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Nature Medicine, ISSN 1078-8956, 03/2016, Volume 22, Issue 3, pp. 298 - 305
Journal Article
Nature medicine, ISSN 1078-8956, 11/2014, Volume 20, Issue 11, pp. 1301 - 1309
Some of the anti-neoplastic effects of anthracyclines in mice originate from the induction of innate and T cell-mediated anticancer immune responses. Here we... 
MEDICINE, RESEARCH & EXPERIMENTAL | DOUBLE-STRANDED-RNA | DENDRITIC CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | INNATE | RIG-I | TUMORS | CELL BIOLOGY | TOLL-LIKE RECEPTOR-3 | BREAST-CANCER | ANTICANCER CHEMOTHERAPY | GENE | IMMUNE-RESPONSES | Doxorubicin - therapeutic use | Breast Neoplasms - immunology | Humans | RNA, Messenger - metabolism | Adaptor Proteins, Vesicular Transport - metabolism | Interferon Type I - biosynthesis | Receptors, Pattern Recognition - metabolism | Neoplasm Metastasis | Anthracyclines - pharmacology | Interferon Type I - metabolism | Female | Neoadjuvant Therapy | Gene Expression Regulation, Neoplastic - drug effects | RNA - metabolism | Receptor, Interferon alpha-beta - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Treatment Outcome | Toll-Like Receptor 3 - metabolism | Breast Neoplasms - drug therapy | Animals | Breast Neoplasms - genetics | Signal Transduction - drug effects | Breast Neoplasms - pathology | Immunocompetence - drug effects | Anthracyclines - therapeutic use | Chemokine CXCL10 - metabolism | Myxovirus Resistance Proteins - metabolism | Doxorubicin - pharmacology | Anthracyclines | Care and treatment | Analysis | Physiological aspects | Interferon | Research | Cancer | Signal transduction | Chemotherapy | Breast cancer | Chemokines | Index Medicus | Life Sciences | Immunology | Immunotherapy
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 10/2015, Volume 211, Issue 2, pp. 339 - 358
Journal Article
The Journal of Pathology, ISSN 0022-3417, 10/2015, Volume 237, Issue 2, pp. 179 - 189
Adenoid cystic carcinoma ( AdCC ) is a rare type of triple‐negative breast cancer ( TNBC ) characterized by the presence of the MYB–NFIB fusion gene. The... 
genomics | MYB–NFIB | adenoid cystic carcinoma | triple‐negative breast cancer | massively parallel sequencing | triple-negative breast cancer | MYB-NFIB | HEAD | HISTOLOGICAL SPECIAL TYPES | MYB EXPRESSION | PATHOLOGY | SALIVARY-GLANDS | MISSENSE MUTATIONS | SOMATIC MUTATIONS | ONCOLOGY | MUTATIONAL LANDSCAPE | COPY-NUMBER ALTERATION | NFIB GENE FUSION | HUMAN CANCER | Genetic Predisposition to Disease | Carcinoma, Adenoid Cystic - pathology | Biomarkers, Tumor - analysis | Gene Frequency | Humans | Gene Expression Regulation, Neoplastic | Carcinoma, Adenoid Cystic - chemistry | In Situ Hybridization, Fluorescence | Gene Dosage | Genes, myb | Reverse Transcriptase Polymerase Chain Reaction | DNA Copy Number Variations | Phenotype | Triple Negative Breast Neoplasms - chemistry | Triple Negative Breast Neoplasms - genetics | Carcinoma, Adenoid Cystic - genetics | DNA Mutational Analysis | Oncogene Proteins, Fusion - genetics | Triple Negative Breast Neoplasms - pathology | Female | Biomarkers, Tumor - genetics | Mutation | Genomics - methods | Real-Time Polymerase Chain Reaction | Ubiquitin | Carcinoma | Multiprocessing | Genomics | Genetic aspects | Breast cancer | Tumor proteins | Chromosomes | Cancer | Index Medicus | Adenoid cystic carcinoma | massively parallel | Oncology | SOMATIC | Pathology | MUTATIONS | Cancer and Oncology | Cancer och onkologi
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 2404 - 10
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 5201 - 14
Deciphering cell-intrinsic mechanisms of metastasis progression in vivo is essential to identify novel therapeutic approaches. Here we elucidate cell-intrinsic... 
