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1. Full Text Hypoxia induces the breast cancer stem cell phenotype by HIF-dependent and ALKBH5-mediated m⁶A-demethylation of NANOG mRNA
by Chuanzhao Zhang and Debangshu Samanta and Haiquan Lu and John W. Bullen and Huimin Zhang and Ivan Chen and Xiaoshun He and Gregg L. Semenza
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2016, Volume 113, Issue 14, pp. E2047 - E2056
⁶-methyladenosine (m⁶A) modification of mRNA plays a role in regulating embryonic stem cell pluripotency. However, the physiological signals that determine the... 
Hypoxia-inducible factors | Self-renewal | Tumorigenesis | Metastasis | Pluripotency factors | Nanog Homeobox Protein - genetics | Basic Helix-Loop-Helix Transcription Factors - physiology | Humans | RNA, Messenger - metabolism | Breast Neoplasms - metabolism | Gene Knockdown Techniques | AlkB Homolog 5, RNA Demethylase - genetics | Cell Hypoxia | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Cell Line, Tumor | Neoplastic Stem Cells - pathology | Female | Catalysis | Methylation | AlkB Homolog 5, RNA Demethylase - physiology | Biological Sciences | PNAS Plus | metastasis | hypoxia-inducible factors | tumorigenesis | self-renewal | pluripotency factors
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2. Full Text Cross-talk between GlcNAcylation and phosphorylation: roles in insulin resistance and glucose toxicity
by Copeland, Ronald J and Bullen, John W and Hart, Gerald W
AJP - Endocrinology and Metabolism, ISSN 0193-1849, 07/2008, Volume 295, Issue 1, pp. E17 - E28
Department of Biological Chemistry, Johns Hopkins University School of Medicine, Baltimore, Maryland Submitted 10 March 2008 ; accepted in final form 25 April... 
O-linked β-N- acetylglucosamine transferase | Diabetes | β-N-acetylglucosaminidase | Hexosamine biosynthesis | O-linked β-N-acetylglucosamine | 3T3-L1 ADIPOCYTES | PHYSIOLOGY | NITRIC-OXIDE SYNTHASE | MUTATIONAL HOT-SPOT | BETA-D-GLUCOSAMINIDASE | beta-N-acetylglucosaminidase | POSTTRANSLATIONAL MODIFICATIONS | O-GLCNAC TRANSFERASE | O-linked beta-N-acetylglucosamine transferase | GLOMERULAR MESANGIAL CELLS | O-linked beta-N-acetylglucosamine | hexosamine biosynthesis | ENDOCRINOLOGY & METABOLISM | PROTEINS IN-VIVO | diabetes | NUCLEOCYTOPLASMIC PROTEINS | LINKED N-ACETYLGLUCOSAMINE | Amino Acid Sequence | Phosphorylation | Signal Transduction | Humans | Diabetes Mellitus, Type 2 - enzymology | Molecular Sequence Data | Diabetes Mellitus, Type 2 - metabolism | Hexosamines - metabolism | N-Acetylglucosaminyltransferases - metabolism | Acetylglucosamine - metabolism | Animals | Acetylglucosaminidase - metabolism | Insulin Resistance - physiology | Acylation | Index Medicus | O-linked β-N-acetylglucosamine transferase | Reviews
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3. Full Text Cross-talk between Two Essential Nutrient-sensitive Enzymes O-GlcNAc TRANSFERASE ( OGT) AND AMP-ACTIVATED PROTEIN KINASE ( AMPK)
by Bullen, John W and Balsbaugh, Jeremy L and Chanda, Dipanjan and Shabanowitz, Jeffrey and Hunt, Donald F and Neumann, Dietbert and Hart, Gerald W
Journal of Biological Chemistry, ISSN 0021-9258, 04/2014, Volume 289, Issue 15, pp. 10592 - 10606
Background: OGT and AMPK collectively target hundreds of intracellular signaling processes, but no study has addressed whether they regulate each other.... 
