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2006, 2nd ed., ISBN 9781841844633, xxi, 370
Book
Neurology, ISSN 0028-3878, 03/2017, Volume 88, Issue 10, pp. 928 - 929
Journal Article
Journal Article
Annals of Neurology, ISSN 0364-5134, 2014, Volume 76, Issue 1, pp. 120 - 133
Journal Article
Science Translational Medicine, ISSN 1946-6234, 10/2018, Volume 10, Issue 465, p. eaau6414
Amyotrophic lateral sclerosis (ALS) is a fatal neurological disease caused by degeneration of motor neurons leading to rapidly progressive paralysis. About 10%... 
NEURODEGENERATIVE DISEASE | NONHUMAN-PRIMATES | MEDICINE, RESEARCH & EXPERIMENTAL | EXTENDS SURVIVAL | HEMOPHILIA-B | MOUSE MODEL | FACTOR-IX | MUTANT SOD1 | GENE-THERAPY | AMYOTROPHIC-LATERAL-SCLEROSIS | MOTOR-NEURON DEATH | CELL BIOLOGY | Index Medicus
Journal Article
Nature Neuroscience, ISSN 1097-6256, 06/2018, Volume 21, Issue 8, pp. 1 - 1138
In the version of this article initially published, the footnote number 17 was missing from the author list for the two authors who contributed equally. Also,... 
Frontotemporal dementia
Journal Article
Genome Research, ISSN 1088-9051, 05/2019, Volume 29, Issue 5, pp. 809 - 818
Large-scale sequencing efforts in amyotrophic lateral sclerosis (ALS) have implicated novel genes using gene-based collapsing methods. However, pathogenic... 
GENETIC-VARIATION | GALECTIN-1 | FUS | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | TDP-43 | BIOCHEMISTRY & MOLECULAR BIOLOGY | FRONTOTEMPORAL DEMENTIA | P0071 | GENETICS & HEREDITY | AMYOTROPHIC-LATERAL-SCLEROSIS | MUTATIONS | GENOME
Journal Article
Annals of Neurology, ISSN 0364-5134, 09/2013, Volume 74, Issue 3, pp. A15 - A16
Journal Article
Neuron, ISSN 0896-6273, 12/2015, Volume 88, Issue 5, pp. 902 - 909
A non-coding hexanucleotide repeat expansion in the gene is the most common mutation associated with familial amyotrophic lateral sclerosis (ALS) and... 
RAN translation | transgenic mice | Amyotrophic lateral sclerosis (ALS) | frontotemporal dementia (FTD) | neurodegeneration | microRNA | repeat expansions | C9ORF72 | RNA foci | Repeat expansions | MicroRNA | Neurodegeneration | Transgenic mice | Frontotemporal dementia (FTD) | DEMENTIA | ALS | AMYOTROPHIC-LATERAL-SCLEROSIS | SPECTRUM | MODEL | FRONTOTEMPORAL LOBAR DEGENERATION | TRANSLATION | SENSE | NEUROSCIENCES | PATHOLOGICAL FEATURES | Age Factors | Amyotrophic Lateral Sclerosis - physiopathology | DNA Repeat Expansion - genetics | Humans | MicroRNAs - metabolism | Frontotemporal Dementia - mortality | Cerebral Cortex - cytology | Brain - metabolism | Chromosomes, Artificial, Bacterial - genetics | Chromosomes, Artificial, Bacterial - metabolism | Neurons - physiology | C9orf72 Protein | Dipeptides - metabolism | Neurons - drug effects | Disease Models, Animal | Frontotemporal Dementia - pathology | Frontotemporal Dementia - genetics | Frontotemporal Dementia - physiopathology | Gene Expression Regulation - genetics | Amyotrophic Lateral Sclerosis - genetics | Cells, Cultured | Genotype | Mice, Transgenic | Amyotrophic Lateral Sclerosis - mortality | Nerve Tissue Proteins - metabolism | Proteins - genetics | Amyotrophic Lateral Sclerosis - pathology | Animals | Brain - pathology | In Vitro Techniques | Dipeptides - genetics | Proteins | Neurosciences | Proline | Genetic engineering | Glycine | Dementia | Analysis | Genetic translation | Plasmids | Neurons | Rodents | Cloning | Amyotrophic lateral sclerosis | Behavior | Artificial chromosomes | Expansion | Index Medicus
Journal Article
Journal Article
Science, ISSN 0036-8075, 7/2012, Volume 337, Issue 6093, pp. 481 - 484
Journal Article