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Immunity, ISSN 1074-7613, 08/2015, Volume 43, Issue 2, pp. 213 - 215
Inhibition of the inflammasome might be beneficial for numerous inflammatory pathologies. In this issue of  , report that the PYRIN domain-only protein (POP1)... 
INTERLEUKIN-1 | DOMAIN | KAPPA-B | IMMUNOLOGY | Cryopyrin-Associated Periodic Syndromes - immunology | Animals | Inflammasomes - metabolism | Dendritic Cells - immunology | Humans | Macrophages, Peritoneal - immunology | Female | Monocytes - immunology | Ribonucleoproteins - metabolism | Peritonitis - immunology | Proteins | Mutation | Feedback | Rodents | Inflammatory diseases | Apoptosis
Journal Article
Journal Article
Immunity, ISSN 1074-7613, 11/2018, Volume 49, Issue 5, pp. 873 - 885.e7
Receptor interacting protein 2 (RIP2) plays a role in sensing intracellular pathogens, but its function in T cells is unclear. We show that RIP2 deficiency in... 
atherosclerosis | Chlamydia pneumoniae | IL-17 | RORα | Th17 | chronic inflammation | RIP2 | T(H)17 CELLS | TGF-BETA | INNATE IMMUNE-RESPONSES | INFLAMMATION | HOST-DEFENSE | ATHEROSCLEROSIS | INFECTION | NOD2 | IMMUNOLOGY | NF-KAPPA-B | T-Lymphocyte Subsets - immunology | Encephalomyelitis, Autoimmune, Experimental - metabolism | T-Lymphocyte Subsets - cytology | Encephalomyelitis, Autoimmune, Experimental - etiology | Receptor-Interacting Protein Serine-Threonine Kinases - chemistry | Nuclear Receptor Subfamily 1, Group F, Member 1 - metabolism | Cell Differentiation - genetics | Inflammation - metabolism | Th17 Cells - metabolism | Atherosclerosis | Th17 Cells - cytology | Gene Expression | Encephalomyelitis, Autoimmune, Experimental - mortality | Immunophenotyping | Interleukin-1beta | Interleukin-17 - biosynthesis | Mice, Knockout | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | T-Lymphocyte Subsets - metabolism | Biomarkers | Inflammation - genetics | Mice | Receptor-Interacting Protein Serine-Threonine Kinases - deficiency | Caspase Activation and Recruitment Domain | Oligomers | Medical research | Pneumonia | Interleukins | Bacterial pneumonia | Communicable diseases | Medicine, Experimental | Inflammation | Research institutes | T cells | Cell differentiation | Encephalomyelitis | Disease | Interleukin | Helper cells | Differentiation (biology) | Chronic infection | Infections | Lymphocytes T | Kinases | Proteins | Sexually transmitted diseases | Lymphocytes | Interleukin 1 | Chronic illnesses | Antigens | Oligomerization | Cytokines | Mortality | Gene expression | Experimental allergic encephalomyelitis | CD4 antigen | Lungs | Arteriosclerosis
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 05/2018, Volume 38, Issue Suppl_1
Journal Article
Cancer Research, ISSN 0008-5472, 08/2015, Volume 75, Issue 15 Supplement, pp. 4057 - 4057
Journal Article
Immunity, ISSN 1074-7613, 03/2012, Volume 36, Issue 3, pp. 401 - 414
Journal Article
Cell Host & Microbe, ISSN 1931-3128, 06/2016, Volume 19, Issue 6, pp. 865 - 873
Journal Article
Immunity, ISSN 1074-7613, 04/2015, Volume 42, Issue 4, pp. 640 - 653
Acute lung injury (ALI) remains a serious health issue with little improvement in our understanding of the pathophysiology and therapeutic approaches. We... 
