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cells (3) 3
chemical and pharmacologic phenomena (3) 3
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expression (3) 3
growth-factor-beta (3) 3
immune system (3) 3
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immunotherapy (3) 3
lymphocytes (3) 3
medicine (3) 3
physiological aspects (3) 3
rna interference (3) 3
rna, small interfering (3) 3
ror-gamma-t (3) 3
suppressor-cells (3) 3
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tgf-beta (3) 3
th17 cells - drug effects (3) 3
transcription factors (3) 3
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[ sdv.can ] life sciences [q-bio]/cancer (2) 2
[ sdv.imm ] life sciences [q-bio]/immunology (2) 2
adenosine (2) 2
adjuvant treatment (2) 2
allergy and immunology (2) 2
animal models (2) 2
anticancer properties (2) 2
antineoplastic agents - therapeutic use (2) 2
antitumor activity (2) 2
antitumor immunity (2) 2
autoimmunity (2) 2
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breast cancer (2) 2
cancer-patients (2) 2
carrier proteins - genetics (2) 2
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Journal of Clinical Investigation, ISSN 0021-9738, 11/2008, Volume 118, Issue 11, pp. 3751 - 3761
Journal Article
X‐Ray Spectrometry, ISSN 0049-8246, 07/2008, Volume 37, Issue 4, pp. 424 - 434
The formation of subsequent layers of calcium carbonate (CaCO3) in karst caves is a well‐known phenomenon observed especially on the prehistoric paintings in... 
X-RAY | MICROPROBE | SPELEOTHEMS | SPECTROSCOPY | MICROSCOPY
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2008, Volume 118, Issue 11, p. 3751
Tumors that progress do so via their ability to escape the antitumor immune response through several mechanisms, including developing ways to induce the... 
Care and treatment | Colon cancer | Immune response | Genetic aspects | Research | T cells | Health aspects
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2008, Volume 118, Issue 11, p. 3751
  Tumors that progress do so via their ability to escape the antitumor immune response through several mechanisms, including developing ways to induce the... 
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2010, Volume 120, Issue 2, pp. 457 - 471
Journal Article
Breast Cancer Research and Treatment, ISSN 0167-6806, 1/2011, Volume 125, Issue 1, pp. 65 - 72
Journal Article
Medecine/Sciences, ISSN 0767-0974, 06/2010, Volume 26, Issue 6-7, pp. 576 - 579
Journal Article
Breast Cancer Research and Treatment, ISSN 0167-6806, 01/2011, Volume 125, Issue 1, p. 65
The Forkhead Box Protein 3 is highly expressed not only in regulatory T cells, but also in tumor cells, acting as a transcriptional repressor of breast... 
Immunohistochemistry | Medical research | Prognosis | Patient outcomes | Analysis | Adjuvant treatment | Medicine, Experimental | Breast cancer | T cells | Cancer
Journal Article
Immunity, ISSN 1074-7613, 03/2012, Volume 36, Issue 3, pp. 362 - 373
Although Th17 cells are known to promote tissue inflammation and autoimmunity, their role during cancer progression remains elusive. Here, we showed that... 
GROWTH-FACTOR-BETA | EFFECTOR T-CELLS | TGF-BETA | T(H)17 CELLS | ROR-GAMMA-T | SIGNALING PATHWAY | IL-17 | ADENOSINE | GENERATION | IMMUNOLOGY | CANCER | 5'-Nucleotidase - genetics | RNA, Small Interfering - genetics | Antigens, CD - genetics | DNA-Binding Proteins - metabolism | Th17 Cells - drug effects | T-Lymphocytes - metabolism | Th17 Cells - metabolism | Lymphocytes, Tumor-Infiltrating - metabolism | Transcription Factors - immunology | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Promoter Regions, Genetic | DNA-Binding Proteins - immunology | Mice, Inbred C57BL | Gene Expression Regulation | Mice, Transgenic | Binding Sites - genetics | Mice, Knockout | Transcription Factors - metabolism | Transforming Growth Factor beta - pharmacology | Animals | Interleukin-6 - pharmacology | Apyrase - genetics | Th17 Cells - immunology | T-Lymphocytes - immunology | Mice | STAT3 Transcription Factor - immunology | STAT3 Transcription Factor - antagonists & inhibitors | Lymphocytes, Tumor-Infiltrating - immunology | Autoimmunity | DNA binding proteins | T cells | Transforming growth factors | Proteins | Transcription factors | Statistical analysis | Plasmids | Rodents | Cytotoxicity | Deoxyribonucleic acid--DNA | Immune system | Cancer | 5'-Nucleotidase | T-Lymphocytes | Interleukin-6 | Life Sciences | Lymphocytes, Tumor-Infiltrating | Immunology | Apyrase | RNA, Small Interfering | STAT3 Transcription Factor | Th17 Cells | Transcription Factors | Binding Sites | Transforming Growth Factor beta | Antigens, CD | DNA-Binding Proteins
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e65181
Bleomycin (BLM) is an anticancer drug currently used for the treatment of testis cancer and Hodgkin lymphoma. This drug triggers cancer cell death via its... 
