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1. Full Text Mutations in early follicular lymphoma progenitors are associated with suppressed antigen presentation
by Michael R. Green and Shingo Kihira and Chih Long Liu and Ramesh V. Nair and Raheleh Salari and Andrew J. Gentles and Jonathan Irish and Henning Stehr and Carolina Vicente-Dueñas and Isabel Romero-Camarero and Isidro Sanchez-Garcia and Sylvia K. Plevritis and Daniel A. Arber and Serafim Batzoglou and Ronald Levy and Ash A. Alizadeh
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2015, Volume 112, Issue 10, pp. E1116 - E1125
Follicular lymphoma (FL) is incurable with conventional therapies and has a clinical course typified by multiple relapses after therapy. These tumors are... 
Exome | Antigen presentation | Hierarchy | CREBBP | Lymphoma | SURVIVAL | TRANSLOCATION | antigen presentation | EPIGENETIC CONTROL | MULTIDISCIPLINARY SCIENCES | lymphoma | hierarchy | SOMATIC MUTATIONS | PATHOGENESIS | B-CELL LYMPHOMA | exome | GENE-EXPRESSION | CREB BINDING-PROTEIN | T-CELLS | EZH2 | CREB-Binding Protein - genetics | Chromatin - metabolism | Flow Cytometry | Lymphoma, Follicular - genetics | Lymphoma, Follicular - immunology | Humans | Polymerase Chain Reaction | Neoplastic Stem Cells - pathology | Histocompatibility Antigens Class II - genetics | Mutation | Antigen-Presenting Cells - immunology | Antigens | Lymphomas | Genetic aspects | Gene mutations | Observations | Health aspects | Biological Sciences | PNAS Plus
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2. Full Text NovelETV6‐RUNX1Mouse Model to Study the Role of ELF‐MF in Childhood B‐Acute Lymphoblastic Leukemia: a Pilot Study
by Campos‐Sanchez, Elena and Vicente‐Dueñas, Carolina and Rodríguez‐Hernández, Guillermo and Capstick, Myles and Kuster, Niels and Dasenbrock, Clemens and Sánchez‐García, Isidro and Cobaleda, César
Bioelectromagnetics, ISSN 0197-8462, 07/2019, Volume 40, Issue 5, pp. 343 - 353
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3. Full Text Identification of cancer initiating cells in K-Ras driven lung adenocarcinoma
by Sara Mainardi and Nieves Mijimolle and Sarah Francoz and Carolina Vicente-Dueñas and Isidro Sánchez-García and Mariano Barbacid
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2014, Volume 111, Issue 1, pp. 255 - 260
Ubiquitous expression of a resident K-RasG12V oncogene in adult mice revealed that most tissues are resistant to K-Ras oncogenic signals. Indeed, K-RasG12V... 
Acinar cells | Adenocarcinoma | Pulmonary alveoli | Cell growth | Lungs | B lymphocytes | Hyperplasia | Giant cells | Adenoma | Tumors | Lung stem cells | Inflammation | Genetically engineered mouse model | Non-small cell lung cancer | non-small cell lung cancer | ONCOGENE | inflammation | MULTIDISCIPLINARY SCIENCES | PANCREATITIS | INDUCTION | EXPRESSION | genetically engineered mouse model | lung stem cells | Cell Proliferation | Adenocarcinoma of Lung | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | ras Proteins - metabolism | Gene Expression Profiling | Stem Cells - cytology | Lung - cytology | Adenocarcinoma - metabolism | Flow Cytometry | Pulmonary Alveoli - metabolism | Adenocarcinoma - genetics | Oncogenes | Lung Neoplasms - genetics | Bronchioles - metabolism | Promoter Regions, Genetic | Signal Transduction | Cell Separation | Carcinoma, Non-Small-Cell Lung - metabolism | Mice, Transgenic | Animals | Cell Transformation, Neoplastic | Alleles | Mice | Adenoviridae - metabolism | Genes, ras | Cell interaction | Lung cancer | Oncology, Experimental | Cancer cells | Physiological aspects | Research | Cancer | Biological Sciences
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4. Full Text A Tumor Suppressor Role for Bank1 in B-Cell Precursor Acute Lymphoblastic Leukemia
by Auer, Franziska and Guillermo, Rodríguez-Hernández and González-Herrero, Inés and Blanco, Oscar and Alonso-López, Diego and De Las Rivas, Javier and Jimenez, Rafael and García-Criado, Francisco J and García-Cenador, Maria B and Vicente-Dueñas, Carolina and Borkhardt, Arndt and Sanchez-Garcia, Isidro and Hauer, Julia
Blood, ISSN 0006-4971, 11/2018, Volume 132, Issue Supplement 1, pp. 1333 - 1333
Abstract Introduction: Since the implementation of next generation sequencing techniques, inherited mutations in malignancy-associated susceptibility genes... 
