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1. Full Text Intratumor lactate levels reflect HER2 addiction status in HER2‐positive breast cancer
by Castagnoli, Lorenzo and Iorio, Egidio and Dugo, Matteo and Koschorke, Ada and Faraci, Simona and Canese, Rossella and Casalini, Patrizia and Nanni, Patrizia and Vernieri, Claudio and Di Nicola, Massimo and Morelli, Daniele and Tagliabue, Elda and Pupa, Serenella M
Journal of Cellular Physiology, ISSN 0021-9541, 02/2019, Volume 234, Issue 2, pp. 1768 - 1779
Despite different molecular tumor profiles indicate that human epidermal growth factor receptor 2 (HER2) messenger RNA (mRNA) levels mirror HER2 addiction and... 
oncogene addiction | breast cancer | HER2 | lactate | glycolysis | ADJUVANT CHEMOTHERAPY | MULTICENTER | PHYSIOLOGY | TRASTUZUMAB | EFFICACY | OPEN-LABEL | HYPOXIA | CELL BIOLOGY | GROWTH | TREATMENT RESPONSE | EXPRESSION | PLUS | Lactates | Breast cancer | DNA microarrays | Analysis | Enrichment | Biotechnology | Nuclear magnetic resonance--NMR | Transcription | Transgenic | Metabolism | Gene expression | Addictions | Ribonucleic acid--RNA | ErbB-2 protein | Magnetic resonance spectroscopy | Epidermal growth factor | Pathways | Addiction | Magnetic permeability | Cell lines | Glycolysis | Biomarkers | Lactic acid | Growth factors | Trastuzumab | Cancer | Tumors | Original | Original s
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2. Full Text The PDGFRβ/ERK1/2 pathway regulates CDCP1 expression in triple-negative breast cancer
by Forte, Luca and Turdo, Federica and Ghirelli, Cristina and Aiello, Piera and Casalini, Patrizia and Iorio, Marilena Valeria and D'Ippolito, Elvira and Gasparini, Patrizia and Agresti, Roberto and Belmonte, Beatrice and Sozzi, Gabriella and Sfondrini, Lucia and Tagliabue, Elda and Campiglio, Manuela and Bianchi, Francesca
BMC Cancer, ISSN 1471-2407, 05/2018, Volume 18, Issue 1, pp. 586 - 11
CDCP1, a transmembrane protein with tumor pro-metastatic activity, was recently identified as a prognostic marker in TNBC, the most aggressive breast cancer... 
PDGFRβ | ERK1/2 | CDCP1 | IHC | FISH | TNBC | PDGF-BB | Becaplermin - pharmacology | Up-Regulation | Humans | Middle Aged | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Gene Expression Regulation, Neoplastic | Neoplasm Proteins - metabolism | Receptor, Platelet-Derived Growth Factor beta - genetics | Cell Adhesion Molecules - metabolism | Antigens, CD - metabolism | Gene Knockdown Techniques | MAP Kinase Signaling System - drug effects | MAP Kinase Signaling System - genetics | Triple Negative Breast Neoplasms - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Triple Negative Breast Neoplasms - pathology | Cell Line, Tumor | Female | Mitogen-Activated Protein Kinase 1 - metabolism | RNA, Small Interfering - metabolism | Receptor, Platelet-Derived Growth Factor beta - metabolism
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3. Full Text FOXP3 Expression and Overall Survival in Breast Cancer
by Andrea Merlo and Patrizia Casalini and Maria Luisa Carcangiu and Chiara Malventano and Tiziana Triulzi and Sylvie Mènard and Elda Tagliabue and Andrea Balsari
Journal of Clinical Oncology, ISSN 0732-183X, 04/2009, Volume 27, Issue 11, pp. 1746 - 1752
Purpose The transcription factor forkhead box P3 (FOXP3) up-or downregulates a large number of genes and has been recently reported to be expressed in tumor... 
