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PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 6, pp. e0157251 - e0157251
The Akt/adenosine monophosphate protein kinase (AMPK)/mammalian target of rapamycin (mTOR) pathway has emerged as a critical signaling nexus for regulating... 
MOLECULAR TARGETS | INHIBITION | MAMMARY-TUMOR GROWTH | RED WINE POLYPHENOLS | PI3K PATHWAY | MULTIDISCIPLINARY SCIENCES | IN-VIVO | AMPK | FLAVONOID QUERCETIN | GRAPE SEED EXTRACT | CELL-LINE | AMP-Activated Protein Kinases - metabolism | Vitis - chemistry | Stilbenes - therapeutic use | TOR Serine-Threonine Kinases - metabolism | Humans | Stilbenes - chemistry | Stilbenes - pharmacology | Breast - metabolism | Breast Neoplasms - metabolism | Neoplasm Metastasis - prevention & control | Resveratrol | Catechin - therapeutic use | Female | Antineoplastic Agents, Phytogenic - therapeutic use | Catechin - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Breast - pathology | Quercetin - chemistry | Quercetin - therapeutic use | Antineoplastic Agents, Phytogenic - chemistry | Mice, SCID | Breast Neoplasms - drug therapy | Animals | Quercetin - pharmacology | Signal Transduction - drug effects | Breast - drug effects | Breast Neoplasms - pathology | Mice, Nude | Neoplasm Metastasis - pathology | Cell Line, Tumor | Cell Proliferation - drug effects | Antineoplastic Agents, Phytogenic - pharmacology | Cell Cycle - drug effects | Catechin - chemistry | Complications and side effects | Care and treatment | Development and progression | Rapamycin | Breast cancer | Metastasis | Research | Health aspects | Risk factors | Energy metabolism | Body weight | AKT protein | Biochemistry | Metastases | Proteins | Angiogenesis | Signal transduction | Cell growth | Polyphenols | Tumorigenesis | Inhibition | Growth factors | AMP | Metabolism | Mammals | Fatty acids | Disease prevention | Ribosomal protein S6 kinase | Energy balance | Flavonoids | Grapes | Protein synthesis | Weight reduction | Quercetin | Mice | Cell migration | Metabolic disorders | TOR protein | Cell proliferation | Surgical implants | Phosphorylation | Homeostasis | Kinases | Cell cycle | G1 phase | Catechin | Adenosine monophosphate | Medicine | Signaling | Protein kinase | Adenosine kinase | Apoptosis | Tumors | Cancer | Index Medicus
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 07/2018, Volume 78, Issue 13 Supplement, pp. 4185 - 4185
Journal Article
Cancer Research, ISSN 0008-5472, 07/2018, Volume 78, Issue 13 Supplement, pp. 1905 - 1905
Journal Article
Nutrition and Cancer, ISSN 0163-5581, 10/2012, Volume 64, Issue 7, pp. 1058 - 1069
Journal Article
Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13 Supplement, pp. 181 - 181
Journal Article
Nutrition and Cancer, ISSN 0163-5581, 10/2012, Volume 64, Issue 7, p. 1058
  We recently reported that a combination of dietary grape polyphenols resveratrol, quercetin, and catechin (RQC), at low concentrations, was effective at... 
Proteins | Rodents | Polyphenols | Breast cancer | Metastasis | Apoptosis | Tumors
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 05/2017, Volume 16, Issue 5, pp. 805 - 818
The Rho GTPases Rac (Ras-related C3 botulinum toxin substrate) and Cdc42 (cell division control protein 42 homolog) regulate cell functions governing cancer... 
P21-ACTIVATED KINASE | EHOP-016 | CELLS | ACTIVATION | ACTIN | ONCOLOGY | RAC | STAT3 | PAK | HUMAN BREAST-CANCER | SMALL-MOLECULE INHIBITOR | Cell Survival - drug effects | Pyrimidines - administration & dosage | Humans | Carbazoles - administration & dosage | Breast Neoplasms - drug therapy | Neovascularization, Pathologic - pathology | Cell Movement - drug effects | Neoplasm Metastasis | Animals | Breast Neoplasms - genetics | Signal Transduction - drug effects | Breast Neoplasms - pathology | Neovascularization, Pathologic - drug therapy | cdc42 GTP-Binding Protein - genetics | cdc42 GTP-Binding Protein - antagonists & inhibitors | rac1 GTP-Binding Protein - antagonists & inhibitors | Cell Line, Tumor | Female | Neovascularization, Pathologic - genetics | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Mice | rac1 GTP-Binding Protein - genetics | Cdc42 protein | Mesenchyme | Epithelial cells | Downstream effects | Homology | AKT protein | Malignancy | Metastasis | Kinases | Cell surface | Metastases | Cell adhesion & migration | Angiogenesis | Actin | Polarity | Mammary gland | Incubation | Stat3 protein | Cell division | p21-activated kinase | MAP kinase | Breast cancer | Botulinum toxin | Substrates | Signaling | Detachment | Inhibitors | Viability | Cell migration | Cancer | Tumors | Apoptosis | anoikis | breast cancer | Rac | Cdc42 inhibitor | Cdc42
Journal Article
Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13 Supplement, pp. 2197 - 2197
Journal Article
Cancer Research, ISSN 0008-5472, 07/2018, Volume 78, Issue 13 Supplement, pp. 4923 - 4923
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2012, Volume 287, Issue 16, pp. 13228 - 13238
Journal Article
Cancer Research, ISSN 0008-5472, 10/2014, Volume 74, Issue 19 Supplement, pp. 4390 - 4390
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 04/2012, Volume 72, Issue 8 Supplement, pp. 2142 - 2142
Journal Article
Molecular Cancer Research, ISSN 1541-7786, 02/2016, Volume 14, Issue 2 Supplement, pp. B29 - B29
Journal Article
Cancer Research, ISSN 0008-5472, 04/2013, Volume 73, Issue 8 Supplement, pp. 4868 - 4868
Journal Article
Translational Oncology, ISSN 1936-5233, 2014, Volume 7, Issue 5, pp. 546 - 555
Abstract Metastatic disease still lacks effective treatments, and remains the primary cause of cancer mortality. Therefore, there is a critical need to develop... 
Oncology | ACTIVATION | INVASION | ONCOLOGY | CYCLIN D1 | CELL-MIGRATION | DOWN-REGULATION | ACTIN CYTOSKELETON | HUMAN BREAST-CANCER | RHO-GTPASES | FACTOR RECEPTOR | SMALL-MOLECULE INHIBITOR
Journal Article
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