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1. Full Text The regenerating family member 3 β instigates IL-17A-mediated neutrophil recruitment downstream of NOD1/2 signalling for controlling colonisation resistance independently of microbiota community structure
by Waldschmitt, Nadine and Kitamoto, Sho and Secher, Thomas and Zacharioudaki, Vassiliki and Boulard, Olivier and Floquet, Emilie and Delacre, Myriam and Lamas, Bruno and Pham, Hang-Phuong and Six, Adrien and Richard, Mathias L and Dagorn, Jean-Charles and Eberl, Gérard and Langella, Philippe and Chatel, Jean-Marc and Ryffel, Bernhard and Iovanna, Juan Lucio and Poulin, Lionel F and Sokol, Harry and Kamada, Nobuhiko and Chamaillard, Mathias
Gut, ISSN 0017-5749, 07/2019, Volume 68, Issue 7, pp. 1190 - 1199
ObjectiveLoss of the Crohn’s disease predisposing NOD2 gene results in an intestinal microenvironment conducive for colonisation by attaching-and-effacing... 
Colonic Microflora | Antibacterial Peptide | Interleukins | Macrophages | Barrier Function | CELLS | PATHOGEN | INNATE IMMUNE-RESPONSES | INFLAMMATION | CARD9 | HOST-DEFENSE | NOD2 | INDUCTION | CITROBACTER-RODENTIUM | GASTROENTEROLOGY & HEPATOLOGY | TH17 | Phosphorylation | Transcription factors | rRNA 16S | Mucosa | Virulence | Infections | Kinases | Antibody response | Recruitment | Proteins | Microbiota | Cell activation | E coli | Intestine | Transcription activation | Bacteria | Feces | Nod1 protein | NOD2 protein | Community structure | Pathogens | NF-κB protein | Carbohydrates | Gnotobiotics | Cytokines | Threonine | Nucleotide sequence | Secretion | Neutrophils | Pancreatitis | Inflammatory bowel disease | Lectins | Protein-serine/threonine kinase | Life Sciences | Immunology
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2. Full Text Card9 mediates susceptibility to intestinal pathogens through microbiota modulation and control of bacterial virulence
by Lamas, Bruno and Michel, Marie-Laure and Waldschmitt, Nadine and Pham, Hang-Phuong and Zacharioudaki, Vassiliki and Dupraz, Louise and Delacre, Myriam and Natividad, Jane M and Costa, Gregory Da and Planchais, Julien and Sovran, Bruno and Bridonneau, Chantal and Six, Adrien and Langella, Philippe and Richard, Mathias L and Chamaillard, Mathias and Sokol, Harry
Gut, ISSN 0017-5749, 10/2018, Volume 67, Issue 10, pp. 1836 - 1844
ObjectiveIn association with innate and adaptive immunity, the microbiota controls the colonisation resistance against intestinal pathogens. Caspase... 
germfree mice | microbiota | citrobacter rodentium | IBD | mono- and polysaccharides | pathogen-specific antibody | CELLS | IMMUNITY | ESCHERICHIA-COLI | GUT MICROBIOTA | IL-22 | RESPONSES | INFLAMMATION | HOST-DEFENSE | INFECTION | CITROBACTER-RODENTIUM | GASTROENTEROLOGY & HEPATOLOGY | Competition | rRNA 16S | Virulence | Homeostasis | Hydrocarbons | Innate immunity | Infections | Genomes | Antibody response | Monosaccharides | Microbiota | Polysaccharides | E coli | Intestine | Rodents | Bacteria | Food | Immune system | Pathogens | Immunoglobulins | Gnotobiotics | Bacterial infections | Cytokines | Lipocalin | Caspase | Inflammation | Adaptive immunity | Gene expression | Immune response (humoral) | Border patrol | Inflammatory bowel disease | Diet | Colitis | Index Medicus | Abridged Index Medicus | Life Sciences | Human health and pathology | Hépatology and Gastroenterology | Infectious diseases | Immunology
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3. Full Text MAVS deficiency induces gut dysbiotic microbiota conferring a proallergic phenotype
by Plantamura, E and Dzutsev, A and Chamaillard, M and Djebali, S and Moudombi, L and Boucinha, L and Grau, M and Macari, C and Bauche, D and Dumitrescu, O and Rasigade, JP and Lippens, S and Plateroti, M and Kress, E and Cesaro, A and Bondu, C and Rothermel, U and Heikenwalder, M and Lina, G and Bentaher-Belaaouaj, A and Marie, JC and Caux, C and Trinchieri, G and Marvel, J and Michallet, MC
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, ISSN 0027-8424, 10/2018, Volume 115, Issue 41, pp. 10404 - 10409
Prominent changes in the gut microbiota (referred to as "dysbiosis") play a key role in the development of allergic disorders, but the underlying mechanisms... 