BACTERIAL LIPOPOLYSACCHARIDE | STERILE INFLAMMATION | SIGNALING PATHWAY | P2X RECEPTORS | ALPHA PRODUCTION | ENDOTHELIAL-CELLS | MOUSE MODEL | IN-VIVO | ACUTE LUNG INJURY | IMMUNOLOGY | INFLAMMATORY RESPONSE | Interleukin-1alpha - immunology | Macrophages, Alveolar - pathology | Calcium - metabolism | Humans | Capillary Permeability - immunology | Neutrophil Infiltration | Necrosis - pathology | Receptors, Purinergic P2X7 - genetics | Necrosis - immunology | Cadherins - immunology | Adenosine Triphosphate - metabolism | HEK293 Cells | Interleukin-1alpha - genetics | Cadherins - genetics | Receptors, Purinergic P2X7 - immunology | Macrophages, Alveolar - immunology | Neutrophils - pathology | Lung - pathology | Protein Precursors - genetics | Signal Transduction | Neutrophils - drug effects | Gene Expression Regulation | Neutrophils - immunology | Lipopolysaccharide Receptors - immunology | Mice, Transgenic | Protein Precursors - immunology | Acute Lung Injury - pathology | Necrosis - chemically induced | Endothelial Cells - immunology | Animals | Acute Lung Injury - chemically induced | Macrophages, Alveolar - drug effects | Lung - drug effects | Lipopolysaccharides - pharmacology | Lipopolysaccharide Receptors - genetics | Acute Lung Injury - immunology | Mice | Endothelial Cells - pathology | Intubation, Intratracheal | Lung - immunology | Endothelial Cells - drug effects | endothelial cells | alveolar macrophage | Ca2+ influx | MyD88 | IL-1α | neutrophil | P2X7R | lung injury | necrosis | ARDS | ALI | sepsis | lung inflammation
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 6, p. e21477
Chlamydia pneumoniae (CP) is an important human pathogen that causes atypical pneumonia and is associated with various chronic inflammatory disorders.... 
APOPTOSIS | INTERLEUKIN-1 | ACTIVATION | MICE DEFICIENT | IFN-GAMMA | IMMUNE-RESPONSES | MULTIDISCIPLINARY SCIENCES | INNATE | INDUCTION | BLOOD MONONUCLEAR-CELLS | NALP3 INFLAMMASOME | Lung - microbiology | Protein Biosynthesis | Chlamydia Infections - enzymology | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Caspase 1 - metabolism | Chlamydia Infections - immunology | Disease Models, Animal | Interleukin-1beta - secretion | Lung - pathology | Macrophages, Alveolar - microbiology | Pneumonia, Bacterial - complications | Pneumonia, Bacterial - enzymology | Chlamydia Infections - complications | Mitochondria - metabolism | Toll-Like Receptor 2 - metabolism | Immunity, Innate | Macrophages, Alveolar - enzymology | Animals | Carrier Proteins - metabolism | Caspase 1 - deficiency | Survival Analysis | Pneumonia, Bacterial - immunology | Mice | Myeloid Differentiation Factor 88 - metabolism | Lung - immunology | Phagocytosis | Chlamydophila pneumoniae - immunology | Nitric Oxide Synthase Type II - metabolism | Pediatrics | Oxidative stress | Reactive oxygen species | Pneumonia | Immune clearance | Infections | Activation | Interleukin 1 receptors | Macrophages | Experiments | Caspase-1 | Sexually transmitted diseases | Mitochondria | Immunology | Chlamydia | Rodents | Interleukin 1 | Toll-like receptors | Bacteria | Alveoli | Immune system | Recombinant | Enzymes | Cytokines | Secretion | Mortality | Caspase | Protein biosynthesis | Pharmacology | Nitric-oxide synthase | Medicine | Signaling | Infectious diseases | Lungs | Protein synthesis | TLR2 protein | MyD88 protein | Potassium | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 9, p. e13027
Inflammatory bowel disease (IBD) is a disorder of chronic inflammation with increased susceptibility to colorectal cancer. The etiology of IBD is unclear but... 
