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American Journal of Physiology-Lung Cellular and Molecular Physiology, ISSN 1040-0605, 09/2019, Volume 317, Issue 3, pp. L361 - L368
In exercising humans, cardiac output (CO) increases, with minor increases in pulmonary artery pressure (PAP). It is unknown if the CO is accommodated via... 
Hypertension | Exercise physiology | Pulmonary arteries | Lung | Blood pressure | Cardiac output | Capillaries | Surface area | Metabolism | Pulmonary artery | Blood flow | Recruitment | Index Medicus
Journal Article
Scientific Reports, 03/2017, Volume 7, p. 44593
Ticks secrete several anti-hemostatic factors in their saliva to suppress the host innate and acquired immune defenses against infestations. Using Ixodes... 
Polymerase chain reaction | Fibrin | Engorgement | Immunodeficiency | Arthropods | Hsp70 protein | Saliva | Feeding
Journal Article
Cytokine, ISSN 1043-4666, 01/2019, Volume 113, pp. 427 - 432
P53 has been recently involved in the defense against inflammation. The “guardian of the genome” appears to orchestrate cellular responses against bacterial... 
Inflammation | Hsp90 inhibitors | Acute Lung Injury | P53 | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | BARRIER FUNCTION | IMMUNOLOGY | CANCER | HEAT-SHOCK-PROTEIN | CELL BIOLOGY | BINDS | SERINES | NF-KAPPA-B | Acute respiratory distress syndrome | Analysis | Genomics | Myosin | Heat shock proteins | Physiological aspects | Tumor proteins | Muscle proteins | Mediation | Endothelium
Journal Article
Immunobiology, ISSN 0171-2985, 07/2019, Volume 224, Issue 4, pp. 532 - 538
The tumor suppressor protein P53 is strongly involved in orchestrating cellular defenses in the diverse variety of human tissues. Anomalies to lung endothelium... 
Inflammation | Hsp90 inhibitors | Acute Lung Injury | Acute Respiratory Distress Syndrome | HSP90 | CELLS | OXIDATIVE STRESS | PROTEIN | PHOSPHORYLATION | MOUSE | VASCULAR-PERMEABILITY | IMMUNOLOGY | EXPRESSION | PROMOTES | BINDING | Acute respiratory distress syndrome | Medical colleges | Pharmacy | Drugstores | Heat shock proteins | Permeability | Tumor proteins | Endothelium | Index Medicus
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 05/2018, Volume 58, Issue 5, pp. 614 - 624
Journal Article
Inhalation Toxicology, ISSN 0895-8378, 03/2019, Volume 31, Issue 4, pp. 147 - 160
Objective: Accidental exposure to hydrochloric acid (HCl) is associated with acute lung injury in humans, development of long-term chronic airway obstruction,... 
Hydrochloric acid exposure | hsp90 | mouse model | acute lung injury | chronic lung injury | pulmonary fibrosis
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 05/2016, Volume 310, Issue 10, pp. L964 - L974
Journal Article
Chest, ISSN 0012-3692, 11/2017, Volume 152, Issue 5, pp. 954 - 962
Journal Article
American Journal of Physiology, ISSN 0363-6143, 06/2012, Volume 302, Issue 12, p. C1786
O-linked β-N-acetylglucosamine (O-GlcNAc) modification of proteins is involved in many important cellular processes. Increased O-GlcNAc has been implicated in... 
Nitric oxide | Heat shock proteins | Biochemistry | Adenosine triphosphatase | Binding sites
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 03/2018, Volume 22, Issue 3, pp. 1792 - 1804
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 2015, Volume 309, Issue 12, pp. L1410 - L1419
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, pp. e99823 - e99823
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 01/2014, Volume 50, Issue 1, pp. 170 - 179
Journal Article
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 2015, Volume 308, Issue 8, pp. L776 - L787
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2014, Volume 34, Issue 8, pp. 1704 - 1715
OBJECTIVE—Pulmonary hypertension (PH) is a progressive disease arising from remodeling and narrowing of pulmonary arteries (PAs) resulting in high pulmonary... 
Adventitia | Fibroblast | NADPH oxidase | Pulmonary artery | pulmonary artery | ACTIVATION | adventitia | PROLIFERATION | FIBROBLASTS | NAD(P)H OXIDASE | fibroblast | CHRONIC HYPOXIA | MODELS | INFLAMMATION | NOX4 | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | MOLECULAR-MECHANISMS | Up-Regulation | Antihypertensive Agents - pharmacology | Fibroblasts - enzymology | Cell Proliferation | Reactive Oxygen Species - metabolism | Humans | Monocrotaline | NADPH Oxidases - metabolism | Pyrroles | Extracellular Matrix - metabolism | Male | Dose-Response Relationship, Drug | Extracellular Matrix - genetics | Transfection | Time Factors | HEK293 Cells | Adventitia - enzymology | NADPH Oxidases - genetics | Hypertrophy, Right Ventricular - pathology | Hypertension, Pulmonary - drug therapy | Indoles | Hypertension, Pulmonary - enzymology | Disease Models, Animal | Signal Transduction | Mice, Inbred C57BL | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Familial Primary Pulmonary Hypertension | Hypertension, Pulmonary - genetics | Hypoxia - complications | Adventitia - drug effects | NADPH Oxidase 4 | Pulmonary Artery - drug effects | Fibroblasts - pathology | Hypertrophy, Right Ventricular - enzymology | Rats, Sprague-Dawley | Pulmonary Artery - enzymology | Animals | Hypertrophy, Right Ventricular - prevention & control | Adventitia - pathology | Mice | Hypertension, Pulmonary - etiology | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Cell Movement | Index Medicus
Journal Article
SCIENTIFIC REPORTS, ISSN 2045-2322, 03/2017, Volume 7, Issue 1, pp. 44593 - 44593
Ticks secrete several anti-hemostatic factors in their saliva to suppress the host innate and acquired immune defenses against infestations. Using Ixodes... 
BORRELIA-BURGDORFERI | VIRUS TRANSMISSION | PROTEIN | IXODES-SCAPULARIS TICKS