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Journal of Clinical Oncology, ISSN 0732-183X, 05/2019, Volume 37, Issue 15_suppl, pp. e14519 - e14519
Journal Article
Human Pathology, ISSN 0046-8177, 2013, Volume 44, Issue 7, pp. 1365 - 1374
Summary The tumor suppressor gene ARID1A encodes BAF250a, a component of human SWI/SNF chromatin-remodeling complexes. Loss of BAF250a expression has recently... 
Pathology | Immunohistochemistry | Tissue microarray | ARID1A/BAF250a | Cervical cancer | Prognostic marker | SWI/SNF COMPLEX | SUPPRESSOR GENE | CHROMATIN | REMODELING GENE ARID1A | HUMAN-PAPILLOMAVIRUS | PATHOLOGY | BREAST-CANCER | PROTEIN EXPRESSION | MUTATIONS | CLEAR-CELL CARCINOMA | Cervical Intraepithelial Neoplasia - pathology | Lymph Nodes - pathology | Tissue Array Analysis | Humans | Gene Expression Regulation, Neoplastic | Uterine Cervical Neoplasms - pathology | Adenocarcinoma - metabolism | Uterine Cervical Neoplasms - metabolism | Biomarkers, Tumor - metabolism | Adult | Cervical Intraepithelial Neoplasia - mortality | Female | Adenocarcinoma - genetics | Nuclear Proteins - genetics | Uterine Cervical Neoplasms - mortality | Nuclear Proteins - metabolism | Survival Rate | Lymphatic Metastasis | Transcription Factors - genetics | Uterine Cervical Neoplasms - genetics | Adenocarcinoma - secondary | Disease Progression | Transcription Factors - metabolism | Cervical Intraepithelial Neoplasia - metabolism | Biomarkers, Tumor - genetics | Cervical Intraepithelial Neoplasia - genetics | HeLa Cells | Adenocarcinoma - mortality | Prognosis | RNA | Analysis | Development and progression | Tumors | Proteins | Human papillomavirus | Hospitals | Radiation therapy | Cancer therapies | Ovarian cancer | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, pp. 4077 - 4093
Adaptation of tumor cells to the host is a major cause of cancer progression, failure of therapy, and ultimately death. Immune selection drives this adaptation... 
TUMOR-INFILTRATING LYMPHOCYTES | MEDICINE, RESEARCH & EXPERIMENTAL | HIGH NUMBER | CELLS | ACTIVATION | VACCINE POTENCY | ENHANCEMENT | RESISTANCE | SELF-RENEWAL | AKT | ANTITUMOR IMMUNITY | Neoplasm Transplantation | Colonic Neoplasms - genetics | Tumor Escape | CD8-Positive T-Lymphocytes - pathology | Cell Proliferation | Homeodomain Proteins - metabolism | Neoplastic Stem Cells - immunology | Apoptosis - genetics | Transplantation, Heterologous | Homeodomain Proteins - immunology | Colonic Neoplasms - metabolism | Proto-Oncogene Proteins c-akt - genetics | Signal Transduction - immunology | Neoplastic Stem Cells - metabolism | Colonic Neoplasms - immunology | Proto-Oncogene Proteins - immunology | CD8-Positive T-Lymphocytes - metabolism | Female | Proto-Oncogene Proteins c-akt - metabolism | Proto-Oncogene Proteins - metabolism | Nanog Homeobox Protein | Proto-Oncogene Proteins - genetics | Signal Transduction - genetics | Mice, SCID | Homeodomain Proteins - genetics | Animals | Apoptosis - immunology | Proto-Oncogene Proteins c-akt - immunology | Colonic Neoplasms - pathology | Cell Line, Tumor | Transcription, Genetic - immunology | Mice, Inbred NOD | Mice | Transcription, Genetic - genetics | CD8-Positive T-Lymphocytes - immunology | Phenotype | Immunogenetics | Cellular signal transduction | Genetic aspects | Research | Identification and classification | Cancer | Index Medicus | Abridged Index Medicus | Adaptations | Cell proliferation | Signal transduction | Animal models | Colon cancer | Transcription | Tumor cells | AKT protein | Evolution | Apoptosis | Tumors
Journal Article
Nature Communications, ISSN 2041-1723, 07/2015, Volume 6, Issue 1, pp. 7769 - 7769
The activity of the phosphatase and tensin homologue (PTEN) is known to be suppressed via post-translational modification. However, the mechanism and... 
