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Current Gastroenterology Reports, ISSN 1522-8037, 9/2017, Volume 19, Issue 9, pp. 1 - 7
Secondary sclerosing cholangitis is an emerging entity. Yet, because of the low incidence and varying etiologies of this disease, the literature is not robust.... 
Obstructive jaundice | Biliary stricture | Medicine & Public Health | Gastroenterology | Cholangitis | Critically ill patient | Secondary sclerosing cholangitis | Medicine, Experimental | Medical research
Journal Article
Gastrointestinal Endoscopy, ISSN 0016-5107, 06/2019, Volume 89, Issue 6, pp. AB162 - AB162
Journal Article
Current gastroenterology reports, 09/2017, Volume 19, Issue 9, p. 44
Secondary sclerosing cholangitis is an emerging entity. Yet, because of the low incidence and varying etiologies of this disease, the literature is not robust.... 
Cholangitis, Sclerosing - therapy | Prognosis | Drug-Related Side Effects and Adverse Reactions - complications | Humans | Critical Illness | Cholangitis, Sclerosing - etiology
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2004, Volume 279, Issue 33, pp. 34530 - 34536
Journal Article
Obesity, ISSN 1930-7381, 06/2013, Volume 21, Issue 6, pp. 1189 - 1199
Background: Alms1 mutant ( foz/foz ) mice develop hyperphagic obesity, diabetes, metabolic syndrome, and fatty liver (steatosis). High‐fat (HF) feeding... 
HEPATOCYTE APOPTOSIS | ALPHA AGONIST | DIABETIC MICE | NUTRITION & DIETETICS | STEATOSIS | ROLES | ACIDS | ENDOCRINOLOGY & METABOLISM | NONALCOHOLIC STEATOHEPATITIS | NF-KAPPA-B | PROGRESSION | ADIPOSE | Obesity - diet therapy | Inflammation - pathology | Up-Regulation | Liver - pathology | CD36 Antigens - genetics | Fatty Liver - complications | Diet, High-Fat - adverse effects | Hepatic Stellate Cells - metabolism | Ion Channels - genetics | Male | NF-kappa B - metabolism | Mitochondrial Proteins - genetics | Tissue Inhibitor of Metalloproteinase-2 - genetics | Adipose Tissue - metabolism | Inflammation - diet therapy | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Tissue Inhibitor of Metalloproteinase-2 - metabolism | Matrix Metalloproteinase 9 - metabolism | CD36 Antigens - metabolism | Matrix Metalloproteinase 9 - genetics | Mitochondrial Proteins - metabolism | Dietary Fats - administration & dosage | Non-alcoholic Fatty Liver Disease | Liver Cirrhosis - diet therapy | Lipogenesis - physiology | Disease Models, Animal | Fatty Liver - diet therapy | Matrix Metalloproteinase 2 - metabolism | Obesity - complications | Liver - metabolism | Lipid Metabolism | Cholesterol - metabolism | Obesity - pathology | Triglycerides - metabolism | Matrix Metalloproteinase 2 - genetics | Tissue Inhibitor of Metalloproteinase-1 - genetics | Animals | Diet | Ion Channels - metabolism | NF-kappa B - genetics | Liver Cirrhosis - pathology | Mice | Uncoupling Protein 2 | Apoptosis - physiology | Obesity | Liver diseases | Pathogenesis | Liver | Lipids | Metabolism | Fatty acids | Nutrition research | Genotype & phenotype | Rodents | Collagen | Gangrene | Insulin resistance | Diabetes | Apoptosis
Journal Article
Journal of Gastroenterology and Hepatology, ISSN 0815-9319, 02/2012, Volume 27, Issue 2, pp. 341 - 350
Background and Aims:  Lipid accumulation precedes hepatocellular injury and liver inflammation in non‐alcoholic steatohepatitis (NASH). The peroxisome... 
