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Handbook of Clinical Neurology, ISSN 0072-9752, 2016, Volume 133, pp. 121 - 143
Over the last decade, neurologic autoimmunity has become a major consideration in the diagnosis and management of patients with many neurologic presentations.... 
Brain | Tissue damage | Autoantibodies | Muscle | Tissue modification | T cells | Channels | Autoimmunity | Autoimmune Diseases of the Nervous System - immunology | Neurology | Animals | Humans | Autoimmune Diseases of the Nervous System - therapy | Disease Models, Animal
Conference Proceeding
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 08/2014, Volume 128, Issue 2, pp. 247 - 266
Recent data suggest that oxidative injury may play an important role in demyelination and neurodegeneration in multiple sclerosis (MS). We compared the extent... 
Peptide Fragments | Comparative Study | Oxidative Stress | Respiratory Burst | Rats, Inbred Lew | CD4 Antigens | Iron | Macrophages | CD8 Antigens | Journal Article | Lipopolysaccharides | Multiple Sclerosis | Cuprizone | Aging | Disease Models, Animal | Coronavirus Infections | Gene Expression | T-Lymphocytes | Mice, Inbred C57BL | Myelin-Oligodendrocyte Glycoprotein | Rats | Microglia | Encephalomyelitis, Autoimmune, Experimental | Animals | Research Support, Non-U.S. Gov't | Inducible nitric oxide synthase (iNOS) | Pathology | Neurosciences | Multiple sclerosis | Experimental autoimmune encephalomyelitis (EAE) | Medicine & Public Health | NADPH oxidase | Oxidative injury | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | MICROGLIAL ACTIVATION | HUMAN BRAIN | MOUSE HEPATITIS-VIRUS | NITRIC-OXIDE SYNTHASE | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | PATHOLOGY | NEUROSCIENCES | CELL-MEDIATED DEMYELINATION | CLINICAL NEUROLOGY | SJL MICE | CENTRAL-NERVOUS-SYSTEM | Myelin-Oligodendrocyte Glycoprotein - immunology | T-Lymphocytes - physiology | Oxidative Stress - physiology | Encephalomyelitis, Autoimmune, Experimental - immunology | Lipopolysaccharides - immunology | T-Lymphocytes - transplantation | Microglia - physiology | Respiratory Burst - physiology | Peptide Fragments - immunology | Microglia - pathology | Macrophages - physiology | Encephalomyelitis, Autoimmune, Experimental - pathology | Macrophages - pathology | Coronavirus Infections - immunology | Iron - metabolism | Aging - pathology | CD8 Antigens - metabolism | Aging - physiology | Coronavirus Infections - pathology | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | CD4 Antigens - metabolism | Oxidases | Membrane lipids | Brain | Nitric oxide | Blood lipids | Comparative analysis | T cells | Gene expression | Original Paper
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 8/2010, Volume 120, Issue 2, pp. 223 - 236
Primary loss and dysfunction of astrocytes may trigger demyelination, as seen in neuromyelitis optica, an inflammatory disease of the central nervous system.... 
Pathology | Neurosciences | Multiple sclerosis | Medicine & Public Health | Brain inflammation | Astrocytes | Demyelination | Neuromyelitis optica | Demyelination neuromyelitis optica | ANTIBODIES | PATHOLOGY | NEUROSCIENCES | AUTOIMMUNE ENCEPHALOMYELITIS | CLINICAL NEUROLOGY | MYELIN OLIGODENDROCYTE GLYCOPROTEIN | GAP-JUNCTIONS | MULTIPLE-SCLEROSIS | NEUROMYELITIS-OPTICA | REACTIVE ASTROCYTES | AQUAPORIN-4 | WATER CHANNEL | TRANSGENIC MICE | Encephalomyelitis, Autoimmune, Experimental - pathology | Gene Expression Regulation - immunology | Humans | Astrocytes - pathology | Cells, Cultured | Encephalomyelitis, Autoimmune, Experimental - etiology | Central Nervous System - pathology | Encephalomyelitis, Autoimmune, Experimental - immunology | Rats | Coculture Techniques - methods | Immunity, Innate | Microscopy, Confocal - methods | Polysaccharides - pharmacology | Neuroglia - drug effects | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Animals | Astrocytes - immunology | Time Factors | Aquaporin 4 - metabolism | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Membrane Proteins - metabolism | Disease Models, Animal | Autoimmunity | Autoantibodies | Analysis | Aquaporins | Central nervous system | Brain damage | Inflammation | Universities and colleges | Mitogens | Original Paper
Journal Article