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5. Full Text Function of oncogenes in cancer development: a changing paradigm
by Vicente‐Dueñas, Carolina and Romero‐Camarero, Isabel and Cobaleda, Cesar and Sánchez‐García, Isidro
The EMBO Journal, ISSN 0261-4189, 05/2013, Volume 32, Issue 11, pp. 1502 - 1513
Tumour‐associated oncogenes induce unscheduled proliferation as well as genomic and chromosomal instability. According to current models, therapeutic... 
oncogenes | cancer therapy | tumoural reprogramming | cancer stem cell | mouse model | Cancer stem cell | INITIATING CELLS | BLADDER-CARCINOMA ONCOGENE | HEMATOPOIETIC STEM/PROGENITOR CELLS | STEM-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | BCR-ABL | C-MYC | SELF-RENEWAL | CHRONIC MYELOID-LEUKEMIA | CELL BIOLOGY | PRIMARY BIOASSAY | TRANSFORMING GENE | Oncogenes - genetics | Cell Proliferation | Genetic Association Studies | Humans | Neoplastic Stem Cells | Oncogenes - physiology | Cell Biology | Neoplasms - therapy | Animals | Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Models, Biological | Mice | Mutation | Cell Transformation, Neoplastic - pathology | Neoplasms - pathology | Chromosomal Instability | Genomics | Tumors | Cancer | Review
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6. Identification of cancer initiating cells in K-Ras driven lung adenocarcinoma
by Sara Mainardi and Nieves Mijimolle and Sarah Francoz and Carolina Vicente-Dueñas and Isidro Sánchez-García and Mariano Barbacid
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 01/2014, Volume 111, Issue 1, p. 255
  Ubiquitous expression of a resident K-Ras^sup G12V^ oncogene in adult mice revealed that most tissues are resistant to K-Ras oncogenic signals. Indeed,... 
Lung cancer | Gene expression | Cells
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7. Full Text Hit-and-run lymphomagenesis by theBcl6oncogene
by Green, Michael R and Vicente-Dueñas, Carolina and Alizadeh, Ash A and Sanchez-García, Isidro
Cell Cycle, ISSN 1538-4101, 06/2014, Volume 13, Issue 12, pp. 1831 - 1832
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8. Full Text Loss of Pax5 Exploits Sca1-BCR-ABLp190Susceptibility to Confer the Metabolic Shift Essential for pB-ALL
by Martín-Lorenzo, Alberto and Auer, Franziska and Chan, Lai N and García-Ramírez, Idoia and González-Herrero, Inés and Rodríguez-Hernández, Guillermo and Bartenhagen, Christoph and Dugas, Martin and Gombert, Michael and Ginzel, Sebastian and Blanco, Oscar and Orfao, Alberto and Alonso-López, Diego and Rivas, Javier De Las and García-Cenador, Maria B and García-Criado, Francisco J and Müschen, Markus and Sánchez-García, Isidro and Borkhardt, Arndt and Vicente-Dueñas, Carolina and Hauer, Julia
Cancer Research, ISSN 0008-5472, 05/2018, Volume 78, Issue 10, pp. 2669 - 2679
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9. Full Text Understanding telomerase in cancer stem cell biology
by Sánchez-García, Isidro
Cell Cycle, ISSN 1538-4101, 04/2012, Volume 11, Issue 8, pp. 1479 - 1480
Comment on: Vicente-Dueñas C, et al. Oncotarget 2012; Epub ahead of print; PMID:22408137. 