allergic skin pathologies | IMMUNITY | PROTEIN | COMMENSAL BACTERIA | MULTIDISCIPLINARY SCIENCES | ATOPIC ECZEMA | CONTACT HYPERSENSITIVITY | CALIBRATE | RIG-like receptors | RESPONSES | MAVS | COLITIS | MICE | dysbiosis | INTESTINAL PERMEABILITY | Intestines - pathology | Signal Transduction | Homeodomain Proteins - metabolism | Mice, Inbred C57BL | Skin Diseases, Bacterial - metabolism | Dysbiosis - complications | Homeodomain Proteins - genetics | Intestines - immunology | Mice, Knockout | Skin Diseases, Bacterial - etiology | Adaptor Proteins, Signal Transducing - physiology | Phenotype | Animals | Intestines - microbiology | Hypersensitivity - metabolism | Hypersensitivity - etiology | Hypersensitivity - pathology | Female | Mice | Gastrointestinal Microbiome - immunology | Skin Diseases, Bacterial - pathology | Disease Models, Animal | Physiological aspects | Mitochondrial diseases | Genetic aspects | Microbiota (Symbiotic organisms) | Health aspects | Translocation | Phenotypes | Dysbacteriosis | Hypersensitivity (delayed) | Digestive system | Hypersensitivity | Organs | Transplantation | Permeability | Dermatitis | Genotype & phenotype | Pathology | Signaling | Microbiota | Mitochondria | Contact dermatitis | Intestine | Skin diseases | Digestive tract | Index Medicus | Biological Sciences
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4. Full Text Nod-like receptor pyrin domain-containing protein 6 (NLRP6) controls epithelial self-renewal and colorectal carcinogenesis upon injury
by Sylvain Normand and Anne Delanoye-Crespin and Aude Bressenot and Ludovic Huot and Teddy Grandjean and Laurent Peyrin-Biroulet and Yves Lemoine and David Hot and Mathias Chamaillard
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2011, Volume 108, Issue 23, pp. 9601 - 9606
The colonic epithelium self-renews every 3 to 5 d, but our understanding of the underlying processes preserving wound healing from carcinogenesis remains... 
Inflammatory bowel diseases | Epithelial cells | Genes | Colorectal cancer | Mice | Inflammation | Colitis | Myofibroblasts | Carcinogenesis | Cancer | Colonic myofibroblasts | ACTIVATION | colorectal cancer | CASPASE-1 | MULTIDISCIPLINARY SCIENCES | ULCERATIVE-COLITIS | RISK | INTESTINAL TUMORIGENESIS | PREVALENCE | BETA | COLON-CANCER | colonic myofibroblasts | INFLAMMATION | MOUSE MODEL | Immunohistochemistry | Intestinal Mucosa - metabolism | Cell Proliferation | Epithelial Cells - metabolism | Oligonucleotide Array Sequence Analysis | Colitis - genetics | Colorectal Neoplasms - genetics | Colitis - pathology | Male | Gene Expression Profiling | Mice, 129 Strain | Cell Movement - genetics | Myofibroblasts - metabolism | Female | Wound Healing - genetics | Colorectal Neoplasms - metabolism | Mice, Inbred C57BL | Receptors, Cell Surface - metabolism | Epithelial Cells - pathology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Animals | Colitis - metabolism | Intestinal Mucosa - injuries | Colorectal Neoplasms - pathology | Receptors, Cell Surface - genetics | Inflammatory bowel disease | Proteins | Rodents | Stem cells | Colon | Gene expression | Index Medicus | Biological Sciences
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5. Full Text Integrin a6 loss promotes colitis-associated colorectal cancer. Response to: “Integrin a6 variants and colorectal cancer” by Beaulieu JF
by De Arcangelis, Adèle and Chamaillard, Mathias and Simon-Assmann, Patricia and Labouesse, Michel
Gut, ISSN 0017-5749, 12/2018, Volume 67, Issue 12, pp. 2227 - 2228
[...]since α6ΔIEC mutant mice develop adenocarcinoma without any additional treatment, Itga6 behaves as a tumour suppressor gene (a gene that protects a cell... 
