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Circulation, ISSN 0009-7322, 11/2015, Volume 132, Issue Suppl_3 Suppl 3, pp. A15199 - A15199
Vascular dysfunction is a major cause of morbidity and mortality in diabetic patients. Diabetes has been linked to elevated arginase and associated with... 
Journal Article
Circulation, ISSN 0009-7322, 11/2016, Volume 134, Issue Suppl_1 Suppl 1, pp. A14793 - A14793
Obesity-induced cardiovascular dysfunction involves pathological expansion of visceral adipose tissue (VAT). Our previous studies have shown that arginase 1... 
Journal Article
European journal of pharmacology, 11/2018, Volume 844, p. 26
Diabetes-induced vascular endothelial dysfunction has been reported to involve hyperglycemia-induced increases in arginase activity. However, upstream... 
Journal Article
Journal Article
Experimental Eye Research, ISSN 0014-4835, 07/2012, Volume 100, p. 101
The goal of this study was to define the role of p38alpha MAP kinase in VEGF-induced vascular permeability increase. Activation of p38 is correlated with... 
Tracers (Biology) | Endothelial growth factors | Analysis | Permeability | Glucose | Pharmaceutical industry | Gene expression | Vascular endothelial growth factor | Dextrose | Endothelium
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2017, Volume 12, Issue 10, pp. e0186734 - e0186734
Background Netrin-1, a secreted laminin-like protein identified as an axon guidance molecule, has been shown to be of critical importance in the cardiovascular... 
ISCHEMIA-REPERFUSION INJURY | APOPTOSIS | OVEREXPRESSION | FEEDFORWARD MECHANISM | MYOCARDIAL-INFARCTION | DCC | MULTIDISCIPLINARY SCIENCES | THERAPEUTIC TARGET | DYSFUNCTION | KIDNEY | BLOOD-BRAIN-BARRIER | Reactive Oxygen Species - metabolism | Oxidative Stress | Netrin-1 | Cells, Cultured | Endothelium, Vascular - physiopathology | Mice, Transgenic | Tumor Suppressor Proteins - physiology | Animals | Cattle | Nerve Growth Factors - genetics | Tumor Suppressor Proteins - genetics | Diabetes Mellitus, Experimental - pathology | Endothelium, Vascular - pathology | Mice | Blood Glucose - metabolism | Nerve Growth Factors - physiology | Nitric Oxide - metabolism | Apoptosis | Diabetes Mellitus, Experimental - physiopathology | Type 2 diabetes | Complications and side effects | Care and treatment | Physiological aspects | Genetic aspects | Glycoproteins | Research | Endothelium | Oxidative stress | Heart attacks | p53 Protein | INK4a protein | Diabetic neuropathy | Biology | mRNA | Kinases | Caspase-3 | Angiogenesis | Toxicology | Laminin | Blood-brain barrier | Ischemia | Rodents | Aorta | DCC protein | Cardiovascular system | Antiinflammatory agents | Diabetes mellitus | Transgenic mice | p16 Protein | Extracellular signal-regulated kinase | Caspase | Pharmacology | Inflammation | Nitric-oxide synthase | Endothelial cells | Nitric oxide | Hypoxia | Diabetes | Cyclooxygenase-2 | Axon guidance | Index Medicus | Stresses | Stress
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 05/2011, Volume 300, Issue 5, pp. 1181 - 1192
Enhanced vascular arginase activity impairs endothelium-dependent vasorelaxation by decreasing l-arginine availability to endothelial nitric oxide (NO)... 
Endothelial nitric oxide synthase dysfunction | PHYSIOLOGY | BLOOD-PRESSURE | P38 MAP KINASE | CELL BIOLOGY | INCREASED ARGINASE ACTIVITY | NITRIC-OXIDE PRODUCTION | INHIBITION | MOUSE MODEL | endothelial nitric oxide synthase dysfunction | CARDIOVASCULAR-DISEASE | SMOOTH-MUSCLE-CELLS | HYPERTENSIVE-RATS | CONTRIBUTES | Phosphorylation | Endothelial Cells | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Endothelium, Vascular - drug effects | Endothelium, Vascular - enzymology | GTP-Binding Protein alpha Subunits, G12-G13 - antagonists & inhibitors | Simvastatin - pharmacology | Angiotensin II Type 1 Receptor Blockers - pharmacology | rho-Associated Kinases - antagonists & inhibitors | Cattle | Arginase - antagonists & inhibitors | Amides - pharmacology | Cell Line | rho-Associated Kinases - physiology | Angiotensin II - pharmacology | p38 Mitogen-Activated Protein Kinases - physiology | RNA, Small Interfering - pharmacology | Endothelium, Vascular - physiopathology | Enzyme Inhibitors - pharmacology | GTP-Binding Protein alpha Subunits, Gq-G11 - antagonists & inhibitors | Imidazoles - pharmacology | Animals | Signal Transduction - drug effects | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Signal Transduction - physiology | Mice | Pyridines - pharmacology | Arginase - physiology | Angiotensin II - physiology | Boronic Acids - pharmacology | Abnormalities | Angiotensin | Physiological aspects | Vascular endothelium | Genetic aspects | Research | Mitogen-activated protein kinases | Proteins | Signal transduction | Nitric oxide | Rodents | Cardiovascular disease | ACE inhibitors | Cells | Index Medicus | Vascular Biology
Journal Article
Journal Article
Journal of cell science, ISSN 0021-9533, 08/2016, Volume 129, Issue 16, pp. 3203 - 3203
Journal Article
The American Journal of Pathology, ISSN 0002-9440, 2011, Volume 178, Issue 5, pp. 2136 - 2145
In diabetic retinopathy (DR), abnormalities in vascular and neuronal function are closely related to the local production of inflammatory mediators whose... 
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2011, Volume 178, Issue 5, pp. 2136 - 2145
In diabetic retinopathy (DR), abnormalities in vascular and neuronal function are closely related to the local production of inflammatory mediators whose... 
Pathology | Regular
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 09/2018, Volume 1864, Issue 9, pp. 2761 - 2768
Journal Article
Nature, ISSN 0028-0836, 10/2006, Volume 443, Issue 7114, pp. 993 - 997
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 10, pp. e0186749 - e0186749
[This corrects the article DOI: 10.1371/journal.pone.0071868.]. 
Retina | Superoxide | Glucose | Diabetes | Vascular endothelial growth factor | Diabetes mellitus
Journal Article
Journal of Cardiovascular Disease Research, ISSN 0975-3583, 2011, Volume 2, Issue 2, pp. 96 - 103
Diabetic retinopathy (DR) is one of the most common complications of diabetes and is a leading cause of blindness in people of the working age in Western... 
Diabetic retinopathy | inflammation | microvascular complications | Complications and side effects | Care and treatment | Development and progression | Inflammation | Diabetes | Research | Diagnosis | Risk factors | Proteins | Studies | Hypertension | Wound healing | Disease | Pathogenesis | Rodents | Retina | Permeability | Chronic illnesses | Invited Review
Journal Article