RELAPSE | METASTASIS | ONCOLOGY | LYMPH-NODES | REGULATORY T-CELLS | PANCREAS | PERIPHERAL-BLOOD | RADIOTHERAPY | PREVALENCE | SUPPRESSION | TUMOR MICROENVIRONMENT | Breast Neoplasms - surgery | Forkhead Transcription Factors - biosynthesis | Prognosis | Breast Neoplasms - pathology | Humans | Middle Aged | Survival Analysis | Breast Neoplasms - mortality | Female | Breast Neoplasms - metabolism
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4. Full Text Defective stromal remodeling and neutrophil extracellular traps in lymphoid tissues favor the transition from autoimmunity to lymphomas
by Sangaletti, Sabina and Tripodo, Claudio and Vitali, Caterina and Portararo, Paola and Guarnotta, Carla and Casalini, Patrizia and Cappetti, Barbara and Miotti, Silvia and Pinciroli, Patrizia and Fuligni, Fabio and Fais, Franco and Piccaluga, Pier Paolo and Colombo, Mario P
Cancer Discovery, ISSN 2159-8274, 01/2014, Volume 4, Issue 1, pp. 110 - 129
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5. Full Text Breast cancer-secreted miR-939 downregulates VE-cadherin and destroys the barrier function of endothelial monolayers
by Di Modica, Martina and Regondi, Viola and Sandri, Marco and Iorio, Marilena V and Zanetti, Adriana and Tagliabue, Elda, Dr and Casalini, Patrizia and Triulzi, Tiziana
Cancer Letters, ISSN 0304-3835, 2016, Volume 384, pp. 94 - 100
Abstract Exosomes-secreted microRNAs play an important role in metastatic spread. During this process breast cancer cells acquire the ability to transmigrate... 
Hematology, Oncology and Palliative Medicine | Breast cancer | Metastasis | miR-939 | Exosomes | Triple-negative breast cancer | CELLS | DIAGNOSIS | ANGIOGENESIS | RISK | MICRORNAS | BIOMARKERS | VESSELS | ONCOLOGY | JUNCTIONS | DISTANT METASTASIS | Exosomes - metabolism | Cadherins - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Antigens, CD - genetics | Antigens, CD - metabolism | Neoplasm Metastasis | Transfection | Time Factors | Transendothelial and Transepithelial Migration | Triple Negative Breast Neoplasms - pathology | Female | Cadherins - genetics | Paracrine Communication | Signal Transduction | Down-Regulation | Proportional Hazards Models | Permeability | Disease-Free Survival | Triple Negative Breast Neoplasms - genetics | Triple Negative Breast Neoplasms - metabolism | Cell Line, Tumor | Human Umbilical Vein Endothelial Cells - pathology | MicroRNAs - genetics | Analysis | Molecular biology | Monomolecular films | Endothelium | Proteins | Cloning | Medical prognosis | Mortality | Conflicts of interest | Binding sites | Methods | Tumors
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6. Full Text Intratumor lactate levels reflect HER2 addiction status in HER2-positive breast cancer
: CASTAGNOLI et al
by Castagnoli, Lorenzo and Iorio, Egidio and Dugo, Matteo and Koschorke, Ada and Faraci, Simona and Canese, Rossella and Casalini, Patrizia and Nanni, Patrizia and Vernieri, Claudio and Di Nicola, Massimo and Morelli, Daniele and Tagliabue, Elda and Pupa, Serenella M
Journal of Cellular Physiology, ISSN 0021-9541, 02/2019, Volume 234, Issue 2, pp. 1768 - 1779
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7. Full Text FOXP3 expression in tumor cells and implications for cancer progression
by Triulzi, Tiziana and Tagliabue, Elda and Balsari, Andrea and Casalini, Patrizia
Journal of Cellular Physiology, ISSN 0021-9541, 01/2013, Volume 228, Issue 1, pp. 30 - 35
FOXP3 is a member of the forkhead/winged‐helix family of transcriptional regulators. Defects in the FOXP3 gene cause an X‐linked autoimmune/immunodeficiency... 
