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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2009, Volume 106, Issue 23, pp. 9373 - 9378
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2013, Volume 110, Issue 42, pp. 16975 - 16980
The activation of STAT3 by tyrosine phosphorylation, essential for normal development and for a normal inflammatory response to invading pathogens, is kept in... 
Hep G2 cells | Phosphorylation | Receptors | Cytokines | Secretion | Antibodies | Washing | Gene expression regulation | Endothelial cells | Cancer | MULTIDISCIPLINARY SCIENCES | EPIDERMAL-GROWTH-FACTOR | IN-VIVO | GENE-EXPRESSION | RESISTANCE | BLASTOCYST IMPLANTATION | LEUKEMIA INHIBITORY FACTOR | IDENTIFICATION | CANCER | T-CELLS | SIGNAL TRANSDUCER | Neoplasms - metabolism | Phosphorylation - physiology | Inflammation - pathology | Receptor, Epidermal Growth Factor - genetics | Cytokine Receptor gp130 - genetics | Humans | Oncostatin M - pharmacology | Antineoplastic Agents - metabolism | Janus Kinase 1 - metabolism | Inflammation - metabolism | Receptor, Epidermal Growth Factor - metabolism | Neoplasms - genetics | Janus Kinase 1 - genetics | Janus Kinase 2 - metabolism | Oncostatin M - metabolism | Antineoplastic Agents - pharmacology | Phosphorylation - drug effects | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Cytokine Receptor gp130 - metabolism | Interleukin-6 - genetics | Janus Kinase 2 - genetics | Gene Expression Regulation - physiology | Suppressor of Cytokine Signaling Proteins - genetics | Hep G2 Cells | Gene Expression Regulation - drug effects | Suppressor of Cytokine Signaling 3 Protein | Interleukin-6 - pharmacology | Inflammation - genetics | Interleukin-6 - biosynthesis | Suppressor of Cytokine Signaling Proteins - metabolism | Neoplasms - pathology | Protein Binding - physiology | Physiological aspects | Epidermal growth factor | Research | Interleukins | Health aspects | Binding sites (Biochemistry) | Biological Sciences
Journal Article
Journal Article
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 10/2014, Volume 74, Issue 19 Supplement, pp. 485 - 485
Journal Article
The EMBO Journal, ISSN 0261-4189, 10/2013, Volume 32, Issue 20, pp. 2751 - 2763
A single high dose of interferon‐β (IFNβ) activates powerful cellular responses, in which many anti‐viral, pro‐apoptotic, and anti‐proliferative proteins are... 
DNA damage resistance | interferon‐β | unphosphorylated interferon‐stimulated gene factor 3 (U‐ISGF3) | anti‐viral genes | interferon-β | anti-viral genes | unphosphorylated interferon-stimulated gene factor 3 (U-ISGF3) | PROTEIN | CANCER CELLS | IKK-EPSILON | BIOCHEMISTRY & MOLECULAR BIOLOGY | IDENTIFICATION | RADIATION | CELL BIOLOGY | BREAST-CANCER | interferon-beta | ALPHA/BETA-INTERFERON-PRODUCTION | GENE-EXPRESSION | ANTIVIRAL ACTIVITY | UP-REGULATION | Tumor Escape - drug effects | Viruses - pathogenicity | Tumor Escape - genetics | Humans | Cercopithecus aethiops | STAT1 Transcription Factor - metabolism | DNA Damage - genetics | Virus Diseases - genetics | Disease Resistance - genetics | Vero Cells | DNA Damage - drug effects | Antiviral Agents - pharmacology | Virus Diseases - immunology | Cells, Cultured | Disease Resistance - drug effects | Gene Expression Regulation - physiology | Viruses - drug effects | Viruses - immunology | Interferon-Stimulated Gene Factor 3, gamma Subunit - genetics | STAT1 Transcription Factor - genetics | Interferon-Stimulated Gene Factor 3, gamma Subunit - metabolism | Gene Expression Regulation - drug effects | Animals | Interferon-beta - pharmacology | STAT2 Transcription Factor - genetics | STAT2 Transcription Factor - metabolism | Virus Diseases - prevention & control
Journal Article
Cold Spring Harbor Perspectives in Biology, 01/2018, Volume 10, Issue 1
Many cytokines and all interferons activate members of a small family of kinases (the Janus kinases [JAKs]) and a slightly larger family of transcription... 
Transducers | Transcription factors | Immune response | Cytokines | Kinases | Gene expression | Proteins | Pathways | Genetics | Janus kinase | Interferon | Mutation | Regulatory proteins | Cancer
Journal Article
Cell Reports, ISSN 2211-1247, 07/2015, Volume 12, Issue 2, pp. 300 - 312
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 06/2009, Volume 106, Issue 23, p. 9373
  In normal human cells treated with interferons (IFNs), the concentration of tyrosine-phosphorylated STAT1 (YP-STAT1), which drives the expression of a large... 
Genes | Genetics | Interferon | Biochemistry | Gene expression | Immune system
Journal Article
International Journal of Cancer, ISSN 0020-7136, 02/2015, Volume 136, Issue 4, pp. E51 - E61
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2017, Volume 114, Issue 52, p. 13792
Journal Article
Journal Article
Cytokine, ISSN 1043-4666, 09/2013, Volume 63, Issue 3, pp. 253 - 254
Journal Article
Cytokine, ISSN 1043-4666, 09/2013, Volume 63, Issue 3, pp. 253 - 254
A single high dose of IFN[beta] activates powerful cellular responses in which many antiviral, pro-apoptotic, and anti-proliferative proteins are highly... 
Antiviral agents
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2013, Volume 110, Issue 42, p. 16975
  The activation of STAT3 by tyrosine phosphorylation, essential for normal development and for a normal inflammatory response to invading pathogens, is kept... 
Pathogens | Phosphorylation | Cytokines | Rheumatoid arthritis | Inflammatory diseases | Cells | Cancer
Journal Article