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2011, Progress in inflammation research, ISBN 3034801475, xiii, 224
Including a range of contributions from numerous leading international experts in the field, this book provides an extensive overview of the current knowledge... 
Immunological aspects | Inflammasomes | Mediators | microbiology | Inflammation | analysis | immunology | Clinical & internal medicine
Book
Journal of Clinical Investigation, ISSN 0021-9738, 12/2007, Volume 117, Issue 12, pp. 3786 - 3799
The molecular mechanisms of acute lung injury resulting in inflammation and fibrosis are not well established. Here we investigate the roles of the IL-1... 
MONOSODIUM URATE CRYSTALS | MEDICINE, RESEARCH & EXPERIMENTAL | INTERLEUKIN-1 RECEPTOR ANTAGONIST | INDUCED CYTOKINE | INDUCED LUNG INJURY | AIRWAY INFLAMMATION | BLEOMYCIN-INDUCED PNEUMONOPATHY | DEFICIENT MICE | ONSET STILLS-DISEASE | NF-KAPPA-B | CUTTING EDGE | Respiratory Distress Syndrome, Adult - pathology | Interleukin-8 - genetics | Sialoglycoproteins - genetics | Interleukin-1beta - pharmacology | Antibiotics, Antineoplastic - toxicity | Humans | Sialoglycoproteins - metabolism | Pneumonia - pathology | Respiratory Distress Syndrome, Adult - metabolism | Receptors, Interleukin-1 Type I - metabolism | Receptors, Interleukin-1 Type I - antagonists & inhibitors | Respiratory Distress Syndrome, Adult - genetics | Lymphokines - genetics | Pneumonia - chemically induced | Bone Marrow Transplantation | Lymphokines - metabolism | Pulmonary Fibrosis - metabolism | Interleukin 1 Receptor Antagonist Protein - pharmacology | Interleukin-8 - metabolism | Bleomycin - toxicity | Disease Models, Animal | Pneumonia - genetics | Receptors, Interleukin-1 Type I - agonists | Respiratory Distress Syndrome, Adult - chemically induced | Myeloid Differentiation Factor 88 - genetics | Pulmonary Fibrosis - pathology | Signal Transduction - genetics | Recombinant Proteins - pharmacology | Mice, Knockout | Transplantation Chimera - genetics | Transplantation Chimera - metabolism | Animals | Signal Transduction - drug effects | Receptors, Interleukin-1 Type I - genetics | Pulmonary Fibrosis - chemically induced | Mice | Pneumonia - metabolism | Chronic Disease | Myeloid Differentiation Factor 88 - metabolism | Complications and side effects | Bleomycin | Pulmonary fibrosis | Dosage and administration | Research | Health aspects | Risk factors | Apoptosis | Pneumonia | Respiratory Distress Syndrome, Adult | Signal Transduction | Interleukin-1beta | Antibiotics, Antineoplastic | Myeloid Differentiation Factor 88 | Recombinant Proteins | Transplantation Chimera | Interleukin-8 | Lymphokines | Life Sciences | Sialoglycoproteins | Immunology | Pulmonary Fibrosis | Interleukin 1 Receptor Antagonist Protein | Receptors, Interleukin-1 Type I
Journal Article
The Journal of Immunology, ISSN 0022-1767, 01/2008, Volume 180, Issue 2, pp. 1169 - 1178
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 2012, Volume 3, p. 414
The NLRP3 inflammasome is a protein complex involved in IL-1 beta and IL-18 processing that senses pathogen- and danger-associated molecular patterns (PAMPs... 
Autophagic cell death | P2R | Danger signal | NLRP3 | Inflammasome | ATP | Purinergic signaling | purinergic signaling | autophagic cell death | inflammasome | danger signal | IMMUNOLOGY
Journal Article
Journal Article
Nature Immunology, ISSN 1529-2908, 2014, Volume 15, Issue 8, pp. 738 - 748
Journal Article
FRONTIERS IN IMMUNOLOGY, ISSN 1664-3224, 04/2019, Volume 10, p. 702
Oral T. gondii infection (30 cysts of 76K strain) induces acute lethal ileitis in sensitive C57BL/6 (B6) mice with increased expression of IL-33 and its... 
CELLS | IMMUNITY | IL-33/ST2 receptor | neutralizing antibody | IMMUNOLOGY | PREVENT | IL-22 | Toxoplasma gondii | innate immunity | COLITIS | PATHWAY | parasite-induced ileitis | T1/ST2 | Complications and side effects | Interleukins | Toxoplasmosis | Gastrointestinal diseases | Colorectal diseases | Cellular signal transduction | Genetic aspects | Research | Gene expression | Risk factors | Life Sciences
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 05/2009, Volume 179, Issue 10, pp. 903 - 913
Rationale Lung injury leads to pulmonary inflammation and fibrosis through myeloid differentiation primary response gene 88 (MyD88) and the IL-1 receptor 1... 
