carcinoma, Non-Small-Cell Lung - metabolism | Mice, Transgenic | Sulfonamides - pharmacology | Gene Knock-In Techniques | Xenograft Model Antitumor Assays | Phenotype | Animals | Sulfonamides - therapeutic use | AC133 Antigen - metabolism | Cell Line, Tumor | Indicators and Reagents | Pyrazines - pharmacology | Usage | Oncology, Experimental | Stem cells | Fluorescent antibody technique | Research | Immunofluorescence | Cancer | Immunohistochemistry | Animal models | Transcription factors | Immunoprecipitation | Mesenchyme | Laboratories | Sox9 protein | Xenotransplantation | Lung cancer | Antibodies | Stem cell transplantation | Selectivity | Metastasis | Quantitation | Drug resistance | Cancer therapies | Metastases | Proteins | Xenografts | Localization | Medical research | Hepatocyte growth factor | Tumor cells | Transgenic mice | Melanoma | Non-small cell lung carcinoma | Mass spectroscopy | Breast cancer | Glycosylation | Epitopes | Molecular chains | Microscopy | Medical prognosis | Pancreatic cancer | Reagents | Biomarkers | Mice | Focal adhesion kinase | Mass spectrometry | Tumors | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2006, Volume 103, Issue 39, pp. 14531 - 14536
Journal Article
Nature, ISSN 0028-0836, 06/2003, Volume 423, Issue 6940, pp. 593 - 595
Starving cancers of oxygen would seem to be a good way of killing them, but the presence of oxygen-deprived areas in tumors appears to correlate with poor... 
Medical research | Oxygen | Molecular biology | Cancer | Tumors
Journal Article
Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13 Supplement, pp. 5530 - 5530
Journal Article
Cancer Research, ISSN 0008-5472, 10/2014, Volume 74, Issue 19 Supplement, pp. 489 - 489
Journal Article