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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2010, Volume 107, Issue 12, pp. 5375 - 5380
Journal Article
BMC Cancer, ISSN 1471-2407, 01/2013, Volume 13, Issue 1, pp. 37 - 37
Journal Article
Blood, ISSN 0006-4971, 04/2018, Volume 131, Issue 16, pp. 1771 - 1771
Polycomb repressive complex 1 (PRC1) and PRC2 are transcriptional repressors that function as key regulators of the self-renewal and differentiation (cell... 
CBP | MOZ-TIF2 | SENESCENCE | ACUTE MYELOID-LEUKEMIA | HEMATOLOGY | Fellowships and Scholarships | Stem Cells | Animals | Histone Acetyltransferases | Polycomb Repressive Complex 1 | Mice | Leukemia, Myeloid, Acute
Journal Article
Nucleic Acids Research, ISSN 0305-1048, 05/2019, Volume 47, Issue 9, pp. 4476 - 4494
Abstract IRF1 (Interferon Regulatory Factor-1) is the prototype of the IRF family of DNA binding transcription factors. IRF1 protein expression is regulated by... 
CELLS | PROTEIN | PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | DEGRADATION | TARGETS | FBW7 | IDENTIFICATION | LIGASE | Gene regulation, Chromatin and Epigenetics
Journal Article
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 07/2013, Volume 38, Issue 8, pp. 1512 - 1520
Drugs that induce psychosis, such as D-amphetamine (AMP), and those that alleviate it, such as antipsychotics, are suggested to exert behavioral effects via... 
antipsychotics | D-amphetamine | receptor | latent inhibition | dopamine D | mice | Dextroamphetamine - pharmacology | Learning - drug effects | Receptors, Dopamine D2 - deficiency | Mice, Inbred C57BL | Male | Mice, Knockout | Learning - physiology | Animals | Time Factors | Female | Mice | Antipsychotic Agents - pharmacology | Inhibition (Psychology)
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, p. e58052
Preferentially expressed antigen in melanoma (PRAME) has been described as a cancer-testis antigen and is associated with leukaemias and solid tumours. Here we... 
BREAST-CANCER | SIGNALING PATHWAYS | PREFERENTIALLY EXPRESSED ANTIGEN | UBIQUITIN LIGASE | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | ACUTE MYELOID-LEUKEMIA | CLINICAL IMPORTANCE | IDENTIFICATION | MINIMAL RESIDUAL DISEASE | MESSENGER-RNA LEVELS | Antigens, Neoplasm - genetics | Cell Line | Transcription, Genetic - drug effects | Bacteria - metabolism | Subcellular Fractions - drug effects | Golgi Apparatus - drug effects | Humans | Protein Transport - drug effects | Subcellular Fractions - metabolism | Elongin | Transcription Factors - metabolism | Up-Regulation - drug effects | Multiprotein Complexes - metabolism | Receptors, Pattern Recognition - metabolism | Models, Biological | Mass Spectrometry | Protein Binding - drug effects | Lipopolysaccharides - pharmacology | Antigens, Neoplasm - metabolism | Golgi Apparatus - metabolism | Protein Biosynthesis - drug effects | Histones - metabolism | Interferon-gamma - pharmacology | Ubiquitin | Care and treatment | Leukemia | Genes | Genetic aspects | Interferon | DNA binding proteins | Genetic transcription | Biological response modifiers | Mitogens | Protein binding | Transcription | Nuclei | Cullin | Proteins | Signal transduction | Rodents | Histones | Bacteria | Polyribosomes | Ubiquitin-protein ligase | Antigens | Peptidoglycans | Melanoma | Breast cancer | RNA polymerase | Gene expression | Golgi apparatus | Signaling | Pharmacy | γ-Interferon | Cell lines | Solid tumors | Position (location) | Histone H3 | Tumors | Cancer
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