Skeletal Muscle | Nutrient Sensing | Nuclear Translocation | Histones | O-GlcNAcylation | AMP-activated Kinase (AMPK) | O-GlcNAc | O-GlcNAc Transferase | GLCNACYLATION | MECHANISM | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLYCOSYLATION | MASS-SPECTROMETRY | COEXPRESSION | METABOLISM | DYNAMICS | INHIBITOR | INSIGHTS | Cell Line | AMP-Activated Protein Kinases - metabolism | Phosphorylation | Cell Proliferation | Signal Transduction | Humans | Substrate Specificity | Cytoplasm - metabolism | Muscle Fibers, Skeletal - metabolism | N-Acetylglucosaminyltransferases - metabolism | Acetylglucosamine - metabolism | Gene Expression Regulation, Enzymologic | Microscopy, Confocal | Animals | Cell Nucleus - metabolism | Mass Spectrometry | HEK293 Cells | Cell Differentiation | Mice | Protein Processing, Post-Translational | HeLa Cells | Glycobiology and Extracellular Matrices
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4. Full Text Chemotherapy triggers HIF-1–dependent glutathione synthesis and copper chelation that induces the breast cancer stem cell phenotype
by Haiquan Lu and Debangshu Samanta and Lisha Xiang and Huimin Zhang and Hongxia Hu and Ivan Chen and John W. Bullen and Gregg L. Semenza
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2015, Volume 112, Issue 33, pp. E4600 - E4609
Triple negative breast cancer (TNBC) accounts for 10–15% of all breast cancer but is responsible for a disproportionate share of morbidity and mortality... 
Tumor-initiating cells | Hypoxia-inducible factor | Chemotherapy resistance | Pluripotency factors | Paclitaxel | paclitaxel | PREDICTOR | MULTIDISCIPLINARY SCIENCES | tumor-initiating cells | PATTERNS | hypoxia-inducible factor | PLURIPOTENCY | pluripotency factors | HYPOXIA-INDUCIBLE FACTORS | HETEROGENEITY | chemotherapy resistance | INHIBITION | GROWTH | RESISTANCE | METABOLIC-REGULATION | ANTIPORTER | Oligonucleotides - genetics | Neoplasm Transplantation | Neoplastic Stem Cells - cytology | Phosphorylation | Glutathione - metabolism | Humans | Chelating Agents - chemistry | Gene Expression Regulation, Neoplastic | Gene Expression Profiling | MAP Kinase Kinase Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Antineoplastic Agents - chemistry | Mice, SCID | RNA, Messenger - metabolism | MAP Kinase Signaling System | Paclitaxel - chemistry | Phenotype | Animals | Copper - chemistry | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Triple Negative Breast Neoplasms - metabolism | Cell Line, Tumor | Female | Mice | Chemotherapy | Cancer cells | Breast cancer | Genetic aspects | Drug therapy | Observations | Health aspects | Methods | Glutathione | Cancer | Biological Sciences | PNAS Plus
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5. Full Text HIF-1 regulates CD47 expression in breast cancer cells to promote evasion of phagocytosis and maintenance of cancer stem cells
by Huimin Zhang and Haiquan Lu and Lisha Xiang and John W. Bullen and Chuanzhao Zhang and Debangshu Samanta and Daniele M. Gilkes and Jianjun He and Gregg L. Semenza
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2015, Volume 112, Issue 45, pp. E6215 - E6223
Increased expression of CD47 has been reported to enable cancer cells to evade phagocytosis by macrophages and to promote the cancer stem cell phenotype, but... 
