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Journal of Clinical Investigation, ISSN 0021-9738, 02/2010, Volume 120, Issue 2, pp. 457 - 471
Journal Article
Immunity, ISSN 1074-7613, 03/2012, Volume 36, Issue 3, pp. 362 - 373
Although Th17 cells are known to promote tissue inflammation and autoimmunity, their role during cancer progression remains elusive. Here, we showed that... 
GROWTH-FACTOR-BETA | EFFECTOR T-CELLS | TGF-BETA | T(H)17 CELLS | ROR-GAMMA-T | SIGNALING PATHWAY | IL-17 | ADENOSINE | GENERATION | IMMUNOLOGY | CANCER | 5'-Nucleotidase - genetics | RNA, Small Interfering - genetics | Antigens, CD - genetics | DNA-Binding Proteins - metabolism | Th17 Cells - drug effects | T-Lymphocytes - metabolism | Th17 Cells - metabolism | Lymphocytes, Tumor-Infiltrating - metabolism | Transcription Factors - immunology | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Promoter Regions, Genetic | DNA-Binding Proteins - immunology | Mice, Inbred C57BL | Gene Expression Regulation | Mice, Transgenic | Binding Sites - genetics | Mice, Knockout | Transcription Factors - metabolism | Transforming Growth Factor beta - pharmacology | Animals | Interleukin-6 - pharmacology | Apyrase - genetics | Th17 Cells - immunology | T-Lymphocytes - immunology | Mice | STAT3 Transcription Factor - immunology | STAT3 Transcription Factor - antagonists & inhibitors | Lymphocytes, Tumor-Infiltrating - immunology | Autoimmunity | DNA binding proteins | T cells | Transforming growth factors | Proteins | Transcription factors | Statistical analysis | Plasmids | Rodents | Cytotoxicity | Deoxyribonucleic acid--DNA | Immune system | Cancer | 5'-Nucleotidase | T-Lymphocytes | Interleukin-6 | Life Sciences | Lymphocytes, Tumor-Infiltrating | Immunology | Apyrase | RNA, Small Interfering | STAT3 Transcription Factor | Th17 Cells | Transcription Factors | Binding Sites | Transforming Growth Factor beta | Antigens, CD | DNA-Binding Proteins
Journal Article
Nature Immunology, ISSN 1529-2908, 07/2015, Volume 16, Issue 8, pp. 859 - 870
The receptor NLRP3 is involved in the formation of the NLRP3 inflammasome that activates caspase-1 and mediates the release of interleukin 1β (IL-1β) and... 
NIH 3T3 Cells | Asthma - metabolism | Inflammasomes - metabolism | Oligonucleotide Array Sequence Analysis | NLR Family, Pyrin Domain-Containing 3 Protein | Interferon Regulatory Factors - metabolism | Gene Expression Profiling | Th2 Cells - immunology | Promoter Regions, Genetic - genetics | CD4-Positive T-Lymphocytes - immunology | Cell Differentiation - genetics | Signal Transduction - immunology | Neoplasms, Experimental - immunology | Asthma - immunology | Neoplasms, Experimental - genetics | Trans-Activators - genetics | Female | Interleukin-4 - genetics | Carrier Proteins - immunology | Trans-Activators - immunology | Interleukin-4 - metabolism | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Interferon Regulatory Factors - genetics | Gene Expression Regulation, Neoplastic - immunology | Signal Transduction - genetics | Protein Binding - immunology | Reverse Transcriptase Polymerase Chain Reaction | Th2 Cells - metabolism | Asthma - genetics | Blotting, Western | Inflammasomes - genetics | Mice, Knockout | Carrier Proteins - genetics | Cell Differentiation - immunology | Interleukin-4 - immunology | Interferon Regulatory Factors - immunology | Animals | Carrier Proteins - metabolism | Inflammasomes - immunology | Cell Line, Tumor | Trans-Activators - metabolism | Mice | Neoplasms, Experimental - metabolism | Promoter Regions, Genetic - immunology
Journal Article
Nature Medicine, ISSN 1078-8956, 01/2013, Volume 19, Issue 1, pp. 57 - 64
Chemotherapeutic agents are widely used for cancer treatment. In addition to their direct cytotoxic effects, these agents harness the host's immune system,... 
MEDICINE, RESEARCH & EXPERIMENTAL | CANCER-CHEMOTHERAPY | ANGIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GEMCITABINE | BETA | CELL BIOLOGY | 5-FLUOROURACIL | INHIBITION | MICROENVIRONMENT | ANTITUMOR IMMUNITY | BEARING MICE | T-CELLS | Neoplasms - metabolism | Cell Proliferation | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Cathepsin B - genetics | Humans | Caspase 1 - metabolism | Deoxycytidine - pharmacology | Signal Transduction - immunology | Neoplasms - genetics | RNA Interference | Female | Antineoplastic Agents - pharmacology | Interleukin-1beta - biosynthesis | Dendritic Cells - metabolism | bcl-2-Associated X Protein - genetics | Interleukin-17 - secretion | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Cathepsin B - metabolism | Mice, Knockout | Carrier Proteins - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Caspase 1 - genetics | Cell Line, Tumor | Fluorouracil - pharmacology | Mice | Mice, Inbred BALB C | RNA, Small Interfering | Receptors, Interleukin-1 - antagonists & inhibitors | Deoxycytidine - analogs & derivatives | Chemotherapy | Gemcitabine | Cathepsins | Physiological aspects | Dosage and administration | Research | Health aspects | Fluorouracil | Cancer | Cells | Inflammatory diseases | Tumors | Engineering Sciences | Automatic
Journal Article
Breast Cancer Research and Treatment, ISSN 0167-6806, 1/2011, Volume 125, Issue 1, pp. 65 - 72
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 02/2013, Volume 133, Issue 2, pp. 499 - 508
Dacarbazine (DTIC) is a cytotoxic drug widely used for melanoma treatment. However, the putative contribution of anticancer immune responses in the efficacy of... 