EVOLUTION | MECHANISM | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | BIOLOGY | RESISTANCE | CLASSIFICATION | THERAPIES | MUTATIONS | EXPRESSION | HISTONE METHYLTRANSFERASE | Proteins | Bone grafts | Animal models | Correlation analysis | Genetic engineering | Metastasis | Prostate cancer | Prostate | Cancer | Metastases | Index Medicus
Journal Article
Cancer Cell, ISSN 1535-6108, 10/2016, Volume 30, Issue 4, pp. 563 - 577
The transition from castration-resistant prostate adenocarcinoma (CRPC) to neuroendocrine prostate cancer (NEPC) has emerged as an important mechanism of... 
castration-resistant prostate adenocarcinoma | prostate cancer organoid | N-Myc | genetically engineered mouse | neuroendocrine prostate cancer | Aurora kinase A | EZH2 | REARRANGEMENT | INACTIVATION | GENOMICS | PROTEIN | ONCOLOGY | ADENOCARCINOMA | MOUSE MODEL | SMALL-CELL CARCINOMA | PTEN | DIFFERENTIATION | IDENTIFICATION | CELL BIOLOGY | Enhancer of Zeste Homolog 2 Protein - antagonists & inhibitors | Prostatic Neoplasms - metabolism | Humans | N-Myc Proto-Oncogene Protein - genetics | Male | Genes, myc | Prostatic Neoplasms, Castration-Resistant - genetics | Prostatic Neoplasms, Castration-Resistant - pathology | Heterografts | Prostatic Neoplasms - genetics | Transcription, Genetic | N-Myc Proto-Oncogene Protein - biosynthesis | Prostatic Neoplasms - drug therapy | Neuroendocrine Tumors - pathology | Prostatic Neoplasms - pathology | Enhancer of Zeste Homolog 2 Protein - genetics | Enhancer of Zeste Homolog 2 Protein - metabolism | Signal Transduction | Neuroendocrine Tumors - metabolism | Mice, Transgenic | Prostatic Neoplasms, Castration-Resistant - drug therapy | Pyrimidines - pharmacology | Prostatic Neoplasms, Castration-Resistant - metabolism | Neuroendocrine Tumors - genetics | Azepines - pharmacology | Animals | N-Myc Proto-Oncogene Protein - metabolism | Neuroendocrine Tumors - drug therapy | Mice | Protein Kinase Inhibitors - pharmacology | Development and progression | Genetic aspects | Genetic engineering | Genetic transcription | Prostate cancer | Genetically modified organisms | Index Medicus
Journal Article
NATURE COMMUNICATIONS, ISSN 2041-1723, 07/2019, Volume 10, Issue 1, pp. 2977 - 11
Upper tract urothelial carcinoma (UTUC) is characterized by a distinctly aggressive clinical phenotype. To define the biological features driving this... 
BLADDER-CANCER | MULTICENTER | SIGNATURES | COMPREHENSIVE MOLECULAR CHARACTERIZATION | MICROSATELLITE INSTABILITY | MULTIDISCIPLINARY SCIENCES | GUIDELINES | SINGLE-ARM | EXPRESSION | ASSOCIATION | GENETIC ALTERATIONS | Phenotypes | Depletion | Bladder | Lymphocytes T | Ribonucleic acid--RNA | Urothelial carcinoma | Fibroblast growth factor receptors | Gene sequencing
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2019, Volume 166, Issue 23, pp. 11428 - 11436
Journal Article
Cancer Research, ISSN 0008-5472, 08/2018, Volume 78, Issue 16 Supplement, pp. IA19 - IA19
Journal Article
Cancer Research, ISSN 0008-5472, 01/2016, Volume 76, Issue 2 Supplement, pp. B41 - B41
Journal Article
Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13 Supplement, pp. 992 - 992
Journal Article