LYSYL OXIDASE | TUMOR HYPOXIA | BONE METASTASES | MULTIDISCIPLINARY SCIENCES | tumor-initiating cells | IDENTIFICATION | antitumor immunity | HEPATOCELLULAR-CARCINOMA | immune evasion | APOPTOTIC CELLS | THERAPEUTIC TARGET | SURFACE CALRETICULIN | tumor microenvironment | "don't eat me" signal | UP-REGULATION | HYPOXIA-INDUCIBLE FACTOR-1 | RNA, Small Interfering - genetics | Humans | Phagocytosis - physiology | Immunoblotting | CD47 Antigen - metabolism | DNA Primers | Tumor Escape - physiology | Hypoxia-Inducible Factor 1 - metabolism | Breast Neoplasms - metabolism | Analysis of Variance | Chromatin Immunoprecipitation | Luciferases | Cell Line, Tumor | Female | Gene Expression Regulation, Neoplastic - drug effects | Gene Expression Regulation, Neoplastic - physiology | Real-Time Polymerase Chain Reaction | Neoplastic Stem Cells - physiology | Hypoxia-Inducible Factor 1 - pharmacology | Transcriptional coactivators | Complications and side effects | Stem cell research | Care and treatment | Analysis | Influence | Breast cancer | Research | Gene expression | “don’t eat me” signal | Biological Sciences | PNAS Plus
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6. Full Text Regulation of adiponectin and its receptors in response to development of diet-induced obesity in mice
by John W. Bullen, Jr and Susann Bluher and Theodoros Kelesidis and Christos S. Mantzoros
American Journal of Physiology - Endocrinology And Metabolism, ISSN 0193-1849, 04/2007, Volume 292, Issue 4, pp. 1079 - 1086
Adiponectin and its receptors play an important role in energy homeostasis and insulin resistance, but their regulation remains to be fully elucidated. We... 
Insulin resistance | Diet-induced obesity | Adiponectin receptor 1 | Adiponectin receptor 2 | Adiponectin | adiponectin | PHYSIOLOGY | adiponectin receptor 1 | ACTIVATED PROTEIN-KINASE | adiponectin receptor 2 | diet-induced obesity | HUMAN SKELETAL-MUSCLE | ZUCKER RATS | TYPE-2 DIABETIC-PATIENTS | PLASMA ADIPONECTIN | FATTY-ACID OXIDATION | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | LEPTIN EXPRESSION | insulin resistance | ADIPOSE-TISSUE | Body Composition | Body Weight | Disease Susceptibility | Mice, Inbred A | Mice, Inbred C57BL | Receptors, Cell Surface - metabolism | Muscle, Skeletal - metabolism | Obesity - physiopathology | Insulin - blood | RNA, Messenger - metabolism | Obesity - metabolism | Adipose Tissue - metabolism | Receptors, Adiponectin | Animals | Leptin - blood | Adiponectin - genetics | Dietary Fats - administration & dosage | Adiponectin - metabolism | Obesity - etiology | Mice | Blood Glucose - metabolism | Aging - metabolism | Receptors, Cell Surface - genetics
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7. Full Text Circulating Adiponectin and Expression of Adiponectin Receptors in Human Skeletal Muscle: Associations with Metabolic Parameters and Insulin Resistance and Regulation by Physical Training
by Blüher, Matthias and Bullen, John W and Lee, Jennifer H and Kralisch, Susan and Fasshauer, Mathias and Klöting, Nora and Niebauer, Josef and Schön, Michael R and Williams, Catherine J and Mantzoros, Christos S
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 06/2006, Volume 91, Issue 6, pp. 2310 - 2316
Context: Adiponectin, an adipocyte-secreted hormone, is associated with insulin resistance and the metabolic syndrome. Objective: The physiological regulation... 
PROTEIN | FATTY-ACID OXIDATION | PLASMA-CONCENTRATIONS | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | SENSITIVITY | INCREASES | IMPAIRED GLUCOSE-TOLERANCE | NECROSIS-FACTOR-ALPHA | EXERCISE | ADIPOSE | Body Mass Index | Glucose Tolerance Test | Cross-Sectional Studies | Humans | Middle Aged | Insulin Resistance | Male | Muscle, Skeletal - metabolism | Adiponectin - blood | Receptors, Adiponectin | Exercise | Leptin - blood | Lipids - blood | Adult | Female | Receptors, Cell Surface - genetics
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8. Full Text Hypoxia-inducible factor 1 mediates TAZ expression and nuclear localization to induce the breast cancer stem cell phenotype
by Xiang, Lisha and Gilkes, Daniele M and Hu, Hongxia and Takano, Naoharu and Luo, Weibo and Lu, Haiquan and Bullen, John W and Samanta, Debangshu and Liang, Houjie and Semenza, Gregg L
Oncotarget, ISSN 1949-2553, 2014, Volume 5, Issue 24, pp. 12509 - 12527
Intratumoral hypoxia, which is associated with breast cancer metastasis and patient mortality, increases the percentage of breast cancer stem cells (BCSCs) but... 
