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Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 137, Issue 5, pp. 1449 - 1456.e4
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2013, Volume 131, Issue 2, pp. 412 - 420
Journal Article
Journal Article
American Journal of Physiology, ISSN 1040-0605, 09/2017, Volume 313, Issue 3, p. L516
Nicotine is a major component of cigarette smoke. It causes addiction and is used clinically to aid smoke cessation. The aim of the present study is to... 
Tetrodotoxin | Animal models | Contractility | Nervous system | Exposure | Smoke | Saline solutions | Cigarettes | Experiments | Respiratory system | Lipopolysaccharides | Respiratory tract | Tobacco | Addiction | Rodents | Methacholine | Electric fields | Cigarette smoke | Nicotine | Long-term effects
Journal Article
Allergy, ISSN 0105-4538, 12/2019, Volume 74, Issue 12, pp. 2499 - 2501
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, p. e98239
Background: The human nasal epithelium is an important physical barrier, and a part of the innate immune defense that protect against pathogens. The epithelial... 
NEUTROPHIL | RNA | RECOGNITION | MULTIDISCIPLINARY SCIENCES | INFECTION | INDUCTION | EXPRESSION | TLR3 | VIRUSES | CHRONIC RHINOSINUSITIS | INNATE IMMUNITY | Cell Line | DEAD Box Protein 58 | Epithelial Cells - metabolism | Epithelium - metabolism | Humans | RNA, Messenger - genetics | Male | RNA, Messenger - metabolism | Nose - cytology | Interferon-Induced Helicase, IFIH1 | Young Adult | DEAD-box RNA Helicases - genetics | Toll-Like Receptors - genetics | Adolescent | Adult | Female | Nasal Mucosa - metabolism | Toll-Like Receptors - metabolism | DEAD-box RNA Helicases - metabolism | Inflammation | Cytokines | Analysis | Enzyme-linked immunosorbent assay | Immunohistochemistry | Biotechnology | Intrusion | Flow cytometry | Rhinitis | Laboratories | Epithelial cells | Pathogenesis | Rhinosinusitis | Mucosa | Reverse transcription | Otolaryngology | Viruses | Stimulation | Infections | Metastasis | Inflammatory diseases | Interleukin 6 | Proteins | Respiratory tract | Receptors | Microorganisms | Immunology | Allergic rhinitis | Penicillin | Toll-like receptors | Respiratory tract diseases | Interleukin 8 | Immune system | CpG islands | Immune response | Granulocyte-macrophage colony-stimulating factor | Melanoma | Poly (I:C) | Pattern recognition | Gene expression | Epithelium | Allergies | TLR3 protein | Asthma | Cytometry | Biopsy | Cell lines | Interferon | Viral infections | Clinical Medicine | Lungmedicin och allergi | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Respiratory Medicine and Allergy
Journal Article
Immunology, ISSN 0019-2805, 11/2011, Volume 134, Issue 3, pp. 314 - 325
Summary NOD‐like receptors (NLRs) and RIG‐I‐like receptors (RLRs) are newly discovered pattern‐recognition receptors. They detect substructures of bacterial... 
allergy | eosinophils | cytokines | innate immunity | Allergy | Innate immunity | Cytokines | Eosinophils | DENDRITIC CELLS | TRAFFICKING | NUCLEOTIDE-BINDING | IMMUNOLOGY | NALP3 | MEDIATED ACTIVATION | INFLAMMASOME | TOLL-LIKE RECEPTORS | ASTHMA | NLRS | ASSOCIATION | Immunohistochemistry | Eosinophils - metabolism | Diaminopimelic Acid - pharmacology | Gene Expression - drug effects | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Nod1 Signaling Adaptor Protein - agonists | NF-kappa B - metabolism | Nod2 Signaling Adaptor Protein - genetics | Nod1 Signaling Adaptor Protein - metabolism | Antigens, CD - metabolism | Interferon-Induced Helicase, IFIH1 | L-Selectin - metabolism | Lectins, C-Type - metabolism | Flow Cytometry | Interleukin-8 - secretion | Nod2 Signaling Adaptor Protein - agonists | DEAD-box RNA Helicases - metabolism | DEAD Box Protein 58 | Eosinophil-Derived Neurotoxin - secretion | Cells, Cultured | Antigens, Differentiation, T-Lymphocyte - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Acetylmuramyl-Alanyl-Isoglutamine - pharmacology | Carrier Proteins - genetics | Cell Movement - drug effects | DEAD-box RNA Helicases - genetics | Carrier Proteins - metabolism | Diaminopimelic Acid - analogs & derivatives | Nod2 Signaling Adaptor Protein - metabolism | Signal Transduction - drug effects | CD11b Antigen - metabolism | Eosinophils - cytology | Nod1 Signaling Adaptor Protein - genetics | Messenger RNA | Macrophage colony stimulating factor | Biological response modifiers | Interferon gamma | Medical research | Index Medicus | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, p. e68701
Background: Pattern-recognition receptors (PRRs), including Toll-like receptors (TLRs), NOD-like receptors (NLRs) and RIG-I-like receptors (RLRs), recognize... 