Inflammatory bowel disease | Adenocarcinoma | Keratin | Microbiota | Carcinoma | Cytokines | Colorectal carcinoma | Colorectal cancer | Extracellular matrix | Inflammation | Colitis | Tumors
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6. Full Text NOD2-mediated dysbiosis predisposes mice to transmissible colitis and colorectal cancer
by Couturier-Maillard, Aurélie and Secher, Thomas and Rehman, Ateequr and Normand, Sylvain and De Arcangelis, Adèle and Haesler, Robert and Huot, Ludovic and Grandjean, Teddy and Bressenot, Aude and Delanoye-Crespin, Anne and Gaillot, Olivier and Schreiber, Stefan and Lemoine, Yves and Ryffel, Bernhard and Hot, David and Nùñez, Gabriel and Chen, Grace and Rosenstiel, Philip and Chamaillard, Mathias
Journal of Clinical Investigation, ISSN 0021-9738, 02/2013, Volume 123, Issue 2, pp. 700 - 711
Instability in the composition of gut bacterial communities (dysbiosis) has been linked to common human intestinal disorders, such as Crohn's disease and... 
MEDICINE, RESEARCH & EXPERIMENTAL | CARCINOGENESIS | CROHNS-DISEASE | FECAL MICROBIOTA | MOUSE MODEL | SUSCEPTIBILITY | RECEPTOR | NOD2 | INFLAMMATORY BOWEL DISEASES | INTESTINAL EPITHELIAL-CELLS | INNATE IMMUNITY | Nod2 Signaling Adaptor Protein - deficiency | Humans | Colitis - pathology | Male | Nod2 Signaling Adaptor Protein - genetics | Colorectal Neoplasms - etiology | Female | Colorectal Neoplasms - metabolism | Disease Models, Animal | Digestive System - metabolism | Risk Factors | Colorectal Neoplasms - microbiology | Mice, Knockout | Digestive System - pathology | Pregnancy | Colitis - etiology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Colitis - microbiology | Colitis - metabolism | Mice | Receptor-Interacting Protein Serine-Threonine Kinases - deficiency | Colorectal Neoplasms - pathology | Metagenome | Digestive System - microbiology | Care and treatment | Microbiota (Symbiotic organisms) | Colorectal cancer | Physiological aspects | Genetic aspects | Research | Colitis | Risk factors | Index Medicus | Abridged Index Medicus | Receptor-Interacting Protein Serine-Threonine Kinases | Nod2 Signaling Adaptor Protein | Life Sciences | Immunology | Digestive System | Colorectal Neoplasms
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7. Full Text Nod2-Dependent Regulation of Innate and Adaptive Immunity in the Intestinal Tract
by Koichi S. Kobayashi and Mathias Chamaillard and Yasunori Ogura and Octavian Henegariu and Naohiro Inohara and Gabriel Nuñez and Richard A. Flavell
Science, ISSN 0036-8075, 2/2005, Volume 307, Issue 5710, pp. 731 - 734
The gene encoding the Nod2 protein is frequently mutated in Crohn's disease (CD) patients, although the physiological function of Nod2 in the intestine remains... 
