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PLoS ONE, ISSN 1932-6203, 01/2016, Volume 11, Issue 1, pp. e0146438 - e0146438
Purpose We evaluated whether orally administered astaxanthin (AST) protects against oxidative damage in the ocular tissues of streptozotocin (STZ)-induced... 
BETA-CAROTENE | IN-VITRO | HUMAN-DISEASE | DB/DB MICE | VITREOUS FLUID | MULTIDISCIPLINARY SCIENCES | EXPERIMENTAL GALACTOSEMIA | HUMAN HEALTH | HEME OXYGENASE-1 | ENDOTOXIN-INDUCED UVEITIS | NF-KAPPA-B | Retina - drug effects | Gene Expression Regulation, Enzymologic - drug effects | Diabetes Mellitus, Experimental - drug therapy | Retina - metabolism | Body Weight | NF-kappa B - metabolism | Retinal Ganglion Cells - metabolism | Aqueous Humor - metabolism | Inflammation Mediators - metabolism | Female | Chemokine CCL2 - metabolism | Diabetes Mellitus, Experimental - metabolism | Electroretinography | Gene Expression | Protective Agents | RNA, Messenger - genetics | Rats | Blood Glucose | Antioxidants - pharmacology | Intercellular Adhesion Molecule-1 - metabolism | Animals | NF-kappa B - genetics | Xanthophylls - pharmacology | Oxidative Stress - drug effects | Lutein - pharmacology | Retina - pathology | Retinal Ganglion Cells - drug effects | Complications and side effects | Oxidative stress | Streptozocin | Astaxanthin | Dosage and administration | Research | Drug therapy | Immunohistochemistry | Neuroprotection | Diabetic retinopathy | Pathogenesis | Retina | Proteins | Antioxidants | Acrolein | Carotenoids | Animal tissues | Heme | Cell adhesion | Fractalkine | Drug dosages | Enzyme-linked immunosorbent assay | Stresses | NF-κB protein | Damage assessment | Cytokines | Intercellular adhesion molecule 1 | Diabetes mellitus | Oral administration | Lutein | Heme oxygenase (decyclizing) | Inflammation | Metabolism | Gene expression | Stress | Medicine | Polymerase chain reaction | Oxygenase | Hospitals | Deoxyguanosine | Monocyte chemoattractant protein | Electrophoretic mobility | Nitrotyrosine | Diabetes | Monocyte chemoattractant protein 1 | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2017, Volume 12, Issue 9, pp. e0184927 - e0184927
Purpose Optic neuritis is highly correlated with multiple sclerosis and is a major cause of acute visual loss and long-term neuronal degeneration. Primary... 
CENTRAL SEROUS CHORIORETINOPATHY | CEREBRAL HYPOPERFUSION | PERIPAPILLARY CHOROIDAL THICKNESS | AXONAL DEGENERATION | MULTIPLE-SCLEROSIS PATIENTS | BLOOD-FLOW-VELOCITY | NERVE-FIBER LAYER | WHITE-MATTER | ENDOTHELIN-1 PLASMA-LEVELS | MULTIDISCIPLINARY SCIENCES | COHERENCE TOMOGRAPHY | Acute Disease | Choroid - diagnostic imaging | Tomography, Optical Coherence | Humans | Middle Aged | Optic Neuritis - diagnostic imaging | Papilledema - pathology | Male | Adrenal Cortex Hormones - therapeutic use | Demyelinating Diseases - metabolism | Young Adult | Choroid - physiology | Regression Analysis | Models, Biological | Optic Neuritis - drug therapy | Adult | Female | Demyelinating Diseases - pathology | Optic Neuritis - pathology | Retina - pathology | Cohort Studies | Glaucoma | Brain | Diabetic retinopathy | Multiple sclerosis | Nuclear magnetic resonance--NMR | Optic neuropathy | Optic neuritis | Myopia | Retina | Optics | Family medical history | Neuropathy | Sclerosis | Inflammatory diseases | Visual perception | Eye | Macular degeneration | Regression models | Neurodegeneration | Demyelination | Neuritis | Eye (anatomy) | Degeneration | Optic nerve | Medical imaging | Swelling | Demyelinating diseases | Customizing | Regression analysis | Patients | Diseases | Medicine | Studies | Neurology | Brain research | Hospitals | Optical Coherence Tomography | Perfusion | Vision | Alzheimers disease | Index Medicus | Nuclear magnetic resonance | NMR
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2016, Volume 11, Issue 8, pp. e0161897 - e0161897
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, pp. e64262 - e64262
Purpose: To evaluate the protective effects of bortezomib (Velcade) on ischemia-reperfusion (IR) injury in the rat retina. Methods: The rats were randomized to... 
