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Nature, ISSN 0028-0836, 11/2014, Volume 515, Issue 7527, pp. 431 - 435
Journal Article
Journal of Molecular Biology, ISSN 0022-2836, 12/2018, Volume 430, Issue 24, pp. 4821 - 4822
Journal Article
Nature, ISSN 0028-0836, 04/2016, Volume 532, Issue 7597, pp. 112 - 116
Brown and beige adipose tissues can dissipate chemical energy as heat through thermogenic respiration, which requires uncoupling protein 1 (UCP1)(1,2).... 
ADIPOCYTES | REDOX STATE | MULTIDISCIPLINARY SCIENCES | DIMEDONE | UNCOUPLING PROTEIN-1 | CHEMICAL PROBES | FAT | BROWN ADIPOSE-TISSUE | MULTIPLE SEQUENCE ALIGNMENT | EXPRESSION | MASS-SPECTROMETRY | Reactive Oxygen Species - metabolism | Humans | Ion Channels - genetics | Male | Mitochondrial Proteins - genetics | Cysteine - genetics | Sulfhydryl Compounds - metabolism | Adipose Tissue, Brown - chemistry | Cell Respiration | Mitochondrial Proteins - metabolism | Female | Cysteine - metabolism | Mitochondrial Proteins - deficiency | Ion Channels - chemistry | Oxidation-Reduction | Mice, Inbred C57BL | Mutant Proteins - genetics | Mutant Proteins - metabolism | Mitochondria - metabolism | Cysteine - chemistry | Mitochondria - drug effects | Adipose Tissue, Brown - cytology | Animals | Ion Channels - metabolism | Mutant Proteins - chemistry | Mitochondrial Proteins - chemistry | Adipose Tissue, Brown - metabolism | Mice | Thermogenesis - drug effects | Uncoupling Protein 1 | Energy Metabolism - drug effects | Ion Channels - deficiency | Physiological aspects | Brown adipose tissue | Energy metabolism | Cell research | Research | Bioenergetics | Cold | Body temperature | Homeostasis | Lipids | Adipocytes | Gene expression | Proteins | Mitochondria | Thermogenesis | Rodents | Physiology | Respiration | Metabolic disorders | Index Medicus
Journal Article
Journal Article
Nature, ISSN 0028-0836, 04/2018, Volume 556, Issue 7699, pp. 113 - 117
Journal Article
Journal Article
Biochemical Journal, ISSN 0264-6021, 08/2010, Volume 430, Issue 1, pp. 49 - 59
The S-nitrosation of mitochondrial proteins as a consequence of NO metabolism is of physiological and pathological significance. We previously developed a... 
S-nitrosation | Mitochondria | Redox signalling | Difference in gel electrophoresis (DIGE) | Nitric oxide (NO) | S-nitrosylation | nitric oxide (NO) | ALDEHYDE DEHYDROGENASE | PEROXYNITRITE | mitochondria | BIOCHEMISTRY & MOLECULAR BIOLOGY | BIOTIN SWITCH ASSAY | ASCORBIC-ACID | MASS-SPECTROMETRY | redox signalling | ISCHEMIA-REPERFUSION INJURY | difference in gel electrophoresis (DIGE) | NITRIC-OXIDE | DETECTING CHANGES | EXPRESSION | NITROSYLATION | Oxidation-Reduction | Mice, Inbred C57BL | Rats | Electrophoresis, Gel, Two-Dimensional | Male | Myocardium - pathology | S-Nitrosothiols - metabolism | Myocardial Reperfusion Injury - metabolism | Mitochondrial Proteins - pharmacology | Myocardial Reperfusion Injury - pathology | Animals | Mitochondrial Proteins - metabolism | Myocardium - metabolism | Membrane Potential, Mitochondrial | Proteomics | Superoxides - metabolism | Mice | In Vitro Techniques | Mitochondria - physiology | Index Medicus | BVA, biological variance analysis | AR, area at risk | RHM, rat heart mitochondria | SA, standardized abundance | DIGE, difference in gel electrophoresis | GSNO, S-nitrosoglutathione | SNO-DIGE, S-nitrosothiol difference in gel electrophoresis | DTT, dithiothreitol | R, ischaemia | RLM, rat liver mitochondria | 2D, two-dimensional | Cy3, indocarbocyanine | ROS, reactive oxygen species | reperfusion | PrSNO, protein S-nitrosothiol | MitoNAP, mito-N-acetylpenicillamine | Cy5, indodicarbocyanine | LAD, left anterior descending coronary artery | NEM, N-ethylmaleimide | LV, left ventricle | SNAP, S-nitroso-N-acetylpenicillamine | MitoSNO, mitochondria-targeted S-nitrosothiol | α-KGDH, α-ketoglutarate dehydrogenase | TPP, triphenylphosphonium | NIH, National Institutes for Health | ALDH2, aldehyde dehydrogenase | MALDI–TOF-TOF, matrix-assisted laser-desorption ionization–time-of-flight time-of-flight
Journal Article
Journal Article
Nature, ISSN 0028-0836, 08/2018, Volume 560, Issue 7716, pp. 102 - 106
Thermogenesis by brown and beige adipose tissue, which requires activation by external stimuli, can counter metabolic disease(1). Thermogenic respiration is... 
ENERGY-EXPENDITURE | NONSHIVERING THERMOGENESIS | METABOLISM | MULTIDISCIPLINARY SCIENCES | MITOCHONDRIA | LACTATE | FAT | UCP1 | DYSFUNCTION | BROWN-ADIPOCYTES | DEFICIENCY | Metabolomics | Reactive Oxygen Species - metabolism | Adipose Tissue, White - metabolism | Adipocytes - enzymology | Male | Adipocytes - drug effects | Thermogenesis - physiology | Adipose Tissue, White - cytology | Oxidation-Reduction - drug effects | Female | Uncoupling Protein 1 - metabolism | Adipose Tissue, Brown - enzymology | Obesity - metabolism | Adipose Tissue, Brown - cytology | Animals | Succinic Acid - pharmacology | Succinic Acid - metabolism | Adipocytes - metabolism | Obesity - prevention & control | Adipose Tissue, White - enzymology | Succinate Dehydrogenase - metabolism | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Mice | Thermogenesis - drug effects | Adipose Tissue, White - drug effects | Adipose tissues | Physiological aspects | Control | Thermogenesis | Succinates | Protein kinase A | Tricarboxylic acid cycle | Reactive oxygen species | Succinate dehydrogenase | Dehydrogenases | Adipose tissue | Body fat | Activation | Adipocytes | Kinases | Dehydrogenase | Accumulation | Proteins | Signal transduction | Energy | Metabolites | Rodents | Oxidation | Adipose tissue (brown) | Oxygen | AMP | Cyclic AMP | Pharmacology | Metabolism | Lipolysis | Glucose tolerance | Signaling | Hypotheses | External stimuli | Respiration | Index Medicus
Journal Article