mouse models | cancer stem cells (CSC) | stem cells | cancer | drug discovery | telomerase inhibitors | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | CELL BIOLOGY | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - enzymology | Promoter Regions, Genetic | Membrane Proteins - genetics | Humans | Antigens, Ly - genetics | Fusion Proteins, bcr-abl - genetics | Animals | Cell Transformation, Neoplastic | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Telomerase - metabolism | Mice | Neoplastic Stem Cells - enzymology | Fusion Proteins, bcr-abl - metabolism | Disease Models, Animal
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10. Full Text Function of the zinc-finger transcription factor SNAI2 in cancer and development
by Cobaleda, Ćesar and Ṕerez-Caro, María and Vicente-Duẽnas, Carolina and Śanchez-García, Isidro
Annual Review of Genetics, ISSN 0066-4197, 2007, Volume 41, Issue 1, pp. 41 - 61
Elucidation of the molecular mechanisms that underlie disease development is still a tremendous challenge for basic science, and a prerequisite to the... 
Biomarkers | Experimental therapeutics and translational oncology | Stem cell niche | Cancer stem cells | Mouse models | Stem cells | mouse models | stem cells | stem cell niche | biomarkers | E-CADHERIN EXPRESSION | C-KIT | EPITHELIAL-MESENCHYMAL TRANSITIONS | GENETICS & HEREDITY | GENE-EXPRESSION | STEM-CELL FACTOR | TYROSINE KINASE RECEPTOR | cancer stem cells | experimental therapeutics and translational oncology | HUMAN BREAST CELLS | GROWTH-FACTOR | NEURAL CREST | SLUG REPRESSOR PROTEIN | Neoplasms, Experimental - pathology | Transcription Factors - physiology | Zinc Fingers | Animals | Neoplasms, Experimental - genetics | Mice | Transcription Factors - genetics | Neoplasms, Experimental - physiopathology | Snail Family Transcription Factors
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11. Full Text Infection exposure is a causal factor in B-cell precursor acute lymphoblastic leukemia as a result of Pax5-inherited susceptibility
by Martín-Lorenzo, Alberto and Hauer, Julia and Vicente-Dueñas, Carolina and Auer, Franziska and González-Herrero, Inés and García-Ramírez, Idoia and Ginzel, Sebastian and Thiele, Ralf and Constantinescu, Stefan N and Bartenhagen, Christoph and Dugas, Martin and Gombert, Michael and Schäfer, Daniel and Blanco, Oscar and Mayado, Andrea and Orfao, Alberto and Alonso-López, Diego and De Las Rivas, Javier and Cobaleda, César and García-Cenador, Maria Begoña and García-Criado, Francisco Javier and Sánchez-García, Isidro and Borkhardt, Arndt
Cancer Discovery, ISSN 2159-8274, 12/2015, Volume 5, Issue 12, pp. 1328 - 1343
Earlier in the past century, infections were regarded as the most likely cause of childhood B-cell precursor acute lymphoblastic leukemia (pB-ALL). However,... 
