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1. Full Text Chronic overexpression of PNPLA3 I148M in mouse liver causes hepatic steatosis
by Li, John Zhong and Huang, Yongcheng and Karaman, Ruchan and Ivanova, Pavlina T and Brown, H. Alex and Roddy, Thomas and Castro-Perez, Jose and Cohen, Jonathan C and Hobbs, Helen H
Journal of Clinical Investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, pp. 4130 - 4144
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2. Full Text PNPLA3, CGI‐58, and Inhibition of Hepatic Triglyceride Hydrolysis in Mice
by Wang, Yang and Kory, Nora and BasuRay, Soumik and Cohen, Jonathan C and Hobbs, Helen H
Hepatology, ISSN 0270-9139, 06/2019, Volume 69, Issue 6, pp. 2427 - 2441
A variant (148M) in patatin‐like phospholipase domain‐containing protein 3 (PNPLA3) is a major risk factor for fatty liver disease. Despite its clinical... 
PROTEIN | LIPOLYSIS | FATTY LIVER-DISEASE | I148M | GASTROENTEROLOGY & HEPATOLOGY | LIPID DROPLETS | DEFICIENCY | Hydrolysis | Phospholipase | Liver diseases | Immunoprecipitation | Fatty liver | Hepatoma | Lipase | Steatosis | Original
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3. Full Text Inactivation of Tm6sf2, a gene defective in fatty liver disease, impairs lipidation but not secretion of very low density lipoproteins
by Smagris, Eriks and Gilyard, Shenise and BasuRay, Soumik and Cohen, Jonathan C and Hobbs, Helen H
Journal of Biological Chemistry, ISSN 0021-9258, 05/2016, Volume 291, Issue 20, pp. 10659 - 10676
A missense mutation (E167K) in TM6SF2 (transmembrane 6 superfamily member 2), a polytopic protein of unknown function, is associated with the full spectrum of... 
TRIGLYCERIDE TRANSFER PROTEIN | CONFERS SUSCEPTIBILITY | SUPERFAMILY MEMBER 2 | HEPATOCELLULAR-CARCINOMA | CHOLESTEROL | BIOCHEMISTRY & MOLECULAR BIOLOGY | RISK | WIDE ASSOCIATION | PNPLA3 RS738409 | HEPATIC STEATOSIS | VARIANT | Fatty Liver - genetics | Fatty Liver - metabolism | Endoplasmic Reticulum - genetics | Lipoylation | Fatty Liver - pathology | Membrane Proteins - genetics | Endoplasmic Reticulum - metabolism | Lipid Droplets - pathology | Golgi Apparatus - pathology | Mutation, Missense | Mice, Knockout | Triglycerides - metabolism | Endoplasmic Reticulum - pathology | Animals | Lipoproteins, VLDL - secretion | Golgi Apparatus - metabolism | Membrane Proteins - metabolism | Mice | Triglycerides - genetics | Lipid Droplets - metabolism | Golgi Apparatus - genetics | Index Medicus | lipid droplet | Lipids | liver injury | triacylglycerol | lipoprotein | cholesterol metabolism
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4. Full Text HSD17B13 and Chronic Liver Disease in Blacks and Hispanics
by Stender, Stefan and Hobbs, Helen H and Kozlitina, Julia and Cohen, Jonathan C
The New England Journal of Medicine, ISSN 0028-4793, 11/2018, Volume 379, Issue 19, pp. 1876 - 1877
The established association between variant HSD17B13 and protection against chronic liver disease has been replicated. A newly discovered, probably damaging... 
POPULATION | MEDICINE, GENERAL & INTERNAL | Gene frequency | Hispanics | Liver diseases | Index Medicus | Abridged Index Medicus
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5. Full Text Accumulation of PNPLA3 on lipid droplets is the basis of associated hepatic steatosis
by BasuRay, S and Wang, Y and Smagris, E and Cohen, JC and Hobbs, HH
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, ISSN 0027-8424, 05/2019, Volume 116, Issue 19, pp. 9521 - 9526
Fatty liver disease (FLD) is a disorder in which accumulation of triglycerides (TGs) in the liver can lead to inflammation, fibrosis, and cirrhosis.... 
