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PLoS Pathogens, ISSN 1553-7366, 02/2019, Volume 15, Issue 2, p. e1007583
CD8 T cells are necessary for the elimination of intracellular pathogens, but during chronic viral infections, CD8 T cells become exhausted and unable to... 
ACTIVATION | VIRUS-INFECTION | MICROBIOLOGY | LPS | PLASMACYTOID DENDRITIC CELLS | T-CELL RESPONSES | VIROLOGY | MEMORY | IN-VIVO | LIPOPOLYSACCHARIDE | MICE | MICROBIAL TRANSLOCATION | PARASITOLOGY | Lymphocytic choriomeningitis virus - immunology | Dendritic Cells | Lymphocytic Choriomeningitis | Virus Diseases - immunology | Signal Transduction | Lymphocyte Activation | Mice, Inbred C57BL | Male | Programmed Cell Death 1 Receptor - antagonists & inhibitors | Toll-Like Receptor 4 - immunology | Adoptive Transfer | Toll-Like Receptor 4 - metabolism | CD8-Positive T-Lymphocytes - physiology | Animals | Lymphocytic choriomeningitis virus - pathogenicity | Lipopolysaccharides - pharmacology | Female | Mice | Programmed Cell Death 1 Receptor - immunology | Chronic Disease | Viral meningitis | Usage | Care and treatment | Dendritic cells | Immunotherapy | Models | Cellular signal transduction | Research | TLR4 | Potentiation | Therapy | Animal models | PD-1 protein | CD8 antigen | Chronic infection | Cytotoxicity | Viruses | Infections | Lymphocytes T | Genomes | B7 antigen | Experiments | Lipopolysaccharides | Cell activation | Immunology | Lymphocytes | Human immunodeficiency virus--HIV | Toll-like receptors | Medical research | Antigens | TLR4 protein | Medicine | Signaling | Cell death | Viral infections | Cancer | Apoptosis | HIV | Human immunodeficiency virus
Journal Article
Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13 Supplement, pp. 5500 - 5500
Journal Article
Blood, ISSN 0006-4971, 12/2016, Volume 128, Issue 22, pp. 1478 - 1478
Abstract The discovery that mRNA splicing factors are frequently mutated in clonal blood disorders represented one of the most unexpected findings from cancer... 
Journal Article
Blood, ISSN 0006-4971, 12/2015, Volume 126, Issue 23, pp. 302 - 302
Abstract Mutations in Addition of Sex Combs Like 1 (ASXL1) are common in patients with myeloid leukemias. More recently, mutations in ASXL2, a paralog of ASXL1... 
Journal Article
Experimental Hematology, ISSN 0301-472X, 08/2014, Volume 42, Issue 8, pp. S29 - S29
Journal Article
Experimental Hematology, ISSN 0301-472X, 2014, Volume 42, Issue 8, pp. S29 - S29
Journal Article
Blood, ISSN 0006-4971, 11/2013, Volume 122, Issue 21, pp. 3627 - 3627
Abstract Background Thrombosis is a major cause of morbidity and mortality in the Philadelphia-chromosome negative myeloproliferative neoplasms (MPN). However,... 
Journal Article
Cancer Research, ISSN 0008-5472, 07/2013, Volume 73, Issue 13 Supplement, pp. A09 - A09
Journal Article
Journal Article
Blood, ISSN 0006-4971, 11/2013, Volume 122, Issue 21, pp. 2876 - 2876
Abstract Background Although treatment with purine analogs is associated with a high response rate, hairy cell leukemia (HCL) remains incurable with a relapse... 
Journal Article
Nature Medicine, ISSN 1078-8956, 06/2016, Volume 22, Issue 6, pp. 672 - 678
Journal Article
Cancer Cell, ISSN 1535-6108, 08/2012, Volume 22, Issue 2, pp. 180 - 193
Journal Article
Journal Article
Cancer Cell, ISSN 1535-6108, 05/2013, Volume 23, Issue 5, pp. 677 - 692
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2013, Volume 19, Issue 7, pp. 924 - 929
During wound healing, stem cells provide functional mature cells to meet acute demands for tissue regeneration(1). Simultaneously, the tissue must maintain a... 
SKIN PIGMENTATION | MEDICINE, RESEARCH & EXPERIMENTAL | HOMEOSTASIS | VITILIGO | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | INVOLVEMENT | NICHE | REPIGMENTATION | CELL BIOLOGY | HORMONE | HAIR-FOLLICLES | PROMOTER | Humans | Male | Melanocytes - metabolism | Receptor, Melanocortin, Type 1 - metabolism | Stem Cells - metabolism | Cell Movement - genetics | Melanocytes - radiation effects | Cell Differentiation - genetics | Epidermis - injuries | Hair Follicle - injuries | Female | Signal Transduction - radiation effects | Wound Healing - genetics | Epidermis - radiation effects | Hair Follicle - radiation effects | Mice, Inbred C57BL | Receptor, Melanocortin, Type 1 - genetics | Cells, Cultured | Stem Cells - radiation effects | Receptor, Melanocortin, Type 1 - physiology | Mice, Transgenic | Signal Transduction - genetics | Melanocytes - physiology | Ultraviolet Rays - adverse effects | Cell Movement - radiation effects | Hair Follicle - cytology | Animals | Models, Biological | Epidermis - physiology | Signal Transduction - physiology | Stem Cells - physiology | Mice | Wound Healing - physiology | Epidermis - cytology | Wound healing | Stem cells | Physiological aspects | Melanocytes | Genetic aspects | Cellular signal transduction | Research | Health aspects | Methods | Clinical trials | Cellular biology | Cell division
Journal Article