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Fertility and Sterility, ISSN 0015-0282, 2015, Volume 105, Issue 3, pp. 815 - 824.e5
Journal Article
Hepatology, ISSN 0270-9139, 05/2012, Volume 55, Issue 5, pp. 1485 - 1494
Journal Article
Peptides, ISSN 0196-9781, 2009, Volume 30, Issue 6, pp. 1098 - 1104
...) modulates insulin signaling via effects on c-Jun N-terminal kinase (JNK). The melanocortin agonist NDP-MSH dose-dependently inhibited JNK activity in HEK293 cells stably expressing the human MC4R... 
Insulin receptor substrate 1 (IRS-1) | NDP-MSH | c-Jun N-terminal kinase (JNK) | AKT | MT II | Hypothalamus | Insulin | Melanocortin receptor | PATHWAYS | PROTEIN | RAT | PHOSPHORYLATION | MAP KINASE | JNK | BIOCHEMISTRY & MOLECULAR BIOLOGY | OBESITY | MELANOCYTE-STIMULATING HORMONE | ENDOCRINOLOGY & METABOLISM | RESISTANCE | PHARMACOLOGY & PHARMACY | Cell Line | Insulin - pharmacology | Phosphorylation | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | alpha-MSH - pharmacology | Signal Transduction | Humans | Rats | JNK Mitogen-Activated Protein Kinases - metabolism | Peptides, Cyclic - pharmacology | Receptor, Melanocortin, Type 4 - metabolism | Receptor, Melanocortin, Type 4 - agonists | Insulin Receptor Substrate Proteins - metabolism | Receptor, Melanocortin, Type 4 - biosynthesis | Rats, Sprague-Dawley | Dose-Response Relationship, Drug | Insulin - metabolism | Animals | Hypothalamus - metabolism | Time Factors | Insulin Receptor Substrate Proteins - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - metabolism | alpha-MSH - analogs & derivatives | Proto-Oncogene Proteins c-akt - drug effects | Phosphatidylinositol | Cell research | Glucose | Intermedin | Peptide hormones | Dextrose | insulin | melanocortin receptor | insulin receptor substrate 1 (IRS-1) | hypothalamus
Journal Article
STEM CELLS, ISSN 1066-5099, 11/2016, Volume 34, Issue 11, pp. 2613 - 2624
... from upregulation of c‐JUN oncogene, which was activated by OCT4 in a positive feedback manner. This finding suggests the positive circuit of OCT4/c‐JUN plays a critical role... 
OCT4 | c‐JUN | Liver cancer | Reprogramming | Cancer stem cells | Positive feedback | Pluripotency | c-JUN | CELLS | DEFINED FACTORS | OVARIAN-CANCER | CELL & TISSUE ENGINEERING | CELL BIOLOGY | BREAST-CANCER | HEPATOCELLULAR-CARCINOMA | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | IN-VIVO | EPIGENETIC REGULATION | DIMERIZATION PROTEIN-2 | HEMATOLOGY | EXPRESSION | Neoplastic Stem Cells - drug effects | Humans | Middle Aged | Transcriptional Activation | Gene Expression Regulation, Neoplastic | Spheroids, Cellular - pathology | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Cellular Reprogramming | Octamer Transcription Factor-3 - genetics | Neoplastic Stem Cells - metabolism | Neoplastic Stem Cells - pathology | Female | Liver Neoplasms - pathology | Antineoplastic Agents - pharmacology | Cell Differentiation | Spheroids, Cellular - drug effects | JNK Mitogen-Activated Protein Kinases - genetics | Induced Pluripotent Stem Cells - metabolism | Induced Pluripotent Stem Cells - pathology | Induced Pluripotent Stem Cells - drug effects | Liver Neoplasms - genetics | Signal Transduction | Spheroids, Cellular - metabolism | Liver Neoplasms - drug therapy | Cisplatin - pharmacology | Mice, SCID | Hep G2 Cells | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Feedback, Physiological | Animals | Tumor Burden - drug effects | Octamer Transcription Factor-3 - metabolism | Liver Neoplasms - metabolism | Aged | Fluorouracil - pharmacology | Mice | Doxorubicin - pharmacology | Development and progression | Stem cells | Transcription factors | Liver | Oct-4 protein | c-Myc protein | Chemoresistance | Stem cell transplantation | Transplantation | Feedback loops | Myc protein | Allografts | Cell fate | Feedback | Xenografts | Tumorigenesis | Tumor markers | Colonies | c-Jun protein | Gene expression | Hepatocytes | Tumors | Cancer
Journal Article
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 06/2003, Volume 20, Issue 6, pp. 603 - 612
...), c-Jun, and the low-affinity neurotrophin receptor (P75) following treatment with these neurotrophic factors was also examined... 
