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Diabetes, ISSN 0012-1797, 05/2018, Volume 67, Issue Supplement 1, p. 2130
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2002, Volume 99, Issue 6, pp. 3884 - 3889
Pancreatic development is a classic example of epithelium-mesenchyme interaction. During embryonic life, signals from the mesenchyme control the proliferation... 
Acinar cells | Biological Sciences | Epithelial cells | Developmental biology | Pancreatic cells | Epithelium | Progenitor cells | Cellular differentiation | Insulin | Endocrine cells | Mesenchymal stem cells | NEURAL STEM-CELLS | MORPHOGENESIS | IN-VITRO | FOREBRAIN | FGF | MULTIDISCIPLINARY SCIENCES | BETA-CELLS | KEY ROLE | FIBROBLAST-GROWTH-FACTOR | IDENTIFICATION | EXOCRINE | Epithelial Cells - metabolism | Receptor, Fibroblast Growth Factor, Type 2 | Epithelial Cells - drug effects | Pancreas - cytology | Stem Cells - cytology | RNA, Messenger - metabolism | Stem Cells - metabolism | Keratins - metabolism | Receptors, Fibroblast Growth Factor - genetics | Flavonoids - pharmacology | Epithelial Cells - cytology | RNA, Messenger - genetics | Biomarkers - analysis | Pancreas - drug effects | Rats | Fibroblast Growth Factors - pharmacology | Pancreas - metabolism | Receptor Protein-Tyrosine Kinases - metabolism | Pancreas - embryology | Cell Division - drug effects | Insulin - metabolism | Animals | MAP Kinase Signaling System - drug effects | Cell Differentiation - drug effects | Receptors, Fibroblast Growth Factor - metabolism | Fibroblast Growth Factor 7 | Morphogenesis - drug effects | Stem Cells - drug effects | Physiological aspects | Fibroblast growth factors | Research | Cytochemistry | Pancreas | Protein kinases
Journal Article
Journal Article
Clinical Diabetes and Endocrinology, ISSN 2055-8260, 2016, Volume 2, Issue 1, p. 11
Intra-islet insulin contributes to alpha-cell suppression. mice carry a toxic-gain-of- function gene mutation encoding proinsulin-C(A7)Y, similar to that... 
Glucagon | C-peptide II | C-peptide I | Exendin-4 | Type 2 diabetes | Hyperglycemia | Gene mutations | Blood sugar | Leptin | Genetic aspects | Insulin | Diabetes therapy
Journal Article
Diabetes, ISSN 0012-1797, 06/2019, Volume 68, Issue Supplement 1, p. 2165
Journal Article