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1998, 1. ed., Agulla, ISBN 9788489936270, Volume 26, 211
Book
2001, ISBN 9789041116642, xv, 272
Book
Nature, ISSN 0028-0836, 04/2012, Volume 484, Issue 7392, pp. 44 - 46
According to this theory4,5, antiangiogenic therapy induces structural and functional changes in tumour blood vessels - which have abnormal features - to make... 
Angiogenesis | Sequential scheduling | Hypoxia | Cytotoxicity | Vascular endothelial growth factor | Cancer therapies | Tumors
Journal Article
Revista Española de Derecho Internacional, ISSN 0034-9380, 6/2019, Volume 71, Issue 1, pp. 17 - 22
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 08/2018, Volume 128, Issue 9, pp. 3887 - 3905
Journal Article
1980, Llavor, ISBN 9788485185214, 89
Book
Nature, ISSN 0028-0836, 04/2012, Volume 484, Issue 7392, pp. 110 - 114
Developing tissues and growing tumours produce vascular endothelial growth factors (VEGFs), leading to the activation of the corresponding receptors in... 
TIP CELL-FORMATION | DLL4 | ACTIVATION | BEVACIZUMAB | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | GROWTH | NETWORK FORMATION | KINASE INHIBITORS | TUMOR ANGIOGENESIS | EXPRESSION | Human Umbilical Vein Endothelial Cells | Neovascularization, Physiologic - drug effects | Up-Regulation | Vascular Endothelial Growth Factor Receptor-3 - biosynthesis | Receptors, Notch - metabolism | Humans | Male | Intracellular Signaling Peptides and Proteins - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor Receptor-2 - genetics | Intracellular Signaling Peptides and Proteins - deficiency | Membrane Proteins - deficiency | Receptors, Notch - antagonists & inhibitors | HEK293 Cells | Female | Transcription, Genetic | Membrane Proteins - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Endothelial Cells - metabolism | Membrane Proteins - genetics | Vascular Endothelial Growth Factor Receptor-2 - deficiency | Cells, Cultured | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Neovascularization, Physiologic - physiology | Animals | Signal Transduction - drug effects | Endothelial Cells - cytology | Models, Biological | Mice | Protein Kinase Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-3 - genetics | Endothelial Cells - drug effects | Cellular control mechanisms | Physiological aspects | Growth factor receptors | Neovascularization | Research | Vascular endothelial growth factor | Endothelium | Angiogenesis | Ligands | Kinases | Rodents | Index Medicus
Journal Article
Journal Article
Current Drug Targets, ISSN 1389-4501, 2016, Volume 17, Issue 15, pp. 1728 - 1734
The mechanisms of tumor growth and progression involve the activation of different processes such as neovascularization and angiogenesis. These processes... 
Anti-angiogenic therapy | Intrinsic resistance | Acquired resistance | Tumor cells | Stromal cells | TARGETED THERAPY | anti-angiogenic therapy | intrinsic resistance | VEGF | TUMOR | DRUG-RESISTANCE | ANTITUMOR-ACTIVITY | RENAL-CELL CARCINOMA | INTERFERON-ALPHA | stromal cells | TYROSINE KINASE INHIBITOR | PHARMACOLOGY & PHARMACY | tumor cells | MOLECULAR-BASIS | KIDNEY CANCER
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2012, Volume 122, Issue 5, pp. 1832 - 1848
Cancer development, progression, and metastasis are highly dependent on angiogenesis. The use of antiangiogenic drugs has been proposed as a novel strategy to... 