Neuronal death | Nitric oxide synthase | Axotomy | Neurotrophic factors | Spinal motoneurons | NITRIC-OXIDE SYNTHASE | LONG-TERM SURVIVAL | AXONAL REGENERATION | axotomy | NEUROSCIENCES | CLINICAL NEUROLOGY | PERIPHERAL-NERVE | RETINAL GANGLION-CELLS | spinal motoneurons | SCIATIC-NERVE | neuronal death | neurotrophic factors | IN-VIVO | nitric oxide synthase | GROWTH-FACTOR | CILIARY NEUROTROPHIC FACTOR | PROGRESSIVE MOTOR NEURONOPATHY | CRITICAL CARE MEDICINE | Proto-Oncogene Proteins c-jun - biosynthesis | Male | Nitric Oxide Synthase - genetics | Motor Neurons - pathology | Nerve Growth Factors - pharmacology | Radiculopathy - pathology | Brain-Derived Neurotrophic Factor - pharmacology | Radiculopathy - drug therapy | Nitric Oxide Synthase Type I | Glial Cell Line-Derived Neurotrophic Factor | Nitric Oxide Synthase - biosynthesis | Radiculopathy - metabolism | Cell Death - drug effects | Brain-Derived Neurotrophic Factor - therapeutic use | Motor Neurons - drug effects | Receptors, Nerve Growth Factor - biosynthesis | Proto-Oncogene Proteins c-jun - genetics | Gene Expression Regulation - physiology | Rats | Rats, Sprague-Dawley | Receptor, Nerve Growth Factor | Receptors, Nerve Growth Factor - genetics | Nerve Growth Factors - therapeutic use | Gene Expression Regulation - drug effects | Motor Neurons - metabolism | Animals | Cell Death - physiology
Journal Article
Cancers, ISSN 2072-6694, 09/2013, Volume 5, Issue 3, pp. 959 - 984
We report here that the Jun dimerization protein 2 (JDP2) plays a critical role as a cofactor for the transcription factors nuclear factor-erythroid 2-related factor 2 (Nrf2... 
Oxidative stress | Jun dimerization protein 2 | Anti oxidation | iPSC-like cells | Medulloblastoma | ROS | Signal transduction | Gene expression | Stem cells | Cancer | anti oxidation | medulloblastoma | oxidative stress
Journal Article
Fertility and Sterility, ISSN 0015-0282, 03/2016, Volume 105, Issue 3, pp. 815 - 824.e5
Objective To test the hypothesis that the c-Jun NH -terminal kinase (JNK) inhibitor (JNKI) bentamapimod (AS602801/PGL5001) can reduce induced endometriosis in baboons... 
endometriotic lesions | endometriosis | baboons | biomarkers | JNK
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, p. e37693
Recently, traditional Chinese medicine and medicinal herbs have attracted more attentions worldwide for its anti-tumor efficacy. Celastrol and Triptolide, two... 
BREAST-CANCER | CROSS-TALK | ANDROGEN RECEPTOR | APOPTOSIS | MOLECULAR-BIOLOGY | DEPENDENT TRANSCRIPTION | PATHWAY | MULTIDISCIPLINARY SCIENCES | GROWTH | KAPPA-B | C-JUN | Diterpenes - pharmacology | Cysteine Endopeptidases | Endopeptidases - metabolism | Prostatic Neoplasms - pathology | Transcription, Genetic - drug effects | Humans | RNA, Messenger - genetics | Male | Down-Regulation - drug effects | RNA, Messenger - metabolism | Disease Progression | Xenograft Model Antitumor Assays | Phenanthrenes - pharmacology | Animals | Endopeptidases - genetics | Epoxy Compounds - pharmacology | Receptors, Androgen - genetics | Cell Line, Tumor | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Mice | Gene Expression Regulation, Neoplastic - drug effects | JNK Mitogen-Activated Protein Kinases - genetics | Medicinal plants | Care and treatment | Sumo | Proteases | Medicine, Botanic | Medicine, Herbal | Prostate cancer | Cancer | Apoptosis | Cell proliferation | Unbalance | Transcription factors | Transcription | Laboratories | Metastasis | Anticancer properties | Proteins | SUMO protein | Cell activation | Chinese medicine | Antitumor agents | Mouse devices | Protease | Triptolide | Cell cycle | Life sciences | Inhibition | Herbs | Enzymes | Lymphatic system | Medicinal herbs | Traditional Chinese medicine | c-Jun protein | Poly(ADP-ribose) polymerase | Pharmacology | Breast cancer | RNA polymerase | Gene expression | Chemical compounds | Herbal medicine | Androgens | Cell death | Natural products | Pancreatic cancer | Lymphomas | Molecular biology | Prostate
Journal Article