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Journal of the National Cancer Institute, ISSN 0027-8874, 2012, Volume 104, Issue 4, pp. 311 - 325
Journal Article
Cancer Research, ISSN 0008-5472, 10/2014, Volume 74, Issue 19 Supplement, pp. 2355 - 2355
Journal Article
by Thorsson, Vesteinn and Thorsson, Vésteinn and Gibbs, Richard A and Gibbs, David L and Brown, Scott D and Wolf, Gregory and Wolf, Denise and Bortone, Dante S and Ou Yang, Tai-Hsien and Porta-Pardo, Eduard and Gao, Galen F and Gao, Jianjiong and Plaisier, Christopher L and Eddy, James A and Ziv, Elad and Culhane, Aedin C and Paull, Evan O and Sivakumar, I.K. Ashok and Gentles, Andrew J and Malhotra, Raunaq and Farshidfar, Farshad and Colaprico, Antonio and Parker, Joel S and Mose, Lisle E and Vo, Nam Sy and Liu, Wenbin and Liu, Jianfang and Liu, Jia and Liu, Yuexin and Liu, Xiuping and Rader, Janet and Dhankani, Varsha and Reynolds, Sheila and Reynolds, Sheila M and Bowlby, Reanne and Califano, Andrea and Cherniack, Andrew D and Anastassiou, Dimitris and Bedognetti, Davide and Mokrab, Younes and Newman, Aaron M and Rao, Arvind and Chen, Amy and Chen, Chu and Chen, Ken and Krasnitz, Alexander and Hu, Jianhong and Hu, Hai and Malta, Tathiane M and Noushmehr, Houtan and Pedamallu, Chandra Sekhar and Bullman, Susan and Ojesina, Akinyemi I and Lamb, Andrew and Zhou, Wanding and Shen, Hui and Choueiri, Toni K and Weinstein, John N and Guinney, Justin and Saltz, Joel and Holt, Robert and Holt, Robert A and Rabkin, Charles S and Caesar-Johnson, Samantha J and Demchok, John A and Felau, Ina and Kasapi, Melpomeni and Ferguson, Martin L and Hutter, Carolyn M and Sofia, Heidi J and Tarnuzzer, Roy and Wang, Jioajiao and Wang, Min and Wang, Linghua and Wang, Zhining and Wang, Timothy and Wang, Jing and Yang, Ju Dong and Yang, Hannah and Yang, Liming and Yang, Ian and Zenklusen, Jean C and Zhang, Jiashan (Julia) and Zhang, Jiexin and Zhang, Lizhi and Zhang, Wei and Zhang, Hongzheng and Zhang, Hailei and Zhang, Hongxin and Chudamani, Sudha and Lolla, Laxmi and Naresh, Rashi and Pihl, Todd and Sun, Yichao and Sun, Qiang and Wan, Yunhu and Wu, Ye and Cho, Juok and DeFreitas, Timothy and Frazer, Scott and ... and Canc Genome Atlas Res Network and Cancer Genome Atlas Research Network
Immunity, ISSN 1074-7613, 04/2018, Volume 48, Issue 4, pp. 812 - 830.e14
We performed an extensive immunogenomic analysis of more than 10,000 tumors comprising 33 diverse cancer types by utilizing data compiled by TCGA. Across... 
tumor immunology | immunotherapy | cancer genomics | integrative network analysis | immuno-oncology | tumor microenvironment | immune subtypes | immunomodulatory | GENOMIC ANALYSES | INFORMATION | T-CELL-RECEPTOR | REGULATORY NETWORK | CLASSIFICATION | IMMUNOLOGY | SEQUENCING DATA | SOMATIC MUTATIONS | COMPREHENSIVE ANALYSIS | PREDICTION | REVEALS | Transforming Growth Factor beta - immunology | Prognosis | Humans | Middle Aged | Th1-Th2 Balance - physiology | Male | Neoplasms - classification | Wound Healing - immunology | Young Adult | Transforming Growth Factor beta - genetics | Neoplasms - genetics | Neoplasms - immunology | Interferon-gamma - immunology | Adolescent | Aged, 80 and over | Adult | Female | Aged | Interferon-gamma - genetics | Wound Healing - genetics | Genomics - methods | Child | Macrophages - immunology | Cell proliferation | Cluster analysis | Transcription | Copy number | p53 Protein | Lung cancer | Genomics | Aneuploidy | Lymphocytes T | Genomes | Cell interactions | Macrophages | Immunotherapy | DNA methylation | Deoxyribonucleic acid--DNA | Immune system | Medical research | Wound healing | Immunomodulation | MiRNA | Stockholders | Inflammation | T cell receptors | Communications networks | Gene expression | White blood cells | Inventors | Medical prognosis | γ-Interferon | Mutation | Cancer | Tumors | Index Medicus | Immune Subtypes | Integrative Network Analysis | Cancer Genomics | Immuno-oncology | Tumor immunology
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2014, Volume 124, Issue 2, pp. 859 - 870
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2015, Volume 10, Issue 10, pp. e0140243 - e0140243
Both oncogenic and tumor-suppressor activities are attributed to the Nuclear Factor kappa B (NF-kB) pathway. Moreover, NF-kB may positively or negatively... 
