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Nature, ISSN 0028-0836, 04/2010, Volume 464, Issue 7292, pp. 1149 - 1154
Journal Article
Nature, ISSN 0028-0836, 03/2019, Volume 567, Issue 7746, p. 43
Journal Article
Journal Article
Journal Article
Diabetes, ISSN 0012-1797, 11/2011, Volume 60, Issue 11, pp. 2872 - 2882
OBJECTIVE-To evaluate whether healthy or diabetic adult mice can tolerate an extreme loss of pancreatic a-cells and how this sudden massive depletion affects... 
INSULIN | HYPERGLUCAGONEMIA | ENDOCRINE PANCREAS | GLUCOSE-HOMEOSTASIS | ENDOCRINOLOGY & METABOLISM | RECEPTOR GENE | SECRETION | DIFFERENTIATION | HYPERPLASIA | ISLET CELLS | EXPRESSION | Insulin-Secreting Cells - secretion | Apoptosis - drug effects | Cell Count | Glucagon - genetics | Male | Diphtheria Toxin - toxicity | Insulin - blood | Glucagon - blood | Diabetes Mellitus, Experimental - blood | Hypoglycemia - prevention & control | Intercellular Signaling Peptides and Proteins - metabolism | Glucagon-Secreting Cells - drug effects | Insulin-Secreting Cells - metabolism | Hyperglycemia - chemically induced | Glucagon-Secreting Cells - metabolism | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Glucagon-Secreting Cells - secretion | Hyperglycemia - prevention & control | Promoter Regions, Genetic | Signal Transduction | Glucagon-Secreting Cells - pathology | Intercellular Signaling Peptides and Proteins - genetics | Pancreas - drug effects | Pancreas - pathology | Receptors, Glucagon - metabolism | Mice, Transgenic | Pancreas - metabolism | Heparin-binding EGF-like Growth Factor | Insulin - metabolism | Animals | Insulin-Secreting Cells - drug effects | Tamoxifen - pharmacology | Diabetes Mellitus, Experimental - pathology | Glucagon - metabolism | Mice | Streptozocin - toxicity | Insulin-Secreting Cells - pathology | Selective Estrogen Receptor Modulators - pharmacology | Index Medicus | Abridged Index Medicus | Islet Studies
Journal Article
Nature, ISSN 0028-0836, 03/2019, Volume 567, Issue 7746, pp. 43 - 3
Cell-identity switches, in which terminally differentiated cells are converted into different cell types when stressed, represent a widespread regenerative... 
Transcription factors | Phosphorylation | Transplants & implants | Glucagon | Medical treatment | Diabetes mellitus | Switches | Insulin | Ablation | Proteins | Plasticity | Stem cells | Proteomics | Plastic properties | Mice | Diabetes | Somatostatin | Apoptosis
Journal Article
Cell Metabolism, ISSN 1550-4131, 03/2017, Volume 25, Issue 3, pp. 622 - 634
Journal Article
Nature, ISSN 0028-0836, 03/2019, Volume 567, Issue 7746, pp. 43 - 48
Cell-identity switches, in which terminally differentiated cells are converted into different cell types when stressed, represent a widespread regenerative... 
SURVIVAL | THERAPY | ISLETS | DECOY SEARCH STRATEGY | CONVERSION | MULTIDISCIPLINARY SCIENCES | TISSUE | PANCREATIC BETA-CELLS | PROTEIN-PHOSPHORYLATION | EXPRESSION | EXOCRINE
Journal Article
Journal Article
Diabetes, ISSN 0012-1797, 11/2011, Volume 60, Issue 11, p. 2872
OBJECTIVE--To evaluate whether healthy or diabetic adult mice can tolerate an extreme loss of pancreatic [alpha]-cells and how this sudden massive depletion... 
Glucagon | Physiological aspects | Development and progression | Cellular signal transduction | Research | Diabetes | Risk factors
Journal Article