MEDICINE, RESEARCH & EXPERIMENTAL | VASCULAR NORMALIZATION | INDUCIBLE FACTOR-1-ALPHA | SQUAMOUS CARCINOGENESIS | PANCREATIC NEUROENDOCRINE TUMORS | MOUSE MODEL | ANTI-ANGIOGENIC THERAPY | GROWTH-FACTOR | K14-HPV16 TRANSGENIC MICE | NF-KAPPA-B | MALIGNANT PROGRESSION | Proto-Oncogene Proteins c-met - metabolism | Genetic Therapy | Pancreatic Neoplasms - metabolism | Uterine Cervical Neoplasms - blood supply | Pancreatic Neoplasms - blood supply | Drug Resistance, Neoplasm | NF-kappa B - metabolism | Uterine Cervical Neoplasms - pathology | Recombinant Proteins - biosynthesis | Semaphorin-3A - biosynthesis | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Pericytes - pathology | Epithelial-Mesenchymal Transition | Angiogenesis Inhibitors - therapeutic use | Female | Indoles - pharmacology | Liver Neoplasms - secondary | Pyrroles - therapeutic use | Uterine Cervical Neoplasms - therapy | Semaphorin-3A - genetics | Neuroendocrine Tumors - pathology | Cell Hypoxia - drug effects | Neoplasm Invasiveness | Neuroendocrine Tumors - metabolism | Pancreatic Neoplasms - pathology | Angiogenesis Inhibitors - pharmacology | Mice, Transgenic | Combined Modality Therapy | Lymphatic Metastasis | Recombinant Proteins - genetics | Tumor Burden | Pyrroles - pharmacology | Animals | Uterine Cervical Neoplasms - virology | Indoles - therapeutic use | Mice | Neuroendocrine Tumors - therapy | Neovascularization, Physiologic | Neuroendocrine Tumors - blood supply | Pancreatic Neoplasms - therapy | Research | Angiogenesis inhibitors | Semaphorins | Cancer cells | Index Medicus | Abridged Index Medicus
Journal Article
Nature communications, ISSN 2041-1723, 11/2018, Volume 9, Issue 1, pp. 4826 - 16
Angiogenesis is a dynamic process relying on endothelial cell rearrangements within vascular tubes, yet the underlying mechanisms and functional relevance are... 
ACTIN POLYMERIZATION | MULTIDISCIPLINARY SCIENCES | NOTCH | VE-CADHERIN | RETINAL ANGIOGENESIS | ADHERENS JUNCTIONS | VESSEL FUSION | SPROUTING ANGIOGENESIS | MYOSIN PHOSPHATASE | DEVELOPING ZEBRAFISH | MASS-SPECTROMETRY | Protein Kinases - metabolism | Protein Kinases - genetics | Retina - metabolism | Retina - growth & development | Human Umbilical Vein Endothelial Cells - metabolism | Embryo, Mammalian | Humans | Gene Expression Profiling | Phosphatidylinositol 3-Kinases - metabolism | Lung - cytology | Phosphoproteins - metabolism | Actomyosin - genetics | Retina - cytology | Gene Expression Regulation, Developmental | Human Umbilical Vein Endothelial Cells - cytology | Myosin-Light-Chain Phosphatase - metabolism | Lung - metabolism | Repressor Proteins - metabolism | Signal Transduction | Neovascularization, Physiologic - genetics | Mice, Inbred C57BL | Myosin-Light-Chain Phosphatase - genetics | Intercellular Junctions - metabolism | Repressor Proteins - genetics | Mice, Transgenic | Zebrafish | Phosphoproteins - genetics | Actomyosin - metabolism | Phosphatidylinositol 3-Kinases - genetics | Animals | Embryo, Nonmammalian | Lung - growth & development | Mice | Intercellular Junctions - ultrastructure | Lung - blood supply | Body Patterning - genetics | Pattern formation | Phosphorylation | Actomyosin | Contractility | Phosphatase | Kinases | Endothelial cells | 1-Phosphatidylinositol 3-kinase | Myosin-light-chain-phosphatase | Proteins | Angiogenesis | Signal transduction | Clonal deletion | Myosin | Deletion | Tubes | Index Medicus | Proteïnes quinases | Regulació genètica | Neovascularization | Genetic regulation | Gene expression | Angiogènesi | Protein kinases | Expressió gènica
Journal Article