KINASE 4/6 INHIBITOR | EXPRESSION PATTERNS | SECRETORY PHENOTYPE | PHOSPHATIDYLINOSITOL 3-KINASE | MULTIDISCIPLINARY SCIENCES | THERAPEUTIC TARGET | CYCLE ARREST | FACTOR-I | HUMAN BREAST-CANCER | NF-KAPPA-B | MAMMARY EPITHELIAL-CELLS | Fallopian Tubes - cytology | Cell Line | Breast - cytology | Interferons - pharmacology | Humans | Retinoblastoma Protein - metabolism | Tumor Suppressor Protein p53 - metabolism | MicroRNAs - metabolism | Interferon-gamma - metabolism | Cyclin-Dependent Kinase 4 - metabolism | Down-Regulation - drug effects | Transcription Factor RelA - metabolism | Breast Neoplasms - pathology | Cell Cycle Checkpoints - drug effects | Female | Cell Proliferation - drug effects | Phosphorylation - drug effects | Epithelial Cells - cytology | Interferon Regulatory Factor-1 - metabolism | Interferon | Genetic transcription | B cells | Biological response modifiers | Analysis | Genomics | Transcription factors | Phosphorylation | Senescence | Transcription | Epithelial cells | Biology | Genomes | Interferon regulatory factor 1 | Kinases | Inactivation | Cyclin-dependent kinase 4 | Transcription activation | Cell cycle | Fibroblasts | Mammary gland | Histochemical analysis | Stat1 protein | Bioinformatics | Medical research | NF-κB protein | Deactivation | Cyclin-dependent kinases | Breast cancer | Gene expression | ErbB-2 protein | Interferon regulatory factor 7 | Pathology | Womens health | γ-Interferon | Breast | RelA protein | Tumors | Cancer | Apoptosis | Index Medicus
Journal Article
PLoS ONE, 10/2015, Volume 10, Issue 10
Both oncogenic and tumor-suppressor activities are attributed to the Nuclear Factor kappa B (NF-kB) pathway. Moreover, NF-kB may positively or negatively... 
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2013, Volume 110, Issue 21, pp. 8632 - 8637
Germ-line mutations in PALB2 lead to a familial predisposition to breast and pancreatic cancer or to Fanconi Anemia subtype N. PALB2 performs its tumor... 
Genomics | Cell lines | Alleles | Breast cancer | Breast neoplasms | Mice | Embryos | Genetic mutation | Tumors | Cancer | Mouse model | Familial breast cancer | EARLY EMBRYONIC LETHALITY | mouse model | SOLID TUMORS | MULTIDISCIPLINARY SCIENCES | CRE RECOMBINASE | FANCONI-ANEMIA | MOUSE MODELS | GENOMIC INSTABILITY | BRCA1 DEFICIENCY | SUSCEPTIBILITY GENE | familial breast cancer | HOMOLOGOUS RECOMBINATION | CELLULAR PROLIFERATION | Neoplastic Syndromes, Hereditary - pathology | Humans | Neoplastic Syndromes, Hereditary - metabolism | Mammary Neoplasms, Experimental - metabolism | Mammary Neoplasms, Experimental - genetics | Tumor Suppressor Protein p53 - genetics | Breast Neoplasms - metabolism | Mice, Mutant Strains | BRCA1 Protein - metabolism | Gene Deletion | Tumor Suppressor Proteins - genetics | Mammary Neoplasms, Experimental - pathology | Female | Neoplastic Syndromes, Hereditary - genetics | Nuclear Proteins - genetics | Fanconi Anemia Complementation Group N Protein | Disease Models, Animal | Rad51 Recombinase - metabolism | Tumor Suppressor Proteins - metabolism | Rad51 Recombinase - genetics | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | BRCA1 Protein - genetics | Animals | BRCA2 Protein - metabolism | Breast Neoplasms - genetics | Breast Neoplasms - pathology | BRCA2 Protein - genetics | Prevention | Tumor suppressor genes | Genetic aspects | Health aspects | DNA damage | Rodents | Genetics | Mutation | Cells | Index Medicus | Biological Sciences
Journal Article