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PLoS ONE, ISSN 1932-6203, 07/2012, Volume 7, Issue 7, pp. e41926 - e41926
Journal Article
Annals of Surgical Oncology, ISSN 1068-9265, 02/2019, Volume 26, Issue 2, pp. 697 - 697
Journal Article
Nature Genetics, ISSN 1061-4036, 09/2013, Volume 45, Issue 9, pp. 1050 - 1054
Journal Article
Science, ISSN 0036-8075, 2/2011, Volume 331, Issue 6018, pp. 768 - 772
Endocrine tumors such as aldosterone-producing adrenal adenomas (APAs), a cause of severe hypertension, feature constitutive hormone production and... 
Depolarization | Hypertension | Somatic mutation | Cell growth | Hyperaldosteronism | REPORTS | Loss of heterozygosity | Adenoma | Genetic mutation | Sequencing | Tumors | REMEDIABLE ALDOSTERONISM | HUMAN BREAST | COLORECTAL CANCERS | ADRENOCORTICAL-CELLS | MULTIDISCIPLINARY SCIENCES | CONDUCTION | GLOMERULOSA CELLS | IDENTIFICATION | MOLECULAR-BASIS | SELECTIVITY | POTASSIUM CHANNEL | Adrenal Glands - pathology | Potassium - metabolism | Cell Proliferation | Humans | Hyperaldosteronism - pathology | Hyperplasia | Protein Multimerization | Zona Glomerulosa - pathology | Male | Hyperaldosteronism - metabolism | Sodium - metabolism | Aldosterone - metabolism | Adrenocortical Adenoma - genetics | G Protein-Coupled Inwardly-Rectifying Potassium Channels - metabolism | Female | Hypertension - genetics | Adrenal Cortex Neoplasms - metabolism | Cell Line | G Protein-Coupled Inwardly-Rectifying Potassium Channels - chemistry | Hyperaldosteronism - genetics | Mutant Proteins - genetics | Mutant Proteins - metabolism | Zona Glomerulosa - metabolism | Adrenal Cortex Neoplasms - pathology | G Protein-Coupled Inwardly-Rectifying Potassium Channels - genetics | Adrenocortical Adenoma - pathology | Adrenocortical Adenoma - metabolism | Hypertension - metabolism | Adrenal Cortex Neoplasms - genetics | Mutant Proteins - chemistry | Mutation | Index Medicus | Medical and Health Sciences | MEDICINE | Medicin och hälsovetenskap | MEDICIN
Journal Article
Journal Article
JOURNAL OF MOLECULAR ENDOCRINOLOGY, ISSN 0952-5041, 05/2019, Volume 62, Issue 4, pp. 179 - 186
Adrenocortical carcinomas are rare tumors with poor prognosis and limited treatment options. Although widely used as in vitro models to test novel therapeutic... 
molecular genetics | MANAGEMENT | carcinoma | LANDSCAPE | GENOMES | ENDOCRINOLOGY & METABOLISM | adrenal cortex | neoplasia | oncology | EVENT | Prognosis | Carcinoma | Wnt protein | p53 Protein | Genes | Genomes | Malignancy | Tumor cell lines | Chromatin remodeling | Cell lines | Mutation | Cancer | DNA sequencing | Tumors
Journal Article
Nature Genetics, ISSN 1061-4036, 2014, Volume 46, Issue 6, pp. 613 - 617
Journal Article
Journal of the American College of Surgeons, ISSN 1072-7515, 06/2018, Volume 226, Issue 6, p. 988
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1016/j.jamcollsurg.2018.01.051... 
Carcinoma | Messenger RNA | Analysis | Genetic engineering | Cancer
Journal Article
Journal of the American College of Surgeons, ISSN 1072-7515, 06/2018, Volume 226, Issue 6, pp. 988 - 995
( ) is a recently described oncogene that promotes tumorigenesis via activation of the Wnt/β-Catenin signaling cascade. Though constitutively active... 
Journal Article
Journal Article
Journal Article
Surgery: Official Journal of the Society of University Surgeons, Central Surgical Association, and the American Association of Endocrine Surgeons, ISSN 0039-6060, 2016, Volume 161, Issue 1, pp. 257 - 263
Journal Article
eLife, ISSN 2050-084X, 04/2015, Volume 4, pp. e06315 - e06315
Many Mendelian traits are likely unrecognized owing to absence of traditional segregation patterns in families due to causation by de novo mutations,... 
human biology | incomplete penetrance | de novo mutation | voltage-gated calcium channel | exome sequencing | genes | chromosomes | adrenal gland | medicine | CaV3.2 | human | CHILDHOOD ABSENCE EPILEPSY | GENETIC-VARIATION | HYPERALDOSTERONISM TYPE-II | GLUCOCORTICOID-REMEDIABLE ALDOSTERONISM | BIOLOGY | WHOLE-GENOME ASSOCIATION | CA2+ CHANNEL | T-TYPE | GLOMERULOSA CELLS | FAMILIAL HYPERALDOSTERONISM | SOMATIC MUTATIONS | Recurrence | Aldosterone - biosynthesis | Calcium - metabolism | Humans | Hyperaldosteronism - pathology | Middle Aged | Hyperaldosteronism - complications | Child, Preschool | Molecular Sequence Data | Zona Glomerulosa - pathology | Infant | Male | Hyperaldosteronism - metabolism | Calcium Channels, T-Type - metabolism | Adult | Female | Calcium Channels, T-Type - genetics | Hypertension - genetics | Child | Calcium Signaling | Amino Acid Sequence | Gene Expression | Hyperaldosteronism - genetics | Aldosterone - secretion | Genotype | Zona Glomerulosa - metabolism | Hypertension - pathology | Hypertension - metabolism | Phenotype | Sequence Alignment | Membrane Potentials | Adolescent | Age of Onset | Hypertension - complications | Heterozygote | Mutation | Hypertension | Haplotypes | Pediatrics | Nephrology | Calcium (intracellular) | Statistical analysis | Genomics | Genes | Calcium channels (voltage-gated) | Genomes | Aldosterone | Morbidity | Calcium signalling | Consortia | Pathology | Children | Age | Index Medicus
Journal Article