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1. Full Text Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease
by Jostins, Luke and Ripke, Stephan and Weersma, Rinse K and Duerr, Richard H and McGovern, Dermot P and Hui, Ken Y and Lee, James C and Schumm, L. Philip and Sharma, Yashoda and Anderson, Carl A and Essers, Jonah and Mitrovic, Mitja and Ning, Kaida and Cleynen, Isabelle and Theatre, Emilie and Spain, Sarah L and Raychaudhuri, Soumya and Goyette, Philippe and Wei, Zhi and Abraham, Clara and Achkar, Jean-Paul and Ahmad, Tariq and Amininejad, Leila and Ananthakrishnan, Ashwin N and Andersen, Vibeke and Anews, Jane M and Baidoo, Leonard and Balschun, Tobias and Bampton, Peter A and Bitton, Alain and Boucher, Gabrielle and Brand, Stephan and Büning, Carsten and Cohain, Ariella and Cichon, Sven and D'Amato, Mauro and de Jong, Dirk and Devaney, Kathy L and Dubinsky, Marla and Edwards, Cathryn and Ellinghaus, David and Ferguson, Lynnette R and Franchimont, Denis and Fransen, Karin and Gearry, Richard and Georges, Michel and Gieger, Christian and Glas, Jürgen and Haritunians, Talin and Hart, Ailsa and Hawkey, Chris and Hedl, Matija and Hu, Xinli and Karlsen, Tom H and Kupcinskas, Limas and Kugathasan, Subra and Latiano, Anna and Laukens, Debby and Lawrance, Ian C and Lees, Charlie W and Louis, Edouard and Mahy, Gillian and Mansfield, John and Morgan, Angharad R and Mowat, Craig and Newman, William and Palmieri, Orazio and Ponsioen, Cyriel Y and Potocnik, Uros and Prescott, Natalie J and Regueiro, Miguel and Rotter, Jerome I and Russell, Richard K and Sanderson, Jeremy D and Sans, Miquel and Satsangi, Jack and Schreiber, Stefan and Simms, Lisa A and Sventoraityte, Jurgita and Targan, Stephan R and Taylor, Kent D and Tremelling, Mark and Verspaget, Hein W and de Vos, Martine and Wijmenga, Cisca and Wilson, David C and Winkelmann, Juliane and Xavier, Ramnik J and Zeissig, Sebastian and Zhang, Bin and Zhang, Clarence K and Zhao, Hongyu and Silverberg, Mark S and Annese, Vito and Hakonarson, Hakon and Brant, Steven R and Radford-Smith, Graham and Mathew, Christopher G and Rioux, John D and Schadt, Eric E and ... and Int IBD Genetics Consortium IIBDGC and International IBD Genetics Consortium (IIBDGC) and The International IBD Genetics Consortium (IIBDGC) and Region Örebro län and Örebro universitet and Institutionen för medicinska vetenskaper
Nature, ISSN 0028-0836, 2012, Volume 491, Issue 7422, pp. 119 - 124
Crohn's disease and ulcerative colitis, the two common forms of inflammatory bowel disease (IBD), affect over 2.5 million people of European ancestry, with... 