inflammation | non‐alcoholic fatty liver disease | non‐alcoholic steatohepatitis experimental | peroxisome proliferator activated receptor alpha | monocyte chemoattractant protein 1 | adipose tissue | experimental | steatosis | PPAR-ALPHA | KAPPA-B ACTIVATION | non-alcoholic fatty liver disease | FATTY LIVER-DISEASE | FENOFIBRATE | non-alcoholic steatohepatitis experimental | INJURY | MODEL | NASH | INSULIN-RESISTANCE | OBESE | GASTROENTEROLOGY & HEPATOLOGY | Diabetes Mellitus - pathology | Liver - pathology | Diabetes Mellitus - genetics | Fatty Liver - pathology | Endoplasmic Reticulum - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Metabolic Syndrome - metabolism | Metabolic Syndrome - drug therapy | Adipose Tissue - metabolism | Liver - drug effects | Time Factors | Endoplasmic Reticulum - drug effects | Inflammation Mediators - metabolism | Non-alcoholic Fatty Liver Disease | Female | Chemokine CCL2 - metabolism | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Fatty Liver - genetics | Fatty Liver - metabolism | Enzyme-Linked Immunosorbent Assay | Liver - metabolism | Diabetes Mellitus - drug therapy | Insulin Resistance | Diabetes Mellitus - metabolism | Pyrimidines - pharmacology | Blotting, Western | Fatty Liver - drug therapy | Gene Expression Regulation - drug effects | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Metabolic Syndrome - genetics | Lipid Metabolism - drug effects | Macrophages - drug effects | Mice, Inbred NOD | NF-kappa B - pharmacology | PPAR alpha - agonists | Mice | Enzyme Activation | Metabolic Syndrome - pathology | Oxidative Stress - drug effects | PPAR alpha - metabolism | Adipose Tissue - drug effects
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 2002, Volume 110, Issue 2, pp. 193 - 202
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2015, Volume 148, Issue 4, pp. S-583 - S-583
Journal Article
Liver International, ISSN 1478-3223, 08/2014, Volume 34, Issue 7, pp. 1084 - 1093
Journal Article
Journal of Gastroenterology and Hepatology (Australia), ISSN 0815-9319, 2009, Volume 24, Issue 3, pp. 443 - 452
We examined extrinsic and intrinsic (endogenous) mitochondrial apoptosis pathways in experimental non-alcoholic steatohepatitis (NASH). To assess extrinsic... 
Cell death pathways | TRAIL-R killer/DR5 | Methionine and choline deficiency | Mitochondria | Insulin-like growth factor-1 | TNF receptors | P53 | HEPATOCYTE APOPTOSIS | ACIDS | DR5 | mitochondria | FATTY LIVER-DISEASE | insulin-like growth factor-1 | NONALCOHOLIC STEATOHEPATITIS | cell death pathways | p53 | PATHOGENESIS | HEPATIC INFLAMMATION | NECROSIS | methionine and choline deficiency | MICE | TNF-ALPHA | GASTROENTEROLOGY & HEPATOLOGY | TRAIL-R killer | NF-KAPPA-B | Liver - pathology | Liver - enzymology | Fatty Liver - pathology | Mitochondria, Liver - metabolism | Caspase 3 - metabolism | Choline Deficiency - complications | Male | Alanine Transaminase - blood | Receptors, TNF-Related Apoptosis-Inducing Ligand - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | RNA, Messenger - metabolism | fas Receptor - metabolism | Tumor Suppressor Protein p53 - genetics | Time Factors | Receptors, Tumor Necrosis Factor, Type II - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Nutritional Status | Receptors, TNF-Related Apoptosis-Inducing Ligand - genetics | BH3 Interacting Domain Death Agonist Protein - metabolism | Disease Models, Animal | Methionine - deficiency | Receptors, Tumor Necrosis Factor - metabolism | Fatty Liver - metabolism | Mitochondria, Liver - pathology | Liver - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Tumor Suppressor Protein p53 - metabolism | Mitochondria, Liver - enzymology | Animals | Mice | bcl-X Protein - metabolism | Insulin-Like Growth Factor I - metabolism | Apoptosis | Fatty Liver - etiology | BH3 Interacting Domain Death Agonist Protein, metabolism | Receptors, Tumor Necrosis Factor, Type II, metabolism | Fatty Liver, pathology | Mitochondria, Liver, metabolism | Receptors, Tumor Necrosis Factor, metabolism | Cyclin-Dependent Kinase Inhibitor p21, metabolism | Insulin-Like Growth Factor I, metabolism | Caspase 3, metabolism | Antigens, CD95, metabolism | RNA, Messenger, metabolism | Tumor Suppressor Protein p53, metabolism | Methionine, deficiency | Choline Deficiency, complications | Tumor Suppressor Protein p53, genetics | bcl-X Protein, metabolism | Mitochondria, Liver, pathology | Liver, pathology | Alanine Transaminase, blood | Liver, metabolism | Fatty Liver, metabolism | Receptors, TNF-Related Apoptosis-Inducing Ligand, metabolism | Liver, enzymology | Fatty Liver, etiology | Mitochondria, Liver, enzymology | Receptors, Tumor Necrosis Factor, Type I, metabolism | Receptors, TNF-Related Apoptosis-Inducing Ligand, genetics | Messenger RNA | Choline | Methionine | Mitochondrial DNA | Tumor proteins | Peptide hormones | Growth factors
Journal Article
Journal of Gastroenterology and Hepatology (Australia), ISSN 0815-9319, 2009, Volume 24, Issue 10, pp. 1658 - 1668
Background and Aims: We previously reported that steatohepatitis develops in obese, hypercholesterolemic, diabetic foz/foz mice fed a high-fat (HF) diet for 12... 
Lipid partitioning | Metabolic syndrome | Non-alcoholic fatty liver disease; non-alcoholic steatohepatitis | Adiponectin | TARGET | adiponectin | non-alcoholic fatty liver disease | lipid partitioning | INJURY | FAILURE | metabolic syndrome | NONALCOHOLIC FATTY LIVER | PLASMA ADIPONECTIN | PIOGLITAZONE | INSULIN-RESISTANCE | DISEASE | TNF-ALPHA | GASTROENTEROLOGY & HEPATOLOGY | SEVERITY | non-alcoholic steatohepatitis | Dietary Fats - metabolism | Liver - pathology | Fatty Liver - pathology | Obesity - genetics | Adipose Tissue - metabolism | Biological Transport | CD36 Antigens - metabolism | Hypercholesterolemia - pathology | Time Factors | Mice, Mutant Strains | Diabetes Complications - genetics | Diabetes Complications - pathology | Lipogenesis | Fatty Acids - metabolism | Disease Models, Animal | Fatty Liver - genetics | Fatty Liver - metabolism | Obesity - complications | Diabetes Complications - metabolism | Liver - metabolism | Adipose Tissue - pathology | Genotype | Biomarkers - blood | DNA-Binding Proteins - genetics | Diabetes Complications - etiology | Disease Progression | Obesity - metabolism | Obesity - pathology | Triglycerides - metabolism | Adiponectin - blood | Phenotype | Animals | Hypercholesterolemia - complications | Mice | Hypercholesterolemia - genetics | Hypercholesterolemia - metabolism | Fatty Liver - etiology | Obesity | Liver diseases | Hypercholesterolemia | Synthesis | Analysis | Dietary fat | Diabetes | Peptide hormones | Fatty acids
Journal Article
Journal of Hospital Medicine, ISSN 1553-5592, 12/2014, Volume 9, Issue 12, pp. 797 - 799
Journal Article
Clinical Journal of Sport Medicine, ISSN 1050-642X, 09/2008, Volume 18, Issue 5, pp. 432 - 437
OBJECTIVE:To characterize differences in the perception and understanding of the placebo effect between sports physicians, coaches, athletes, and sports... 
Perception | Athlete | Coach | Placebo effect | Sports medicine | SPORT SCIENCES | PHYSIOLOGY | ERGOGENIC AID | ENDURANCE | athlete | PERFORMANCE | EXERCISE | perception | MAMMARY-ARTERY LIGATION | placebo effect | sports medicine | ORTHOPEDICS | coach | Young Adult | Placebo Effect | Sports Medicine | Humans | Middle Aged | Adolescent | Adult | Female | Male | Surveys and Questionnaires | Australia | Health Knowledge, Attitudes, Practice
Journal Article
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