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Radiation Oncology, ISSN 1748-717X, 11/2015, Volume 10, Issue 1, p. 223
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2011, Volume 108, Issue 23, pp. 9595 - 9600
Journal Article
The Journal of Pathology, ISSN 0022-3417, 08/2013, Volume 230, Issue 4, pp. 410 - 419
Chemotherapeutic drugs kill cancer cells, but it is unclear why this happens in responding patients but not in non‐responders. Proteomic profiles of patients... 
tumour response | COX7A2 | MALDI imaging mass spectrometry | chemotherapy | oesophageal adenocarcinoma | IMAGING MASS-SPECTROMETRY | PERIOPERATIVE CHEMOTHERAPY | BARRETTS ADENOCARCINOMA | ANTICANCER AGENTS | PATHOLOGY | CYTOCHROME-C-OXIDASE | ALPHA-TOCOPHERYL SUCCINATE | VITAMIN-E ANALOGS | NEOADJUVANT CHEMOTHERAPY | ONCOLOGY | MULTIMODAL THERAPY | ESOPHAGOGASTRIC JUNCTION | Mitochondria - enzymology | Adenocarcinoma - pathology | Humans | Middle Aged | Drug Resistance, Neoplasm | Cisplatin - administration & dosage | Electron Transport Complex IV - metabolism | Esophageal Neoplasms - pathology | Tandem Mass Spectrometry | Fluorouracil - administration & dosage | Transfection | RNA Interference | Biomarkers, Tumor - metabolism | Chromatography, Liquid | Neoadjuvant Therapy | Adenocarcinoma - genetics | Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization | Chemotherapy, Adjuvant | Proteomics - methods | Precision Medicine | Down-Regulation | Adenocarcinoma - enzymology | Treatment Outcome | Electron Transport Complex IV - genetics | Mitochondria - drug effects | Mitochondria - pathology | Adenocarcinoma - drug therapy | Esophageal Neoplasms - enzymology | Esophageal Neoplasms - genetics | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Biopsy | Cell Line, Tumor | Aged | Biomarkers, Tumor - genetics | Esophageal Neoplasms - drug therapy | Adenocarcinoma | Cytochrome oxidase | Liquid chromatography | Mitochondrial DNA | Antineoplastic agents | Proteins | Antimitotic agents | Cytochrome c | Chemotherapy | Mass spectrometry | Esophageal cancer | Cancer | Tumors
Journal Article
Scientific Reports, ISSN 2045-2322, 07/2016, Volume 6, Issue 1, p. 28890
PC4 is an abundant single-strand DNA binding protein that has been implicated in transcription and DNA repair. Here, we show that PC4 is involved in the... 
RNA-POLYMERASE-II | B-CELLS | DAMAGE SITES | SOMATIC HYPERMUTATION | MULTIDISCIPLINARY SCIENCES | IMMUNOGLOBULIN GENE CONVERSION | TRANSCRIPTIONAL COACTIVATOR PC4 | INTERMEDIATE | C-TERMINAL DOMAIN | URACIL | NUCLEOTIDE EXCISION-REPAIR
Journal Article
International Journal of Cancer, ISSN 0020-7136, 02/2018, Volume 142, Issue 3, pp. 573 - 583
Ionizing radiation is a well‐recognized risk factor for the development of breast cancer. However, it is unknown whether radiation‐specific molecular oncogenic... 
Chernobyl | breast cancer | hsa‐miR‐26b‐5p | TRPS1 | radiation‐associated | hsa-miR-26b-5p | radiation-associated | APOPTOSIS | DNA-DAMAGE | CYTOSCAPE | MESENCHYMAL TRANSITION | BIOMARKERS | GENE | ONCOLOGY | CELL-CYCLE | PROMOTES | PROGRESSION | MICRORNA | Humans | Middle Aged | Neoplasms, Radiation-Induced - etiology | Breast Neoplasms - etiology | Chernobyl Nuclear Accident | MicroRNAs - biosynthesis | Transcription Factors - biosynthesis | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Neoplasms, Radiation-Induced - metabolism | Breast Neoplasms - metabolism | Paraffin Embedding | Neoplasms, Radiation-Induced - genetics | Breast Neoplasms - genetics | Adult | Female | MicroRNAs - genetics | DNA-Binding Proteins - biosynthesis | Breast cancer | MicroRNA | Genetic transcription | Analysis | Risk factors | Radiation | Cell culture | Deregulation | Transcription | Mitosis | Genes | DNA damage | Occupational exposure | Tissues | DNA repair | Carcinogenesis | Cleanup | Cell adhesion & migration | Angiogenesis | Carcinogens | Ionizing radiation | Pathways | Cell cycle | Repair | Deoxyribonucleic acid--DNA | Radiation effects | Markers | Health risks | MiRNA | Exposure | siRNA | Gene expression | Ribonucleic acids | Cell lines | Breast | Health risk assessment | Radiation damage | Cell migration | Apoptosis | Cancer | Index Medicus
Journal Article
International Journal of Cancer, ISSN 0020-7136, 09/2018, Volume 143, Issue 6, pp. 1505 - 1515
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, p. e47185
Radiotherapy is a powerful cure for several types of solid tumours, but its application is often limited because of severe side effects in individual patients.... 