HYPER-IGE SYNDROME | RISK LOCI | NETWORK | NUMBER | METAANALYSIS | TUBERCULOSIS | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | MUTATIONS | EXPRESSION | Crohn Disease - genetics | Humans | Inflammatory Bowel Diseases - immunology | Colitis, Ulcerative - genetics | Mycobacterium tuberculosis - immunology | Inflammatory Bowel Diseases - physiopathology | Colitis, Ulcerative - immunology | Host-Pathogen Interactions - immunology | Mycobacterium tuberculosis - pathogenicity | Haplotypes - genetics | Mycobacterium - pathogenicity | Mycobacterium Infections - genetics | Inflammatory Bowel Diseases - genetics | Crohn Disease - microbiology | Mycobacterium Infections - microbiology | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Reproducibility of Results | Mycobacterium - immunology | Crohn Disease - immunology | Genome, Human - genetics | Phenotype | Colitis, Ulcerative - microbiology | Host-Pathogen Interactions - genetics | Polymorphism, Single Nucleotide - genetics | Inflammatory Bowel Diseases - microbiology | Crohn Disease - physiopathology | Colitis, Ulcerative - physiopathology | Inflammatory bowel disease | Candidates | Tuberculosis | Architecture | Genetics | Genomes | Diabetes | Gene expression | Health risk assessment | Immune system | Index Medicus | Rheumatology and Autoimmunity | Basic Medicine | Medical Genetics | Medicinsk genetik | Clinical Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Reumatologi och inflammation | Medicinska och farmaceutiska grundvetenskaper | Klinisk medicin
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2. Full Text Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci
by Franke, Ane and McGovern, Dermot P. B and Barrett, Jeffrey C and Wang, Kai and Radford-Smith, Graham L and Ahmad, Tariq and Lees, Charlie W and Balschun, Tobias and Lee, James and Roberts, Rebecca and Anderson, Carl A and Bis, Joshua C and Bumpstead, Suzanne and Ellinghaus, David and Festen, Eleonora M and Georges, Michel and Green, Todd and Haritunians, Talin and Jostins, Luke and Latiano, Anna and Mathew, Christopher G and Montgomery, Grant W and Prescott, Natalie J and Raychaudhuri, Soumya and Rotter, Jerome I and Schumm, Philip and Sharma, Yashoda and Simms, Lisa A and Taylor, Kent D and Whiteman, David and Wijmenga, Cisca and Baldassano, Robert N and Barclay, Murray and Bayless, Theodore M and Brand, Stephan and Büning, Carsten and Cohen, Albert and Colombel, Jean-Frederick and Cottone, Mario and Stronati, Laura and Denson, Ted and de Vos, Martine and D'Inca, Renata and Dubinsky, Marla and Edwards, Cathryn and Florin, Tim and Franchimont, Denis and Gearry, Richard and Glas, Jürgen and van Gossum, Ane and Guthery, Stephen L and Halfvarson, Jonas and Verspaget, Hein W and Hugot, Jean-Pierre and Karban, Amir and Laukens, Debby and Lawrance, Ian and Lemann, Marc and Levine, Arie and Libioulle, Cecile and Louis, Edouard and Mowat, Craig and Newman, William and Panés, Julián and Phillips, Anne and Proctor, Deborah D and Regueiro, Miguel and Russell, Richard and Rutgeerts, Paul and Sanderson, Jeremy and Sans, Miquel and Seibold, Frank and Steinhart, A. Hillary and Stokkers, Pieter C. F and Torkvist, Leif and Kullak-Ublick, Gerd and Wilson, David and Walters, Thomas and Targan, Stephan R and Brant, Steven R and Rioux, John D and D'Amato, Mauro and Weersma, Rinse K and Kugathasan, Subra and Griffiths, Anne M and Mansfield, John C and Vermeire, Severine and Duerr, Richard H and Silverberg, Mark S and Satsangi, Jack and Schreiber, Stefan and Cho, Judy H and Annese, Vito and Hakonarson, Hakon and Daly, Mark J and Parkes, Miles and Hälsoakademin and Örebro universitet
Nature genetics, ISSN 1061-4036, 2010, Volume 42, Issue 12, pp. 1118 - 1125
We undertook a meta-analysis of six Crohn's disease genome-wide association studies (GWAS) comprising 6,333 affected individuals (cases) and 15,056 controls... 