CALRETICULIN EXPOSURE | LUNG | RESPONSES | MESSENGER-RNA | MULTIDISCIPLINARY SCIENCES | VINBLASTINE | MECHANISMS | SUPPRESSOR-CELLS | EXPRESSION | PULMONARY-FIBROSIS | CUTTING EDGE | Immunity, Innate - drug effects | Humans | T-Lymphocytes, Regulatory - pathology | T-Lymphocytes, Regulatory - immunology | Immunity - drug effects | T-Lymphocytes, Regulatory - drug effects | Animals | CD8-Positive T-Lymphocytes - drug effects | Cell Line, Tumor | Bleomycin - pharmacology | Female | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Mice | Cell Death - drug effects | CD8-Positive T-Lymphocytes - immunology | Transforming Growth Factor beta - metabolism | Care and treatment | Bleomycin | Immune response | Cell death | Biochemistry | Interferon | B cells | Non-Hodgkin's lymphomas | Transforming growth factors | T cells | Cancer | Cell proliferation | Disease | CD8 antigen | Transforming growth factor-b | Cytotoxicity | Lymphocytes T | Chaperones | Cancer therapies | Immunity | Anticancer properties | Calreticulin | Lymphocytes | Rodents | Foxp3 protein | Growth factors | Antigens | Dendritic cells | Immunoregulation | Secretion | Tumor cells | Mortality | Radiation therapy | Lymphoma | Chemotherapy | Pulmonary fibrosis | Immunogenicity | Antitumor activity | Hodgkin's disease | Phagocytosis | Apoptosis | Life Sciences | Immunology
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 02/2013, Volume 133, Issue 2, pp. 499 - 508
Dacarbazine (DTIC) is a cytotoxic drug widely used for melanoma treatment. However, the putative contribution of anticancer immune responses in the efficacy of... 
METASTATIC MELANOMA | IMMUNOGENICITY | IMMUNOTHERAPY | RECEPTOR | ANTITUMOR IMMUNITY | SUPPRESSOR-CELLS | EXPRESSION | CYTOTOXICITY | RADIATION | CHEMOTHERAPY | DERMATOLOGY | Skin Neoplasms - drug therapy | Humans | Melanoma, Experimental - drug therapy | NK Cell Lectin-Like Receptor Subfamily K - immunology | Antineoplastic Agents, Alkylating - pharmacology | Interferon-gamma - metabolism | Melanoma, Experimental - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Dacarbazine - pharmacology | CD8-Positive T-Lymphocytes - metabolism | Killer Cells, Natural - immunology | Disease Models, Animal | Skin Neoplasms - pathology | Skin Neoplasms - immunology | Mice, Inbred C57BL | Melanoma, Experimental - pathology | Up-Regulation - drug effects | Animals | Up-Regulation - immunology | Mice, Nude | CD8-Positive T-Lymphocytes - drug effects | Interferon-gamma - immunology | Cell Line, Tumor | Ligands | Killer Cells, Natural - drug effects | Mice | Killer Cells, Natural - metabolism | CD8-Positive T-Lymphocytes - immunology | Animal models | CD8 antigen | Tumor cells | Melanoma | Cytotoxicity | Lymphocytes T | Immunosuppressive agents | Dacarbazine | NKG2 antigen | Cell activation | Immunosuppression | Major histocompatibility complex | Immunogenicity | Natural killer cells | Cancer | Up-Regulation | Interferon-gamma | NK Cell Lectin-Like Receptor Subfamily K | Killer Cells, Natural | CD8-Positive T-Lymphocytes | Life Sciences | Immunology | Melanoma, Experimental | Skin Neoplasms | Antineoplastic Agents, Alkylating
Journal Article
Nature Medicine, ISSN 1078-8956, 01/2013, Volume 19, Issue 1, pp. 57 - 64
Chemotherapeutic agents are widely used for cancer treatment. In addition to their direct cytotoxic effects, these agents harness the host's immune system,... 
MEDICINE, RESEARCH & EXPERIMENTAL | CANCER-CHEMOTHERAPY | ANGIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GEMCITABINE | BETA | CELL BIOLOGY | 5-FLUOROURACIL | INHIBITION | MICROENVIRONMENT | ANTITUMOR IMMUNITY | BEARING MICE | T-CELLS | Neoplasms - metabolism | Cell Proliferation | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Cathepsin B - genetics | Humans | Caspase 1 - metabolism | Deoxycytidine - pharmacology | Signal Transduction - immunology | Neoplasms - genetics | RNA Interference | Female | Antineoplastic Agents - pharmacology | Interleukin-1beta - biosynthesis | Dendritic Cells - metabolism | bcl-2-Associated X Protein - genetics | Interleukin-17 - secretion | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Cathepsin B - metabolism | Mice, Knockout | Carrier Proteins - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Caspase 1 - genetics | Cell Line, Tumor | Fluorouracil - pharmacology | Mice | Mice, Inbred BALB C | RNA, Small Interfering | Receptors, Interleukin-1 - antagonists & inhibitors | Deoxycytidine - analogs & derivatives | Chemotherapy | Gemcitabine | Cathepsins | Physiological aspects | Dosage and administration | Research | Health aspects | Fluorouracil | Cancer | Cells | Inflammatory diseases | Tumors | Engineering Sciences | Automatic
Journal Article
Cancer Research, ISSN 0008-5472, 06/2013, Volume 73, Issue 12, pp. 3578 - 3590
Journal Article