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Journal of Neuro-Oncology, ISSN 0167-594X, 3/2018, Volume 137, Issue 1, pp. 215 - 218
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s11060-017-2705-4 
Medicine & Public Health | Neurology | Oncology | BREAST-CANCER | ONCOLOGY | CLINICAL NEUROLOGY | Neurosciences | Meninges
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 3, p. e18200
Targeting pro-inflammatory events to reduce seizures is gaining momentum. Experimentally, antagonism of inflammatory processes and of blood-brain barrier (BBB)... 
RASMUSSENS-SYNDROME | IN-VITRO | INFANTILE SPASMS | DISRUPTION | TEMPORAL-LOBE EPILEPSY | HMPAO SPECT | PILOCARPINE | BIOLOGY | BLOOD-BRAIN-BARRIER | EPILEPTOGENESIS | CHILDHOOD | Seizures - blood | Humans | Seizures - drug therapy | Adrenocorticotropic Hormone - pharmacology | Electroencephalography | Pilocarpine | Adrenal Cortex Hormones - therapeutic use | Seizures - diagnostic imaging | Seizures - physiopathology | Dexamethasone - pharmacology | Time Factors | Anti-Inflammatory Agents - therapeutic use | Child | Disease Models, Animal | Acute Disease | Adrenocorticotropic Hormone - therapeutic use | Anti-Inflammatory Agents - pharmacology | Adrenal Cortex Hormones - pharmacology | Rats | Treatment Outcome | Drug Resistance - drug effects | Blood-Brain Barrier - drug effects | Radiography | Blood-Brain Barrier - pathology | Dexamethasone - therapeutic use | Animals | Dexamethasone | Epilepsy | ACTH | Brain damage | Inflammation | Seizures (Medicine) | Children | Drug resistance | Drug therapy | Health aspects | Pediatrics | Disorders | Lithium | Biology | Hydrocortisone | Methylprednisolone | Antagonism | Convulsions & seizures | Blood-brain barrier | Ischemia | Rodents | Extravasation | Seizures | Seizing | Edema | Encephalitis | Cytokines | Mortality | IL-1β | Spasms | Studies | Hypotheses | Brain research | Adrenocorticotropic hormone | Brain injury | Integrity
Journal Article
ANTICANCER RESEARCH, ISSN 0250-7005, 07/2019, Volume 39, Issue 7, pp. 3803 - 3808
Background: Platinum-based therapy represents the main pharmacological treatment for ovarian carcinoma. Since molecular targeting of receptor tyrosine kinases... 
SURVIVAL | Ovarian carcinoma | ONCOLOGY | AXL | KINASE | RESISTANCE | cisplatin | p38 MAPK | CANCER | Tyrosine | Transcription | Toxicity | Downstream effects | Drug interactions | Pharmacology | Tumor cell lines | Kinases | Cisplatin | Axl protein | Cell death | Platinum | Cell lines | Down-regulation | Drug interaction | Combined treatment | Synergistic effect | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 12, p. e15183
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2011, Volume 6, Issue 12, p. e28321
Background: Mesenchymal stromal cells may represent an ideal candidate to deliver anti-cancer drugs. In a previous study, we demonstrated that exposure of... 