ACTIVATION | ONCOLOGY | GENE-EXPRESSION | MECHANISMS | PAX5 | DIFFERENTIATION | NORMALIZATION | CHILDHOOD | LINEAGE | CANCER | JAK3 | Precursor B-Cell Lymphoblastic Leukemia-Lymphoma - pathology | Precursor B-Cell Lymphoblastic Leukemia-Lymphoma - etiology | Interleukin-7 - pharmacology | Male | Gene Expression Profiling | Exome | Janus Kinase 3 - antagonists & inhibitors | STAT5 Transcription Factor - genetics | Receptors, Interleukin-7 - genetics | Bone Marrow Transplantation | Female | Interleukin-7 - metabolism | Disease Models, Animal | Disease Susceptibility | PAX5 Transcription Factor - genetics | Genotype | Precursor B-Cell Lymphoblastic Leukemia-Lymphoma - therapy | Virus Integration | Mice, Knockout | Host-Pathogen Interactions | Drug Resistance, Neoplasm - genetics | Phenotype | Animals | Cell Transformation, Neoplastic | Janus Kinase 3 - genetics | Cell Line, Tumor | High-Throughput Nucleotide Sequencing | Mice | Protein Kinase Inhibitors - pharmacology | Mutation | Cluster Analysis
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12. Full Text Crebbp loss cooperates with Bcl2 overexpression to promote lymphoma in mice
by García-Ramírez, Idoia and Tadros, Saber and González-Herrero, Inés and Martín-Lorenzo, Alberto and Rodríguez-Hernández, Guillermo and Moore, Dalia and Ruiz-Roca, Lucía and Blanco, Oscar and Alonso-López, Diego and De Las Rivas, Javier and Hartert, Keenan and Duval, Romain and Klinkebiel, David and Bast, Martin and Vose, Julie and Lunning, Matthew and Fu, Kai and Greiner, Timothy and Rodrigues-Lima, Fernando and Jiménez, Rafael and Criado, Francisco Javier García and Cenador, María Begoña García and Brindle, Paul and Vicente-Dueñas, Carolina and Alizadeh, Ash and Sánchez-García, Isidro and Green, Michael R
Blood, ISSN 0006-4971, 05/2017, Volume 129, Issue 19, pp. 2645 - 2656
CREBBP is targeted by inactivating mutations in follicular lymphoma (FL) and diffuse large B-cell lymphoma (DLBCL). Here, we provide evidence from transgenic... 
B-CELL LYMPHOMA | ANALYSES REVEAL | MYC | P300 | GENE-EXPRESSION | GENOME-WIDE | FOLLICULAR LYMPHOMA | BINDING PROTEIN | HEMATOLOGY | HISTONE ACETYLATION | SOMATIC MUTATIONS | Lymphoma, Large B-Cell, Diffuse - pathology | Epigenesis, Genetic | Humans | Gene Expression Regulation, Neoplastic | Mice, Transgenic | CREB-Binding Protein - genetics | Animals | Lymphoma, Follicular - genetics | Gene Deletion | Mice | B-Lymphocytes - pathology | Mutation | Lymphoma, Large B-Cell, Diffuse - genetics | Proto-Oncogene Proteins c-bcl-2 - genetics | Lymphoid Neoplasia
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13. Full Text Transient expression of Bcl6 is sufficient for oncogenic function and induction of mature B-cell lymphoma
by Green, Michael R and Vicente-Dueñas, Carolina and Romero-Camarero, Isabel and Long Liu, Chih and Dai, Bo and González-Herrero, Inés and García-Ramírez, Idoia and Alonso-Escudero, Esther and Iqbal, Javeed and Chan, Wing C and Campos-Sanchez, Elena and Orfao, Alberto and Pintado, Belén and Flores, Teresa and Blanco, Oscar and Jiménez, Rafael and Martínez-Climent, Jose Angel and Criado, Francisco Javier García and Cenador, María Begoña García and Zhao, Shuchun and Natkunam, Yasodha and Lossos, Izidore S and Majeti, Ravindra and Melnick, Ari and Cobaleda, César and Alizadeh, Ash A and Sánchez-García, Isidro
Nature Communications, ISSN 2041-1723, 06/2014, Volume 5, Issue 1, p. 3904
Diffuse large B-cell lymphoma (DLBCL) is the most common lymphoma and can be separated into two subtypes based upon molecular features with similarities to... 
