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Clinical Cancer Research, ISSN 1078-0432, 05/2018, Volume 24, Issue 9, pp. 2029 - 2031
Breast and gynecologic cancers harboring PIK3CA mutations showed no significant responses to AZD5363, a pan-AKT catalytic inhibitor, in contrast with previous... 
ONCOLOGY | Biomarkers | AKT protein | Bioindicators | Catalysis | Mutation | Cancer | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Cancer and Oncology | Medicin och hälsovetenskap | Cancer och onkologi
Journal Article
Nature Medicine, ISSN 1078-8956, 03/2016, Volume 22, Issue 3, pp. 262 - 269
Journal Article
Nature Communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, p. 6377
Tyrosine kinase inhibitors are effective treatments for non-small-cell lung cancers (NSCLCs) with epidermal growth factor receptor (EGFR) mutations. However,... 
GROWTH-FACTOR RECEPTOR | DRUG-SENSITIVITY | GEFITINIB | AMPLIFICATION | MODELS | GENE | MULTIDISCIPLINARY SCIENCES | MUTATION | ACQUIRED-RESISTANCE | KINASE INHIBITORS | CHEMOTHERAPY | Lung Neoplasms - drug therapy | Recurrence | Adenocarcinoma - pathology | Adenocarcinoma of Lung | Humans | Lung Neoplasms - metabolism | ErbB Receptors - genetics | Gene Expression Regulation, Neoplastic | bcl-X Protein - genetics | Lung Neoplasms - pathology | Small Cell Lung Carcinoma - drug therapy | Small Cell Lung Carcinoma - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Adenocarcinoma - metabolism | Adenocarcinoma - genetics | Antineoplastic Agents - pharmacology | Lung - metabolism | Gefitinib | ErbB Receptors - deficiency | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Lung - pathology | Aniline Compounds - pharmacology | ErbB Receptors - antagonists & inhibitors | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Retinoblastoma Protein - deficiency | Small Cell Lung Carcinoma - genetics | Sulfonamides - pharmacology | Adenocarcinoma - drug therapy | Disease Progression | Drug Resistance, Neoplasm - genetics | Small Cell Lung Carcinoma - pathology | Afatinib | Retinoblastoma Protein - genetics | Erlotinib Hydrochloride - pharmacology | Lung - drug effects | Cell Line, Tumor | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Quinazolines - pharmacology | bcl-X Protein - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Drug Resistance, Neoplasm - drug effects
Journal Article
The Lancet, ISSN 0140-6736, 2017, Volume 389, Issue 10075, pp. 1229 - 1237
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2013, Volume 110, Issue 52, pp. 21124 - 21129
Journal Article
Current Topics in Microbiology and Immunology, ISSN 0070-217X, 2010, Volume 346, Issue 1, pp. 171 - 181
Recently, it has been reported that some members of the PI3K family might have a "double identity"; in other words, PI3K have been found to act not only as... 
Animals | Cell Proliferation | Neoplasms - etiology | Myocytes, Cardiac - physiology | Humans | Phosphatidylinositol 3-Kinases - physiology | Blood Platelets - physiology | Endothelial Cells - physiology
Journal Article
Cancer Research, ISSN 0008-5472, 07/2016, Volume 76, Issue 14 Supplement, pp. 3846 - 3846
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2015, Volume 112, Issue 11, pp. E1288 - E1296
BH3 mimetics such as ABT-263 induce apoptosis in a subset of cancer models. However, these drugs have shown limited clinical efficacy as single agents in... 