BREAST-CANCER | CARCINOMA-CELLS | LUNG-CANCER | ACTIVATION | ONCOGENE | PHYSIOLOGY | INHIBITION | ACETYLATION | REGULATORY T-CELLS | REPRESSOR | ASSOCIATION | CELL BIOLOGY | Neoplasm Metastasis | Animals | Forkhead Transcription Factors - metabolism | T-Lymphocyte Subsets - metabolism | Humans | Forkhead Transcription Factors - genetics | Gene Expression Regulation, Neoplastic - physiology | Development and progression | Metastasis | Genetic transcription | Cancer
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8. Full Text Conversion to stem-cell state in response to microenvironmental cues is regulated by balance between epithelial and mesenchymal features in lung cancer cells
by Andriani, Francesca and Bertolini, Giulia and Facchinetti, Federica and Baldoli, Erika and Moro, Massimo and Casalini, Patrizia and Caserini, Roberto and Milione, Massimo and Leone, Giorgia and Pelosi, Giuseppe and Pastorino, Ugo and Sozzi, Gabriella and Roz, Luca
Molecular Oncology, ISSN 1574-7891, 02/2016, Volume 10, Issue 2, pp. 253 - 271
Cancer cells within a tumor are functionally heterogeneous and specific subpopulations, defined as cancer initiating cells (CICs), are endowed with higher... 
SNAI2 | Microenvironment | CDH1 | Lung cancer | Cancer initiating cells | Plasticity | DOWN-REGULATION | POPULATIONS | E-CADHERIN | MIR-200 FAMILY | TUMOR HETEROGENEITY | FIBROBLASTS | TRANSITION | ZEB1 | ONCOLOGY | EXPRESSION | Cadherins - metabolism | Epigenesis, Genetic | Humans | Lung Neoplasms - metabolism | Transforming Growth Factor beta1 - metabolism | Glycoproteins - metabolism | Lung Neoplasms - pathology | Epithelial-Mesenchymal Transition - genetics | Antigens, CD - metabolism | Peptides - metabolism | Tumor Microenvironment - genetics | Neoplastic Stem Cells - metabolism | Neoplastic Stem Cells - pathology | Female | Snail Family Transcription Factors | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | AC133 Antigen | Transcription Factors - metabolism | Phenotype | Animals | Mice, Nude | Cell Line, Tumor | Cell Plasticity | Mice | Primary Cell Culture | Epigenetic inheritance | Analysis | Cancer cells | Stem cells | Transforming growth factors | Tumors | Subpopulations | Phenotypes | Mesenchyme | Laboratories | Breast cancer | Metastasis | Gene expression | E-cadherin | Cell adhesion & migration | Proteins | External stimuli | Medical prognosis | Cell lines | DNA methylation | Fibroblasts | Epigenetics | Snail protein
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9. Full Text Oncosuppressive role of p53-induced miR-205 in triple negative breast cancer
by Piovan, Claudia and Palmieri, Dario and Di Leva, Gianpiero and Braccioli, Luca and Casalini, Patrizia and Nuovo, Gerard and Tortoreto, Monica and Sasso, Marianna and Plantamura, Ilaria and Triulzi, Tiziana and Taccioli, Cristian and Tagliabue, Elda and Iorio, Marilena V and Croce, Carlo M
Molecular Oncology, ISSN 1574-7891, 08/2012, Volume 6, Issue 4, pp. 458 - 472
An increasing body of evidence highlights an intriguing interaction between microRNAs and transcriptional factors involved in determining cell fate, including... 