INDUCED DNA-DAMAGE | GEL-ELECTROPHORESIS | B-CELLS | ALKALINE COMET ASSAY | MULTIDISCIPLINARY SCIENCES | BREAST-CANCER PATIENTS | DOUBLE-STRAND BREAKS | LYMPHOBLASTOID CELL-LINES | RADIATION SENSITIVITY | NORMAL TISSUE COMPLICATIONS | H2AX PHOSPHORYLATION | Lymphocytes - metabolism | DNA Damage - physiology | Radiation Tolerance - physiology | Humans | Biomarkers - analysis | Cells, Cultured | Lymphocytes - radiation effects | Histones - metabolism | Comet Assay | Dose-Response Relationship, Radiation | Radiation, Ionizing | Care and treatment | Usage | Head and neck cancer | Breast cancer | Research | Biological markers | Radiotherapy | Health aspects | Biotechnology | Side reactions | Phosphorylation | Radiosensitivity | Laboratories | Toxicity | DNA damage | Sex | Biology | Genomes | Order parameters | DNA repair | Epidemiology | Cancer therapies | Risk factors | Comet assay | Ionizing radiation | Lymphocytes | Peripheral blood | Bioindicators | Repair | Deoxyribonucleic acid--DNA | Age | Medical research | Head | Sensitivity analysis | Bioassay | Bioassays | Lymphoblastoid cell lines | Environmental health | I.R. radiation | Radiation therapy | Gene expression | Patients | Studies | Side effects | Damage detection | Cell death | Predictions | Irradiation | Biomarkers | Solid tumors | In vitro methods and tests | Tumors | Deoxyribonucleic acid | DNA
Journal Article
Cancer Letters, ISSN 0304-3835, 2016, Volume 386, pp. 87 - 99
Abstract Radio (chemo) therapy is a crucial treatment modality for head and neck squamous cell carcinoma (HNSCC), but relapse is frequent, and the underlying... 
Hematology, Oncology and Palliative Medicine | DNA damage repair | Head and neck squamous cell carcinoma | Radioresistance | Senescence | Fanconi anemia | Radiotherapy | SECRETORY PHENOTYPE | DNA-DAMAGE RESPONSE | DOWN-REGULATION | RADIATION SENSITIVITY | CANCER | I INTERFERON | ONCOLOGY | PATHWAY | NF-KAPPA-B | EXPRESSION | Oligonucleotide Array Sequence Analysis | Keratinocytes - radiation effects | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | Gene Expression Regulation, Neoplastic | Carcinoma, Squamous Cell - radiotherapy | Gene Regulatory Networks | RNA, Messenger - metabolism | Fanconi Anemia Complementation Group A Protein - genetics | Tumor Suppressor Protein p53 - genetics | Squamous Cell Carcinoma of Head and Neck | Carcinoma, Squamous Cell - mortality | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Transfection | RNA Interference | Time Factors | Treatment Failure | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Cellular Senescence - genetics | RNA, Messenger - genetics | Kaplan-Meier Estimate | Tumor Suppressor Protein p53 - metabolism | Radiation Tolerance - genetics | Gene Expression Profiling - methods | Genotype | Head and Neck Neoplasms - pathology | Disease-Free Survival | Head and Neck Neoplasms - radiotherapy | Keratinocytes - pathology | Phenotype | Gene Amplification | Cell Line, Tumor | Head and Neck Neoplasms - genetics | Biomarkers, Tumor - genetics | Head and Neck Neoplasms - mortality | Chemotherapy | Squamous cell carcinoma | DNA microarrays | Radiation | Cytogenetics | Interferon | Biological response modifiers | Cells | Cancer | Fanconi's anemia | Cell culture | Anemia | Genes | Radiation therapy | Cancer therapies | Patients | DNA repair | Studies | Cell cycle | Deoxyribonucleic acid--DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2016, Volume 11, Issue 6, p. e0156599
Histone demethylases have recently gained interest as potential targets in cancer treatment and several histone demethylases have been implicated in the DNA... 
CHROMATIN COMPACTION | CANCER-CELLS | LYSINE DEMETHYLASES | GASTRIC-CANCER | MULTIDISCIPLINARY SCIENCES | DEACETYLASE INHIBITORS | TRANSCRIPTIONAL REPRESSION | VALPROIC ACID | H3K4 DEMETHYLASE | DAMAGE RESPONSE PROTEIN | ONCOMETABOLITE 2-HYDROXYGLUTARATE | Chromatin - metabolism | Humans | DNA Repair - radiation effects | Down-Regulation - radiation effects | Plasmids - metabolism | DNA Breaks, Double-Stranded - radiation effects | Gene Knockdown Techniques | Radiation Tolerance - radiation effects | Retinoblastoma-Binding Protein 2 - metabolism | MCF-7 Cells | Lysine - metabolism | Acetylation | HeLa Cells | Histones - metabolism | Genes, Reporter | Cell Proliferation - radiation effects | Radiation, Ionizing | Ionizing radiation | DNA damage | Histones | Physiological aspects | Causes of | Genetic aspects | Research | Chromatin | Phosphorylation | Radiosensitivity | Transcription | Laboratories | Radiation | Oncology | Genomes | DNA repair | Proteins | Cooperation | DNA methylation | Repair | Damage accumulation | Deoxyribonucleic acid--DNA | Radiation effects | BRCA1 protein | Breast cancer | siRNA | RNA polymerase | Tumor cell lines | Radiation therapy | Double-strand break repair | Sensitivity | Depletion | Lysine | Cell lines | Stem cells | Cytogenetics | Mutation | Head & neck cancer | Charged particles | Radiation damage | Histone H3 | Cancer | Deoxyribonucleic acid | DNA
Journal Article