MOLECULAR SUBTYPES | INTEGRATIVE ANALYSIS REVEALS | STEM-CELLS | DNA METHYLATION | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | CHRONIC LYMPHOCYTIC-LEUKEMIA | NON-HODGKIN-LYMPHOMA | MYELOID-LEUKEMIA | SOMATIC MUTATION | COPY-NUMBER ALTERATION | Doxorubicin - therapeutic use | Prognosis | Epigenesis, Genetic | Humans | Gene Expression Regulation, Neoplastic | Male | Lymphoma, Large B-Cell, Diffuse - metabolism | Cyclophosphamide - therapeutic use | DNA Copy Number Variations | DNA-Binding Proteins - metabolism | DNA Methylation | Female | B-Lymphocytes - metabolism | Lymphoma, Large B-Cell, Diffuse - drug therapy | Mice, Transgenic | Hematopoietic Stem Cells - metabolism | DNA-Binding Proteins - genetics | Phenotype | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Vincristine - therapeutic use | Mice | Lymphoma, Large B-Cell, Diffuse - genetics | Prednisone - therapeutic use | Antibodies, Monoclonal, Murine-Derived - therapeutic use | Proto-Oncogene Proteins c-bcl-6
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14. Full Text Dnmt1 links BCR-ABLp210 to epigenetic tumor stem cell priming in myeloid leukemia
by Vicente-Dueñas, Carolina and González-Herrero, Inés and Sehgal, Lalit and García-Ramírez, Idoia and Rodríguez-Hernández, Guillermo and Pintado, Belén and Blanco, Oscar and Criado, Francisco Javier García and Cenador, María Begoña García and Green, Michael R and Sánchez-García, Isidro
Leukemia, ISSN 0887-6924, 01/2019, Volume 33, Issue 1, pp. 249 - 278
INDUCTION | ONCOLOGY | HEMATOLOGY | EXPRESSION | IMATINIB | Myelocytic leukemia | Stem cells | Cancer cells | Genetic aspects | Nonlymphoid leukemia | Research | Gene expression | Priming | Myeloid leukemia | Leukemia | DNMT1 protein | Brief Communication
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15. Full Text Tumoral stem cell reprogramming as a driver of cancer: Theory, biological models, implications in cancer therapy
by Vicente-Dueñas, Carolina and Hauer, Julia and Ruiz-Roca, Lucía and Ingenhag, Deborah and Rodríguez-Meira, Alba and Auer, Franziska and Borkhardt, Arndt and Sánchez-García, Isidro
Seminars in Cancer Biology, ISSN 1044-579X, 2014, Volume 32, pp. 3 - 9
Abstract Cancer is a clonal malignant disease originated in a single cell and characterized by the accumulation of partially differentiated cells that are... 
Hematology, Oncology and Palliative Medicine | Cancer stem cell | Mouse model | Oncogenes | Tumoral reprogramming | Stem cells | BLADDER-CARCINOMA ONCOGENE | HEMATOPOIETIC STEM/PROGENITOR CELLS | RAS | PERSISTENCE | HARVEY | KINASE | CHRONIC MYELOID-LEUKEMIA | ONCOLOGY | PRIMARY BIOASSAY | TRANSFORMING GENE | EXPRESSION | Cell Differentiation - genetics | Oncogenes - genetics | Neoplasms - therapy | Animals | Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Models, Biological | Humans | Mice | Neoplastic Stem Cells | Cellular Reprogramming - genetics | Disease Models, Animal | Antimitotic agents | Care and treatment | Automobile driving | Analysis | Antineoplastic agents | Health aspects | Cancer | Motor vehicle driving
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16. Full Text Infection exposure promotes ETV6-RUNX1 precursor B-cell leukemia via impaired H3K4 demethylases
by Rodríguez-Hernández, Guillermo and Hauer, Julia and Martín-Lorenzo, Alberto and Schäfer, Daniel and Bartenhagen, Christoph and García-Ramírez, Idoia and Auer, Franziska and Gonzalez-Herrero, Inés and Ruiz-Roca, Lucia and Gombert, Michael and Okpanyi, Vera and Fischer, Ute and Chen, Cai and Dugas, Martin and Bhatia, Sanil and Linka, René Martin and Garcia-Suquia, Marta and Rascón-Trincado, María Victoria and Garcia-Sanchez, Angel and Blanco, Oscar and García-Cenador, Maria Begoña and García-Criado, Francisco Javier and Cobaleda, César and Alonso-López, Diego and De Las Rivas, Javier and Müschen, Markus and Vicente-Dueñas, Carolina and Sánchez-García, Isidro and Borkhardt, Arndt
Cancer Research, ISSN 0008-5472, 08/2017, Volume 77, Issue 16, pp. 4365 - 4377
ETV6-RUNX1 is associated with the most common subtype of childhood leukemia. As few ETV6-RUNX1 carriers develop precursor B-cell acute lymphocytic leukemia... 
