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1. Full Text Haploinsufficiency of TNFAIP3 ( A20 ) by germline mutation is involved in autoimmune lymphoproliferative syndrome
by Takagi, Masatoshi, MD, PhD and Ogata, Shohei, MD, PhD and Ueno, Hiroo, MD and Yoshida, Kenichi, MD, PhD and Yeh, Tzuwen, BSc and Hoshino, Akihiro, MD, PhD and Piao, Jinhua, DDS, PhD and Yamashita, Motoy, MD and Nanya, Mai, PhD and Okano, Tsubasa, MD and Kajiwara, Michiko, MD, PhD and Kanegane, Hirokazu, MD, PhD and Muramatsu, Hideki, MD, PhD and Okuno, Yusuke, MD, PhD and Shiraishi, Yuichi, MD, PhD and Chiba, Kenichi, BA and Tanaka, Hiroko, BS and Bando, Yuki, MD, PhD and Kato, Motohiro, MD, PhD and Hayashi, Yasuhide, MD, PhD and Miyano, Satoru, PhD and Imai, Kohsuke, MD, PhD and Ogawa, Seishi, MD, PhD and Kojima, Seiji, MD, PhD and Morio, Tomohiro, MD, PhD
Journal of allergy and clinical immunology, ISSN 0091-6749, 2016, Volume 139, Issue 6, pp. 1914 - 1922
Allergy and Immunology | TNFAIP3 (A20) | Autoimmune lymphoproliferative syndrome | Allergy | Immunology | Life Sciences & Biomedicine | Science & Technology | Humans | Cells, Cultured | NF-kappa B - immunology | Infant | Male | Haploinsufficiency | Tumor Necrosis Factor alpha-Induced Protein 3 - genetics | Phenotype | Autoimmune Lymphoproliferative Syndrome - genetics | Leukocytes, Mononuclear - immunology | Autoimmune Lymphoproliferative Syndrome - immunology | Germ-Line Mutation | Tumor Necrosis Factor alpha-Induced Protein 3 - immunology | Gene mutations | Developmental biology | Analysis | Genomics | Genetic research | Genetic aspects | Information management | Children | Health aspects | Diseases | Pediatrics | Fas antigen | Disease | Transcription | Genes | Science | Frameshift mutation | Genomes | Kinases | Gene sequencing | Proteins | Signal transduction | Alterations | Functional anatomy | Databases | Pathways | Lymphocytes | Immunoglobulins | Patients | Tumor necrosis factor | Mutation | Autoimmune diseases | Apoptosis | Index Medicus | Abridged Index Medicus
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2. Full Text Integrated molecular analysis of clear-cell renal cell carcinoma
by Sato, Yusuke and Yoshizato, Tetsuichi and Shiraishi, Yuichi and Maekawa, Shigekatsu and Okuno, Yusuke and Kamura, Takumi and Shimamura, Teppei and Sato-Otsubo, Aiko and Nagae, Genta and Suzuki, Hiromichi and Nagata, Yasunobu and Yoshida, Kenichi and Kon, Ayana and Suzuki, Yutaka and Chiba, Kenichi and Tanaka, Hiroko and Niida, Atsushi and Fujimoto, Akihiro and Tsunoda, Tatsuhiko and Morikawa, Teppei and Maeda, Daichi and Kume, Haruki and Sugano, Sumio and Fukayama, Masashi and Aburatani, Hiroyuki and Sanada, Masashi and Miyano, Satoru and Homma, Yukio and Ogawa, Seishi
Nature genetics, ISSN 1061-4036, 08/2013, Volume 45, Issue 8, pp. 860 - 867
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Cell Line | Kidney Neoplasms - genetics | Signal Transduction | Genomics | Humans | Gene Expression Regulation, Neoplastic | Carcinoma, Renal Cell - genetics | Gene Expression Profiling | Kidney Neoplasms - metabolism | Transcription Factors - genetics | Kidney Neoplasms - mortality | Hypoxia-Inducible Factor 1 - metabolism | DNA Copy Number Variations | Elongin | Exome | Transcription Factors - metabolism | Carcinoma, Renal Cell - metabolism | Carcinoma, Renal Cell - mortality | DNA Methylation | High-Throughput Nucleotide Sequencing | Mutation | Genome, Human | Cluster Analysis | Gene mutations | Physiological aspects | Genetic aspects | Research | Carcinoma, Renal cell | Methylation | Gene expression | Risk factors | Pathogenesis | DNA methylation | Epigenetics | Genomes | Deoxyribonucleic acid--DNA | Index Medicus
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3. Full Text Mutational landscape and clonal architecture in grade II and III gliomas
