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Methods in Molecular Biology, ISSN 1064-3745, 2017, Volume 1543, pp. 259 - 270
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2006, Volume 103, Issue 10, pp. 3799 - 3804
Journal Article
Nature Medicine, ISSN 1078-8956, 09/2017, Volume 23, Issue 9, pp. 1046 - 1054
It is generally assumed that recurrent mutations within a given cancer driver gene elicit similar drug responses. Cancer genome studies have identified... 
MEDICINE, RESEARCH & EXPERIMENTAL | BROMODOMAIN INHIBITORS | ENDOMETRIAL CARCINOMAS | UBIQUITIN LIGASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | TUMORS | CELL BIOLOGY | BREAST-CANCER | GENOMIC LANDSCAPE | PROSTATE-CANCER | RESISTANCE | MUTATIONS | Immunohistochemistry | Neoplasms, Cystic, Mucinous, and Serous - metabolism | Neoplasm Transplantation | Prostatic Neoplasms - metabolism | Immunoprecipitation | Apoptosis - drug effects | Heterocyclic Compounds, 3-Ring - pharmacology | Epigenesis, Genetic | Humans | Endometrial Neoplasms - metabolism | Drug Resistance, Neoplasm | Immunoblotting | Male | Molecular Targeted Therapy | Carcinoma, Endometrioid - metabolism | Neoplasms, Cystic, Mucinous, and Serous - genetics | Ubiquitination | Prostatic Neoplasms - genetics | Endometrial Neoplasms - genetics | Mass Spectrometry | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Chromatography, Liquid | Cullin Proteins - metabolism | Female | Nuclear Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | RNA-Binding Proteins - antagonists & inhibitors | Acetanilides - pharmacology | Carcinoma, Endometrioid - genetics | Carcinosarcoma - metabolism | Adenocarcinoma, Clear Cell - metabolism | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Reverse Transcriptase Polymerase Chain Reaction | Azepines - pharmacology | Transcription Factors - metabolism | Triazoles - pharmacology | Animals | Mice, Nude | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Carcinosarcoma - genetics | Cell Proliferation - drug effects | Mutation | Adenocarcinoma, Clear Cell - genetics | RNA-Binding Proteins - metabolism | Care and treatment | Gene mutations | Proteolysis | Physiological aspects | Genetic aspects | Research | Cancer | Ubiquitin | Biodegradation | Inhibitor drugs | Endometrial cancer | Genomes | Pharmacology | Substrates | Mutants | Degradation | Proteins | Sensitivity | Missense mutation | Inhibitors | Bet protein | Gene mapping | Prostate cancer | Prostate | Endometrium | Ubiquitin-protein ligase
Journal Article
Nature Genetics, ISSN 1061-4036, 02/2018, Volume 50, Issue 2, pp. 219 - 228
The mechanisms by which mitochondrial metabolism supports cancer anabolism remain unclear. Here, we found that genetic and pharmacological inactivation of... 
OXIDATIVE STRESS | METABOLIC SWITCH | EPITHELIAL-CELLS | TCA CYCLE | STEROL REGULATORY ELEMENT | MITOCHONDRIA | GENETICS & HEREDITY | ZINC INHIBITION | LIPOPROTEIN RECEPTOR PROMOTER | NUCLEAR-PROTEIN | HISTONE ACETYLATION | Pyruvate Dehydrogenase Complex - metabolism | Prostatic Neoplasms - metabolism | Prostatic Neoplasms - pathology | Humans | Protein Processing, Post-Translational - genetics | Cells, Cultured | Cytoplasm - metabolism | Male | Cytoplasm - pathology | Pyruvate Dehydrogenase (Lipoamide) - metabolism | Mice, Knockout | Cell Compartmentation - physiology | Pyruvate Dehydrogenase Complex - physiology | Animals | Cell Nucleus - metabolism | Prostatic Neoplasms - genetics | Cell Nucleus - genetics | Lipogenesis - genetics | Cell Nucleus - pathology | Cell Line, Tumor | Cytoplasm - genetics | Mice | Pyruvate Dehydrogenase (Lipoamide) - genetics | Phosphatases | Pyruvate dehydrogenase complex | Genetic aspects | Research | Genetic transcription | Diagnosis | Prostate cancer | Animal models | Phosphorylation | Dehydrogenases | Xenotransplantation | Pyruvic acid | Lipids | Biosynthesis | Regulatory sequences | Kinases | Dehydrogenase | Inactivation | Datasets | Proteins | Mitochondria | Cell growth | Xenografts | Tumorigenesis | Acetylation | Lipogenesis | Deactivation | Pyruvate dehydrogenase (lipoamide) | Pharmacology | Metabolism | Gene expression | Citric acid | Respiration | Prostate | Cancer | Apoptosis | Tumors
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2012, Volume 7, Issue 3, p. e33571
Journal Article
Journal Article
EMBO Reports, ISSN 1469-221X, 02/2003, Volume 4, Issue 2, pp. 166 - 171
Journal Article
AGING-US, ISSN 1945-4589, 10/2019, Volume 11, Issue 19, pp. 8036 - 8038
Journal Article
Molecular & Cellular Oncology, ISSN 2372-3556, 09/2019, Volume 6, Issue 5, p. e1644598
The emergence of therapy-resistant cancer stem cells (CSCs) limit the efficacy of prostate cancer treatment. Using genetic knockdown and chemical inhibitors,... 
bromodomain and extra-terminal domain proteins | cancer stem cells | mitochondrial fission | MFF | Prostate cancer | epigenetics | BRD4
Journal Article
Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13 Supplement, pp. 5080 - 5080
Journal Article