INFLAMMATORY-BOWEL-DISEASE | TGF-BETA | DENDRITIC CELLS | EPITHELIAL-CELLS | NITRIC-OXIDE SYNTHASE | COLORECTAL-CANCER | IN-VIVO | BIOLOGY | REGULATORY T-CELLS | SPONTANEOUS INTESTINAL TUMORIGENESIS | CUTTING EDGE | Aberrant Crypt Foci - etiology | Toll-Like Receptor 2 - genetics | Colitis - genetics | Humans | Colitis - complications | Male | Aberrant Crypt Foci - immunology | Colorectal Neoplasms - etiology | Female | Colitis - immunology | Aberrant Crypt Foci - prevention & control | Disease Models, Animal | Signal Transduction | Mice, Inbred C57BL | Colorectal Neoplasms - prevention & control | Aberrant Crypt Foci - pathology | Immunity, Innate | Mice, Knockout | Neoplastic Processes | Animals | Colorectal Neoplasms - immunology | Interleukin-6 - immunology | Toll-Like Receptor 2 - immunology | Mice | Colorectal Neoplasms - pathology | Gastrointestinal diseases | Oncology, Experimental | Colorectal cancer | Colitis | Research | Risk factors | Cancer | Diseases | Immunohistochemistry | Haplotypes | Pediatrics | Inflammatory bowel diseases | Epithelial cells | Physicians | Colorectal carcinoma | Homeostasis | Innate immunity | Genomes | Immunity | Interleukin 6 | Proteins | Microorganisms | Histopathology | Immunology | Lymphocytes | Etiology | Intestine | Rodents | Toll-like receptors | Bacteria | Tumorigenesis | Immune system | Medical research | Immune response | Cytokines | Stat3 protein | Inflammation | Epithelium | Crohns disease | Medicine | Inflammatory bowel disease | Signaling | Infectious diseases | TLR2 protein | Regulation | Aberration | Immunofluorescence | Tumors
Journal Article
Journal Article
PLoS Pathogens, ISSN 1553-7366, 04/2009, Volume 5, Issue 4, pp. e1000379 - e1000379
Here we investigated the role of the Nod/Rip2 pathway in host responses to Chlamydophila pneumoniae-induced pneumonia in mice. Rip2(-/-) mice infected with C.... 
IFN-GAMMA | RECEPTOR-INTERACTING PROTEIN-2 | MICROBIOLOGY | INTRACELLULAR BACTERIAL-INFECTION | CHLAMYDIA-TRACHOMATIS INFECTION | VIROLOGY | FOAM CELL-FORMATION | IMMUNE-RESPONSES | IN-VIVO | TOLL-LIKE RECEPTORS | NITRIC-OXIDE | NF-KAPPA-B | PARASITOLOGY | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Nitric Oxide Synthase Type II - biosynthesis | Nod Signaling Adaptor Proteins - immunology | RNA, Messenger - analysis | Chlamydophila Infections - pathology | Pneumonia, Bacterial - metabolism | Flow Cytometry | Chlamydophila pneumoniae - metabolism | Bone Marrow Cells - immunology | Nod Signaling Adaptor Proteins - metabolism | Receptor-Interacting Protein Serine-Threonine Kinases - immunology | Pneumonia, Bacterial - pathology | Macrophages - immunology | Chemokines - immunology | Neutrophil Infiltration - immunology | Nitric Oxide - biosynthesis | Enzyme-Linked Immunosorbent Assay | Chlamydophila Infections - metabolism | Chlamydophila Infections - immunology | Immunity, Innate | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Pneumonia, Bacterial - immunology | Signal Transduction - physiology | Chemokines - metabolism | Mice | Bone Marrow Cells - metabolism | Chlamydophila pneumoniae - immunology | Chlamydia infections | Pneumonia | Immune response | Bacterial pneumonia | Physiological aspects | Causes of | Development and progression | Genetic aspects | Research | Gene expression | Cell culture | Lungs | Rodents | Bone marrow | Bacteria | Experiments | Recruitment
Journal Article