by Suzuki, Hiromichi and Aoki, Kosuke and Chiba, Kenichi and Sato, Yusuke and Shiozawa, Yusuke and Shiraishi, Yuichi and Shimamura, Teppei and Niida, Atsushi and Motomura, Kazuya and Ohka, Fumiharu and Yamamoto, Takashi and Tanahashi, Kuniaki and Ranjit, Melissa and Wakabayashi, Toshihiko and Yoshizato, Tetsuichi and Kataoka, Keisuke and Yoshida, Kenichi and Nagata, Yasunobu and Sato-Otsubo, Aiko and Tanaka, Hiroko and Sanada, Masashi and Kondo, Yutaka and Nakamura, Hideo and Mizoguchi, Masahiro and Abe, Tatsuya and Muragaki, Yoshihiro and Watanabe, Reiko and Ito, Ichiro and Miyano, Satoru and Natsume, Atsushi and Ogawa, Seishi
Nature genetics, ISSN 1061-4036, 05/2015, Volume 47, Issue 5, pp. 458 - 468
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Genetic Predisposition to Disease | Glioma - mortality | Genetic Association Studies | Gene Frequency | Humans | Kaplan-Meier Estimate | Brain Neoplasms - genetics | DNA Copy Number Variations | Glioma - genetics | Neoplasm Grading | High-Throughput Nucleotide Sequencing | Brain Neoplasms - mortality | Mutation | Development and progression | Genetic aspects | Gene mutations | Gliomas | Identification and classification | Health aspects | Studies | Architecture | Pathogenesis | Brain cancer | Medical prognosis | Mortality | Genomes | Histology | Tumors | Cancer | Index Medicus
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4. Full Text Aberrant PD-L1 expression through 3′-UTR disruption in multiple cancers
by Kataoka, Keisuke and Shiraishi, Yuichi and Takeda, Yohei and Sakata, Seiji and Matsumoto, Misako and Nagano, Seiji and Maeda, Takuya and Nagata, Yasunobu and Kitanaka, Akira and Mizuno, Seiya and Tanaka, Hiroko and Chiba, Kenichi and Ito, Satoshi and Watatani, Yosaku and Kakiuchi, Nobuyuki and Suzuki, Hiromichi and Yoshizato, Tetsuichi and Yoshida, Kenichi and Sanada, Masashi and Itonaga, Hidehiro and Imaizumi, Yoshitaka and Totoki, Yasushi and Munakata, Wataru and Nakamura, Hiromi and Hama, Natsuko and Shide, Kotaro and Kubuki, Yoko and Hidaka, Tomonori and Kameda, Takuro and Masuda, Kyoko and Minato, Nagahiro and Kashiwase, Koichi and Izutsu, Koji and Takaori-Kondo, Akifumi and Miyazaki, Yasushi and Takahashi, Satoru and Shibata, Tatsuhiro and Kawamoto, Hiroshi and Akatsuka, Yoshiki and Shimoda, Kazuya and Takeuchi, Kengo and Seya, Tsukasa and Miyano, Satoru and Ogawa, Seishi
Nature (London), ISSN 0028-0836, 05/2016, Volume 534, Issue 7607, pp. 402 - 406
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Up-Regulation | Clonal Selection, Antigen-Mediated | Tumor Escape - genetics | Humans | Gene Expression Regulation, Neoplastic | 3' Untranslated Regions - genetics | Programmed Cell Death 1 Receptor - antagonists & inhibitors | RNA, Messenger - metabolism | Neoplasms - genetics | Programmed Cell Death 1 Receptor - biosynthesis | Female | Adenocarcinoma - genetics | Stomach Neoplasms - genetics | Antibodies - therapeutic use | Leukemia-Lymphoma, Adult T-Cell - genetics | RNA, Messenger - genetics | RNA Stability | Antibodies - pharmacology | Animals | Genetic Markers - genetics | CRISPR-Cas Systems | Cell Line, Tumor | Mice | Neoplasms - pathology | Lymphoma, Large B-Cell, Diffuse - genetics | Programmed Cell Death 1 Receptor - genetics | Molecular targeted therapy | Care and treatment | Genetic variation | Ligands (Biochemistry) | Innovations | Genetic aspects | Gene expression | Properties | Health aspects | Cancer | Proteins | Genes | Immunotherapy | Cytotoxicity | Ligands | Genomes | Lymphoma | Apoptosis | Index Medicus
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5. Full Text Age-related remodelling of oesophageal epithelia by mutated cancer drivers
