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1. Full Text L’espace d’une pensée. Lucie Roy, Le pouvoir de l’oubliée : la perception au cinéma, Paris, L’Harmattan, coll. « Esthétiques », 2015, 235 p
by Carrier-Lafleur, Thomas
Cinémas (Montréal), ISSN 1181-6945, 2016, Volume 27, Issue 1, p. 157
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2. Full Text MYBPC3 Gene Therapy for Neonatal Sarcomeric Cardiomyopathies
by Carrier, Lucie
Biophysical journal, ISSN 0006-3495, 01/2015, Volume 108, Issue 2, pp. 34a - 34a
Infants (Newborn) | Gene therapy | Care and treatment
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3. Full Text Cardiomyopathy phenotypes in human-induced pluripotent stem cell-derived cardiomyocytes—a systematic review
by Eschenhagen, Thomas and Eschenhagen, Thomas and Carrier, Lucie and Carrier, Lucie
Pflügers Archiv, ISSN 0031-6768, 5/2019, Volume 471, Issue 5, pp. 755 - 768
Quantitative phenotypes | Neurosciences | Receptors | Biomedicine | Human Physiology | hiPSC | Cardiomyopathy | Disease modelling | Molecular Medicine | Cell Biology | Cardiomyopathy, Dilated - genetics | Myocardial Contraction | Cardiomyopathy, Dilated - pathology | Cardiomyopathy, Hypertrophic - genetics | Myocytes, Cardiac - cytology | Cardiomyopathy, Hypertrophic - metabolism | Humans | Cardiomyopathy, Dilated - metabolism | Phenotype | Myocytes, Cardiac - physiology | Myocytes, Cardiac - metabolism | Cell Differentiation | Mutation | Induced Pluripotent Stem Cells - cytology | Cardiomyopathy, Hypertrophic - pathology | Induced Pluripotent Stem Cells - metabolism | Medical research | Heart cells | T cells | Muscle proteins | Gene expression | Analysis | Stem cells | Myosin | Medicine, Experimental | Genetic aspects | Heart diseases | Phenotypes | Transcription factors | Cardiomyocytes | NF-AT protein | Coronary artery disease | Dilated cardiomyopathy | Cell size | Localization | Pluripotency | Index Medicus | Invited Review
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4. Full Text Gene therapy strategies in the treatment of hypertrophic cardiomyopathy
by Prondzynski, Maksymilian and Mearini, Giulia and Carrier, Lucie
Pflügers Archiv, ISSN 0031-6768, 5/2019, Volume 471, Issue 5, pp. 807 - 815
MYBPC3 | Gene replacement | Neurosciences | Human Physiology | CRISPR/Cas9 | Cell Biology | trans -splicing | Receptors | Biomedicine | Exon skipping | Hypertrophic cardiomyopathy | Molecular Medicine | Gene therapy | trans-splicing | Life Sciences & Biomedicine | Physiology | Science & Technology | Cardiomyopathy, Hypertrophic - genetics | Gene Editing - methods | Animals | Cardiomyopathy, Hypertrophic - therapy | Humans | Genetic Therapy - methods | Proteins | Medical research | Anopheles | Care and treatment | Cardiomyopathy, Hypertrophic | RNA | Analysis | Genes | Genomics | Stem cells | Medicine, Experimental | Cell culture | Splicing | Cardiomyopathy | RNA-mediated interference | Genomes | Ribonucleic acid--RNA | Gene silencing | Fibrosis | Mutation | Ventricle | Pluripotency | Hypertrophy | Index Medicus
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5. Full Text The ubiquitin–proteasome system in cardiomyopathies
by Schlossarek, Saskia and Carrier, Lucie
Current opinion in cardiology, ISSN 0268-4705, 05/2011, Volume 26, Issue 3, pp. 190 - 195
cardiomyopathy | hypertrophy | ubiquitin-proteasome system | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Molecular Chaperones - metabolism | Humans | Ubiquitin - metabolism | Cardiomegaly - pathology | Endoplasmic Reticulum | Heart Failure - pathology | Molecular Chaperones - physiology | Proteasome Endopeptidase Complex - physiology | Ubiquitin - physiology | Protein Folding | Myocardial Ischemia - pathology | Myocytes, Cardiac - pathology | Proteasome Endopeptidase Complex - metabolism | Index Medicus
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6. Full Text Cardiac myosin-binding protein C (MYBPC3) in cardiac pathophysiology
by Carrier, Lucie and Mearini, Giulia and Stathopoulou, Konstantina and Cuello, Friederike
Gene, ISSN 0378-1119, 12/2015, Volume 573, Issue 2, pp. 188 - 197