Aldefluor assay | Targeted therapy | Basal-like breast cancer | Mammospheres | Triple-negative breast cancer | Tumor Suppressor Proteins - metabolism | Signal Transduction | Humans | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - biosynthesis | Transcription Factors - genetics | Mice, SCID | Hypoxia-Inducible Factor 1 - metabolism | Transcription Factors - metabolism | Neoplasm Metastasis | Phenotype | Animals | Cell Nucleus - metabolism | Heterografts | Transfection | MCF-7 Cells | Breast Neoplasms - pathology | Tumor Suppressor Proteins - genetics | Neoplastic Stem Cells - pathology | Female | Mice | Protein-Serine-Threonine Kinases - metabolism
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9. Full Text HIF-1α and TAZ serve as reciprocal co-activators in human breast cancer cells
by Xiang, Lisha and Gilkes, Daniele M and Hu, Hongxia and Luo, Weibo and Bullen, John W and Liang, Houjie and Semenza, Gregg L
Oncotarget, ISSN 1949-2553, 2015, Volume 6, Issue 14, pp. 11768 - 11778
MCF-7 cells | Hypoxia-inducible factor 1 | MDA-MB-231 cells | Breast cancer progression
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10. Full Text Protein kinase A-dependent phosphorylation stimulates the transcriptional activity of hypoxia-inducible factor
by Bullen, John W and Tchernyshyov, Irina and Holewinski, Ronald J and Devine, Lauren and Wu, Fan and Venkatraman, Vidya and Kass, David L and Cole, Robert N and Van Eyk, Jennifer and Semenza, Gregg L
Science Signaling, ISSN 1945-0877, 05/2016, Volume 9, Issue 430
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11. Cross-talk between GlcNAcylation and phosphorylation: roles in insulin resistance and glucose toxicity
by Ronald J Copeland and John W Bullen and Gerald W Hart
American Journal of Physiology, ISSN 0193-1849, 07/2008, Volume 295, Issue 1, p. E17
  O-linked β-N-acetylglucosamine (O-GlcNAc) is a dynamic posttranslational modification that, analogous to phosphorylation, cycles on and off serine and/or... 
Proteins | Toxicity | Proteomics | Glucose | Metabolism | Insulin | Endocrinology
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12. Full Text Protein kinase A-dependent phosphorylation stimulates the transcriptional activity of hypoxia-inducible factor 1
by Bullen, JW and Tchernyshyov, I and Holewinski, RJ and DeVine, L and Wu, F and Venkatraman, V and Kass, DL and Cole, RN and Van Eyk, J and Semenza, GL
SCIENCE SIGNALING, ISSN 1945-0877, 05/2016, Volume 9, Issue 430, pp. ra56 - ra56
Hypoxia-inducible factor 1 (HIF-1) activates the transcription of genes encoding proteins that enable cells to adapt to reduced O-2 availability. Proteins... 
