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2010, Oxford American neurology library, ISBN 0195393384, viii, 252
Book
Physiological Genomics, ISSN 1094-8341, 04/2008, Volume 33, Issue 2, pp. 240 - 256
Journal Article
Journal of the American Geriatrics Society, ISSN 0002-8614, 01/2019, Volume 67, Issue 1, pp. 192 - 193
This letter comments on the letter by Luis Aguera‐Ortiz et al. 
Cognitive ability
Journal Article
PLoS Genetics, ISSN 1553-7390, 09/2008, Volume 4, Issue 9, pp. e1000193 - e1000193
Journal Article
Journal Article
Neurology, ISSN 0028-3878, 07/2015, Volume 85, Issue 3, pp. 298 - 299
Johansson et al.[1] described the influence of personality features and long-standing distress on the risk of Alzheimer disease (AD) dementia, which remained... 
Alzheimer Disease - diagnosis | Follow-Up Studies | Time Factors | Humans | Middle Aged | Alzheimer Disease - epidemiology | Female | Personality | Personality Disorders - psychology | Personality Disorders - diagnosis | Alzheimer Disease - psychology | Personality Disorders - epidemiology
Journal Article
Pediatric Blood & Cancer, ISSN 1545-5009, 10/2014, Volume 61, Issue 10, pp. 1739 - 1742
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2001, Volume 98, Issue 6, pp. 3334 - 3339
Cross-sectional positron emission tomography (PET) studies find that cognitively normal carriers of the apolipoprotein E (APOE) ε4 allele, a common Alzheimer's... 
Biological Sciences | Transcriptional regulatory elements | Disorders | Heterozygotes | Placebos | Prefrontal cortex | Alzheimers disease | Positron emission tomography | Family history | Genotypes | Dementia
Journal Article
Medical Image Analysis, ISSN 1361-8415, 01/2017, Volume 35, p. 517
Landmark curves were widely adopted in neuroimaging research for surface correspondence computation and quantified morphometry analysis. However, most of the... 
Neuroimaging | Brain | Euclidean geometry | Medical imaging | Deformities | Deformation | Neurodegenerative diseases | Abnormalities | Cortex | Data processing | Disease control | Patients | Invariants | Isometric | Neurology | Magnetic resonance imaging | Biomarkers | Bioindicators | Alzheimers disease | Morphometry | Surface area | Alzheimer's disease
Journal Article
Neuron, ISSN 0896-6273, 08/2017, Volume 95, Issue 4, pp. 808 - 816.e9
Amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD) are age-related neurodegenerative disorders with shared genetic etiologies and... 
low-complexity domain | liquid-liquid phase separation | TDP-43 | T cell-restricted intracellular antigen-1 | stress granules | frontotemporal dementia | amyotrophic lateral sclerosis | membrane-less organelle | frontotemporal lobar degeneration | MULTISYSTEM PROTEINOPATHY | DISTAL MYOPATHY | MESSENGER-RNA | LIQUID DROPLETS | SEQUENCING DATA | HEXANUCLEOTIDE REPEAT | DOMAINS | C9ORF72 | NEUROSCIENCES | FAMILIAL ALS | Humans | Middle Aged | Family Health | Male | Green Fluorescent Proteins - genetics | DNA-Binding Proteins - metabolism | Transfection | Time Factors | Adult | Female | T-Cell Intracellular Antigen-1 | Frontotemporal Dementia - pathology | Frontotemporal Dementia - genetics | Stress, Physiological - physiology | Green Fluorescent Proteins - metabolism | Poly(A)-Binding Proteins - genetics | Amyotrophic Lateral Sclerosis - genetics | Heterogeneous-Nuclear Ribonucleoprotein Group A-B - metabolism | RNA-Binding Protein FUS - metabolism | Mutation - genetics | Heterogeneous Nuclear Ribonucleoprotein A1 | Microscopy, Confocal | Amyotrophic Lateral Sclerosis - pathology | Aged | HeLa Cells | Nervous system diseases | RNA | Analysis | Genetic research | Development and progression | Amyotrophic lateral sclerosis | Genetic aspects | T cells | Binding proteins | Protein binding | Dementia | Disease | Pathogenesis | Genes | Disorders | Lymphocytes T | Phase transitions | Proteins | Consortia | DNA-binding protein | Etiology | Dementia disorders | Age | Deoxyribonucleic acid--DNA | Phase transformations | Neurodegenerative diseases | Metabolism | Ribonucleic acid--RNA | Pathology | Phase separation | Mutation | Frontotemporal dementia | Dismantling | Phase transition | T-cell-restricted intracellular antigen-1
Journal Article
Alzheimer's & Dementia: The Journal of the Alzheimer's Association, ISSN 1552-5260, 2016, Volume 12, Issue 7, pp. P287 - P288
Journal Article
JAMA Neurology, ISSN 2168-6149, 10/2015, Volume 72, Issue 10, p. 1124
  β-Amyloid peptide (Aβ) plaques are a cardinal neuropathologic feature of Alzheimer disease (AD), yet more than one-third of apolipoprotein E ...4 (APOE4)... 
Peptides | Alzheimers disease | Medical diagnosis | Patients | Dementia
Journal Article
NeuroImage: Clinical, ISSN 2213-1582, 2019, Volume 22, p. 101744
Apolipoprotein E (APOE) e4 is the major genetic risk factor for late-onset Alzheimer's disease (AD). The dose-dependent impact of this allele on hippocampal... 
Magnetic resonance imaging (MRI) | APOE-e4 | Hippocampal morphometry | Alzheimer's disease | Cognitively unimpaired
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 12/2008, Volume 17, Issue 23, pp. 3631 - 3642
Journal Article
Alzheimer's & Dementia: The Journal of the Alzheimer's Association, ISSN 1552-5260, 2014, Volume 10, Issue 4, pp. P687 - P687
Journal Article
Journal Article
Medicine and Science in Sports and Exercise, ISSN 0195-9131, 01/2007, Volume 39, Issue 1, pp. 199 - 207
Journal Article
JAMA Neurology, ISSN 2168-6149, 03/2015, Volume 72, Issue 3, p. 333
  There is a deficit of pituitary adenylate cyclase-activating polypeptide (PACAP) in patients with neuropathologically confirmed Alzheimer dementia. However,... 
Neurology | Brain | Polypeptides | Cognition & reasoning | Correlation analysis | Alzheimers disease | Dementia
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 01/2013, Volume 33, Issue s1, pp. S405 - S416
Studies of asymptomatic carriers of genes that are known to predispose to Alzheimers disease (AD) have facilitated the characterization of preclinical AD. The... 
Journal Article
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