RECOMBINATION | READ ALIGNMENT | TEL/AML1 | SPECIFICITY | ONCOLOGY | ACUTE LYMPHOBLASTIC-LEUKEMIA | TEL-AML1 | MECHANISMS | NORMALIZATION | EXPRESSION | BURROWS-WHEELER TRANSFORM | Oncogene Proteins, Fusion - metabolism | Core Binding Factor Alpha 2 Subunit - metabolism | Precursor B-Cell Lymphoblastic Leukemia-Lymphoma - metabolism | Humans | Mice, Inbred C57BL | Oncogene Proteins, Fusion - biosynthesis | Male | Mice, Transgenic | Precursor B-Cell Lymphoblastic Leukemia-Lymphoma - microbiology | Precursor B-Cell Lymphoblastic Leukemia-Lymphoma - genetics | Histone Demethylases - metabolism | Animals | Core Binding Factor Alpha 2 Subunit - biosynthesis | Oncogene Proteins, Fusion - genetics | Mice, Inbred CBA | Mice | Core Binding Factor Alpha 2 Subunit - genetics | Disease Models, Animal | Hematopoietic Stem Cells | Recombination | Leukemia | Biological evolution | Stem cell transplantation | Runx1 protein | Infections | Exposure | Lymphatic leukemia | Alterations | RAG2 protein | Lymphocytes B | Lysine | Precursors | RAG1 protein | Stem cells | Children | Mutation | Cancer
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17. Full Text Novel ETV6‐RUNX1 Mouse Model to Study the Role of ELF‐MF in Childhood B‐Acute Lymphoblastic Leukemia: a Pilot Study
by Campos‐Sanchez, Elena and Vicente‐Dueñas, Carolina and Rodríguez‐Hernández, Guillermo and Capstick, Myles and Kuster, Niels and Dasenbrock, Clemens and Sánchez‐García, Isidro and Cobaleda, César
Bioelectromagnetics, ISSN 0197-8462, 07/2019, Volume 40, Issue 5, pp. 343 - 353
Exposure to extremely low‐frequency magnetic fields (ELF‐MFs) has been classified by the International Agency for Research on Cancer (IARC) as “possibly... 
ARIMMORA project | magnetic field | cancer susceptibility | exposure | FUSION | FREQUENCY MAGNETIC-FIELDS | MECHANISMS | CHILDREN | PRENATAL ORIGIN | TEL/AML1 | BIOPHYSICS | DISEASE | BIOLOGY | INFECTION | TEL-AML1 | Animal experimentation | Oncology, Experimental | Disease susceptibility | Research | Children | Acute lymphocytic leukemia | Magnetic fields | Cancer | Diseases | Flow cytometry | Animal models | Statistical analysis | Acute lymphatic leukemia | Leukemia | Runx1 protein | Exposure | Malignancy | Lymphatic leukemia | Carcinogens | Ataxin | Lymphocytes B | Etiology | Magnetic permeability | Peripheral blood | Mice | Fusion protein | Index Medicus
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