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by Hoshino, Ayuko and Costa-Silva, Bruno and Shen, Tang-Long and Rodrigues, Goncalo and Hashimoto, Ayako and Tesic Mark, Milica and Molina, Henrik and Kohsaka, Shinji and Di Giannatale, Angela and Ceder, Sophia and Singh, Swarnima and Williams, Caitlin and Soplop, Nadine and Uryu, Kunihiro and Pharmer, Lindsay and King, Tari and Bojmar, Linda and Davies, Alexander E and Ararso, Yonathan and Zhang, Tuo and Zhang, Haiying and Hernandez, Jonathan and Weiss, Joshua M and Dumont-Cole, Vanessa D and Kramer, Kimberly and Wexler, Leonard H and Narendran, Aru and Schwartz, Gary K and Healey, John H and Sandstrom, Per and Jørgen Labori, Knut and Kure, Elin H and Grandgenett, Paul M and Hollingsworth, Michael A and De Sousa, Maria and Kaur, Sukhwinder and Jain, Maneesh and Mallya, Kavita and Batra, Surinder K and Jarnagin, William R and Brady, Mary S and Fodstad, Oystein and Muller, Volkmar and Pantel, Klaus and Minn, Andy J and Bissell, Mina J and Garcia, Benjamin A and Kang, Yibin and Rajasekhar, Vinagolu K and Ghajar, Cyrus M and Matei, Irina and Peinado, Hector and Bromberg, Jacqueline and Lyden, David and Lawrence Berkeley National Lab. (LBNL), Berkeley, CA (United States) and Weill Cornell Medicine, New York, NY (United States) and Memorial Sloan Kettering Cancer Center, New York, NY (United States) and Medicinska fakulteten and Region Östergötland and Kirurgiska kliniken US and Linköpings universitet and Institutionen för klinisk och experimentell medicin and Avdelningen för kliniska vetenskaper and Centrum för kirurgi, ortopedi och cancervård
Nature, ISSN 0028-0836, 11/2015, Volume 527, Issue 7578, pp. 329 - 335
Ever since Stephen Paget's 1889 hypothesis, metastatic organotropism has remained one of cancer's greatest mysteries. Here we demonstrate that exosomes from... 
CELLS | BONE METASTASES | VESICLES | BREAST-CANCER METASTASIS | MULTIDISCIPLINARY SCIENCES | IN-VIVO | GROWTH | NICHE | MICROVESICLES | PROTEINS | PROTEOMICS | Exosomes - metabolism | Tropism | Phosphorylation | Epithelial Cells - metabolism | Receptors, Vitronectin - antagonists & inhibitors | Humans | Lung - cytology | Integrin beta4 - metabolism | Integrins - metabolism | Brain - metabolism | Integrin alpha6beta1 - metabolism | S100 Proteins - genetics | Neoplasm Metastasis - prevention & control | Female | Kupffer Cells - metabolism | Lung - metabolism | Receptors, Vitronectin - metabolism | Epithelial Cells - cytology | Integrin alpha6beta4 - metabolism | Fibroblasts - metabolism | Biomarkers - metabolism | Brain - cytology | Kupffer Cells - cytology | Endothelial Cells - metabolism | Liver - metabolism | Mice, Inbred C57BL | Integrin alpha6beta4 - antagonists & inhibitors | Organ Specificity | Animals | Endothelial Cells - cytology | Neoplasm Metastasis - pathology | Cell Line, Tumor | Integrin beta Chains - metabolism | Fibroblasts - cytology | Liver - cytology | Mice | Genes, src | Integrins - antagonists & inhibitors | Complications and side effects | Development and progression | Cell organelles | Metastasis | Health aspects | Integrins | Tumors | Studies | Microscopy | Liver | Melanoma | Fibroblasts | Breast cancer | Chemokines | Index Medicus | mechanisms of disease | cancer microenvironment | BASIC BIOLOGICAL SCIENCES | metastasis | 60 APPLIED LIFE SCIENCES | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 05/2019, Volume 37, Issue 15_suppl, pp. e14712 - e14712
Journal Article
Clinical Genetics, ISSN 0009-9163, 09/2019, Volume 96, Issue 3, pp. 216 - 225
Pathogenic germline TP53 variants predispose to a wide range of early onset cancers, often recognized as the Li‐Fraumeni syndrome (LFS). They are also... 
mutation | germline | hereditary breast cancer | Li‐Fraumeni syndrome | TP53 | p53 activity | ONSET BREAST-CANCER | IMAGING SURVEILLANCE | MUTATION CARRIERS | GENETICS & HEREDITY | MUTANT P53 | germline TP53 mutation | RISK | BRCA2 | BINDING | Li-Fraumeni syndrome | Tumor proteins | Breast cancer | Localization | p53 Protein | Apoptosis | Index Medicus
Journal Article
Autophagy, ISSN 1554-8627, 02/2013, Volume 9, Issue 2, pp. 14 - 13
Multiple myeloma (MM) comprises 1% of all malignancies and 10% of all hematological malignancies. MM is a malignancy of plasma cells in the bone marrow where... 
Proteins | Binding | Landes | Calcium | Biology | Bioscience | Cell | Cycle | Organogenesis | Cancer
Journal Article
Journal of molecular cell biology, ISSN 1674-2788, 02/2019, Volume 11, Issue 4, pp. 330 - 341
The TP53 tumor suppressor gene encodes a DNA-binding transcription factor that regulates multiple cellular processes including cell growth and cell death. The... 
redox regulation | TUMOR | p53 | CELL BIOLOGY | mutation | cancer therapy | HOT-SPOT MUTANTS | Nrf2 | FERROPTOSIS | FERREDOXIN REDUCTASE | thiols | NF-KAPPA-B | EXPRESSION | PROMOTES | BINDING | oxidative stress
Journal Article
Frontiers in oncology, ISSN 2234-943X, 2016, Volume 6, p. 21
TP53 is the most frequently mutated gene in cancer. The p53 protein activates transcription of genes that promote cell cycle arrest or apoptosis, or regulate... 
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 2015, Volume 6, pp. 66 - 66
Tumors must evade the immune system to survive and metastasize, although the mechanisms that lead to tumor immunoediting and their evasion of immune... 
Innate immunity | Exosomes | Tumor surveillance | Tumor progression | secreted vesicles | tumor progression | Tumor Surveillance | innate immunity
Journal Article
Frontiers in Oncology, ISSN 2234-943X, 2016, Volume 6, pp. 21 - 21
TP53 is the most frequently mutated gene in cancer. The p53 protein activates transcription of genes that promote cell cycle arrest or apoptosis, or regulate... 
Mutant p53 | Cancer therapy | Redox balance | Clinical trial | APR-246 | Thioredoxin reductase | Apoptosis | Glutathione | Care and treatment | Gene mutations | Observations | Methods | Cancer | cancer therapy | mutant p53 | Clinical Trial | thioredoxin reductase
Journal Article
Journal Article
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