METASTATIC MELANOMA | IMMUNOGENICITY | IMMUNOTHERAPY | RECEPTOR | ANTITUMOR IMMUNITY | SUPPRESSOR-CELLS | EXPRESSION | CYTOTOXICITY | RADIATION | CHEMOTHERAPY | DERMATOLOGY | Skin Neoplasms - drug therapy | Humans | Melanoma, Experimental - drug therapy | NK Cell Lectin-Like Receptor Subfamily K - immunology | Antineoplastic Agents, Alkylating - pharmacology | Interferon-gamma - metabolism | Melanoma, Experimental - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Dacarbazine - pharmacology | CD8-Positive T-Lymphocytes - metabolism | Killer Cells, Natural - immunology | Disease Models, Animal | Skin Neoplasms - pathology | Skin Neoplasms - immunology | Mice, Inbred C57BL | Melanoma, Experimental - pathology | Up-Regulation - drug effects | Animals | Up-Regulation - immunology | Mice, Nude | CD8-Positive T-Lymphocytes - drug effects | Interferon-gamma - immunology | Cell Line, Tumor | Ligands | Killer Cells, Natural - drug effects | Mice | Killer Cells, Natural - metabolism | CD8-Positive T-Lymphocytes - immunology | Animal models | CD8 antigen | Tumor cells | Melanoma | Cytotoxicity | Lymphocytes T | Immunosuppressive agents | Dacarbazine | NKG2 antigen | Cell activation | Immunosuppression | Major histocompatibility complex | Immunogenicity | Natural killer cells | Cancer | Up-Regulation | Interferon-gamma | NK Cell Lectin-Like Receptor Subfamily K | Killer Cells, Natural | CD8-Positive T-Lymphocytes | Life Sciences | Immunology | Melanoma, Experimental | Skin Neoplasms | Antineoplastic Agents, Alkylating
Journal Article
Bulletin du Cancer, ISSN 0007-4551, 2014, Volume 101, Issue 6, pp. 605 - 607
Journal Article
Medecine/Sciences, ISSN 0767-0974, 2014, Volume 30, Issue 4, pp. 429 - 435
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e65181
Bleomycin (BLM) is an anticancer drug currently used for the treatment of testis cancer and Hodgkin lymphoma. This drug triggers cancer cell death via its... 
CALRETICULIN EXPOSURE | LUNG | RESPONSES | MESSENGER-RNA | MULTIDISCIPLINARY SCIENCES | VINBLASTINE | MECHANISMS | SUPPRESSOR-CELLS | EXPRESSION | PULMONARY-FIBROSIS | CUTTING EDGE | Immunity, Innate - drug effects | Humans | T-Lymphocytes, Regulatory - pathology | T-Lymphocytes, Regulatory - immunology | Immunity - drug effects | T-Lymphocytes, Regulatory - drug effects | Animals | CD8-Positive T-Lymphocytes - drug effects | Cell Line, Tumor | Bleomycin - pharmacology | Female | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Mice | Cell Death - drug effects | CD8-Positive T-Lymphocytes - immunology | Transforming Growth Factor beta - metabolism | Care and treatment | Bleomycin | Immune response | Cell death | Biochemistry | Interferon | B cells | Non-Hodgkin's lymphomas | Transforming growth factors | T cells | Cancer | Cell proliferation | Disease | CD8 antigen | Transforming growth factor-b | Cytotoxicity | Lymphocytes T | Chaperones | Cancer therapies | Immunity | Anticancer properties | Calreticulin | Lymphocytes | Rodents | Foxp3 protein | Growth factors | Antigens | Dendritic cells | Immunoregulation | Secretion | Tumor cells | Mortality | Radiation therapy | Lymphoma | Chemotherapy | Pulmonary fibrosis | Immunogenicity | Antitumor activity | Hodgkin's disease | Phagocytosis | Apoptosis | Life Sciences | Immunology
Journal Article
Cancer Research, ISSN 0008-5472, 06/2013, Volume 73, Issue 12, pp. 3578 - 3590
Journal Article
Genomics Data, ISSN 2213-5960, 09/2015, Volume 5, Issue C, pp. 314 - 315
The Nod-like receptor NLRP3 is involved in the formation of NLRP3. Up to now, the immunological functions of NLRP3 independently of inflammasome is unclear. In... 
Th2 differentiation | NLRP3 | Transcriptome
Journal Article
Nature immunology, ISSN 1529-2908, 08/2014, Volume 15, Issue 8, pp. 758 - 766
Journal Article
M S-MEDECINE SCIENCES, ISSN 0767-0974, 04/2014, Volume 30, Issue 4, pp. 429 - 435
Immunosuppression is a mechanism developed by cancer cells to help them escape the immune system. Immunosuppression involves expansion of various cell types... 
MEDICINE, RESEARCH & EXPERIMENTAL | SUBPOPULATIONS | DENDRITIC CELLS | INFLAMMATION | MECHANISMS | SUPPRESSOR-CELLS | GROWTH-FACTOR | INTERLEUKIN-6 | CANCER | TH17 | T-CELLS | Immune Tolerance | Tumor Microenvironment | T-Lymphocytes - immunology | Neoplasms - immunology | Humans | Cytokines - physiology
Journal Article