ISCHEMIA/REPERFUSION INJURY | LYMPHOCYTE RECIRCULATION | LEUKOCYTE INFILTRATION | BRAIN-INJURY | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | GANGLION-CELLS | GENE-EXPRESSION | NECROSIS-FACTOR-ALPHA | NF-KAPPA-B | TUMOR-SUPPRESSOR P53 | Retina - drug effects | Retina - metabolism | Apoptosis - drug effects | Chymotrypsin - metabolism | Cell Count | Male | NF-kappa B - metabolism | RNA, Messenger - metabolism | Antigens, CD - metabolism | Retinal Ganglion Cells - pathology | Inflammation Mediators - metabolism | Reperfusion Injury - metabolism | Biomarkers - metabolism | Bortezomib | Proteasome Inhibitors - pharmacology | Reperfusion Injury - pathology | RNA, Messenger - genetics | Retina - physiopathology | Rats | Rats, Sprague-Dawley | Apoptosis Regulatory Proteins - metabolism | Gene Expression Regulation - drug effects | Animals | Reperfusion Injury - prevention & control | Antigens, Differentiation, Myelomonocytic - metabolism | Reperfusion Injury - physiopathology | Proteasome Endopeptidase Complex - metabolism | Pyrazines - pharmacology | Retina - pathology | Retinal Ganglion Cells - drug effects | Boronic Acids - pharmacology | Nitric Oxide Synthase Type II - metabolism | Ischemia | Tumor proteins | Thioredoxin | Heme | Apoptosis | Peroxiredoxin | Oxidative stress | Diabetic retinopathy | Bax protein | p53 Protein | Staining | Retina | Cell density | Proteins | Acrolein | Reperfusion | Rodents | Cell cycle | Pretreatment | Drug dosages | Electroretinography | Medical research | NF-κB protein | Free radicals | Intercellular adhesion molecule 1 | Neurons | Retinal cells | Markers | Emigration | Inflammation | Gene expression | ErbB-2 protein | Nitric-oxide synthase | Electroretinograms | Endothelium | Studies | Oxygenase | Retinal ganglion cells | Injury prevention | Tumor necrosis factor | Nitric oxide | Nitrotyrosine | Immunofluorescence | Monocyte chemoattractant protein 1 | Index Medicus
Journal Article
Scientific reports, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 529 - 529
This study evaluated the characteristic changes in optical coherence tomography (OCT) biomarkers in neovascular age-related macular degeneration (nAMD) treated... 
VISUAL-ACUITY | VISION LOSS | THERAPY | MULTIDISCIPLINARY SCIENCES | PIGMENT EPITHELIAL DETACHMENT | DOSING REGIMEN | RANIBIZUMAB | INTRAVITREAL AFLIBERCEPT
Journal Article
Scientific reports, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 595 - 595
This study analyze the morphological characteristics of branching vascular networks (BVN) in polypoidal choroidal vasculopathy (PCV) using optical coherence... 
DIAGNOSIS | THERAPY | SPECIFICITY | SUBTYPES | MULTIDISCIPLINARY SCIENCES | MACULAR DEGENERATION | CLASSIFICATION | THICKNESS
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, pp. e77323 - e77323
This study investigated the therapeutic potential and mechanisms of chitosan oligosaccharides (COS) for oxidative stress-induced retinal diseases. Retinal... 
PROGRAMMED CELL-DEATH | CHITOOLIGOSACCHARIDES | WATER-SOLUBLE CHITOSAN | APOPTOSIS | BCL-2 | IN-VITRO | MULTIDISCIPLINARY SCIENCES | NECROSIS-FACTOR-ALPHA | NF-KAPPA-B | TRANSCRIPTION FACTOR | DAMAGE | Apoptosis - drug effects | Glutathione - metabolism | Antioxidants - metabolism | Cell Count | Cell Survival - genetics | NF-kappa B - metabolism | RNA, Messenger - metabolism | Retinal Degeneration - prevention & control | Retinal Ganglion Cells - pathology | Oligosaccharides - pharmacology | Chitosan - therapeutic use | Chitosan - pharmacology | Free Radicals - metabolism | Chemokine CCL2 - metabolism | Electroretinography | Cell Line | Cell Survival - drug effects | Retinal Degeneration - drug therapy | Oligosaccharides - therapeutic use | RNA, Messenger - genetics | Oxidative Stress - genetics | Rats | Chemokine CCL2 - genetics | Retinal Degeneration - physiopathology | Rats, Sprague-Dawley | Gene Expression Regulation - drug effects | Animals | Oxidative Stress - drug effects | Retinal Degeneration - pathology | Retinal Ganglion Cells - drug effects | Nitric Oxide Synthase Type II - metabolism | Physiological aspects | Free radicals (Chemistry) | Health aspects | Apoptosis | Oxidative stress | Diabetic retinopathy | Reactive oxygen species | Transcription factors | Staining | Retina | Superoxide dismutase | Kinases | Acrolein | Signal transduction | Catalase | Rodents | Attenuation | Tumor necrosis factor-TNF | Degeneration | Deoxyribonucleic acid--DNA | Stresses | NF-κB protein | Damage assessment | Free radicals | Neurons | Superoxide | Chemiluminescence | Oligosaccharides | Injection | Gene expression | Chemical damage | Electroretinograms | Retinal ganglion cells | Deoxyguanosine | Retinal degeneration | Paraquat | Nitrotyrosine | Chitosan | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
American Journal of Ophthalmology, ISSN 0002-9394, 2012, Volume 154, Issue 5, pp. 872 - 880.e2
Journal Article