COMMON VARIANTS | ACTIVATION | INFLAMMATORY-BOWEL-DISEASE | PROTEIN | NEDD4 FAMILY | GENE | GENETICS & HEREDITY | SEQUENCE VARIANTS | RECEPTOR | IDENTIFICATION | ASSOCIATION | Genetic Variation | Crohn Disease - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Reproducibility of Results | Humans | Computational Biology | Genetic Loci - genetics | Crohn Disease - etiology | Genome, Human - genetics | Genetic Linkage | Care and treatment | Methyltransferases | Physiological aspects | Disease susceptibility | Genetic aspects | Crohn's disease | Research | Risk factors | Inflammatory bowel disease | Genetics | Genomics | Meta-analysis | Index Medicus | Medicine | Medicin | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
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3. Full Text Deep resequencing of GWAS loci identifies independent rare variants associated with inflammatory bowel disease
by Rivas, Manuel A and Beaudoin, Mélissa and Gardet, Agnes and Stevens, Christine and Sharma, Yashoda and Zhang, Clarence K and Boucher, Gabrielle and Ripke, Stephan and Ellinghaus, David and Burtt, Noel and Fennell, Tim and Kirby, Anew and Latiano, Anna and Goyette, Philippe and Green, Todd and Halfvarson, Jonas and Haritunians, Talin and Korn, Joshua M and Kuruvilla, Finny and Lagacé, Caroline and Neale, Benjamin and Lo, Ken Sin and Schumm, Phil and Törkvist, Leif and Dubinsky, Marla C and Brant, Steven R and Silverberg, Mark S and Duerr, Richard H and Altshuler, David and Gabriel, Stacey and Lettre, Guillaume and Franke, Ane and D'Amato, Mauro and McGovern, Dermot P. B and Cho, Judy H and Rioux, John D and Xavier, Ramnik J and Daly, Mark J and Parkes, Miles and Lee, James and Zhang, Hu and Bredin, Francesca and Ahmad, Tariq and Satsangi, Jack and Nimmo, Elaine and mmond, Hazel and Lees, Charlie and Mansfield, John and Mathew, Christopher G and Prescott, Natalie and Harrison, Kristina and Sanderson, Jeremy and Newman, William and Phillips, Anne and Mowat, Craig and Edwards, Cathryn and Wilson, David C and Barrett, Jeffrey and Anderson, Carl and Gray, Emma and Edkins, Sarah and Russell, Richard K and Henderson, Paul and Anderson, Carl A and Annese, Vito and Baldassano, Robert N and Balschun, Tobias and Barclay, Murray and Barrett, Jeffrey C and Bayless, Theodore M and Bis, Joshua C and Brand, Stephan and Bumpstead, Suzanne and Buning, Carsten and Cohen, Albert and Colombel, Jean-Frederick and Cottone, Mario and D'Inca, Renata and Denson, Ted and Dubinsky, Marla and Florin, Tim and Franchimont, Denis and Gearry, Richard and Georges, Michel and Glas, Jurgen and van Gossum, Ane and Griffiths, Anne M and Guthery, Stephen L and Hakonarson, Hakon and Hugot, Jean-Pierre and de Jong, Dirk J and Jostins, Luke and Kugathasan, Subra and Kullack-Ublick, Gerd and Laukens, Debby and Lawrance, Ian and Lees, Charlie W and Lemann, Marc and Levine, Arie and Libioulle, Cecile and ... and United Kingdom Inflammatory Bowel and Natl Inst Diabet Digestive Kidney and Int Inflammatory Bowel Dis and United Kingdom Inflammatory Bowel Disease Genetics Consortium and National Institute of Diabetes and Digestive Kidney Diseases Inflammatory Bowel Disease Genetics Consortium (NIDDK IBDGC) and International Inflammatory Bowel Disease Genetics Consortium and Hälsoakademin and Örebro universitet
Nature genetics, ISSN 1061-4036, 2011, Volume 43, Issue 11, pp. 1066 - 1073
More than 1,000 susceptibility loci have been identified through genome-wide association studies (GWAS) of common variants; however, the specific genes and... 
RECRUITMENT | RISK LOCI | CROHNS-DISEASE | GENE | FREQUENCY | GENETICS & HEREDITY | MURAMYL DIPEPTIDE RECOGNITION | SUSCEPTIBILITY LOCI | CARD15 MUTATIONS | NOD2 | GENOME-WIDE ASSOCIATION | Cell Line | Genetic Predisposition to Disease | Genome-Wide Association Study | RNA Splicing | Humans | Inflammatory Bowel Diseases - genetics | Nod2 Signaling Adaptor Protein - genetics | Receptors, Interleukin - genetics | Sequence Analysis, DNA | Case-Control Studies | Inflammatory bowel disease | Studies | Design | Medical research | Genealogy | Genes | Genetics | Data processing | Genomes | Grants | Diabetes | Twins | Index Medicus | Medicin | Medicine | Medical and Health Sciences | Medicin och hälsovetenskap
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4. Full Text Deep resequencing of GWAS loci identifies rare variants in CARD9, IL23R and RNF186 that are associated with ulcerative colitis
by Beaudoin, M and Goyette, P and Boucher, G and Lo, K.S and Rivas, M.A and Stevens, C and Alikashani, A and Ladouceur, M and Ellinghaus, D and Torkvist, L and Goel, G and Lagace, C and Annese, V and Bitton, A and Begun, J and Brant, S.R and Bresso, F and Cho, J.H and Duerr, R.H and Halfvarson, J and McGovern, D.P and Radford-Smith, G and Schreiber, S and Schumm, P.L and Sharma, Y and Silverberg, M.S and Weersma, R.K and Quebec, I.B.D.G.C and Consortium, N.I.G and D'Amato, M and Vermeire, S and Franke, A and Lettre, G and Xavier, R.J and Daly, M.J and Rioux, J.D and Jong, D.J. de and Quebec IBD Genetics Consortium and NIDDK IBD Genetics Consortium and Int IBD Genetics Consortium and International IBD Genetics Consortium and Region Örebro län and Örebro universitet and Institutionen för hälsovetenskap och medicin
PLOS Genetics, ISSN 1553-7390, 2013, Volume 9, Issue 9, pp. e1003723 - e1003723
Genome-wide association studies and follow-up meta-analyses in Crohn's disease (CD) and ulcerative colitis (UC) have recently identified 163 disease-associated... 