STEM-CELLS | IN-VITRO | HUMAN BONE-MARROW | MULTIDISCIPLINARY SCIENCES | GENE-THERAPY | UMBILICAL-CORD BLOOD | MULTIDRUG-RESISTANCE | MELANOMA GROWTH | LARGE ANIMALS | ADIPOSE-TISSUE | DELIVERY | Neoplasms - metabolism | Paclitaxel - metabolism | Paclitaxel - pharmacology | Humans | ATP-Binding Cassette, Sub-Family B, Member 1 - metabolism | Antineoplastic Agents - therapeutic use | Antineoplastic Agents - metabolism | Biological Transport | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Tumor Microenvironment - drug effects | Mesenchymal Stromal Cells - drug effects | Mesenchymal Stromal Cells - metabolism | Neoplasms - blood supply | Paclitaxel - therapeutic use | Neoplasms - drug therapy | Xenograft Model Antitumor Assays | Animals | Neovascularization, Pathologic - drug therapy | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Kinetics | Mesenchymal Stromal Cells - pathology | Endothelial Cells - pathology | Neoplasms - pathology | Endothelial Cells - drug effects | Anthracyclines | Chemotherapy | Analysis | Paclitaxel | Genetic engineering | Health aspects | Hematopoietic stem cells | Cancer | Cell culture | Therapy | Syngeneic grafts | Cytotoxicity | Confocal | Drug development | Cancer therapies | Doxorubicin | Anticancer properties | Angiogenesis | Antitumor agents | Ischemia | Coinjection | Bone marrow | Aorta | Conditioning | Public health | Melanoma | Immunodeficiency | Liquid chromatography | Studies | Stromal cells | Stem cells | Umbilical cord | Cell proliferation | Drugs | Flow cytometry | Neurosciences | Drug delivery systems | Mesenchyme | Stem cell transplantation | Confocal microscopy | Allografts | Cell cycle | Tumor cell lines | High-performance liquid chromatography | Myogenesis | Endothelial cells | Virology | Microscopy | Coronary vessels | In vivo methods and tests | Bone | Chemokines | Apoptosis | Tumors
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2014, Volume 9, Issue 9, p. e108403
The pathogenetic role of vascular endothelial growth factor (VEGF) in long-term retinal and kidney complications of diabetes has been demonstrated. Conversely,... 
PATHOGENESIS | APOPTOSIS | PERIPHERAL-NERVE REGENERATION | HYPERGLYCEMIA | INHIBITION | GANGLION NEURONS | MULTIDISCIPLINARY SCIENCES | MECHANISMS | EXPRESSION | NEUROTOXICITY | ENDOTHELIAL GROWTH-FACTOR | Diabetes Mellitus, Experimental - drug therapy | Streptozocin | Coculture Techniques | Diabetes Mellitus, Experimental - genetics | Male | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | Hyperglycemia - drug therapy | Hyperglycemia - genetics | Bevacizumab | Vascular Endothelial Growth Factor A - secretion | Diabetic Neuropathies - genetics | Hyperglycemia - chemically induced | Antibodies, Monoclonal, Humanized - pharmacology | Hyperglycemia - pathology | Nociception - drug effects | Diabetes Mellitus, Experimental - chemically induced | Neural Conduction - drug effects | Sensory Receptor Cells - metabolism | Schwann Cells - secretion | Neurites - drug effects | Schwann Cells - drug effects | Vascular Endothelial Growth Factor Receptor-1 - genetics | Sensory Receptor Cells - pathology | Signal Transduction | Gene Expression Regulation | Angiogenesis Inhibitors - pharmacology | Rats | Schwann Cells - pathology | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Diabetic Neuropathies - drug therapy | Neurites - metabolism | Rats, Sprague-Dawley | Neurites - pathology | Animals | Sensory Receptor Cells - drug effects | Diabetic Neuropathies - pathology | Ganglia, Spinal - pathology | Diabetes Mellitus, Experimental - pathology | Ganglia, Spinal - drug effects | Diabetic Neuropathies - chemically induced | Ganglia, Spinal - metabolism | Diabetic retinopathy | Conduction | Syngeneic grafts | Pathogenesis | Impairment | Headaches | Retina | Diabetic neuropathy | mRNA | Glucose | Neuropathy | Experiments | Nerve conduction | Receptors | Hyperglycemia | Dorsal root ganglia | Rodents | Vascular endothelial growth factor | Pain perception | Kidneys | Neurons | Complications | Axonogenesis | Diabetes mellitus | Schwann cells | Cultures | Gene expression | Signaling | Hypoxia | Diabetes | Intracellular | Laboratory animals | Apoptosis | Neutralization
Journal Article
Journal Article
Anti-cancer agents in medicinal chemistry, ISSN 1871-5206, 11/2017, Volume 17, Issue 11, pp. 1578 - 1585
Journal Article
Journal Article
Scientific reports, ISSN 2045-2322, 11/2018, Volume 8, Issue 1, pp. 17060 - 1
A correction to this article has been published and is linked from the HTML and PDF versions of this paper. The error has not been fixed in the paper. 
Spermiogenesis
Journal Article