Targeted therapies | Small-cell lung cancer | BH3 mimetics | BIM | Apoptosis | DRUG-SENSITIVITY | small-cell lung cancer | targeted therapies | ANTAGONIST ABT-737 | MCL-1 | MULTIDISCIPLINARY SCIENCES | apoptosis | KINASE INHIBITORS | OBATOCLAX GX15-070 | CAP-DEPENDENT TRANSLATION | BCL-2 FAMILY INHIBITOR | CHRONIC LYMPHOCYTIC-LEUKEMIA | PHASE-I | Lung Neoplasms - drug therapy | TOR Serine-Threonine Kinases - metabolism | Apoptosis - drug effects | Humans | Myeloid Cell Leukemia Sequence 1 Protein - metabolism | Lung Neoplasms - pathology | Mechanistic Target of Rapamycin Complex 2 | Small Cell Lung Carcinoma - drug therapy | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - antagonists & inhibitors | Dose-Response Relationship, Drug | TOR Serine-Threonine Kinases - antagonists & inhibitors | Multiprotein Complexes - metabolism | Bcl-2-Like Protein 11 | Inhibitory Concentration 50 | Membrane Proteins - metabolism | Morpholines - therapeutic use | Proto-Oncogene Proteins - metabolism | Aniline Compounds - pharmacology | Morpholines - pharmacology | Sulfonamides - pharmacology | Remission Induction | Apoptosis Regulatory Proteins - metabolism | Xenograft Model Antitumor Assays | Small Cell Lung Carcinoma - pathology | Animals | Genetic Engineering | Sulfonamides - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Cell Line, Tumor | Mice | Aniline Compounds - therapeutic use | Antimitotic agents | Molecular targeted therapy | Care and treatment | Lung cancer, Small cell | Genetic aspects | Antineoplastic agents | Methods | Testing | Biological Sciences | PNAS Plus
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 1 - 12
The lipid sensor oleoylethanolamide (OEA), an endogenous high-affinity agonist of peroxisome proliferator-activated receptor-α (PPAR-α) secreted in the... 
Appetite | Phenotypes | Nutrient deficiency | Immune response | Cytokines | Digestive system | Cognitive ability | Inflammation | Anti-inflammatory agents | Lipolysis | Mimicry | High fat diet | Polysaccharides | Microbiota | Intestine | Oleic acid | Low fat diet | Oxidation | Intestinal microflora | Peroxisome proliferator-activated receptors | Digestive tract
Journal Article
Cancer Research, ISSN 0008-5472, 07/2018, Volume 78, Issue 13 Supplement, pp. 5502 - 5502
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 12/2015, Volume 14, Issue 12 Supplement 2, pp. A77 - A77
Journal Article
Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13 Supplement, pp. 3026 - 3026
Journal Article
LANCET, ISSN 0140-6736, 03/2017, Volume 389, Issue 10075, pp. 1229 - 1237
Background In 2011, WHO member states signed up to the 25 x 25 initiative, a plan to cut mortality due to noncommunicable diseases by 25% by 2025. However,... 
UNITED-STATES | MEDICINE, GENERAL & INTERNAL | INEQUALITIES | EUROPEAN COUNTRIES | INCOME | NONCOMMUNICABLE DISEASES | SMOKING | HEALTH | BURDEN | ASSOCIATION | LIFE EXPECTANCY | Health aspects | Social classes | Mortality | United Kingdom | France
Journal Article
Cancer Cell, ISSN 1535-6108, 07/2014, Volume 26, Issue 1, pp. 136 - 149
Activation of the phosphoinositide 3-kinase (PI3K) pathway occurs frequently in breast cancer. However, clinical results of single-agent PI3K inhibitors have... 
DRUG | INACTIVATION | ADVANCED SOLID TUMORS | MAMMARY-TUMORS | GENE | ONCOLOGY | PATHWAY INHIBITORS | CYCLIN D1 EXPRESSION | GROWTH | PHASE-I | PRECLINICAL EVALUATION | CELL BIOLOGY | Cyclin-Dependent Kinase 6 - antagonists & inhibitors | Phosphorylation | Phosphatidylinositol Phosphates - metabolism | Cyclin-Dependent Kinase 4 - genetics | Humans | Drug Resistance, Neoplasm | Phosphatidylinositol 3-Kinases - metabolism | Molecular Targeted Therapy | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Dose-Response Relationship, Drug | Breast Neoplasms - enzymology | Transfection | MCF-7 Cells | RNA Interference | Time Factors | Female | Cyclin-Dependent Kinase 4 - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - metabolism | Cell Survival - drug effects | Genetic Predisposition to Disease | Retinoblastoma Protein - metabolism | Cyclin-Dependent Kinase 6 - genetics | Treatment Outcome | Cyclin-Dependent Kinase 6 - metabolism | Cyclin-Dependent Kinase 4 - metabolism | Mice, SCID | Breast Neoplasms - drug therapy | Drug Synergism | Phosphatidylinositol 3-Kinases - genetics | Xenograft Model Antitumor Assays | Protein Kinase Inhibitors - administration & dosage | Phenotype | Animals | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | Signal Transduction - drug effects | Breast Neoplasms - pathology | Mice, Nude | Cell Proliferation - drug effects | Mice | Mutation | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Medical colleges | Breast cancer
Journal Article