by Yokoyama, Akira and Kakiuchi, Nobuyuki and Yoshizato, Tetsuichi and Nannya, Yasuhito and Suzuki, Hiromichi and Suzuki, Hiromichi and Takeuchi, Yasuhide and Shiozawa, Yusuke and Sato, Yusuke and Sato, Yusuke and Aoki, Kosuke and Aoki, Kosuke and Kim, Soo Ki and Kim, Soo Ki and Fujii, Yoichi and Yoshida, Kenichi and Yoshida, Kenichi and Kataoka, Keisuke and Kataoka, Keisuke and Nakagawa, Masahiro M and Nakagawa, Masahiro M and Inoue, Yoshikage and Inoue, Yoshikage and Hirano, Tomonori and Shiraishi, Yuichi and Shiraishi, Yuichi and Chiba, Kenichi and Chiba, Kenichi and Tanaka, Hiroko and Tanaka, Hiroko and Sanada, Masashi and Sanada, Masashi and Nishikawa, Yoshitaka and Nishikawa, Yoshitaka and Amanuma, Yusuke and Ohashi, Shinya and Ohashi, Shinya and Aoyama, Ikuo and Aoyama, Ikuo and Horimatsu, Takahiro and Miyamoto, Shin'ichi and Miyamoto, Shinichi and Tsunoda, Shigeru and Sakai, Yoshiharu and Sakai, Yoshiharu and Narahara, Maiko and Brown, J.B and Sato, Yoshitaka and Sato, Yoshitaka and Sawada, Genta and Mimori, Koshi and Mimori, Koshi and Minamiguchi, Sachiko and Haga, Hironori and Haga, Hironori and Seno, Hiroshi and Seno, Hiroshi and Miyano, Satoru and Miyano, Satoru and Makishima, Hideki and Muto, Manabu and Muto, Manabu and Ogawa, Seishi and Ogawa, Seishi
Nature (London), ISSN 0028-0836, 01/2019, Volume 565, Issue 7739, pp. 312 - 317
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Life Style | Cell Count | Humans | Middle Aged | Child, Preschool | Infant | Male | Gene-Environment Interaction | DNA Copy Number Variations | Esophageal Neoplasms - pathology | Young Adult | Aging - genetics | Cell Transformation, Neoplastic - genetics | Aged, 80 and over | Adult | Female | Single-Cell Analysis | Child | Mutation Accumulation | Epithelium - pathology | Epithelium - metabolism | Risk Factors | Smoking - genetics | Clone Cells - metabolism | Precancerous Conditions - genetics | Sequence Analysis, DNA | Alcohol Drinking - genetics | Genome, Human - genetics | Aging - pathology | Clone Cells - pathology | Esophageal Neoplasms - genetics | Biopsy | Adolescent | Aged | Mutation | Protein Phosphatase 2C - genetics | Receptor, Notch1 - genetics | Evolution, Molecular | Risk factors (Health) | Gene mutations | Demographic aspects | Analysis | Epithelial tumors | Development and progression | Models | Esophageal cancer | Automobile drivers | Medical colleges | Squamous cell carcinoma | Genetic aspects | Comparative analysis | Health aspects | Risk factors | Sample size | Genomics | Alcohol | Genomes | Remodeling | Older people | Dependence | Aging | Tumorigenesis | Chronology | Children | Adolescents | Signatures | Expansion | Age | Drinking behavior | Cloning | Health risks | Epithelium | Esophagus | Alcoholic beverages | Lifestyles | Cancer | Smoking | Geriatrics | Polymorphism | Index Medicus
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6. Full Text Somatic RHOA mutation in angioimmunoblastic T cell lymphoma
by Sakata-Yanagimoto, Mamiko and Enami, Terukazu and Yoshida, Kenichi and Shiraishi, Yuichi and Ishii, Ryohei and Miyake, Yasuyuki and Muto, Hideharu and Tsuyama, Naoko and Sato-Otsubo, Aiko and Okuno, Yusuke and Sakata, Seiji and Kamada, Yuhei and Nakamoto-Matsubara, Rie and Tran, Nguyen Bich and Izutsu, Koji and Sato, Yusuke and Ohta, Yasunori and Furuta, Junichi and Shimizu, Seiichi and Komeno, Takuya and Sato, Yuji and Ito, Takayoshi and Noguchi, Masayuki and Noguchi, Emiko and Sanada, Masashi and Chiba, Kenichi and Tanaka, Hiroko and Suzukawa, Kazumi and Nanmoku, Toru and Hasegawa, Yuichi and Nureki, Osamu and Miyano, Satoru and Nakamura, Naoya and Takeuchi, Kengo and Ogawa, Seishi and Chiba, Shigeru
Nature genetics, ISSN 1061-4036, 2014, Volume 46, Issue 2, pp. 171 - 175
