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Journal of Cell Biology, ISSN 0021-9525, 07/2011, Volume 194, Issue 2, pp. 209 - 227
Journal Article
Journal Article
Biochimica et Biophysica Acta - Molecular Basis of Disease, ISSN 0925-4439, 2014, Volume 1842, Issue 9, pp. 1668 - 1680
Huntington's disease (HD) is an autosomal dominant neurological disorder that is induced by a CAG trinucleotide expansion in exon 1 of the Huntingtin (HTT)... 
N-acetyl-l-cysteine (NAC) | Huntington's disease (HD) | AMP-activated protein kinase (AMPK) | Reactive oxygen species (ROS) | Index Medicus
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 09/2014, Volume 1842, Issue 9, p. 1668
Journal Article
NATURE COMMUNICATIONS, ISSN 2041-1723, 08/2019, Volume 10, Issue 1, p. 3473
Huntington's disease (HD) is a neurodegenerative disorder that manifests with movement dysfunction. The expression of mutant Huntingtin (mHTT) disrupts the... 
BIOMARKERS | ACTIVATION | GENE | MULTIDISCIPLINARY SCIENCES | MUTANT HUNTINGTIN | MOUSE MODEL | CELL-CYCLE | NLRP3 INFLAMMASOME | NF-KAPPA-B | EXPRESSION | CONTRIBUTES
Journal Article
Nature Communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 1 - 18
Huntington’s disease (HD) is a neurodegenerative disorder that manifests with movement dysfunction. The expression of mutant Huntingtin (mHTT) disrupts the... 
Brain | Level (quantity) | Cell survival | Huntingtin | Disease | Neurodegenerative diseases | Pathogenesis | Lysosomes | Inflammation | Galectin-3 | Huntington's disease | Microglia | Mice
Journal Article
MOLECULAR NEUROBIOLOGY, ISSN 0893-7648, 12/2018, Volume 55, Issue 12, pp. 8936 - 8952
Alzheimer's disease (AD) is a neurodegenerative disorder characterized by cognitive impairment and synaptic dysfunction. Adenosine is an important homeostatic... 
Glutamate receptors | Adenosine | PHOSPHORYLATION | ENT1 | TRANSGENIC MOUSE | PKA | DEPENDENT PROTEIN-KINASE | COGNITIVE IMPAIRMENT | AMYOTROPHIC-LATERAL-SCLEROSIS | SYNAPTIC PLASTICITY | NEUROSCIENCES | BETA-AMYLOID DEPOSITION | NUCLEOSIDE TRANSPORTER | A(2A) RECEPTOR | Alzheimer's disease | BRAIN | Memory Disorders - physiopathology | Cerebral Cortex - pathology | Neuronal Plasticity - drug effects | Cerebral Cortex - metabolism | Cognitive Dysfunction - prevention & control | Alzheimer Disease - pathology | Cerebral Cortex - physiopathology | Adenosine - therapeutic use | Amyloid beta-Protein Precursor - metabolism | Synaptic Transmission - drug effects | Disease Models, Animal | Cognitive Dysfunction - drug therapy | Cyclic AMP-Dependent Protein Kinases - metabolism | Alzheimer Disease - physiopathology | Plaque, Amyloid - pathology | Mice, Transgenic | Equilibrative Nucleoside Transporter 1 - metabolism | Hippocampus - pathology | Adenosine - pharmacology | Receptor, Adenosine A2A - metabolism | Cognitive Dysfunction - physiopathology | Glycogen Synthase Kinase 3 beta - metabolism | Hippocampus - metabolism | Animals | Memory Disorders - drug therapy | Plaque, Amyloid - physiopathology | Presenilin-1 - metabolism | Equilibrative Nucleoside Transporter 1 - antagonists & inhibitors | Hippocampus - physiopathology | Astrocytes - metabolism | Proteins | Epigenetic inheritance | Neurosciences | Neurons | Analysis | Glutamate
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2016, Volume 96, pp. 47 - 53
Journal Article
Movement Disorders, ISSN 0885-3185, 11/2017, Volume 32, Issue 11, pp. 1600 - 1609
ABSTRACT Background: Disruptions in gamma‐aminobutyric (GABA) acid signaling are believed to be involved in Huntington's disease pathogenesis, but the... 
Huntington's disease | potassium‐chloride cotransporter‐2 | GABAergic signaling | GABAA receptors | potassium-chloride cotransporter-2 | GABA | receptors | IN-VIVO MICRODIALYSIS | EXTRASYNAPTIC GABA(A) RECEPTORS | GLOBUS-PALLIDUS | LOCOMOTOR-ACTIVITY | BENZODIAZEPINE-RECEPTOR | CLINICAL NEUROLOGY | GABA(A) receptors | K+-CL-COTRANSPORTER | POSTMORTEM BRAIN-TISSUE | TONIC INHIBITION | CREATINE-KINASE | TRANSGENIC MICE | Male | Mice, Transgenic | Corpus Striatum - metabolism | Huntington Disease - metabolism | Cerebral Cortex - metabolism | Diazepam - pharmacology | Isoxazoles - pharmacology | Animals | GABA Agonists - pharmacology | GABA Modulators - pharmacology | Receptors, GABA-A - drug effects | Corpus Striatum - drug effects | Behavior, Animal - drug effects | Female | Mice | Receptors, GABA-A - metabolism | Cerebral Cortex - drug effects | Disease Models, Animal | Brain | Medical research | Neurons | Creatine kinase | Analysis | Medicine, Experimental | Genetic engineering | Creatine | Huntingtons disease | Animal models | γ-Aminobutyric acid A receptors | Immunocytochemistry | Transgenic mice | Homeostasis | Potassium-chloride cotransporter | Polymerase chain reaction | γ-Aminobutyric acid | Chloride | Molecular modelling | Chlorides | Rodents | Neostriatum | Receptor mechanisms | Potassium | Locomotor activity | Diazepam
Journal Article
Journal Article