CELLS | DATABASE | TANDEM | PATHWAY | TRANSACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | DEGRADATION | HIF-1-ALPHA | BINDING | MASS-SPECTROMETRY | STATISTICAL-MODEL | CELL BIOLOGY | Cyclic AMP-Dependent Protein Kinases - metabolism | Phosphorylation | Humans | Apyrase - metabolism | Glutathione Transferase - metabolism | Disease Progression | GPI-Linked Proteins - metabolism | Antigens, CD - metabolism | Immunosuppression | Animals | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Myocytes, Cardiac - metabolism | Protein Binding | Transcription, Genetic | Mice | HeLa Cells | 5'-Nucleotidase - metabolism | Cyclic AMP - metabolism
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13. Hypoxia induces the breast cancer stem cell phenotype by HIF-dependent and ALKBH5-mediated m^sup 6^A-demethylation of NANOG mRNA
by Chuanzhao Zhang and Debangshu Samanta and Haiquan Lu and John W Bullen and Huimin Zhang and Ivan Chen and Xiaoshun He and Gregg L Semenza
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 04/2016, Volume 113, Issue 14, p. E2047
  N...-methyladenosine (m...A) modification of mRNA plays a role in regulating embryonic stem cell pluripotency. However, the physiological signals that... 
Genotype & phenotype | Stem cells | Hypoxia | Breast cancer | Ribonucleic acid--RNA | Gene expression
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14. Chemotherapy triggers HIF-1-dependent glutathione synthesis and copper chelation that induces the breast cancer stem cell phenotype
by Haiquan Lu and Debangshu Samanta and Lisha Xiang and Huimin Zhang and Hongxia Hu and Ivan Chen and John W Bullen and Gregg L Semenza
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 08/2015, Volume 112, Issue 33, p. E4600
  Triple negative breast cancer (TNBC) accounts for 10-15% of all breast cancer but is responsible for a disproportionate share of morbidity and mortality... 
Genotype & phenotype | Chemotherapy | Stem cells | Breast cancer | Biosynthesis | Metastasis | Tumors
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15. Chemotherapy triggers HIF-1-dependent glutathione synthesis and copper chelation that induces the breast cancer stem cell phenotype
by Lu, Haiquan and Samanta, Debangshu and Xiang, Lisha and Zhang, Huimin and Hu, Hongxia and Chen, Ivan and Bullen, John W and Semenza, Gregg L
Proceedings of the National Academy of Sciences, USA, ISSN 0027-8424, 08/2015, Volume 112, Issue 33, pp. E4600 - E4600
Triple negative breast cancer (TNBC) accounts for 10-15% of all breast cancer but is responsible for a disproportionate share of morbidity and mortality... 
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16. Full Text Circulating resistin in lean, obese, and insulin-resistant mouse models: lack of association with insulinemia and glycemia
by Jennifer H. Lee and John W. Bullen, Jr and Violeta L. Stoyneva and Christos S. Mantzoros
American Journal of Physiology - Endocrinology And Metabolism, ISSN 0193-1849, 03/2005, Volume 288, Issue 3, pp. 625 - 632
Resistin is an adipocyte-secreted hormone proposed to link obesity with insulin resistance and diabetes, but no previous study has performed a joint... 
Uncoupling protein-diphtheria toxin A chain | Obesity | Diet-induced obesity | MTII | Ciliary neurotrophic factor analog | Tumor necrosis factor-α | SYSTEM | PHYSIOLOGY | HYPOTHALAMIC NEUROPEPTIDE-Y | PROTEIN | SERUM RESISTIN | RECEPTOR | diet-induced obesity | tumor necrosis factor-alpha | BODY-FAT | ciliary neurotrophic factor analog | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | uncoupling protein-diphtheria toxin A chain | MICE | obesity | ADIPOSE-TISSUE | Blood Glucose - physiology | Multivariate Analysis | alpha-MSH - pharmacology | Ion Channels | Gene Expression - genetics | Tumor Necrosis Factor-alpha - genetics | Body Weight - drug effects | Male | Insulin - blood | Mitochondrial Proteins | RNA, Messenger - metabolism | Hormones, Ectopic - blood | Adipose Tissue - metabolism | Insulin Resistance - physiology | Ciliary Neurotrophic Factor - pharmacology | Peptide Fragments - genetics | Diphtheria Toxin - genetics | Disease Models, Animal | Resistin | Obesity - chemically induced | Membrane Proteins - genetics | Mice, Inbred C57BL | RNA, Messenger - genetics | Mice, Transgenic | Energy Intake - drug effects | Hormones, Ectopic - physiology | Ciliary Neurotrophic Factor - analogs & derivatives | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Obesity - metabolism | Blood Glucose - drug effects | Carrier Proteins - genetics | Eating - drug effects | Animals | Diet | Hormones, Ectopic - genetics | Mice, Obese | Adipose Tissue - chemistry | Mice | Uncoupling Protein 1 | alpha-MSH - analogs & derivatives | Adipose Tissue - drug effects | alpha-MSH - agonists
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17. Full Text Responsiveness to Peripherally Administered Melanocortins in Lean and Obese Mice
by Susann Blüher and Mary Ziotopoulou and John W. Bullen, Jr and Stergios J. Moschos and Linda Ungsunan and Efi Kokkotou and Eleftheria Maratos-Flier and Christos S. Mantzoros
Diabetes, ISSN 0012-1797, 01/2004, Volume 53, Issue 1, pp. 82 - 90
Responsiveness to Peripherally Administered Melanocortins in Lean and Obese Mice Susann Blüher 1 , Mary Ziotopoulou 1 , John W. Bullen, Jr 1 , Stergios J.... 