Spleen | Bacterial infections | Liver | Crypts | Ligands | Bacteria | Reports | Infections | Health savings accounts | Macrophages | Genetic mutation | ALPHA-DEFENSIN | NOD2 GENE | ACTIVATION | INFLAMMATORY-BOWEL-DISEASE | CROHNS-DISEASE | MULTIDISCIPLINARY SCIENCES | MICE | PANETH CELLS | MUTATIONS | AVIUM SUBSPECIES PARATUBERCULOSIS | NF-KAPPA-B | Oligonucleotide Array Sequence Analysis | Immunity, Mucosal | Liver - microbiology | Male | Intestinal Diseases - immunology | Listeria monocytogenes - immunology | Ileum - immunology | Spleen - microbiology | alpha-Defensins - biosynthesis | alpha-Defensins - genetics | Toll-Like Receptors | Membrane Glycoproteins - physiology | Serum Albumin - immunology | Intestinal Mucosa - immunology | Listeriosis - immunology | Female | Receptors, Cell Surface - physiology | Interleukins - biosynthesis | Immunoglobulins - biosynthesis | Macrophages - immunology | Gene Targeting | Gene Expression | Lipopolysaccharides - toxicity | Protein Precursors - genetics | Signal Transduction | Listeria monocytogenes - isolation & purification | Listeria monocytogenes - growth & development | Nod2 Signaling Adaptor Protein | Intestinal Mucosa - microbiology | Immunity, Innate | Animals | Protein Precursors - biosynthesis | Intestinal Diseases - microbiology | Acetylmuramyl-Alanyl-Isoglutamine - immunology | Antibody Formation | Listeriosis - microbiology | Mice | Intracellular Signaling Peptides and Proteins - physiology | Ileum - microbiology | Tumor Necrosis Factor-alpha - biosynthesis | Proteins | Care and treatment | Crohn's disease | Research | Genetic variation | Analysis | Mutation | Digestive system | Genes | Bowel disease | Medical immunity | Index Medicus
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8. Full Text Defining dysbiosis threatens Koch's postulates and current dogma on the role of Paneth cells in Crohn's disease
by Chamaillard, Mathias and Radulovic, Katarina
Gut, ISSN 0017-5749, 02/2016, Volume 65, Issue 2, pp. 190 - 191
  [...]genome-wide association studies have revealed a large set of inborn genetic errors in several regulatory factors involved in the maintenance of Paneth... 
GASTROENTEROLOGY & HEPATOLOGY | Animals | Intestines - microbiology | Dysbiosis - complications | Crohn Disease - etiology | Ileitis - etiology | Cell proliferation | Anti-infective agents | Care and treatment | Dosage and administration | Research | Dysbiosis | Studies | Researchers | Metabolism | Small intestine | Experiments | Crohns disease | Index Medicus | Abridged Index Medicus
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9. Full Text Time for epithelial sensing of vitamin D to step into the limelight
by Waldschmitt, Nadine and Chamaillard, Mathias
Gut, ISSN 0017-5749, 12/2014, Volume 64, Issue 7, pp. 1013 - 1014
  3 4 Remarkably, reciprocal transplantation of faecal microbiota from Nod2-deficient mice enhanced the vulnerability of control animals to colonic injury. 3... 
CIRCADIAN CLOCK | HOMEOSTASIS | INFLAMMATION | FECAL MICROBIOTA | LINKS | AUTOPHAGY | D-3 | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | GENE ATG16L1 | Intestinal Mucosa - metabolism | Animals | Colitis - physiopathology | Humans | Receptors, Calcitriol - physiology | Female | Autophagy - physiology | Researchers | Vitamin D | Pathogenesis | Genes | Rodents | Autophagy | Crohns disease | Index Medicus | Abridged Index Medicus
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10. Full Text Enterococcus hirae and Barnesiella intestinihominis Facilitate Cyclophosphamide-Induced Therapeutic Immunomodulatory Effects
by Daillère, Romain and Vétizou, Marie and Waldschmitt, Nadine and Yamazaki, Takahiro and Isnard, Christophe and Poirier-Colame, Vichnou and Duong, Connie P.M and Flament, Caroline and Lepage, Patricia and Roberti, Maria Paula and Routy, Bertrand and Jacquelot, Nicolas and Apetoh, Lionel and Becharef, Sonia and Rusakiewicz, Sylvie and Langella, Philippe and Sokol, Harry and Kroemer, Guido and Enot, David and Roux, Antoine and Eggermont, Alexander and Tartour, Eric and Johannes, Ludger and Woerther, Paul-Louis and Chachaty, Elisabeth and Soria, Jean-Charles and Golden, Encouse and Formenti, Silvia and Plebanski, Magdalena and Madondo, Mutsa and Rosenstiel, Philip and Raoult, Didier and Cattoir, Vincent and Boneca, Ivo Gomperts and Chamaillard, Mathias and Zitvogel, Laurence
Immunity, ISSN 1074-7613, 10/2016, Volume 45, Issue 4, pp. 931 - 943
The efficacy of the anti-cancer immunomodulatory agent cyclophosphamide (CTX) relies on intestinal bacteria. How and which relevant bacterial species are... 