Inflammasome | Lung inflammation | Bleomycin | Uric acid | Fibrosis | bleomycin | DENDRITIC CELLS | CASPASE-1 | INNATE IMMUNE-RESPONSE | CUTTING EDGE | MONOSODIUM URATE CRYSTALS | RECEPTOR-DEFICIENT MICE | ADAPTIVE IMMUNITY | DYING CELLS | RESPIRATORY SYSTEM | inflammasome | APOPTOTIC CELLS | fibrosis | uric acid | PULMONARY-FIBROSIS | lung inflammation | CRITICAL CARE MEDICINE | Bleomycin - antagonists & inhibitors | NLR Family, Pyrin Domain-Containing 3 Protein | Bronchoalveolar Lavage Fluid - immunology | Lung Injury - metabolism | Myeloid Differentiation Factor 88 - immunology | Pneumonia - immunology | Uric Acid - administration & dosage | Interleukin-1beta - biosynthesis | Lung - metabolism | Pulmonary Fibrosis - metabolism | Carrier Proteins - immunology | Disease Models, Animal | Biomarkers - metabolism | Lung - pathology | Uric Acid - metabolism | Enzyme-Linked Immunosorbent Assay | Biomarkers - analysis | Pulmonary Fibrosis - immunology | Interleukin-1beta - immunology | Bleomycin - administration & dosage | Uric Acid - immunology | Animals | Carrier Proteins - metabolism | Lung - drug effects | Allopurinol - pharmacology | Mice | Lung Injury - chemically induced | Pneumonia - metabolism | Lung Injury - immunology | Lung - immunology | Index Medicus | Abridged Index Medicus | Bronchoalveolar Lavage Fluid | Pneumonia | Biological Markers | Allopurinol | Lung | Interleukin-1beta | Myeloid Differentiation Factor 88 | Life Sciences | Immunology | Uric Acid | Pulmonary Fibrosis | Lung Injury | Carrier Proteins
Journal Article
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 10/2011, Volume 45, Issue 4, pp. 731 - 739
Tumor Necrosis Factor (TNF) is a pleiotropic cytokine consisting of soluble and transmembrane forms, with distinct roles in inflammation and immunity. TNF is... 
Airway hyperreactivity | XENP1595 | Etanercept | Asthma | RHEUMATOID-ARTHRITIS | COLLAGEN-INDUCED ARTHRITIS | airway hyperreactivity | MEMBRANE TNF | etanercept | ASTHMA MODEL | TUBERCULOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | asthma | FACTOR-ALPHA BLOCKADE | CELL BIOLOGY | TRANSMEMBRANE TNF | RESPIRATORY SYSTEM | AIRWAY HYPERRESPONSIVENESS | MICE | MYCOBACTERIAL INFECTION | Tumor Necrosis Factor-alpha - metabolism | Pneumonia - physiopathology | Bronchial Hyperreactivity - immunology | Humans | Hyperplasia | Pulmonary Eosinophilia - genetics | Tumor Necrosis Factor-alpha - genetics | Pneumonia - pathology | Pneumonia - prevention & control | Goblet Cells - drug effects | Biological Products - pharmacology | Tumor Necrosis Factor-alpha - deficiency | Bronchial Hyperreactivity - genetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Goblet Cells - immunology | Bronchial Hyperreactivity - physiopathology | Inflammation Mediators - metabolism | Immunoglobulin G - pharmacology | Pneumonia - immunology | Disease Models, Animal | Pneumonia - genetics | Pulmonary Eosinophilia - immunology | Ovalbumin | Lung - pathology | Pulmonary Eosinophilia - prevention & control | Cytokines - metabolism | Immunoglobulin E - metabolism | Mice, Inbred C57BL | Mice, Transgenic | Lung - physiopathology | Bronchial Hyperreactivity - prevention & control | Mice, Knockout | Tumor Necrosis Factor-alpha - pharmacology | Bronchial Hyperreactivity - pathology | Animals | Receptors, Tumor Necrosis Factor | Lung - drug effects | Mice | Mice, Inbred BALB C | Goblet Cells - pathology | Mutation | Lung - immunology | Tumor Necrosis Factor-alpha - antagonists & inhibitors
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 8, pp. e23185 - e23197
Background: Idiopathic pulmonary fibrosis is a devastating as yet untreatable disease. We demonstrated recently the predominant role of the NLRP3 inflammasome... 