E2F1 | LAMC1 | miR-205 | p53 | MiR-205 | P53 | PROSTATE | EXPRESSION ANALYSIS | PROTEIN | LAMININ | DOWN-REGULATION | CELL-PROLIFERATION | SUPPRESSION | INVASION | ONCOLOGY | CARCINOMAS | SENESCENCE | Cell Cycle - genetics | Cellular Senescence - genetics | Cell Proliferation | Humans | Gene Expression Regulation, Neoplastic | Tumor Suppressor Protein p53 - metabolism | Molecular Sequence Data | MicroRNAs - metabolism | E2F1 Transcription Factor - metabolism | Mice, SCID | Down-Regulation - genetics | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Base Sequence | Cell Line, Tumor | Protein Binding | Female | Transcription, Genetic | Mice | MicroRNAs - genetics | Response Elements - genetics | Laminin - metabolism | MicroRNA | Emissions (Pollution) | Genomics | Stem cells | Development and progression | Breast cancer | Genetic transcription | Tumor proteins | Cells | Tumors | Cell proliferation | Transcription factors | Transcription | Mesenchyme | p53 Protein | Genomes | Kinases | Experiments | Cell adhesion & migration | Cell fate | Penicillin | Cell adhesion | Cell cycle | Extracellular matrix | Phenotypes | Cloning | MiRNA | Mutagenesis | Plasmids | MicroRNAs | Cell lines | Tumor suppressor genes | Regulation | Mutation | Binding sites | Cell migration | miRNA | p53 protein | Papers | Paper
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10. Full Text Neutrophil extracellular traps mediate transfer of cytoplasmic neutrophil antigens to myeloid dendritic cells toward ANCA induction and associated autoimmunity
by Sangaletti, Sabina and Tripodo, Claudio and Chiodoni, Claudia and Guarnotta, Carla and Cappetti, Barbara and Casalini, Patrizia and Piconese, Silvia and Parenza, Mariella and Guiducci, Cristiana and Vitali, Caterina and Colombo, Mario P
Blood, ISSN 0006-4971, 10/2012, Volume 120, Issue 15, pp. 3007 - 3018
Antineutrophil cytoplasmic antibodies (ANCAs) target proteins normally retained within neutrophils, indicating that cell death is involved in the autoimmunity... 
ANTIBODIES | AUTOANTIBODIES | IMMUNITY | SYSTEMIC-LUPUS-ERYTHEMATOSUS | NETTING NEUTROPHILS | DYING CELLS | RECOGNITION | APOPTOTIC NEUTROPHILS | CLINICAL ASSOCIATIONS | VASCULITIS | HEMATOLOGY
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11. Full Text Dasatinib reduces FAK phosphorylation increasing the effects of RPI-1 inhibition in a RET/PTC1-expressing cell line
by Caccia, Dario and Miccichè, Francesca and Cassinelli, Giuliana and Mondellini, Piera and Casalini, Patrizia and Bongarzone, Italia
Molecular Cancer, ISSN 1476-4598, 10/2010, Volume 9, Issue 1, pp. 278 - 278
Background: TPC-1 is a papillary thyroid carcinoma (PTC)-derived cell line that spontaneously expresses the oncogene RET/PTC1. TPC-1 treated with the RET/PTC1... 
RET | SURVIVAL | BREAST-CANCER CELLS | ACTIVATION | ONCOLOGY | SIGNALING PATHWAY | MAP KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | PAPILLARY THYROID-CARCINOMA | FOCAL ADHESION KINASE | RET/PTC1 ONCOPROTEIN | ANTITUMOR-ACTIVITY | Dasatinib | Proto-Oncogene Proteins c-ret - metabolism | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Humans | Receptor, EphA2 - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Pyrimidines - pharmacology | Integrin beta1 - metabolism | Indoles - metabolism | Thyroid Neoplasms - drug therapy | Cell Line, Tumor | Protein Kinase Inhibitors - pharmacology | Thiazoles - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Indoles - antagonists & inhibitors | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | STAT3 Transcription Factor - metabolism | Thyroid Neoplasms - metabolism | Thyroid cancer | Genes | Physiological aspects | Genetic aspects | Research | Drug therapy | Health aspects | Protein kinases | Proteins | Medical research | Enzymes | Phosphorylation | Cell cycle | Clinical medicine | Kinases | Experiments | Manuscripts | Molecular weight | Cancer
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12. Full Text The PDGFR[beta]/ERK1/2 pathway regulates CDCP1 expression in triple-negative breast cancer
by Forte, Luca and Turdo, Federica and Ghirelli, Cristina and Aiello, Piera and Casalini, Patrizia and Iorio, Marilena Valeria and D'Ippolito, Elvira and Gasparini, Patrizia and Agresti, Roberto and Belmonte, Beatrice and Sozzi, Gabriella and Sfondrini, Lucia and Tagliabue, Elda and Campiglio, Manuela and Bianchi, Francesca
BMC Cancer, ISSN 1471-2407, 05/2018, Volume 18, Issue 1
CDCP1, a transmembrane protein with tumor pro-metastatic activity, was recently identified as a prognostic marker in TNBC, the most aggressive breast cancer... 
