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Cell Death and Differentiation, ISSN 1350-9047, 01/2019, Volume 26, Issue 2, pp. 199 - 212
Journal Article
Carcinogenesis, ISSN 0143-3334, 11/2012, Volume 33, Issue 11, pp. 2007 - 2017
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 01/2017, Volume 24, Issue 1, pp. 15 - 25
One of the most defining features of cancer is aberrant cell communication; therefore, a molecular understanding of the intricate network established among... 
GRAFT ARTERIOSCLEROSIS | BLADDER-CANCER | HEPATOCELLULAR-CARCINOMA | TUMOR-SUPPRESSOR DAB2IP | COLORECTAL-CANCER | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROSTATE-CANCER | ENDOPLASMIC-RETICULUM STRESS | MESENCHYMAL TRANSITION | ABERRANT PROMOTER METHYLATION | PROTEIN HDAB2IP GENE | CELL BIOLOGY | Review
Journal Article
ONCOGENE, ISSN 0950-9232, 08/2019, Volume 38, Issue 34, pp. 6184 - 6195
Missense mutations in the TP53 gene are frequent in human cancers, giving rise to mutant p53 proteins that can acquire oncogenic properties. Gain of function... 
SURVIVAL | APOPTOSIS | METASTASIS | ONCOLOGY | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | PROTEOLYSIS | TRANSLATION | ER | TRANSMEMBRANE PROTEIN | CELL BIOLOGY | Genetic aspects | Tumor proteins | Cancer cells | Cancer | Phenotypes | Cell survival | p53 Protein | Chemoresistance | Breast cancer | Drug delivery | Metastases | Proteins | Chemotherapy | Missense mutation | Protein folding | Mutation | Endoplasmic reticulum | Apoptosis | Tumors
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e35051
Mutations in PARK7/DJ-1 gene are associated to autosomal recessive early onset forms of Parkinson's disease (PD). Although large gene deletions have been... 
PROTEIN | OXIDATIVE DAMAGE | UBIQUITIN | MULTIDISCIPLINARY SCIENCES | MITOCHONDRIAL-FUNCTION | PROTEASOME | DOPAMINERGIC-NEURONS | ALPHA-SYNUCLEIN | DYSFUNCTION | STRESS | LEWY BODIES | Oncogene Proteins - genetics | Cell Fractionation | Cell Proliferation | Humans | Intracellular Signaling Peptides and Proteins - metabolism | Substantia Nigra - metabolism | RNA, Ribosomal - genetics | Lewy Bodies - genetics | Cell Nucleolus - metabolism | Parkinson Disease - metabolism | Lewy Bodies - metabolism | TNF Receptor-Associated Factor 6 - genetics | Nuclear Proteins - genetics | Intracellular Signaling Peptides and Proteins - genetics | Cell Nucleolus - genetics | Oncogene Proteins - metabolism | RNA, Ribosomal - metabolism | Nuclear Proteins - metabolism | Parkinson Disease - genetics | Transcription Factors - genetics | Proteasome Endopeptidase Complex - genetics | Transcription Factors - metabolism | Protein Deglycase DJ-1 | TNF Receptor-Associated Factor 6 - metabolism | Cell Line, Tumor | Proteasome Endopeptidase Complex - metabolism | Genetic aspects | Biosynthesis | RNA | Ligases | Tumor necrosis factor receptors | Brain | Neurosciences | Parkinson's disease | Yeast | Neuropathology | Parkinsons disease | Neurobiology | Neuroblastoma cells | Recruitment | Proteins | Nucleoli | Signal transduction | Missense mutation | Neurodegeneration | Protein folding | Evolution | Physiology | Life sciences | Trends | Stress response | Localization | Ubiquitin-protein ligase | Deoxyribonucleic acid--DNA | Movement disorders | TRAF6 protein | Neurodegenerative diseases | rRNA | MAP kinase | JNK protein | Lewy bodies | PARK7 protein | Molecular modelling | Aggregates | Tumor necrosis factor | Quality control | Mutation | Alzheimers disease | Endoplasmic reticulum | Cytoplasm | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article
Breast Cancer: Targets and Therapy, ISSN 1179-1314, 06/2018, Volume 10, pp. 101 - 112
Breast cancer is the most frequently diagnosed malignancy in women, and mutations in the tumor suppressor p53 are commonly detected in the most aggressive... 
Mutant p53 gain of function | Targeted therapy | Protein-protein interactions | Cancer-cell homeostasis | Cell growth | Breast cancer | Mutation | Kinases | Apoptosis | targeted therapy | cancer cell homeostasis | mutant p53 gain-of-function | protein-protein interactions
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2010, Volume 107, Issue 14, pp. 6322 - 6327
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 07/2017, Volume 114, Issue 29, p. 7623
Obesity and type 2 diabetes are significant risk factors for malignancies, being associated with chronic inflammation and hyperinsulinemia. In this context,... 
Cell proliferation | Obesity | Hyperinsulinemia | AKT1 protein | Cell survival | p53 Protein | Diabetes mellitus | Health risks | Stimulation | Breast cancer | Risk analysis | Insulin | Survival | Synergism | Risk factors | Metastases | Point mutation | Tumor suppressor genes | Diabetes | Inhibition | Prostate cancer | Prostate | Cytoplasm | Cancer
Journal Article
Molecular & Cellular Oncology, ISSN 2372-3556, 10/2015, Volume 2, Issue 4, p. e1002719
Inflammation and mutation of the tumor suppressor p53 are two apparently unrelated conditions that are strongly associated with cancer initiation and... 
cancer cell invasivity | basal-like breast cancer | DAB2IP | tumor necrosis factor | AIP1
Journal Article