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | NIH 3T3 Cells | Immunoblastic Lymphadenopathy - genetics | Jurkat Cells | Humans | Lymphoma, T-Cell, Peripheral - genetics | Molecular Sequence Data | Bromodeoxyuridine | Isocitrate Dehydrogenase - genetics | Proto-Oncogene Proteins - genetics | rhoA GTP-Binding Protein - genetics | DNA-Binding Proteins - genetics | Sequence Analysis, DNA | Mutation, Missense - genetics | DNA (Cytosine-5-)-Methyltransferases - genetics | Exome - genetics | Animals | Base Sequence | Lymphoma, T-Cell, Peripheral - pathology | Mice | Genetic aspects | Non-Hodgkin's lymphomas | Research | Gene mutations | Health aspects | Risk factors | Medical research | Lymphocytes | Genomes | Mutation | Gene expression | Lymphoma | Tumors | Index Medicus
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7. Full Text Dynamics of clonal evolution in myelodysplastic syndromes
by Makishima, Hideki and Yoshizato, Tetsuichi and Yoshida, Kenichi and Sekeres, Mikkael A and Radivoyevitch, Tomas and Suzuki, Hiromichi and Przychodzen, Bartlomie J and Nagata, Yasunobu and Meggendorfer, Manja and Sanada, Masashi and Okuno, Yusuke and Hirsch, Cassandra and Kuzmanovic, Teodora and Sato, Yusuke and Sato-Otsubo, Aiko and Laframboise, Thomas and Hosono, Naoko and Shiraishi, Yuichi and Chiba, Kenichi and Haferlach, Claudia and Kern, Wolfgang and Tanaka, Hiroko and Shiozawa, Yusuke and Gómez-Seguí, Inés and Husseinzadeh, Holleh D and Thota, Swapna and Guinta, Kathryn M and Dienes, Brittney and Nakamaki, Tsuyoshi and Miyawaki, Shuichi and Saunthararajah, Yogen and Chiba, Shigeru and Miyano, Satoru and Shih, Lee-Yung and Haferlach, Torsten and Ogawa, Seishi and MacIejewski, Jaroslaw P
Nature genetics, ISSN 1061-4036, 01/2017, Volume 49, Issue 2, pp. 204 - 212
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Exome - genetics | Humans | Myelodysplastic Syndromes - genetics | Clonal Evolution - genetics | Mutation - genetics | Clone Cells - metabolism | Disease Progression | Leukemia, Myeloid, Acute - genetics | Care and treatment | Gene mutations | Development and progression | Genetic aspects | Gene expression | Health aspects | Myelodysplastic syndromes | Data analysis | Research & development--R&D | Cloning | Science | Genomes | Mutation | Grants | Tumors | Cancer | Index Medicus
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8. Full Text Clinical utility of next-generation sequencing for inherited bone marrow failure syndromes
by Muramatsu, Hideki and Okuno, Yusuke and Yoshida, Kenichi and Shiraishi, Yuichi and Doisaki, Sayoko and Narita, Atsushi and Sakaguchi, Hirotoshi and Kawashima, Nozomu and Wang, Xinan and Xu, Yinyan and Chiba, Kenichi and Tanaka, Hiroko and Hama, Asahito and Sanada, Masashi and Takahashi, Yoshiyuki and Kanno, Hitoshi and Yamaguchi, Hiroki and Ohga, Shouichi and Manabe, Atsushi and Harigae, Hideo and Kunishima, Shinji and Ishii, Eiichi and Kobayashi, Masao and Koike, Kenichi and Watanabe, Kenichiro and Ito, Etsuro and Takata, Minoru and Yabe, Miharu and Ogawa, Seishi and Miyano, Satoru and Kojima, Seiji
Genetics in medicine, ISSN 1098-3600, 07/2017, Volume 19, Issue 7, pp. 796 - 802
nextgeneration sequencing | target sequencing | Fanconi anemia | inherited bone marrow failure | whole-exome sequencing | Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Bone Marrow Diseases - diagnosis | High-Throughput Nucleotide Sequencing - utilization | Genetic Testing | Hemoglobinuria, Paroxysmal - genetics | Humans | Male | Mutation - genetics | Whole Exome Sequencing - methods | Bone Marrow Diseases - genetics | Anemia, Aplastic - diagnosis | Exome - genetics | Female | Hemoglobinuria, Paroxysmal - diagnosis | High-Throughput Nucleotide Sequencing - methods | Sequence Analysis, DNA - methods | Anemia, Aplastic - genetics | Bone marrow | Index Medicus
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