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Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 01/2000, Volume 292, Issue 1, pp. 156 - 163
Journal Article
Journal of Materials Science: Materials in Medicine, ISSN 0957-4530, 6/2014, Volume 25, Issue 6, pp. 1483 - 1493
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e33031
Apoptosis, a form of programmed cell death, is a critical defence mechanism against the formation and progression of cancer and acts by eliminating potentially... 
INHIBITION | INFLAMMATION | SPONGE FASCIOSPONGIA-CAVERNOSA | BIOLOGY | SECRETORY PHOSPHOLIPASE A | KAPPA-B | CANCER | EXPRESSION | Cell Survival - drug effects | NF-kappa B p50 Subunit - metabolism | Apoptosis - drug effects | Humans | 4-Butyrolactone - pharmacology | Homosteroids - pharmacology | Apoptosis Regulatory Proteins - metabolism | Animals | Cell Nucleus - metabolism | Transcription Factor RelA - metabolism | Active Transport, Cell Nucleus - drug effects | Cell Line, Tumor | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Sesterterpenes - pharmacology | 4-Butyrolactone - analogs & derivatives | Aquatic Organisms - chemistry | DNA Fragmentation - drug effects | Cell Nucleus - drug effects | Porifera - chemistry | Pyrans - pharmacology | Antimitotic agents | Proteins | Chemotherapy | Colon cancer | Carcinoma | Development and progression | Research | Antineoplastic agents | Cancer | Apoptosis | Cell proliferation | Drugs | Oxidative stress | Inflammatory response | Cytotoxicity | Drug development | Cancer therapies | Fragmentation | Breast carcinoma | Signal transduction | Mitochondria | Antitumor agents | Metabolites | DNA fragmentation | Membrane potential | Colon | Cervical carcinoma | Deoxyribonucleic acid--DNA | NF-κB protein | Defense programs | Incubation | Cystic fibrosis | Inflammation | Breast cancer | Tumor cell lines | Cervix | Sesterterpenoids | Signaling | Chemistry | Microscopy | Councils | Cell death | Natural products | Morphology | Cell lines | Viability | Cell migration | Deoxyribonucleic acid | DNA
Journal Article
International Journal of Pharmaceutics, ISSN 0378-5173, 01/2013, Volume 440, Issue 2, pp. 179 - 187
Journal Article
Nature Cell Biology, ISSN 1465-7392, 06/2008, Volume 10, Issue 6, pp. 676 - 687
Journal Article
Cell Cycle, ISSN 1538-4101, 05/2009, Volume 8, Issue 10, pp. 1571 - 1576
The oncosuppressor protein p53 regulates autophagy in a dual fashion. The pool of cytoplasmic  p53 protein represses autophagy in a transcription-independent... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Dram | Sestrins | Autophagy | p53 | PROGRAMMED CELL-DEATH | APOPTOSIS | autophagy | dram | STRESS | sestrins | CANCER-THERAPY | MTOR | CELL BIOLOGY | RNA, Small Interfering - genetics | Humans | Tumor Suppressor Protein p53 - metabolism | Cell Line, Tumor | Nuclear Proteins - metabolism | Tumor Suppressor Protein p53 - genetics
Journal Article
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 12/2011, Volume 15, Issue 12, pp. 2664 - 2674
Journal Article
FRONTIERS IN PHARMACOLOGY, ISSN 1663-9812, 07/2019, Volume 10, p. 775
The ecto-5'-nucleotidase (ecto-5'NT/CD73) represents a crucial enzyme for endogenous adenosine generation. Several findings have shown that CD73 plays an... 
adenosine | CELLS | CD73 | MCP-1 | rat | inflammation | PATHWAY | ecto-5 '-nucleotidase | CD39 | PHARMACOLOGY & PHARMACY | MODEL | pleurisy | ecto-5’-nucleotidase
Journal Article
Mediators of Inflammation, ISSN 0962-9351, 2013, Volume 2013, pp. 584715 - 14
Journal Article
Autophagy, ISSN 1554-8627, 11/2012, Volume 8, Issue 11, pp. 1657 - 1672
Channel activators (potentiators) of cystic fibrosis (CF) transmembrane conductance regulator (CFTR), can be used for the treatment of the small subset of CF... 
cystamine | CFTR potentiators | autophagy | cystic fibrosis | therapy | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Therapy | Cystic fibrosis | Autophagy | Cystamine | GAMMA DOWN-REGULATION | OXIDATIVE STRESS | F508DEL-CFTR | PLASMA-MEMBRANE | INTESTINAL EPITHELIAL-CELLS | CELL BIOLOGY | PROTEIN-QUALITY CONTROL | MUTATION | TISSUE TRANSGLUTAMINASE | LUNG INFLAMMATION | CFTR | Inflammation - pathology | Epithelial Cells - metabolism | Sequestosome-1 Protein | Epithelial Cells - drug effects | Humans | Cystic Fibrosis - pathology | Male | Nasal Mucosa - pathology | Molecular Targeted Therapy | Autophagy - drug effects | Salicylates - pharmacology | Cystamine - therapeutic use | Organometallic Compounds - therapeutic use | Cystamine - pharmacology | Female | Cell Membrane - metabolism | Membrane Proteins - metabolism | Child | Cell Membrane - drug effects | Nasal Mucosa - drug effects | Beclin-1 | Epithelium - drug effects | Epithelium - pathology | Lung - pathology | Nasal Polyps - pathology | Epithelium - metabolism | Heat-Shock Proteins - metabolism | Epithelial Cells - pathology | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Antioxidants - pharmacology | Salicylates - therapeutic use | Apoptosis Regulatory Proteins - metabolism | Animals | Lung - drug effects | Adolescent | Genistein - pharmacology | Lipopolysaccharides - pharmacology | Mice | Adaptor Proteins, Signal Transducing - metabolism | Cystic Fibrosis - drug therapy | Organometallic Compounds - pharmacology | Translational Research Paper
Journal Article
European Journal of Pharmaceutics and Biopharmaceutics, ISSN 0939-6411, 2008, Volume 70, Issue 1, pp. 7 - 18
Nuclear factor-κB (NF-κB) is involved in several pathological processes, such as inflammation. Pro-inflammatory genes expression can be down-regulated by using... 
Decoy oligonucleotide | Cationic liposomes | RAW 264.7 macrophages | Nuclear factor-κB | CELLS | COMPLEX | LIPOFECTION | GENE DELIVERY | TRANSFECTION | nuclear factor-kappa B | SERUM | IN-VITRO | DNA | cationic liposomes | PHARMACOLOGY & PHARMACY | TRANSCRIPTION FACTOR | PHOSPHOROTHIOATE OLIGONUCLEOTIDES | decoy oligonucleotide
Journal Article