Immunohistochemistry | Breast cancer | Prognosis | Platelet-derived growth factor | Metastasis
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13. Full Text HER2 Overexpression and Doxorubicin in Adjuvant Chemotherapy for Resectable Breast Cancer
by Angela Moliterni and Sylvie Ménard and Pinuccia Valagussa and Elia Biganzoli and Patrizia Boracchi and Andrea Balsari and Patrizia Casalini and Gorana Tomasic and Ettore Marubini and Silvana Pilotti and Gianni Bonadonna
Journal of Clinical Oncology, ISSN 0732-183X, 02/2003, Volume 21, Issue 3, pp. 458 - 462
Purpose: Human epidermal growth factor receptor 2 (HER2) overexpression was found to predict a good response in breast carcinoma patients treated with... 
WOMEN | ERBB-2 | THERAPY | ONCOLOGY | CYCLOPHOSPHAMIDE | RESIDUALS | METHOTREXATE | TUMOR | REGRESSION-MODEL | FLUOROURACIL | C-ERBB-2 EXPRESSION | Immunohistochemistry | Doxorubicin - therapeutic use | Predictive Value of Tests | Cyclophosphamide - administration & dosage | Receptor, ErbB-2 - biosynthesis | Antineoplastic Combined Chemotherapy Protocols - administration & dosage | Prognosis | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Antineoplastic Agents - therapeutic use | Breast Neoplasms - drug therapy | Survival | Breast Neoplasms - genetics | Fluorouracil - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Breast Neoplasms - pathology | Adult | Female | Methotrexate - administration & dosage | Aged | Chemotherapy, Adjuvant | Receptor, ErbB-2 - analysis
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14. Full Text Defective Stromal Remodeling and Neutrophil Extracellular Traps in Lymphoid Tissues Favor the Transition from Autoimmunity to Lymphoma
by Sangaletti, S and Tripodo, C and Vitali, C and Portararo, P and Guarnotta, C and Casalini, P and Cappetti, B and Miotti, S and Pinciroli, P and Fuligni, F and Fais, F and Piccaluga, PP and Colombo, MP
CANCER DISCOVERY, ISSN 2159-8274, 01/2014, Volume 4, Issue 1, pp. 110 - 129
Altered expression of matricellular proteins can become pathogenic in the presence of persistent perturbations in tissue homeostasis. Here, we show that... 
INFLAMMATORY DISORDERS | SYSTEMIC-LUPUS-ERYTHEMATOSUS | KAPPA-B ACTIVATION | IMMUNE-SYSTEM | DENDRITIC CELLS | ONCOLOGY | MARGINAL ZONE | MATRICELLULAR PROTEIN | GENE-EXPRESSION | CHRONIC LYMPHOCYTIC-LEUKEMIA | INHIBITORY RECEPTOR LAIR-1 | Autoimmunity | Osteonectin - immunology | Humans | Cells, Cultured | Extracellular Matrix - immunology | Lymphoma - immunology | NF-kappa B - immunology | Neutrophils - immunology | Lymphoma - genetics | Lymphoid Tissue - immunology | Lymphoid Tissue - cytology | Animals | B-Lymphocytes - immunology | Osteonectin - genetics | Mice, Mutant Strains | CD5 Antigens - immunology | fas Receptor - genetics | Mice
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15. Full Text Toll-like receptor 3 as a new marker to detect high risk early stage Non-Small-Cell Lung Cancer patients
by Bianchi, F and Milione, M and Casalini, P and Centonze, G and Le Noci, VM and Storti, C and Alexiadis, S and Truini, M and Sozzi, G and Pastorino, U and Balsari, A and Tagliabue, E and Sfondrini, L
SCIENTIFIC REPORTS, ISSN 2045-2322, 10/2019, Volume 9, Issue 1, pp. 1 - 10
Immune and epithelial cells express TLR3, a receptor deputed to respond to microbial signals activating the immune response. The prognostic value ofTLR3 in... 
APOPTOSIS | PROGNOSIS | MULTIDISCIPLINARY SCIENCES | TLR3 | Immunohistochemistry | Cell survival | Immune response | Small cell lung carcinoma | PD-1 protein | Tumor cells | Epithelial cells | Stroma | Non-small cell lung carcinoma | Macrophages | Patients | TLR3 protein | Medical prognosis | Surgery | Toll-like receptors
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