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细胞研究:英文版, ISSN 1001-0602, 2012, Volume 22, Issue 10, pp. 1479 - 1501
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2012, Volume 122, Issue 5, pp. 1832 - 1848
Cancer development, progression, and metastasis are highly dependent on angiogenesis. The use of antiangiogenic drugs has been proposed as a novel strategy to... 
MEDICINE, RESEARCH & EXPERIMENTAL | VASCULAR NORMALIZATION | INDUCIBLE FACTOR-1-ALPHA | SQUAMOUS CARCINOGENESIS | PANCREATIC NEUROENDOCRINE TUMORS | MOUSE MODEL | ANTI-ANGIOGENIC THERAPY | GROWTH-FACTOR | K14-HPV16 TRANSGENIC MICE | NF-KAPPA-B | MALIGNANT PROGRESSION | Proto-Oncogene Proteins c-met - metabolism | Genetic Therapy | Pancreatic Neoplasms - metabolism | Uterine Cervical Neoplasms - blood supply | Pancreatic Neoplasms - blood supply | Drug Resistance, Neoplasm | NF-kappa B - metabolism | Uterine Cervical Neoplasms - pathology | Recombinant Proteins - biosynthesis | Semaphorin-3A - biosynthesis | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Pericytes - pathology | Epithelial-Mesenchymal Transition | Angiogenesis Inhibitors - therapeutic use | Female | Indoles - pharmacology | Liver Neoplasms - secondary | Pyrroles - therapeutic use | Uterine Cervical Neoplasms - therapy | Semaphorin-3A - genetics | Neuroendocrine Tumors - pathology | Cell Hypoxia - drug effects | Neoplasm Invasiveness | Neuroendocrine Tumors - metabolism | Pancreatic Neoplasms - pathology | Angiogenesis Inhibitors - pharmacology | Mice, Transgenic | Combined Modality Therapy | Lymphatic Metastasis | Recombinant Proteins - genetics | Tumor Burden | Pyrroles - pharmacology | Animals | Uterine Cervical Neoplasms - virology | Indoles - therapeutic use | Mice | Neuroendocrine Tumors - therapy | Neovascularization, Physiologic | Neuroendocrine Tumors - blood supply | Pancreatic Neoplasms - therapy | Research | Angiogenesis inhibitors | Semaphorins | Cancer cells | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Molecular Cancer, ISSN 1476-4598, 12/2009, Volume 8, Issue 1, pp. 118 - 118
Background: Osteosarcoma (OS) is the most common primary bone tumour in children and young adults. Despite improved prognosis, metastatic or relapsed OS... 
ACTIVATION | SOLID TUMORS | ONCOLOGY | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOTHELIAL-CELLS | ITALIAN-SARCOMA-GROUP | MAP-KINASES | MUTATIONS | FACTOR RECEPTOR | EXPRESSION | CANCER | PROGRESSION | Niacinamide - analogs & derivatives | Osteosarcoma - drug therapy | Vascular Endothelial Growth Factor A - biosynthesis | Cytoskeletal Proteins - antagonists & inhibitors | Apoptosis - drug effects | Humans | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Male | Antineoplastic Agents - therapeutic use | Benzenesulfonates - therapeutic use | Phenylurea Compounds | Extracellular Signal-Regulated MAP Kinases - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Proto-Oncogene Proteins c-bcl-2 - metabolism | Benzenesulfonates - pharmacology | Neoplasm Metastasis - prevention & control | Osteosarcoma - blood supply | Cytoskeletal Proteins - metabolism | Female | Neovascularization, Pathologic - prevention & control | Antineoplastic Agents - pharmacology | Matrix Metalloproteinase 2 - biosynthesis | Osteosarcoma - metabolism | Pyridines - therapeutic use | Down-Regulation - drug effects | Cell Division - drug effects | Animals | Myeloid Cell Leukemia Sequence 1 Protein | Cell Line, Tumor | Matrix Metalloproteinase Inhibitors | Proto-Oncogene Proteins c-bcl-2 - antagonists & inhibitors | Mice | Pyridines - pharmacology | Osteosarcoma - pathology | Development and progression | Diagnosis | Drug therapy | Osteosarcoma | Risk factors | Studies | Proteins | Medical research | Kinases | Index Medicus
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 9/2005, Volume 170, Issue 6, pp. 993 - 1004
During angiogenic remodeling, Ang-1, the ligand of Tie2 tyrosine kinase, is involved in vessel sprouting and stabilization through unclear effects on nascent... 
Journal Article
Journal Article
Nature cell biology, ISSN 1465-7392, 10/2014, Volume 16, Issue 10, pp. 8 - 941
The mechanism by which angiogenic endothelial cells break the physical barrier of the vascular basement membrane and consequently sprout to form new vessels in... 
F-ACTIN | INVADOPODIA | TIP CELLS | INTEGRIN | BLOOD-VESSELS | EXTRACELLULAR-MATRIX | MICE | GROWTH-FACTOR | TRANSFORMED FIBROBLASTS | BASEMENT-MEMBRANE | CELL BIOLOGY | Neoplasms - metabolism | Cell Membrane Structures - physiology | Tetradecanoylphorbol Acetate - pharmacology | Humans | Lung Neoplasms - metabolism | Male | Melanoma, Experimental - metabolism | Green Fluorescent Proteins - genetics | Vascular Endothelial Growth Factor A - metabolism | Lung Neoplasms - physiopathology | Integrin alpha6beta1 - metabolism | Melanoma, Experimental - physiopathology | Cell Membrane Structures - metabolism | RNA Interference | HEK293 Cells | Integrin alpha6beta1 - genetics | Neovascularization, Pathologic - physiopathology | Neoplasms - physiopathology | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Lung Neoplasms - blood supply | Green Fluorescent Proteins - metabolism | Cell Membrane Structures - drug effects | Melanoma, Experimental - blood supply | Endothelial Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Neoplasms - blood supply | Matrix Metalloproteinase 14 - metabolism | Basement Membrane - metabolism | Mice, Knockout | Microscopy, Confocal | Animals | Cell Line, Tumor | Neovascularization, Pathologic - metabolism | HeLa Cells | Laminin - metabolism | Microscopy, Fluorescence | Endothelial Cells - drug effects | Blood vessels | Physiological aspects | Cell research | Neovascularization | Research | Index Medicus
Journal Article
Acta Biomaterialia, ISSN 1742-7061, 02/2017, Volume 49, pp. 152 - 166
Journal Article
Angiogenesis, ISSN 0969-6970, 9/2007, Volume 10, Issue 3, pp. 227 - 228
Journal Article
CELL DEATH & DISEASE, ISSN 2041-4889, 01/2018, Volume 9, Issue 2, pp. 45 - 15
Somatic activating mutations within the PIK3CA gene have been recently detected in sporadic lymphatic and venous malformations, and in vascular malformations... 
MIGRATION | SECRETORY PHENOTYPE | PI3K | ANGIOGENESIS | PIK3CA CAUSE | SENESCENCE | LYMPHATIC ENDOTHELIAL-CELLS | SOMATIC MUTATIONS | ACTIVATING MUTATIONS | MTOR | CELL BIOLOGY | TOR protein | Cell proliferation | Senescence | AKT protein | Inflammation | Gene expression | Hemorrhage | Endothelial cells | Cell density | 1-Phosphatidylinositol 3-kinase | Signal transduction | Angiogenesis | Rodents | Mutation | Veins & arteries
Journal Article