TRANSLOCATION | BACTERIA | DENDRITIC CELLS | RICHNESS | INTESTINAL MICROBIOTA | INFLAMMATION | INFECTION | ANTITUMOR IMMUNITY | GUT MICROBIOTA | IMMUNOLOGY | CANCER | Immunotherapy - methods | Intestine, Small - microbiology | Mice, Inbred C57BL | Monitoring, Immunologic | Immunologic Factors - immunology | Nod2 Signaling Adaptor Protein - immunology | Colon - immunology | Th1 Cells - immunology | Enterococcus hirae - immunology | Neoplasms - drug therapy | Animals | Neoplasms - immunology | Cyclophosphamide - pharmacology | Interferon-gamma - immunology | Colon - microbiology | Mice | Immunologic Memory - immunology | Intestine, Small - immunology | Cyclophosphamide | Microbiota (Symbiotic organisms) | T cells | Health aspects | Ovarian cancer | Antigens | Medical research | Antibiotics | Lymphocytes | Cloning | Bacteria | Experiments | Cancer | Index Medicus | Life Sciences | Immunology
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11. Full Text Lactobacillus acidophilus modulates intestinal pain and induces opioid and cannabinoid receptors
by Barnich, Nicolas and Rousseaux, Christel and Thuru, Xavier and Geboes, Karen and Chamaillard, Mathias and Leyer, Greg and Merour, Emilie and Gelot, Agathe and Ardid, Denis and Neut, Christel and Dubuquoy, Caroline and Carcano, Didier and Dubuquoy, Laurent and Colombel, Jean-Frédéric and Desreumaux, Pierre and Ouwehand, Arthur
Nature Medicine, ISSN 1078-8956, 01/2007, Volume 13, Issue 1, pp. 35 - 37
Abdominal pain is common in the general population and, in patients with irritable bowel syndrome, is attributed to visceral hypersensitivity. We found that... 
MEDICINE, RESEARCH & EXPERIMENTAL | VISCERAL PAIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | NCFM | RATS | COLON INFLAMMATION | EXPRESSION | IRRITABLE-BOWEL-SYNDROME | HYPERSENSITIVITY | CELL BIOLOGY | Intestines - drug effects | Probiotics - administration & dosage | Abdominal Pain - prevention & control | Narcotic Antagonists - administration & dosage | Receptor, Cannabinoid, CB2 - physiology | Cannabinoid Receptor Antagonists | Humans | Colon - drug effects | Receptors, Cannabinoid - physiology | Analgesics, Opioid - pharmacology | Male | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Receptor, Cannabinoid, CB2 - biosynthesis | Indoles - administration & dosage | Receptors, Opioid, mu - biosynthesis | Dose-Response Relationship, Drug | Receptors, Opioid - physiology | Rectum - drug effects | Colon - physiopathology | Indoles - pharmacology | Receptors, Cannabinoid - biosynthesis | Rectum - microbiology | Morphine - pharmacology | Administration, Oral | Lactobacillus acidophilus - physiology | Abdominal Pain - physiopathology | Rats | Probiotics - pharmacology | Receptors, Opioid, mu - antagonists & inhibitors | Rats, Sprague-Dawley | HT29 Cells | Animals | Intestines - microbiology | Rectum - physiopathology | Analgesics, Opioid - administration & dosage | Naloxone - pharmacology | Narcotic Antagonists - pharmacology | Receptors, Opioid - biosynthesis | Colon - microbiology | Mice | Mice, Inbred BALB C | Intestines - physiopathology | Naloxone - administration & dosage | Morphine - administration & dosage | Receptors, Opioid, mu - physiology | Bacteria | Pain | Microbiology | Gastrointestinal diseases | Irritable bowel syndrome | Index Medicus
