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Nature Medicine, ISSN 1078-8956, 04/2010, Volume 16, Issue 4, pp. 460 - 465
Journal Article
PLoS Pathogens, ISSN 1553-7366, 10/2011, Volume 7, Issue 10, p. e1002314
Journal Article
Clinical Infectious Diseases, ISSN 1058-4838, 1/2009, Volume 48, Issue 2, pp. 229 - 238
Journal Article
PLoS Pathogens, ISSN 1553-7366, 04/2016, Volume 12, Issue 4, p. e1005545
Journal Article
Antiviral Therapy, ISSN 1359-6535, 2012, Volume 17, Issue 2, pp. 355 - 364
Background: Residual viraemia is a major obstacle to HIV-1 eradication in subjects receiving HAART. The intensification with raltegravir could impact latent... 
INFECTIOUS DISEASES | VIRAL SUPPRESSION | INFECTED INDIVIDUALS | CD4 | REPLICATION | PLASMA | VIROLOGY | ACTIVE ANTIRETROVIRAL THERAPY | T-CELL-ACTIVATION | IN-VIVO | IMMUNE ACTIVATION | PHARMACOLOGY & PHARMACY | BURDEN
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2014, Volume 9, Issue 12, p. e114142
Background: HIV-1 infection increases plasma levels of inflammatory markers. Combination antiretroviral therapy (cART) does not restore inflammatory markers to... 
CONTROLLED-TRIAL | ACTIVATION | CD4 | T-CELL RECOVERY | MULTIDISCIPLINARY SCIENCES | ANTIRETROVIRAL THERAPY | MICROBIAL TRANSLOCATION | SIV INFECTION | RALTEGRAVIR INTENSIFICATION | PROTEASE INHIBITORS | D-DIMER | HIV Infections - blood | Prospective Studies | Humans | Middle Aged | Raltegravir Potassium - administration & dosage | Raltegravir Potassium - therapeutic use | Male | Biomarkers - blood | HIV Integrase Inhibitors - administration & dosage | Inflammation - blood | HIV Infections - immunology | HIV Integrase Inhibitors - therapeutic use | Adult | Female | HIV Infections - drug therapy | Usage | HIV patients | Inflammation | T cells | HIV (Viruses) | Infection | Highly active antiretroviral therapy | Protease inhibitors | Proteases | Analysis | Hepatitis C virus | Health aspects | HIV infection | Antiretroviral drugs | Composition | CD8 antigen | Markers | Lymphocytes T | Proteinase inhibitors | T cell receptors | Infections | Patients | Antiretroviral therapy | CD4 antigen | Amplification | Cell activation | Acquired immune deficiency syndrome--AIDS | Lymphocytes | Antiretroviral agents | Integrals | Human immunodeficiency virus--HIV | Replication | Sampling methods | Plasma levels | Dimers | Antiretrovirals | HIV infections | Inflamació | Marcadors bioquímics | Infeccions per VIH | Biochemical markers | Acquired immune deficiency syndrome | AIDS | HIV | Human immunodeficiency virus
Journal Article
Journal Article
FRONTIERS IN IMMUNOLOGY, ISSN 1664-3224, 04/2019, Volume 10, p. 825
Antigen presenting cells from the cervical mucosa are thought to amplify incoming HIV-1 and spread infection systemically without being productively infected.... 
HIV-1 | cervix | myeloid cells | VAGINAL TRANSMISSION | IMMUNE PROTECTION | INDUCE | Siglec-1 | IMMUNOLOGY | HUMAN-IMMUNODEFICIENCY-VIRUS | trans-infection | DC-SIGN | INFECTION | INTRAVAGINAL INOCULATION | TYPE-1 | RECEPTORS | EXPRESSION | Interferon | Dendritic cells | Biological response modifiers | HIV (Viruses)
Journal Article