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1997, Counterpoints, ISBN 0820438650, Volume 67, xxxv, 358
Book
2000, 1st Vintage Contemporaries ed., ISBN 0375708421, 260
Book
1995, 1st ed., ISBN 9780679431787, 260
Book
Journal Article
1978, General technical report (Pacific Northwest Forest and Range Experiment Station (Portland, Or.)), Volume PNW-GTR-67., 21
Book
Journal of Neurology, Neurosurgery & Psychiatry, ISSN 0022-3050, 12/2019, Volume 90, Issue 12, p. e11
BackgroundWhen patients with functional neurological disorders are followed up, another neurological disease rarely better explains the initial symptoms in... 
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2011, Volume 108, Issue 32, pp. 13253 - 13257
Journal Article
Lancet Oncology, The, ISSN 1470-2045, 2008, Volume 9, Issue 12, pp. 1166 - 1172
Journal Article
Journal of Neurology, ISSN 0340-5354, 8/2019, Volume 266, Issue 8, pp. 2018 - 2026
Several studies have shown that when patients with functional neurological disorders are followed up, it is rare to find another neurological condition that... 
Neurology | Neurosciences | Conversion disorder | Functional neurological disorders | Medicine & Public Health | Misdiagnosis | Psychogenic | Neuroradiology | STROKE | DIAGNOSIS | MULTIPLE-SCLEROSIS | PSYCHOGENIC NONEPILEPTIC SEIZURES | SYMPTOMS | EPILEPSY | CLINICAL NEUROLOGY | Neurological diseases | Diagnosis | Primary care | Neurological disorders | Original Communication
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2015, Volume 10, Issue 6, p. e0129867
Autoimmune diabetes mellitus (DM) results from the destruction of pancreatic islet cells by activated T lymphocytes, which have been primed by activated... 
ACTIVATION | NOD MICE | STIMULATION | INHIBITION | CD127 EXPRESSION | AUTOIMMUNITY | MULTIDISCIPLINARY SCIENCES | VITAMIN-D-3 | PREVENTION | T-CELLS | INNATE IMMUNITY | T-Lymphocyte Subsets - immunology | Leukocytes, Mononuclear - metabolism | Dendritic Cells - immunology | Diabetes Mellitus, Type 1 - metabolism | Adoptive Transfer | Diabetes Mellitus, Type 1 - therapy | Lymphocyte Activation - immunology | T-Lymphocyte Subsets - drug effects | Leukocytes, Mononuclear - immunology | Inflammation Mediators - metabolism | Dendritic Cells - drug effects | Female | Diabetes Mellitus, Type 1 - immunology | Dendritic Cells - metabolism | Disease Models, Animal | Toll-Like Receptor 7 - genetics | Gene Expression | Leukocytes, Mononuclear - drug effects | Toll-Like Receptor 7 - metabolism | Cytokines - metabolism | Immunomodulation | Diabetes Mellitus, Type 1 - genetics | Immune Tolerance | Immunity, Innate | B7-H1 Antigen - genetics | Mice, Knockout | B7-H1 Antigen - metabolism | Phenotype | Animals | T-Lymphocyte Subsets - metabolism | Ligands | Mice, Inbred NOD | Mice | Pancreatic beta cells | B cells | Dendritic cells | T cells | Type 1 diabetes | Diabetes therapy | Insulitis | CD86 antigen | Interleukin | Medical services | Lymphocytes T | Arthritis | Diabetes mellitus (insulin dependent) | Lymph nodes | Cell activation | Hyperglycemia | Lymphocytes | Polyethylene glycol | Rodents | Modulation | Interleukin 1 | Toll-like receptors | Bone marrow | Pancreas | CD11c antigen | Antigens | Phenotypes | Polyethylene | TLR7 protein | Cytokines | Immunoregulation | Diabetes mellitus | CD80 antigen | Inflammation | T cell receptors | Major histocompatibility complex | PD-L1 protein | Islet cells | Islets of Langerhans | Diabetes | Autoimmune diseases | Laboratory animals | Apoptosis | Cancer
Journal Article
PloS one, ISSN 1932-6203, 2009, Volume 4, Issue 12, p. e8294
Background: Aberrant activation of Wnt/beta-catenin signaling promotes the development of several cancers. It has been demonstrated that the Wnt signaling... 
MULTIPLE-MYELOMA | COLON-CANCER | APOPTOSIS | WNT PROTEINS | SIGNALING PATHWAY | CYCLIN D1 | MULTIDISCIPLINARY SCIENCES | BETA-CATENIN | NF-KAPPA-B | EXPRESSION | LEF-1 | Cell Survival - drug effects | Cyclin D1 - metabolism | Ethacrynic Acid - toxicity | Receptors, G-Protein-Coupled - metabolism | Humans | Lymphoid Enhancer-Binding Factor 1 - genetics | Frizzled Receptors - metabolism | Gene Expression Regulation, Leukemic - drug effects | Leukemia, Lymphocytic, Chronic, B-Cell - genetics | Wnt Proteins - metabolism | Lymphoid Enhancer-Binding Factor 1 - metabolism | beta Catenin - metabolism | Fibronectins - metabolism | Leukemia, Lymphocytic, Chronic, B-Cell - pathology | Frizzled Receptors - genetics | Cyclin D1 - genetics | Signal Transduction - drug effects | Leukemia, Lymphocytic, Chronic, B-Cell - metabolism | Protein Binding - drug effects | Acetylcysteine - pharmacology | Cell Line, Tumor | Fibronectins - genetics | Receptors, G-Protein-Coupled - genetics | Drug Screening Assays, Antitumor | Drugs | Wnt protein | Toxicity | Leukemia | Oxidizing agents | Multiple myeloma | Acetylcysteine | Cytotoxicity | Activation | Malignancy | Kinases | Assaying | Cyclin D1 | Drug resistance | Ethacrynic acid | Fibronectin | Immunosuppressive agents | Proteins | Antioxidants | β-catenin | Signal transduction | Oxidants | Lymphocytes | Inhibition | Library collections | Chronic lymphatic leukemia | Medical treatment | Lymphatic leukemia | Bioavailability | Gene expression | LEF protein | Signaling | Acids | Plasmids | Cell death | LEF-1 protein | N-Acetyl-L-cysteine | Lymphomas | Aberration | Viability | Apoptosis | Cancer
Journal Article
Stroke, ISSN 0039-2499, 03/2018, Volume 49, Issue 3, pp. 556 - 564
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2016, Volume 113, Issue 46, pp. 13150 - 13155
Journal Article