lipid droplet | proteasome | autophagy | FATTY LIVER-DISEASE | MULTIDISCIPLINARY SCIENCES | fatty liver disease | TM6SF2 | CONFERS SUSCEPTIBILITY | OVEREXPRESSION | GENE VARIANT | INHIBITION | PROTEIN-DEGRADATION | PNPLA3 | MICE | WIDE ASSOCIATION | Ubiquitin | Inflammation | Proteolysis | Liver cirrhosis | Liver | Liver diseases | Deactivation | Sucrose | Hydrolase | Lipids | Triglycerides | Inactivation | Accumulation | Steatosis | Proteins | Cirrhosis | Phospholipase | Fatty liver | Enzymatic activity | Fibrosis | Mice | Droplets | Sugars | Biological Sciences
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6. Full Text Patatin-like phospholipase domain–containing protein 3 promotes transfers of essential fatty acids from triglycerides to phospholipids in hepatic lipid droplets
by Mitsche, Michael A and Hobbs, Helen H and Cohen, Jonathan C
The Journal of Biological Chemistry, ISSN 0021-9258, 6/2018, Volume 293, Issue 24, pp. 9232 - 9232
Additions and s
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7. Full Text Reply
by Smagris, Eriks and BasuRay, Soumik and Gromada, Jesper and Cohen, Jonathan C and Hobbs, Helen H
Hepatology, ISSN 0270-9139, 02/2016, Volume 63, Issue 2, pp. 677 - 677
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8. Full Text Reply
by Smagris, Eriks and Basuray, Soumik and Gromada, Jesper and Cohen, Jonathan C and Hobbs, Helen H
Hepatology, ISSN 0270-9139, 02/2016, Volume 63, Issue 2, p. 677
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9. Full Text Reply
by Eriks Smagris and Soumik BasuRay and Jesper Gromada and Jonathan C Cohen and Helen H Hobbs
Hepatology, ISSN 0270-9139, 02/2016, Volume 63, Issue 2, p. 677
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10. Full Text The PNPLA3 variant associated with fatty liver disease (I148M) accumulates on lipid droplets by evading ubiquitylation
: Basuray et al
by BasuRay, Soumik and Smagris, Eriks and Cohen, Jonathan C and Hobbs, Helen H
Hepatology, ISSN 0270-9139, 10/2017, Volume 66, Issue 4, pp. 1111 - 1124
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11. Full Text Genetic Variation at PPP1R3B and Changes in Levels of Liver Glycogen and Triglyceride
by Stender, Stefan and Smagris, Eriks and Lauridsen, Bo K and Kofoed, Klaus F and Nordestgaard, Børge G and Tybjærg-Hansen, Anne and Pennacchio, Len A and Dickel, Diane E and Cohen, Jonathan C and Hobbs, Helen H
Hepatology (Baltimore, Md.), ISSN 0270-9139, 6/2018, Volume 67, Issue 6, pp. 2182 - 2195
Genetic variation at rs4240624 on chromosome 8 is associated with an attenuated signal on hepatic computerized tomography (CT), which has been attributed to... 