INFLAMMATORY-BOWEL-DISEASE | CROHNS-DISEASE | LIVER | GENETICS & HEREDITY | SUSCEPTIBILITY LOCI | GENETIC-VARIANTS | NUCLEAR FACTOR 4-ALPHA | UBIQUITIN LIGASES | EXPRESSION | INTESTINAL EPITHELIAL-CELLS | GENOME-WIDE ASSOCIATION | Crohn Disease - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Humans | Colitis, Ulcerative - genetics | Colitis, Ulcerative - pathology | Receptors, Interleukin - genetics | CARD Signaling Adaptor Proteins - genetics | Canada | Ethnic Groups | Crohn Disease - pathology | High-Throughput Nucleotide Sequencing | Polymorphism, Single Nucleotide | Ubiquitin-Protein Ligases - genetics | Genomics | Genetic aspects | Crohn's disease | Research | Nucleotide sequencing | Ulcerative colitis | DNA sequencing | Index Medicus | Medical and Health Sciences | Medicin och hälsovetenskap | Inflammatory bowel disease | Medical research | Hospitals | Genes
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5. Full Text Comparative performances of machine learning methods for classifying Crohn Disease patients using genome-wide genotyping data
by Romagnoni, A and Jegou, S and Van Steen, K and Wainrib, G and Hugot, JP and Peyrin-Biroulet, L and Chamaillard, M and Colombel, JF and Cottone, M and D'Amato, M and D'Inca, R and Halfvarson, J and Henderson, P and Karban, A and Kennedy, NA and Khan, MA and Lemann, M and Levine, A and Massey, D and Milla, M and Ng, SME and Oikonomou, I and Peeters, H and Proctor, DD and Rahier, JF and Rutgeerts, P and Seibold, F and Stronati, L and Taylor, KM and Torkvist, L and Ublick, K and Van Limbergen, J and Van Gossum, A and Vatn, MH and Zhang, H and Zhang, W and Andrews, JM and Bampton, PA and Barclay, M and Florin, TH and Gearry, R and Krishnaprasad, K and Lawrance, IC and Mahy, G and Montgomery, GW and Radford-Smith, G and Roberts, RL and Simms, LA and Hanigan, K and Croft, A and Amininijad, L and Cleynen, I and Dewit, O and Franchimont, D and Georges, M and Laukens, D and Theatre, E and Vermeire, S and Aumais, G and Baidoo, L and Barrie, AM and Beck, K and Bernard, EJ and Binion, DG and Bitton, A and Brant, SR and Cho, JH and Cohen, A and Croitoru, K and Daly, MJ and Datta, LW and Deslandres, C and Duerr, RH and Dutridge, D and Ferguson, J and Fultz, J and Goyette, P and Greenberg, GR and Haritunians, T and Jobin, G and Katz, S and Lahaie, RG and McGovern, DP and Nelson, L and Ng, SM and Ning, K and Pare, P and Regueiro, MD and Rioux, JD and Ruggiero, E and Schumm, LP and Schwartz, M and Scott, R and Sharma, Y and Silverberg, MS and Spears, D and Steinhart, AH and Stempak, JM and Swoger, JM and Tsagarelis, C and ... and Int Inflammatory Bowel Dis Genetic and International Inflammatory Bowel Disease Genetics Consortium (IIBDGC)
SCIENTIFIC REPORTS, ISSN 2045-2322, 07/2019, Volume 9, Issue 1, pp. 1 - 18
Crohn Disease (CD) is a complex genetic disorder for which more than 140 genes have been identified using genome wide association studies (GWAS). However, the... 