TRANSLATIONAL MINIREVIEW SERIES | ACID | TOLL-LIKE-RECEPTOR-2 | RECOGNITION | RNA | PATHWAY | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | TOLL-LIKE RECEPTOR | ASTHMA | EXPRESSION | RNA, Messenger - immunology | Nod Signaling Adaptor Proteins - immunology | Cell Proliferation | Lipopeptides - pharmacology | Humans | Trachea - drug effects | Nod Signaling Adaptor Proteins - agonists | Myocytes, Smooth Muscle - drug effects | Aminoquinolines - pharmacology | Myocytes, Smooth Muscle - cytology | Cytokines - immunology | Biomarkers - metabolism | Poly I-C - pharmacology | Signal Transduction | Trachea - cytology | Gene Expression Regulation - immunology | RNA, Messenger - genetics | Toll-Like Receptors - immunology | Immunity, Innate | Toll-Like Receptors - agonists | Trachea - immunology | Gene Expression Regulation - drug effects | Nod Signaling Adaptor Proteins - genetics | Toll-Like Receptors - genetics | Lipopolysaccharides - pharmacology | Myocytes, Smooth Muscle - immunology | Cytokines - biosynthesis | Smooth muscle | Gene expression | Analysis | Pattern recognition receptors | Flow cytometry | Cell culture | Immunocytochemistry | Stimulation | mRNA | Infections | Activation | Cell surface | Myosin-light-chain kinase | Eotaxin | Lipopolysaccharides | Interleukin 6 | Proteins | Respiratory tract | Signal transduction | Receptors | Microorganisms | Rodents | Myosin | Tumor necrosis factor-TNF | Bacteria | Respiratory tract diseases | Gram-negative bacteria | Nod1 protein | Growth factors | Interleukin 8 | Deoxyribonucleic acid--DNA | NOD2 protein | Immune system | Intercellular adhesion molecule 1 | Granulocyte-macrophage colony-stimulating factor | Immunomodulation | Muscles | Inflammation | Pattern recognition | Disease control | Muscle contraction | Asthma | Polymerase chain reaction | Cytometry | Ligands | Adrenergic receptors | Histocompatibility antigen HLA | Surface markers | Immunologi inom det medicinska området | Lungmedicin och allergi | Klinisk medicin | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Deoxyribonucleic acid | DNA
Journal Article
Allergy, ISSN 0105-4538, 12/2019, Volume 74, Issue 12, pp. 2499 - 2501
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2014, Volume 9, Issue 8, p. e104114
It is well-established that bacterial and viral infections have an exacerbating effect on allergic asthma, particularly aggravating respiratory symptoms, such... 
RESPIRATORY-INFECTIONS | RESPONSES | TOLL-LIKE-RECEPTOR-4 | SMOOTH-MUSCLE | BRONCHIAL HYPERRESPONSIVENESS | FUNCTIONAL PLASTICITY | ASTHMA EXACERBATIONS | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | RHINOVIRUS | INNATE IMMUNITY | Asthma - metabolism | Bronchial Hyperreactivity - immunology | Ovalbumin - immunology | Bronchoalveolar Lavage Fluid - immunology | Toll-Like Receptor 3 - immunology | Inflammation - immunology | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Inflammation - metabolism | Animals | Ovalbumin - administration & dosage | Poly I-C - metabolism | Asthma - immunology | Respiratory System - drug effects | Bronchial Hyperreactivity - metabolism | Female | Ligands | Mice | Poly I-C - immunology | Respiratory System - immunology | Infection | Inflammation | Research | Health aspects | Analysis | Asthma | Inflammatory response | Smooth muscle | Lymphocytes T | Infections | Lipopolysaccharides | Streptococcus infections | Proteins | Respiratory tract | Receptors | Microorganisms | Rodents | Methacholine | Toll-like receptors | Respiratory tract diseases | Immune system | Cellular manufacture | Ovalbumin | Ventilators | Bacterial infections | Cytokines | Hypersensitivity | Environmental health | Poly (I:C) | TLR4 protein | Eosinophilia | Allergies | TLR3 protein | Ostomy | Respiratory syncytial virus | Lungs | Neutrophilia | Mechanics | Viral infections
Journal Article