MELANOCYTE-STIMULATING HORMONE | FOOD-INTAKE | ENDOCRINOLOGY & METABOLISM | RATS | FAT | DIET-INDUCED OBESITY | ARCUATE NUCLEUS | LEPTIN RESISTANCE | EXPRESSION | NEUROPEPTIDE-Y | AGONIST | alpha-MSH - pharmacology | Mice, Inbred C57BL | Body Weight - drug effects | Thinness | Energy Intake - drug effects | Mice, Inbred Strains | Gene Expression Regulation - drug effects | Neuropeptide Y - metabolism | Receptors, Adiponectin | Animals | Liver - drug effects | Receptors, Cell Surface - drug effects | Time Factors | Liver - physiology | Mice, Obese | Leptin - pharmacology | Mice | Receptors, Cell Surface - genetics | Obesity | Insulin resistance | Diabetes | Research | Analysis | Weight loss
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18. Full Text HIF-1 regulates CD47 expression in breast cancer cells to promote evasion of phagocytosis and maintenance of cancer stem cells
by Zhang, Huimin and Lu, Haiquan and Xiang, Lisha and Bullen, John W and Zhang, Chuanzhao and Samanta, Debangshu and Gilkes, Daniele M and He, Jianjun and Semenza, Gregg L
Proceedings of the National Academy of Sciences of the United States, ISSN 0027-8424, 11/2015, Volume 112, Issue 45, p. 13761
Care and treatment | Usage | Cancer cells | Hypoxia (Aquatic ecology) | Breast cancer | Research | Risk factors | Phagocytosis
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19. HIF-1 regulates CD47 expression in breast cancer cells to promote evasion of phagocytosis and maintenance of cancer stem cells
by Huimin Zhang and Haiquan Lu and Lisha Xiang and John W Bullen and Chuanzhao Zhang and Debangshu Samanta and Daniele M Gilkes and Jianjun He and Gregg L Semenza
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2015, Volume 112, Issue 45, p. E6215
  Increased expression of CD47 has been reported to enable cancer cells to evade phagocytosis by macrophages and to promote the cancer stem cell phenotype, but... 
Genotype & phenotype | Correlation analysis | Stem cells | Hypoxia | Breast cancer | Gene expression
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20. HIF-1 regulates CD47 expression in breast cancer cells to promote evasion of phagocytosis and maintenance of cancer stem cells
by Zhang, Huimin and Lu, Haiquan and Xiang, Lisha and Bullen, John W and Zhang, Chuanzhao and Samanta, Debangshu and Gilkes, Daniele M and He, Jianjun and Semenza, Gregg L
Proceedings of the National Academy of Sciences, USA, ISSN 0027-8424, 11/2015, Volume 112, Issue 45, pp. E6215 - E6215
Increased expression of CD47 has been reported to enable cancer cells to evade phagocytosis by macrophages and to promote the cancer stem cell phenotype, but... 
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