liver injury | metabolism | hepatic glycogen | hepatic steatosis
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12. Full Text Global, regional, and national age–sex specific all-cause and cause-specific mortality for 240 causes of death, 1990–2013: a systematic analysis for the Global Burden of Disease Study 2013
by Naghavi, Mohsen and Wang, Haidong and Lozano, Rafael and Davis, Aian and Liang, Xiaofeng and Zhou, Maigeng and Vollset, Stein Emil and Ozgoren, Ayse Abbasoglu and Abdalla, Safa and Abd-Allah, Foad and Aziz, Muna I. Abdel and Abera, Semaw Ferede and Aboyans, Victor and Abraham, Biju and Abraham, Jerry P and Abuabara, Katrina E and Abubakar, Ibrahim and Abu-Raddad, Laith J and Abu-Rmeileh, Niveen M. E and Achoki, Tom and Adelekan, Ademola and Ademi, Zanfi Na and Adofo, Koranteng and Adou, Arsene Kouablan and Adsuar, Jose C and Aernlov, Johan and Agardh, Emilie Elisabet and Akena, Dickens and Al Khabouri, Mazin J and Alasfoor, Deena and Albittar, Mohammed and Alegretti, Miguel Angel and Aleman, Alicia V and Alemu, Zewdie Aderaw and Alfonso-Cristancho, Rafael and Alhabib, Samia and Ali, Mohammed K and Ali, Raghib and Alla, Francois and Al Lami, Faris and Allebeck, Peter and AlMazroa, Mohammad A and Salman, Rustam Al-Shahi and Alsharif, Ubai and Alvarez, Elena and Alviz-Guzman, Nelson and Amankwaa, Adansi A and Amare, Azmeraw T and Ameli, Omid and Amini, Hassan and Ammar, Walid and Anderson, H. Ross and Anderson, Benjamin O and Antonio, Carl Abelardo T and Anwari, Palwasha and Apfel, Henry and Cunningham, Solveig Argeseanu and Arsenijevic, Valentina S. Arsic and Al Artaman, Ali and Asad, Majed Masoud and Asghar, Rana J and Assadi, Reza and Atkins, Lydia S and Atkinson, Charles and Badawi, Alaa and Bahit, Maria C and Bakfalouni, Talal and Balakrishnan, Kalpana and Balalla, Shivanthi and Banerjee, Amitava and Barber, Ryan M and Barker-Collo, Suzanne L and Barquera, Simon and Barregard, Lars and Barrero, Lope H and Barrientos-Gutierrez, Tonatiuh and Basu, Arindam and Basu, Sanjay and Basulaiman, Mohammed Omar and Beardsley, Justin and Bedi, Neeraj and Beghi, Ettore and Bekele, Tolesa and Bell, Michelle L and Benjet, Corina and Bennett, Derrick A and Bensenor, Isabela M and Benzian, Habib and Bertozzi-Villa, Amelia and Beyene, Tariku Jibat and Bhala, Neeraj and Bhalla, Ashish and Bhutta, Zulfiqar A and Bikbov, Boris and Bin Abdulhak, Aref and Biryukov, Stan and Blore, Jed D and Blyth, Fiona M and Bohensky, Megan A and Borges, Guilherme and ... and GBD Mortal 2013 Causes Death Colla and GBD 2013 Mortality and Causes of Death Collaborators and Högskolan Dalarna and Akademin Utbildning, hälsa och samhälle and Medicinsk vetenskap
The Lancet, ISSN 0140-6736, 01/2015, Volume 385, Issue 9963, pp. 117 - 171
Summary Background Up-to-date evidence on levels and trends for age-sex-specific all-cause and cause-specific mortality is essential for the formation of... 
Internal Medicine | PNEUMOCOCCAL CONJUGATE VACCINE | CHILDHOOD PNEUMONIA | MEDICINE, GENERAL & INTERNAL | INFLUENZAE TYPE-B | RISK-FACTORS | DOUBLE-BLIND | CHILDREN YOUNGER | POPULATION HEALTH | 187 COUNTRIES | INTEGRATED APPROACH | ENTERIC MULTICENTER | Databases, Factual - standards | Humans | Middle Aged | Child, Preschool | Infant | Male | Mortality - trends | Life Tables | Models, Statistical | Life Expectancy - trends | Young Adult | Global Health - trends | Adolescent | Global Health - statistics & numerical data | Aged, 80 and over | Sex Distribution | Adult | Cause of Death - trends | Female | Aged | Child | Child Mortality - trends | Infant, Newborn | Medicine, Experimental | Medical research | Analysis | Mortality | Global health | Disease | Malaria | Time series | Diarrhea | Epidemiology | Tuberculosis | Acquired immune deficiency syndrome--AIDS | Human immunodeficiency virus--HIV | Meningitis | Trends | Injuries | Index Medicus | Abridged Index Medicus | Global Health | Cause of Death | Child Mortality | Life Sciences | Life Expectancy | Santé publique et épidémiologie | Databases, Factual | Clinical Medicine | Hälsa och välfärd | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Health and Welfare | Nutrition and Disease | HNE Nutrition and Disease | Chair Nutrition and Disease | Voeding en Ziekte | VLAG | HNE Voeding en Ziekte
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