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12. Full Text Meta-analysis identifies 29 additional ulcerative colitis risk loci, increasing the number of confirmed associations to 47
by Anderson, Carl A and Boucher, Gabrielle and Lees, Charlie W and Franke, Ane and D'Amato, Mauro and Taylor, Kent D and Lee, James C and Goyette, Philippe and Imielinski, Marcin and Latiano, Anna and Lagacé, Caroline and Scott, Regan and Amininejad, Leila and Bumpstead, Suzannah and Baidoo, Leonard and Baldassano, Robert N and Barclay, Murray and Bayless, Theodore M and Brand, Stephan and Büning, Carsten and Colombel, Jean-Frédéric and Denson, Lee A and de Vos, Martine and Dubinsky, Marla and Edwards, Cathryn and Ellinghaus, David and Fehrmann, Rudolf S. N and Floyd, James A. B and Florin, Timothy and Franchimont, Denis and Franke, Lude and Georges, Michel and Glas, Jürgen and Glazer, Nicole L and Guthery, Stephen L and Haritunians, Talin and Hayward, Nicholas K and Hugot, Jean-Pierre and Jobin, Gilles and Laukens, Debby and Lawrance, Ian and Lémann, Marc and Levine, Arie and Libioulle, Cecile and Louis, Edouard and McGovern, Dermot P and Milla, Monica and Montgomery, Grant W and Morley, Katherine I and Mowat, Craig and Ng, Aylwin and Newman, William and Ophoff, Roel A and Papi, Laura and Palmieri, Orazio and Peyrin-Biroulet, Laurent and Panés, Julián and Phillips, Anne and Prescott, Natalie J and Proctor, Deborah D and Roberts, Rebecca and Russell, Richard and Rutgeerts, Paul and Sanderson, Jeremy and Sans, Miquel and Schumm, Philip and Seibold, Frank and Sharma, Yashoda and Simms, Lisa A and Seielstad, Mark and Steinhart, A. Hillary and Targan, Stephan R and van den Berg, Leonard H and Vatn, Morten and Verspaget, Hein and Walters, Thomas and Wijmenga, Cisca and Wilson, David C and Westra, Harm-Jan and Xavier, Ramnik J and Zhao, Zhen Z and Ponsioen, Cyriel Y and Andersen, Vibeke and Torkvist, Leif and Gazouli, Maria and Anagnou, Nicholas P and Karlsen, Tom H and Kupcinskas, Limas and Sventoraityte, Jurgita and Mansfield, John C and Kugathasan, Subra and Silverberg, Mark S and Halfvarson, Jonas and Rotter, Jerome I and Mathew, Christopher G and Griffiths, Anne M and Gearry, Richard and Ahmad, Tariq and Brant, Steven R and Chamaillard, Mathias and ... and Hälsoakademin and Örebro universitet
Nature genetics, ISSN 1061-4036, 2011, Volume 43, Issue 3, pp. 246 - 252
Genome-wide association studies and candidate gene studies in ulcerative colitis have identified 18 susceptibility loci. We conducted a meta-analysis of six... 
COMMON VARIANTS | CELLS | INFLAMMATORY-BOWEL-DISEASE | PROTEIN | CROHNS-DISEASE | GENETICS & HEREDITY | SUSCEPTIBILITY LOCI | GENE-EXPRESSION | ENRICHMENT | MULTIPLE | GENOME-WIDE ASSOCIATION | Crohn Disease - genetics | Risk | Genome-Wide Association Study | Humans | Colitis, Ulcerative - genetics | Genetic aspects | Disease susceptibility | Research | Risk factors | Ulcerative colitis | Meta-analysis | Studies | Inflammatory bowel disease | Celiac disease | Genetics | Genomics | Genes | Index Medicus | Medicine | Medicin | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
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