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Human Mutation, ISSN 1059-7794, 01/2013, Volume 34, Issue 1, pp. 79 - 82
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 1 - 10
Soluble amyloid-β (Aβ) is considered to be a critical component in the pathogenesis of Alzheimer’s disease (AD). Evidence suggests that these non-fibrillar Aβ... 
Animal models | Microscopy | Cell death | Neurodegeneration | Autopsy | Amyloid | Alzheimer's disease | Plaques
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2013, Volume 8, Issue 9, pp. e74203 - e74203
Journal Article
by Sims, Rebecca and Van Der Lee, Sven J and Naj, Adam C and Bellenguez, Céline and Badarinarayan, Nandini and Jakobsdottir, Johanna and Kunkle, Brian W and Boland, Anne and Raybould, Rachel and Bis, Joshua C and Martin, Eden R and Grenier-Boley, Benjamin and Heilmann-Heimbach, Stefanie and Chouraki, Vincent and Kuzma, Amanda B and Sleegers, Kristel and Vronskaya, Maria and Ruiz, Agustin and Graham, Robert R and Olaso, Robert and Hoffmann, Per and Grove, Megan L and Vardarajan, Badri N and Hiltunen, Mikko and Nöthen, Markus M and White, Charles C and Hamilton-Nelson, Kara L and Epelbaum, Jacques and Maier, Wolfgang and Choi, Seung-Hoan and Beecham, Gary W and Dulary, Cécile and Herms, Stefan and Smith, Albert V and Funk, Cory C and Derbois, Céline and Forstner, Andreas J and Ahmad, Shahzad and Li, Hongdong and Bacq, Delphine and Harold, Denise and Satizabal, Claudia L and Valladares, Otto and Squassina, Alessio and Thomas, Rhodri and Brody, Jennifer A and Qu, Liming and Sánchez-Juan, Pascual and Morgan, Taniesha and Wolters, Frank J and Zhao, Yi and Garcia, Florentino Sanchez and Denning, Nicola and Fornage, Myriam and Malamon, John and Naranjo, Maria Candida Deniz and Majounie, Elisa and Mosley, Thomas H and Dombroski, Beth and Wallon, David and Lupton, Michelle K and Dupuis, Josée and Whitehead, Patrice and Fratiglioni, Laura and Medway, Christopher and Jian, Xueqiu and Mukherjee, Shubhabrata and Keller, Lina and Brown, Kristelle and Lin, Honghuang and Cantwell, Laura B and Panza, Francesco and McGuinness, Bernadette and Moreno-Grau, Sonia and Burgess, Jeremy D and Solfrizzi, Vincenzo and Proitsi, Petra and Adams, Hieab H and Allen, Mariet and Seripa, Davide and Pastor, Pau and Cupples, L Adrienne and Price, Nathan D and Hannequin, Didier and Frank-García, Ana and Levy, Daniel and Chakrabarty, Paramita and Caffarra, Paolo and Giegling, Ina and Beiser, Alexa S and Giedraitis, Vilmantas and Hampel, Harald and Garcia, Melissa E and Wang, Xue and Lannfelt, Lars and Mecocci, Patrizia and Eiriksdottir, Gudny and Crane, Paul K and Pasquier, Florence and Boccardi, Virginia and ... and EADI and GERAD PERADES and ARUKConsortium and CHARGE and ADGC and GERAD/PERADES, CHARGE, ADGC, EADI and ARUK Consortium and Stockholms universitet and Centrum för forskning om äldre och åldrande (ARC), (tills m KI) and Samhällsvetenskapliga fakulteten
Nature Genetics, ISSN 1061-4036, 09/2017, Volume 49, Issue 9, pp. 1373 - 1384
Journal Article
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 2/2013, Volume 125, Issue 2, pp. 201 - 213
Autosomal-dominant Alzheimer disease (ADAD) is a genetic disorder caused by mutations in Amyloid Precursor Protein (APP) or Presenilin (PSEN) genes. Studies... 
Pathology | Neurosciences | Autosomal-dominant Alzheimer disease | Medicine & Public Health | β -Site APP-cleaving enzyme | Presenilin | β -Amyloid | Amyloid precursor protein | β-Amyloid | β-Site APP-cleaving enzyme | BETA-SECRETASE ACTIVITY | PATHOLOGY | CEREBROSPINAL-FLUID | NEUROSCIENCES | CEREBRAL-CORTEX | CLINICAL NEUROLOGY | SENILE PLAQUES | PRESENILIN-1 MUTATIONS | beta-Site APP-cleaving enzyme | BRAIN INTERSTITIAL FLUID | IN-VIVO | beta-Amyloid | EXPRESSION | TRANSGENIC MICE | Immunohistochemistry | Central Nervous System - metabolism | Humans | Middle Aged | Male | Apolipoproteins E - metabolism | Amyloid beta-Protein Precursor - cerebrospinal fluid | Amyloid beta-Protein Precursor - metabolism | Adult | Female | Presenilin-1 - cerebrospinal fluid | Presenilin-1 - genetics | Alzheimer Disease - cerebrospinal fluid | Mutation - genetics | Amyloid Precursor Protein Secretases - cerebrospinal fluid | Blotting, Western | Neurites - pathology | Amyloid Precursor Protein Secretases - metabolism | Aspartic Acid Endopeptidases - cerebrospinal fluid | Apolipoproteins E - genetics | Aspartic Acid Endopeptidases - metabolism | Alzheimer Disease - metabolism | Heterozygote | Aged | Alzheimer Disease - genetics | Physiological aspects | Enzymes | Alzheimer's disease | Amyloid beta-protein | Index Medicus | Brain | Neurodegenerative diseases | BACE protein | Central nervous system | Clinical trials | Data processing | beta -Amyloid | Cerebrospinal fluid | beta -Site APP cleaving enzyme | Amyloid | Mutation | Original Paper
Journal Article
Current Genomics, ISSN 1389-2029, 12/2013, Volume 14, Issue 8, pp. 560 - 567
Breakthroughs in genetics over the last decade have radically advanced our understanding of the etiological basis of Parkinson's disease (PD). Although much... 
Mendelian genes | Genetic risk factor | Clinical genetics | Parkinson's disease | Genetics | Genetic testing | PARK | RISK-FACTORS | DEMENTIA | BIOCHEMISTRY & MOLECULAR BIOLOGY | SUSCEPTIBILITY | PINK1 MUTATIONS | ALPHA-SYNUCLEIN | PLA2G6 | GLUCOCEREBROSIDASE MUTATIONS | LOCUS | GENETICS & HEREDITY | JUVENILE PARKINSONISM | GENOME-WIDE ASSOCIATION
Journal Article