LOCI | INFLAMMATORY-BOWEL-DISEASE | RISK PREDICTION | ASSOCIATION | MULTIDISCIPLINARY SCIENCES | Inflammatory bowel diseases | Complementarity | Epistasis | Genomes | Genetic variance | Learning algorithms | Genetic markers | Genotyping | Intestine | Neural networks | Machine learning | Quality control | Artificial intelligence | Genotypes
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6. Full Text A Method to Exploit the Structure of Genetic Ancestry Space to Enhance Case-Control Studies
by Bodea, Corneliu A and Neale, Benjamin M and Ripke, Stephan and Barclay, Murray and Peyrin-Biroulet, Laurent and Chamaillard, Mathias and Colombel, Jean-Frederick and Cottone, Mario and Croft, Anthony and D’Incà, Renata and Halfvarson, Jonas and Hanigan, Katherine and Henderson, Paul and Hugot, Jean-Pierre and Karban, Amir and Kennedy, Nicholas A and Khan, Mohammed Azam and Lémann, Marc and Levine, Arie and Massey, Dunecan and Massey, Dunecan C.O and Milla, Monica and Montgomery, Grant W and Ng, Sok Meng and Ng, Sok Meng Evelyn and Oikonomou, Ioannis and Peeters, Harald and Proctor, Deborah D and Rahier, Jean-Francois and Roberts, Rebecca and Roberts, Rebecca L and Rutgeerts, Paul and Seibold, Frank and Stronati, Laura and Taylor, Kirstin M and Törkvist, Leif and Ublick, Kullak and Van Limbergen, Johan and Van Gossum, André and Van Gossum, Andre and Vatn, Morten H and Zhang, Clarence and Zhang, Hu and Zhang, Wei and Andrews, Jane M and Bampton, Peter A and Florin, Timothy H and Gearry, Richard and Krishnaprasad, Krupa and Lawrance, Ian C and Mahy, Gillian and Radford-Smith, Graham and Simms, Lisa A and Amininijad, Leila and Cleynen, Isabelle and Dewit, Olivier and Franchimont, Denis and Georges, Michel and Laukens, Debby and Theatre, Emilie and Vermeire, Severine and Aumais, Guy and Baidoo, Leonard and Barrie, Arthur M and Beck, Karen and Bernard, Edmond-Jean and Binion, David G and Bitton, Alain and Brant, Steve R and Cho, Judy H and Cohen, Albert and Croitoru, Kenneth and Daly, Mark J and Datta, Lisa W and Deslandres, Colette and Duerr, Richard H and Dutridge, Debra and Ferguson, John and Fultz, Joann and Goyette, Philippe and Greenberg, Gordon R and Haritunians, Talin and Jobin, Gilles and Katz, Seymour and Lahaie, Raymond G and McGovern, Dermot P and Nelson, Linda and Ning, Kaida and Paré, Pierre and Regueiro, Miguel D and Rioux, John D and Ruggiero, Elizabeth and Schumm, L. Philip and Schwartz, Marc and Scott, Carol E and Scott, Richard and Scott, Regan and Sharma, Yashoda and Silverberg, Mark S and Spears, Denise and ... and International IBD Genetics Consortium
The American Journal of Human Genetics, ISSN 0002-9297, 05/2016, Volume 98, Issue 5, pp. 857 - 868
One goal of human genetics is to understand the genetic basis of disease, a challenge for diseases of complex inheritance because risk alleles are few relative... 
Genetics, Population | Genetic Predisposition to Disease | Disease - genetics | Gene Frequency | Humans | Bayes Theorem | Heredity - genetics | Polymorphism, Single Nucleotide - genetics | Genotype | Software | Case-Control Studies | Genetic Linkage | Genetic research | Usage | Methods | Gaussian processes | Genetics | Disease | Genes | Risk factors | Index Medicus | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Gastroenterology and Hepatology | Gastroenterologi
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