APOPTOSIS | CELLS | GENE | TUMOR SUPPRESSION | PATHWAY | MULTIDISCIPLINARY SCIENCES | GERMLINE MUTATIONS | DEGRADATION | POSTTRANSLATIONAL REGULATION | CANCER | LIGASE | Immunohistochemistry | Phosphorylation | Adenocarcinoma - pathology | Prognosis | TOR Serine-Threonine Kinases - metabolism | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Gene Expression Regulation, Neoplastic | Phosphatidylinositol 3-Kinases - metabolism | Receptor, Epidermal Growth Factor - metabolism | Ubiquitination | Adenocarcinoma - metabolism | Uterine Cervical Neoplasms - metabolism | Phosphoproteins | Female | Adenocarcinoma - genetics | Carcinoma - pathology | Proto-Oncogene Proteins c-akt - metabolism | PTEN Phosphohydrolase - genetics | Carcinogenesis - genetics | PTEN Phosphohydrolase - metabolism | Ribonucleoproteins - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Uterine Cervical Neoplasms - genetics | Nerve Tissue Proteins - metabolism | Phosphatidylinositol 3-Kinases - genetics | Feedback, Physiological | Cell Line, Tumor | Carcinoma - genetics | Cervical Intraepithelial Neoplasia - metabolism | Cervical Intraepithelial Neoplasia - genetics | Protein Processing, Post-Translational | Carcinoma - metabolism | HeLa Cells | Mutation | In Vitro Techniques | Cell Movement | Index Medicus
Journal Article
Journal Article
International journal of clinical oncology, ISSN 1341-9625, 07/2019
Forkhead box protein O1 (FOXO1) and paired box gene 3 (PAX3) have been reported to play an imported role in human cancers, but their role in cervical cancer... 
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 08/2015, Volume 21, Issue 16 Supplement, pp. POSTER-BIOL-1351 - POSTER-BIOL-1351
Journal Article
Cancer Research, ISSN 0008-5472, 09/2017, Volume 77, Issue 18, pp. 5039 - 5053
Cancer immunoediting drives the adaptation of tumor cells to host immune surveillance. Immunoediting driven by antigen (Ag)-specific T cells enriches NANOG... 
UBIQUITINATION | HETEROGENEITY | LUNG-CANCER | ACTIVATION | REPRESSION | ONCOLOGY | MCL-1 | DRUG-RESISTANCE | LARGE GENE LISTS | INHIBITOR | PLASTICITY | Cell Proliferation | Prognosis | Breast Neoplasms - immunology | Neoplastic Stem Cells - drug effects | Drug Resistance, Multiple - drug effects | Epigenesis, Genetic | Humans | Transcriptional Activation | Neoplastic Stem Cells - immunology | Uterine Cervical Neoplasms - pathology | Biomarkers, Tumor | Breast Neoplasms - metabolism | Neoplastic Stem Cells - metabolism | Uterine Cervical Neoplasms - metabolism | Neoplastic Stem Cells - pathology | Female | Acetylation | Tumor Cells, Cultured | Histone Deacetylase 1 - genetics | Drug Resistance, Multiple - immunology | Nanog Homeobox Protein - genetics | Uterine Cervical Neoplasms - immunology | Uterine Cervical Neoplasms - drug therapy | Combined Modality Therapy | Drug Resistance, Neoplasm - immunology | Mice, SCID | Breast Neoplasms - drug therapy | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - pathology | Mice, Inbred NOD | Mice | Histones - metabolism | Neoplasm Staging | Nanog Homeobox Protein - metabolism | Apoptosis | Drug Resistance, Neoplasm - drug effects | Histone Deacetylase 1 - metabolism | Histone deacetylase | Tumor cells | Immunomodulation | Multidrug resistance | Immunosurveillance | Lymphocytes T | Drug resistance | Mcl-1 protein | Modulators | Genotype & phenotype | Lymphocytes | Immunotherapy | Epigenetics | Histone H3 | Cancer | Index Medicus
Journal Article
Human Pathology, ISSN 0046-8177, 2013, Volume 44, Issue 4, pp. 472 - 479
Journal Article
BMC Cancer, ISSN 1471-2407, 12/2014, Volume 14, Issue 1, pp. 957 - 957
Background: NKG2D (natural killer group 2, member D) is thought to play an important role in mediating the activation of anticancer immune response. Expression... 
Immunohistochemistry | Tissue microarray | NKG2D ligands | Cervical cancer | NATURAL-KILLER-CELLS | RECEPTOR | CHAIN | WORLDWIDE | RETINOIC ACID | ONCOLOGY | POOR-PROGNOSIS | EXPRESSION | CYTOTOXICITY | CARCINOMA | DNA microarrays | Prognosis | Comparative analysis | Index Medicus
Journal Article
International Journal of Hyperthermia, ISSN 0265-6736, 01/2019, Volume 36, Issue 1, pp. 9 - 20
Purpose: Modulated electro-hyperthermia (mEHT), known as oncothermia, is an anticancer therapy that induces radiofrequency thermal damage to the cancer... 
autophagy inhibitor | anti-cancer effects | Modulated electro-hyperthermia | ovarian cancer | cervical cancer | SURVIVAL | APOPTOSIS | HEAT-SHOCK | INDUCTION | CHEMOTHERAPY | PROGRAMMED CELL-DEATH | ONCOLOGY | RECURRENT | GENE-EXPRESSION | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | AGGREGATION
Journal Article
Biomarkers, ISSN 1354-750X, 05/2009, Volume 14, Issue 3, pp. 161 - 170
Journal Article
Tumor Biology, ISSN 1010-4283, 10/2018, Volume 40, Issue 10, pp. 1010428318808678 - 1010428318808678
Journal Article