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The Journal of pharmacology and experimental therapeutics, 08/2019
Adenosine signaling is associated with ethanol-related behaviors. We previously found that adenosine A2A receptor (A2AR) activation dampens ethanol-drinking... 
Journal Article
Frontiers in Molecular Neuroscience, ISSN 1662-5099, 05/2018, Volume 11, p. 158
Journal Article
Molecular & Cellular Proteomics, ISSN 1535-9476, 12/2017, Volume 16, Issue 12, pp. 2268 - 2280
The intrinsic nature of glycosylation, namely nontemplate encoded, stepwise elongation and termination with a diverse range of isomeric glyco-epitopes... 
Brain | Transformation | Computational neuroscience | Data acquisition | Data processing | Workflow | Mass spectroscopy | Glycosylation | Mapping | N-glycans | Data mining | Optimization | Computer programs | Polysaccharides | Reproducibility | Coding | Computer applications | Diagnostic systems | Mass spectrometry | Elongation | Technological Innovation and Resources
Journal Article
Nature Communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 1 - 18
Huntington’s disease (HD) is a neurodegenerative disorder that manifests with movement dysfunction. The expression of mutant Huntingtin (mHTT) disrupts the... 
Brain | Level (quantity) | Cell survival | Huntingtin | Disease | Neurodegenerative diseases | Pathogenesis | Lysosomes | Inflammation | Galectin-3 | Huntington's disease | Microglia | Mice
Journal Article
JOURNAL OF BIOMEDICAL SCIENCE, ISSN 1021-7770, 10/2018, Volume 25, Issue 1, pp. 71 - 14
Journal Article
Cellular and Molecular Life Sciences (CMLS), ISSN 1420-682X, 12/2012, Volume 69, Issue 24, p. 4107
Huntington's disease (HD) is an autosomal dominant neurodegenerative disease caused by a CAG trinucleotide expansion in the Huntingtin (Htt) gene. When the... 
Nervous system diseases | Mitochondrial DNA | Huntington's chorea | Creatine | Creatine kinase | Movement disorders | Proteins | Huntingtons disease | Mitochondria | Kinases | Neurodegeneration
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 8, p. e22924
We investigated the therapeutic potential of human bone marrow-derived mesenchymal stem cells (hBM-MSCs) in Huntington's disease (HD) mouse models. Ten weeks... 
STROKE | QUINOLINIC ACID | RAT STRIATUM | ADULT BRAIN | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | EXCITOTOXIC LESION | NEURAL TRANSPLANTATION | FETAL STRIATAL TRANSPLANTATION | EXPRESSION | MARROW STROMAL CELLS | Apoptosis - drug effects | Humans | Huntington Disease - pathology | Caspase 3 - metabolism | Male | Cell Movement - physiology | Huntington Disease - therapy | Mesenchymal Stromal Cells - cytology | Motor Neurons - cytology | Bone Marrow Transplantation | Quinolinic Acid - toxicity | Cell Differentiation - physiology | Mesenchymal Stromal Cells - physiology | Motor Neurons - drug effects | Disease Models, Animal | Microglia - cytology | Microglia - drug effects | Mice, Inbred C57BL | Cells, Cultured | Huntington Disease - metabolism | Cell Movement - drug effects | Animals | Cell Differentiation - drug effects | Cell Proliferation - drug effects | Mice | Mesenchymal Stem Cell Transplantation | Genetically modified animals | Analysis | Von Willebrand factor | Stem cells | Bone marrow | Animal genetic engineering | Transplantation | Neurophysiology | Huntingtons disease | Animal models | Transplants & implants | Bax protein | Mesenchyme | Bone marrow transplantation | Stem cell transplantation | Biochemistry | Caspase-3 | Laminin | Transgenic animals | Neuroblasts | Rodents | Neostriatum | Tomography | Life sciences | Quinolinic acid | Genetic modification | Antigens | Cell survival | Extracellular signal-regulated kinase | Caspase | Huntington's disease | SDF-1 protein | Survival | Patients | CXCR4 protein | Microglia | Studies | Molecular biology | Differentiation | Cell migration | Apoptosis
Journal Article