COLLAGEN-INDUCED ARTHRITIS | TGF-BETA | ROR-GAMMA-T | HELPER-CELLS | BIOLOGY | LUNG INJURY | DELTA-T-CELLS | NEUTROPHIL RECRUITMENT | RECEPTOR ANTAGONIST | AIRWAY NEUTROPHILIA | TH17 CELLS | Transforming Growth Factor beta1 - metabolism | Pneumonia - pathology | Male | Pulmonary Fibrosis - genetics | Interleukin-1beta - genetics | Lung Injury - etiology | Interleukin-23 - genetics | Interleukin-23 Subunit p19 - metabolism | Flow Cytometry | Interleukin-23 - metabolism | Interleukin-1beta - metabolism | T-Lymphocyte Subsets - drug effects | Interleukin-23 Subunit p19 - genetics | Lung Injury - metabolism | Female | Lung - metabolism | Pulmonary Fibrosis - metabolism | Pneumonia - genetics | Lung - pathology | Enzyme-Linked Immunosorbent Assay | Mice, Inbred C57BL | Interleukin-17 - genetics | Mice, Transgenic | Pulmonary Fibrosis - pathology | Bleomycin - administration & dosage | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Interleukin-17 - metabolism | Animals | Interleukin-1beta - administration & dosage | T-Lymphocyte Subsets - metabolism | Lung - drug effects | Mice | Pneumonia - metabolism | Collagen | Analysis | Lung diseases | Fibrosis | Bone morphogenetic proteins | Inflammation | T cells | Transforming growth factors | Flow cytometry | Traumatic brain injury | Laboratories | Antibodies | Homeostasis | Inflammatory response | Interleukin 23 | Infections | Lymphocytes T | Bleomycin | E coli | Lymphocytes | Rodents | Interleukin 1 | Chronic obstructive pulmonary disease | Drug dosages | Medical research | Wound healing | Bacterial infections | Cytokines | Neutrophils | Interleukin 17 | Signaling | Infectious diseases | Pulmonary fibrosis | Rheumatoid arthritis
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 09/2010, Volume 182, Issue 6, pp. 774 - 783
Journal Article
SCIENTIFIC REPORTS, ISSN 2045-2322, 04/2019, Volume 9, Issue 1, pp. 5788 - 10
Unconventional protein secretion represents an important process of the inflammatory response. The release of the pro-inflammatory cytokine interleukin (IL)-1... 
ACTIVE CASPASE-1 | EEA1 | AUTOANTIBODIES | PROTEIN | MECHANISM | INTERLEUKIN-1-BETA | MULTIDISCIPLINARY SCIENCES | NLRP3 INFLAMMASOME | SECRETION | PYROPTOSIS | PLASMA-MEMBRANE | Proteins | Cytokines | Secretion | Caspase | Inflammation | Lactic acid | IL-1β | L-Lactate dehydrogenase | Membrane proteins | Caspase-1
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, pp. e99383 - e99383
The exact implication of innate immunity in granuloma formation and irreversible lung fibrosis remains to be determined. In this study, we examined the lung... 
FIBROSIS | TUMOR-NECROSIS-FACTOR | INDUCED PULMONARY INFLAMMATION | NMRI MICE | INDUCED LUNG INFLAMMATION | C57BL/6 MICE | MULTIDISCIPLINARY SCIENCES | RECEPTOR ANTAGONIST | MURINE SILICOSIS | T-LYMPHOCYTES | NALP3 INFLAMMASOME | Immunohistochemistry | Inflammation - chemically induced | T-Lymphocytes, Regulatory - metabolism | Granuloma - metabolism | Interleukin-1alpha - immunology | Receptors, Interleukin-1 - genetics | Immunity, Innate - genetics | Interleukin-1alpha - metabolism | Pulmonary Fibrosis - genetics | Interleukin-1beta - genetics | Silicosis - genetics | T-Lymphocytes, Regulatory - immunology | Signal Transduction - immunology | Flow Cytometry | Th17 Cells - metabolism | Interleukin-1beta - metabolism | Myeloid Differentiation Factor 88 - immunology | Interleukin-1alpha - genetics | Cytokines - genetics | Silicosis - immunology | Cytokines - immunology | Granuloma - genetics | Silicosis - etiology | Cytokines - metabolism | Receptors, Interleukin-1 - immunology | Silicon Dioxide - immunology | Mice, Inbred C57BL | Myeloid Differentiation Factor 88 - genetics | Pulmonary Fibrosis - immunology | Interleukin-1beta - immunology | Inflammation - immunology | Signal Transduction - genetics | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Immunity, Innate - immunology | Animals | Silicon Dioxide - toxicity | Inflammation - genetics | Pulmonary Fibrosis - chemically induced | Th17 Cells - immunology | Granuloma - immunology | Myeloid Differentiation