MYBPC3 | Cardiomyopathies | Gene therapy | Cardiac myosin-binding protein C | Hypertrophy postranslational modifications | Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Carrier Proteins - physiology | Cardiomyopathies - genetics | Founder Effect | Genetic Therapy | Animals | Humans | Protein Processing, Post-Translational | Mutation | Cardiomyopathies - therapy | Protein C | Gene mutations | Muscle proteins | Cardiac patients | Myosin | Protein binding | Index Medicus | gene therapy | hypertrophy postranlational modifications | cardiomyopathies | cardiac myosin-binding protein C
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7. Full Text Ubiquitin-proteasome system and hereditary cardiomyopathies
by Schlossarek, Saskia and Frey, Norbert and Carrier, Lucie
Journal of molecular and cellular cardiology, ISSN 0022-2828, 2013, Volume 71, pp. 25 - 31
Cardiovascular | Dilated cardiomyopathy | Hypertrophic cardiomyopathy | Ubiquitin-proteasome system | Heart - physiopathology | Sarcomeres - metabolism | Humans | Ubiquitin - metabolism | Ubiquitin-Protein Ligases - metabolism | Myocardium - pathology | Autophagy - physiology | Cardiomyopathies - genetics | Cardiomyopathies - physiopathology | Animals | Cardiomyopathies - metabolism | Myocardium - metabolism | Proteasome Endopeptidase Complex - metabolism | Ubiquitin | Index Medicus
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8. Full Text How do MYBPC3 mutations cause hypertrophic cardiomyopathy?
by Marston, Steven and Marston, Steven and Copeland, O’Neal and Copeland, O’Neal and Gehmlich, Katja and Gehmlich, Katja and Schlossarek, Saskia and Schlossarek, Saskia and Carrrier, Lucie and Carrrier, Lucie
Journal of muscle research and cell motility, ISSN 0142-4319, 5/2012, Volume 33, Issue 1, pp. 75 - 80
Life Sciences | MYBPC3 | Ca 2+ -regulation | Nonsense mediated RNA decay | Proteomics | Hypertrophic cardiomyopathy | Ubiquitin-proteasome system | Animal Anatomy / Morphology / Histology | MyBP-C | Biomedicine general | Cell Biology | Haploinsufficiency | regulation | Life Sciences & Biomedicine | Science & Technology | Cardiomyopathy, Hypertrophic - genetics | Frameshift Mutation | Calcium - metabolism | Exons | Cardiomyopathy, Hypertrophic - metabolism | Humans | RNA, Messenger - genetics | Peptide Chain Termination, Translational | Myocardium - pathology | RNA, Messenger - metabolism | Nonsense Mediated mRNA Decay | Carrier Proteins - genetics | RNA Splicing | Animals | Carrier Proteins - metabolism | Myocardium - metabolism | Codon, Nonsense - metabolism | Codon, Nonsense - genetics | Cardiomyopathy, Hypertrophic - pathology | Proteins | Cardiomyopathy, Hypertrophic | RNA | Peptides | Cardiomyopathy | Genetic aspects | Heart diseases | Index Medicus
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9. Full Text The miRNA-212/132 family regulates both cardiac hypertrophy and cardiomyocyte autophagy
by Ucar, Ahmet and Gupta, Shashi K and Fiedler, Jan and Erikci, Erdem and Kardasinski, Michal and Batkai, Sandor and Dangwal, Seema and Kumarswamy, Regalla and Bang, Claudia and Holzmann, Angelika and Remke, Janet and Caprio, Massimiliano and Jentzsch, Claudia and Engelhardt, Stefan and Geisendorf, Sabine and Glas, Carolina and Hofmann, Thomas G and Nessling, Michelle and Richter, Karsten and Schiffer, Mario and Carrier, Lucie and Napp, L. Christian and Bauersachs, Johann and Chowdhury, Kamal and Thum, Thomas
Nature communications, ISSN 2041-1723, 2012, Volume 3, Issue 1, pp. 1078 - 1078
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Oligonucleotides - genetics | Cells, Cultured | Rats | Male | Mice, Transgenic | Reverse Transcriptase Polymerase Chain Reaction | Animals | Calcineurin - genetics | Myocytes, Cardiac - metabolism | Mice | MicroRNAs - genetics | Autophagy - genetics | Cardiomegaly - genetics | Index Medicus
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10. Full Text Targeting the population for gene therapy with MYBPC3
by Carrier, Lucie
Journal of molecular and cellular cardiology, ISSN 0022-2828, 01/2021, Volume 150, pp. 101 - 108
Gene replacement | MYBPC3 | Hypertrophic cardiomyopathy | Gene therapy | Index Medicus
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